Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

7110 > Neuro > Flashcards

Neuro Flashcards

1
Q

pneumonic for alt LOC

A

AEIOUTIPS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

A in AEIOUTIPS

A

acidosis, alcohol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

E in AEIOUTIPS

A

Epilepsy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

I in AEIOUTIPS

A

Insulin reaction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

O in AEIOUTIPS

A

overdose

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

U in AEIOUTIPS

A

Uremia, underdose

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

T in AEIOUTIPS

A

Trauma, Tumor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

I in AEIOUTIPS

A

Infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

P in AEIOUTIPS

A

Psychosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

S in AEIOUTIPS

A

Stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

CVA

A

sudden loss of brain fx, neuro deficits that last 24h or more

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what are the main risks with decreased LOC

A

Aspiration, inability to protect airway, seizure, death

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what is ICP

A

pressure exerted on ventricles by CSF

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Equation for CPP

A

Map-ICP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

% of brain matter

A

80

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

% of CSF

A

10

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

% of blood in brain

A

10

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Monro Kellie hypothesis

A

Postulates there is reciprocal compensation between intracranial compartments. Increase in one means decrease in other

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What happens first when ICP increases

A

CSF moves down spinal cord
CSF production decreased/ reabsorption increased
Small amount of distension in dura mater

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

what happens second in increased ICP

A

Pressure builds, venous system is compressed. ICP rises as brain is compressed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

What happens last in increased ICP

A

compensation fails. Increased CO2 causes cerebral vasodilation, increasing blood flow and increasing ICP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

what does CO2 do in brain vasculature

A

Causes increased ICP by increasing perfusion to brain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Why does the venous system fail when ICP increases

A

it is compressed, outflow of blood is blocked and beings to accumulate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Clinical symptoms of increased ICP

A

headache
Nausea/vomitting
Alt LOC

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Clinical signs of impending brain herniation

A

Significant pupillary asymmetry
Unilateral or bilateral fixed and dilated pupils
Decorticate or decebrate posturing
Resp depression

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

What are signs of impending brain herniation (cushings triad)

A

HTN with widening pulse pressure, Bradycardia, irregular respirations

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Normal ICP

A

5-15mmhg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

when is cerebral autoregulation lost

A

MAP <50, >100 or ICP >35 for 20-30m

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

why is maintaining MAP important in neuro patients

A

to ensure CBF and prevent ischemia

30
Q

how to maintain MAP to ensure CBF

A

Inotropes, vaspressors

31
Q

monitoring/treatments for patients with increased ICP

A
  • Ventilation status
  • Drainage of CSF with ICP monitoring system
  • Maintain MAP >90
  • IV bolus or albumin
  • Pressors to maintain MAP
  • Sedation/analgesia to decrease workload
  • Maintain Sats
32
Q

nursing interventions for increased ICP

A
  1. Dim lights
  2. Decompress stomach to reduce gastric pressure
  3. Hyperventilate to blow off Co2
  4. Mannitol
  5. Avoid hip flexion (increases P in abd cavity)
  6. Manage BP
  7. Increase HOB to drain jugular veins
33
Q

3 layers of meninges

A

Dura mater
Arachnoid mater
Pia mater

34
Q

Outmost layer of brain

A

dura mater

35
Q

middle layer of brain

A

arachnoid mater

36
Q

which layer of brain is deepest

37
Q

characteristic of dura mater

A

thick spongey layer, protects

38
Q

characteristics of arachnoid mater

A

thin and delicate, weblike and semitransparent

39
Q

Where is the subaarachnoid space

A

between arachnoid mater and pia matter

40
Q

Characteristics of pia mater

A

attached to brain

41
Q

occipital lobe

A

visual cortex

42
Q

parietal lobe

A

somatosensory cortex

43
Q

temporal lobe

A

hearing, equilibrium, emotion, memory

44
Q

frontal lobe

A

motor cortex and association areas, complex thought, ethical behaviour

45
Q

limbic structures

A

emotions, short term memory, smell

46
Q

basal ganglia

A

initiation and planning of learned motor activities

47
Q

what is RAS

A

reticulatar activating system

48
Q

what does RAS do

A

maintains consciousness, vital regulation for CV, Resp

49
Q

how do brain injuries manifest

A

LOC, cranial nerve reflexes, GCS, brain hemodynamics

50
Q

where is an epidrual hematoma

A

epidural space-between skull surface and dura mater (extradural)

51
Q

what injury increases risk of epidural hematoma

A

skull fracture

52
Q

source of most epidural bleeds (art or venous)

53
Q

where do subdural hematoma form

A

between dura and outer arachnoid membrane

54
Q

what type of blood is in subdural hematoma

55
Q

why may symptoms of a venous bleed appear later

A

slower bleeding

56
Q

where does CSF live

A

subarachnoid space

57
Q

where does a subarachnoid bleed occur

A

between pia mater and outer arachnoid membrane

58
Q

what type of bleed is subarachnoid

A

venous (like subdural) or arterial (anyerusm rupture)

59
Q

what happens when blood is in the CSF

A

meningial irritation
bloody spinal tap
sever headache

60
Q

Common stroke symptoms

A

numbness and weakness on one side of body
confusion, trouble speaking/understanding
visual disturbance
Dizziness, loss of balance
Severe headache

61
Q

characteristic of TIA

A

clot is lysed before permanent tissue. damage occurs

62
Q

how long can a TIA last

A

minutes to hours

63
Q

goals of care for ischemic stroke

A

minimize infarct size and perserve function. Aspirin stat or thrombolytic therapy

64
Q

goals of care for hemorrhagic stroke

A

manage increased ICP

65
Q

Origins of meningitis

A

viral, fungal, bacterial

66
Q

why does meningitis move quickly in CNS

A

present in subarachnoid space, moves through CSF

67
Q

S/S meningitis

A

headache

fever stiff neck, alt LOC. Occasionally, Increased HR, seizures

68
Q

Encephalitis

A

inflammation of the brain

69
Q

common causes of encephalitis

A

HSV, Equine virus, West nile virus

70
Q

S/S encephalitis

A

fever, headache, seizure, confusion, stupor, coma, hallucinations, personality changes

71
Q

characteristics of concussion

A

alteration or loss of consciousness with no brain damage on CT. Symtpoms present immediatly and resolve quickly

7110 (5 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2025 Bold Learning Solutions. Terms and Conditions