Neck Flashcards

1
Q

Discuss zone 1 of the neck and contents (10)

A

Zone 1 Extends superiorly from the sternal notch and clalicle to the cricoid cartilatge

Contents

  • Proximal common carotid artery
  • vertebral artery
  • subclavian artery
  • major vessels of upper mediastinum
  • apices of the lung
  • oesophagus
  • trachea
  • thyroids
  • thoracic duct
  • spinal cord
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2
Q

Discuss zone 2 of the neck and contents `(10)

A

Zone 2 extends from the cricoid cartilage to the angle of the mandible

Contents

  • carotid artery
  • vertebral artery
  • larynx
  • trachea
  • oeosphagus
  • pharynx
  • jugular vein
  • vagus snerve
  • recurrent laryngeal nerve – vocal cords
  • spinal nerve
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3
Q

Discuss zone 3 of the neck and its contents (6)

A

Zone 3 extends from the angle of the mandible to the base of the skull

Contents

  • distal carotid artery
  • vertebral artery
  • distal jugular vein
  • salivary and parotid glands
  • cranial nerves 9-`12
  • spinal cord
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4
Q

Discuss the fasical layers of the neck

A

Two layres
-Superficial layer covers the platysma muscle and is located just below the skin

The deep layer is divided into three parts

1) the investing layer surrounds the neck and splits to encase the sternocleridomastoid and trap
2) the pretracheal layer adheres to the cricoid and thyroids cartilages and travels deep to the sternum to insert into the pericardium
3) The prevertebral layer envelopes the cervical prevertebal muscles and extends to form the axillary sheath in which the subclavian artery runs

The pretracheal layer is important because of its connection of the neck to the anterior mediastinum. Missed aerodigestive injury can cause mediastinitis due to this anatomic continuity,

Injury to the platysma should raise questions about injury to deep structures

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5
Q

Discuss broadly the four groups of neck injury patients

A

1) those who are HD unstable
2) Those are HD stable with hard signs
3) Those who are HD stable with soft signs
4) asymptomatic patients

1 and 2 likely need OR for exploration
Diagnositic testing is indicated in all who who soft or hard signs

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6
Q

List soft signs for penetrating neck injury

A
Minor haemoptysis 
Haematemesis
Dysphonia, dysphagia 
Subcutaneous or mediastinal air 
Nonexpanding haematoma 
Oropharyngeal bleeding 
Neurological findings 
Proximity to the wound
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7
Q

List hard signs of penetrating neck injury (9)

A
Rapidly expanding pulsatile haematoma 
Massive haemoptysis 
Air bubbling from wound 
Severe haemorrhage 
Shock not responding to fluids 
Decreased or absent radial pulse 
Vascular bruit or thrill 
Stridor/hoarseness or airway compromise 
Cerebral ischaemia 
\+- massive subcut emphysema
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8
Q

Discuss management of neck trauma in general

A

A: highest priority– BVM can be harmful as can force air into injured tissue planes – resulting in massive subcut emphysema and subsequent airwya distortion or rarely air embolus.
ETT with RSI remains standard with inline stabilization – relative contraindication include significant anatomical disruption of the face or neck massive haematemesis or concern that RSI will not succeed- If time allows fibreoptic laryngscopy may be preferable – Dont use nasal intubation due to false passages and poor outcomes – cric if needed

B: As standard with attention for pneumothorax in zone 1 injuries
- Indication for resuscitiative thoracotomy is CPR for less than 15 minutes wiht penetrating neck trauma or less than 10 minutes of CPR with any blunt trauma

c: direct pressure without occlusion of airway

D: Attention to c-spine

G: placement of NGT is relatively contrainicated in patient with penetrating neck trauma

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9
Q

Discuss venous or artieral air embolism in neck trauma

A

Venous air embolism causes profound shock or arrest unresponsive to fluids or thoracotomy.

As the circulatory pressure in most arteries and veins is greater than atmospheric pressure an embolus does not often happen when a vessel is injured. In veins above the heart such as in the head and neck this pressure is often lower than the atmosphere with potentional risk for air embolism

Placing the patientin a head down left lateral will cause intracardiac air to accumulate in the apex of the right ventricle. If shock still persist aspiration of air form the right ventricle can eb attempted either via US or under direct vision after thoracotomy

Neurological sequela or any otherwise unexplained stroke like syndrome should prompt consideration of arterial air embolis, If arterial embolus is suscpected stroke can cause cerebral oedema and brain protective measures should be undertaken

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10
Q

Discuss pharyngoesophaegeal trauma (epi, signs, ix, mx)

A

Rare usually associated with penetrating trauma involving the cervical segement. Injury from blunt trauma is associated with hyperextension or cervical fractures

Early identification is necessary as can lead to bacteraila contamination and mediastinitis

Air leaking out through the owund site is the most compelling indicator of an underlying oesophageal or airway injury

Investigation

  • contrast oesophagraphy can be used with a co-operative patient. Typically water soluble contrast is used as barium is more dangerous when extravasation occurs
  • CTA without oral contrast is currently not supported by literature– non oral contrast CT depends on visualistaion of idnirect signs such as paraeosophageal air or fluid, wall thickening or oeedmea.

If suspected broad spectrum AB with anerobic coverage (piptaz) should be used and patient should be kept NBM
Emergent theatre is likley necessary

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11
Q

Discuss laryngotracheal trauma (epi, anatomy)

A

Account for only 1% of neck trauma but a high percentage of immediate morality
The cricoid cartilage is the only complete ring int he larynx – fractures can lead to daeth through acute airway obstruction
The degree of airway obstruction after blunt trauma to the larynx is inversely related to the degree of cartilage calcification – children are therefore at the highest risk of respiratory compromise after injury

Pain on movement of the toungue or rotation of the neck may indicate damage to the hyioid bone

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12
Q

Discuss laryngotracheal trauma investigations

A

Laryngoscopy or flexible nosopharyngoscopy allows direct evaluation of laryngeal integrity. – Usually is well tolerated with appropriate local anaethetics – preparation for emergeny intubation should always be perform prior to laryngoscopy

CT has almost 100% sensitivity – less useful for detection of mucosal perforation degloving injyuries or the cartilage and some types of minor trauma especially in the peadiatric population

Ultrasound can be considered as a bedside test to detect blunt laryngotracheal injuries such as laryngotracheal separation.

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13
Q

Discuss management of laryngotracheal trauma

A

Early laryngoscopy to identify injury with low threshold for intubation. – Ideally awake fibreoptic intubation is the choice
If not available awake video intubation
If not available double set up – sinlge attempt by best provider than surgical access

Most patient will be able to be intubated normally however it is not possible to visualise the airway inferior to the cords unless a flexi scope is used. Tube must be placed gently and guided down the airway with the least friction possible to reduce further injury. (completing laryngeal tracheal seperation or false passage)

If seperation has occured – proximal intubation as above can be tried – if surgical option tracheal hooks can be used to retrieve the distal segment.

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14
Q

Discuss vascular injury (blunt)

A

Half of all patient with dissection from blunt trauma are neurologically asymptomatic. – if present carotid injuries cause either transient of fixed contraleratl sensory or motor deficitis, aphasia dysphasia and Horners syndrome.

Vertebral injuries can cause ataxia vertigo, emesis and visual field deficits. –

If present these symptoms are often delayed. Most develop 10-72 hours post injury with a median of 12.5 hours.

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15
Q

Discuss Western Trauma Association criteria for imaging of blunt cerebrovascular imaging

A

Symptomatic

  • Arterial haemorrhage form the neck mouth, nose or ears
  • Large or expanding cervical haemartoma
  • Cervical bruit in patient <50 years
  • Focal neuroglocail deficit
  • evidence of cerebrla infarct
  • Neurolgocial deficit incongruous with CT/MRI

Asymptomatic
-Cervical hyperextension or flexion with rotation
- Direct cervical trauma
-intraoral trauma
-basilar skull fracutre involving the carotid canal
-Lefort 2 or 3
- head injury with GCS <6
Fracture of c1-3
-vertebral body or transverse foramen fracutre
-hanging with cerebral anoxia
-seatbelt sign of clothesline injury with significant cervical pain, swelling

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16
Q

Discuss Eastern Trauma Association criteria for imaging of blunt cerebrovascular imaging

A

Symptomatic

  • unexplained neurological injury
  • Arterial epistaxis

Asymtpoamtic

  • GCS<9
  • Petrous bone fracutre
  • Diffuse axonal injury
  • C-spine injury
  • Lefort
17
Q

Discuss IX in blunt and penetrating neck trauma `

A

CTA for penetrating injury close to 100% spec and sens

CTA standard for blunt trauma with similar sensitivty and specficity

CT should inculde the intracranial portion of the carotid artery wtih zone 3 injury or suspected blunt cervical trauma

Zone 1 inury should inlcude the aortic arch wtih its branches

US can be considered for evulation of vascular injury in neck trauma

MRA has distinct disadvantages in neck trauma icnluding

    • inavlability
  • possiblility of retained metalic fragments from injury
  • length
  • cost
  • inability to visualise the neck during the procesdure
18
Q

Discuss grading of vascular injury to the neck

A

They are typically graded as follows
-Grade 1: intimal irregularity with <25% narrowing
-Grade 2: dissection or intramural haematoma with >25% narrowing
Grade 3: pseudoaneurysm
Grade 4: Occlusion
Grade 5: transection with extravasation

19
Q

Discuss management of blunt neck injury

A

Treatment depend in part on the mechanism, type and location of the lesion

Anticoagulation is used for dissection wihtout clear evidence of benefit – antiplatelets comparable to heparin

Surgical option include ligation, resection thrombectomy and endovascular stent placement

Endovasuclar stent hterapy is less invasive than surgery and considerably easier to perform. used in higher grade lesions with comparible outcomes to medical management alone
Stent placement is usually followed by 6 months of aspirin or DAPT

Low grade lesions have better outcomes with long term anticoagulation with reduction of stroke rates from close to 25% to 3.9%

Repeat imaging in 7 days is advised to assess for pseudoaneurysms and recannulation

20
Q

Discuss management of penetrating neck injury

A

Goals are twofold – repair of acute life threatening haemorrhage and to prevent stroke
Penetrating take to theatre

21
Q

Discuss nervous system injury

A

The brachail plexus, peripheral nerve roots, symptathetic chain, spinal cord and CN 7-12 (excluding 10) are all vulnerable to trauma

Neurological injury can also result from vascular injury with subsequent cerebral ischaemia.

Phrenic nerve can be injured leading to impaired respiration

Glandular injury is rare

22
Q

Discuss hanging and strangulation

A

Complete haning – freely suspended
Incomplete – partial suspension

Typical hanging refers to the ligature knot being midline direcftly under the occiput – maximal force is apoplied immediatly opposite the placement of the knot and typical hanging has the highest risk of arterial occlusion.,

Survivors of haning can develop multiple sequaele

  • hypoxic brain injury
  • pulmonary oedema
  • –neurogenic from centrally mediated masses sympathetic discharge
  • –postobstructive - relief of the negative intrapleural pressure generated by forceful inspiraotry effort
  • – cardiogenic as a result of hanging associated takotsubo cardiomyopathy
23
Q

Discuss clinical features of hanging

A
External trauma may or may not be present 
Tardieu spots ( petechial haemorrhages seen in the conjunctiva and mucous membranes) are highly correleated with asphyxial deaths 

Thyroid fracutre and/or hyoid are seen in half of all non judicial hanging

Cricoid cartilage fractures are rarely reporting in sucidal hanging and are more common in manual strangulation

Vascular injury leading to delayed neurological sequeal after near haning is rare,

CTA anyone with possibility of injuyr