Myocardial Infarction Flashcards

1
Q

Myocardial Infarction (Acute Coronary Syndrome)

A

limited blood supply to the myocardial tissue resulting in necrosis

Characteristics:

  • Unstable angina
  • Non ST elevation myocardial infarct (NSTEMI)
  • ST elevation myocardial infarct (STEMI)
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2
Q

Etiology

A
  • Blockage in the coronary artery from CAD
  • Coronary spasms (vasospasms) from illicit drug use like cocaine, which causes constriction of the coronary artery and stop blood flowing to the heart muscle
  • Blood loss from anemia
  • Increases in oxygen demand and supply as with thyrotoxicosis and cocaine use
    (-) manifested by increased heart rate
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3
Q

Pathophysiology

A
  1. Development of the unstable plaque that ruptures and forms a thrombus
  2. Broken pieces of this unstable plaque can circulate in the blood, reaching the heart and creating an obstruction which can also result from a spasm, constriction, dysfunction or adrenergic stimuli
  3. Severe narrowing of the coronary lumen
  4. Ischemia results from the coronary artery being starved of oxygen because of the obstructed artery
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4
Q

What happens after an MI?

A

Early signs

  • No physical changes to heart muscle until 6 - 8 hours
  • When myocytes die, cardiac enzymes are released - CK-MB, troponin and myoglobin

24 - 36 hours

  • Inflammation sets in and neutrophils come on the scene and congregate at the damaged tissue site
  • Within 24 hours, heart fails to pump efficiently (cardiogenic shock) and arrhythmias can develop

10 days

  • Macrophages arrive to the site to clean up dead cells
  • New tissue formed from granulation is not well formed and is weak, increasing changes of cardiac rupture

2 months
- Scarring occurs, heart size and functionality is affected due to increased collagen

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5
Q

Clinical Manifestations

A

“CRUSHING”

  • Chest pain
  • Radiating chest pain that goes to left arm, jaw, back
  • Unrelieved by nitroglycerin or rest
  • Sweating
  • Hard to breath (SoB)
  • Increased heart rate, blood pressure or irregular heart rate
  • Nausea with vomiting
  • Going to be anxious and scared
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6
Q

Diagnostic tests

A

BLOOD TESTS

  • Troponin: Protein released from the heart when damage is present
  • Myoglobin: Early cardiac marker released after heart injury
  • CK- MB (creatine kinase): form of enzyme released when their is muscle damage to the heart specifically

OTHER TEST:

  • Echocardiogram
  • Heart Cath
  • Stress test
  • ECG (Obtain a 12 lead ECG ASAP)
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7
Q

What are the roles of medical treatment

A
  • Reduce cardiac damage
  • Preserve myocardial function
  • Prevent complications
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8
Q

Pharmacology treatment

A

Acute Angina Means Nasty Artery Blockages And Cardiac Complications

  • Antithrombotic agents
  • Antiplatelet e.g., Aspirin (162- 325mg) or Plavix (Clopidogrel)
  • Morphine
  • Nitrates
  • ACE inhibitors
  • Beta blockers
  • ARBs receptors
  • Cholesterol lowering medications
  • Calcium channel blockers
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9
Q

Cardiac rehabilitation 1

A
  • Diagnosis of atherosclerosis
  • Education lifestyle changes
  • Early low level mobilization
  • Medication
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10
Q

Cardiac rehabilitation 2

A
  • Last about 4-6 weeks 6 months
  • Scheduled visits
  • Monitored ECG exercise programmes ( stress tests)
  • Support programmes
  • Lifestyle changes
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11
Q

Cardiac rehabilitation 3

A
  • The patient is usually stable in this stage

- Knows signs and symptoms and can manage lifestyle

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12
Q

Nursing Diagnosis

A
  • Ineffective tissue perfusion
  • Risk for imbalances tissue perfusion
  • Fluid volume deficit
  • Death and Anxiety
  • Deficient Knowledge
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13
Q

Complications

A
  • Acute pulmonary edema
  • Heart failure
  • Cardiogenic Shock
  • Dysrhythmias and cardiac arrest
  • Cardiac tamponade
  • Pericardial effusion
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14
Q

Nursing Intervention

A
Relieving pain 
- Oxygen @ 2 - 4 l/min
- Monitor vital signs 
- Bed rest 
    (-) Elevate the HOB 
    (-) Venous return to the heart decreases

Improving respiratory function

  • Oxygen administration
  • Chest auscultation
  • Monitor cyanosis
  • Pursed lip breathing

Promoting Adequate tissue function

  • Vital signs
  • Checking skin temperature
  • Monitoring pulses
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15
Q

Patient teaching

A
  • Avoid activity that brings on symptoms
  • Avoid extremes of heat and cold
  • Lose weight
  • Stop smoking, avoid second hand smoke
  • Develop positive coping methods
  • Modify diet
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16
Q

What are you looking for on EKG?

A
  • ST-segment depression
  • ST- segment elevation
  • T-wave inversion
  • T-wave hyperacute
  • Pathological Q-wave
17
Q

NSTEMI vs STEMI

A

Persons without ST- segment elevation on ECG are those in whom thrombotic coronary occlusion is subtotal or intermittent whereas, those with ST- segment elevation are usually found to have complete coronary occlusion and many ultimately have Q-wave myocardial infarction

18
Q

Transmural infarcts

A

involve the full thickness of the ventricular wall and most commonly occur when there is an obstruction of a single artery

19
Q

Subendocardial infarcts

A

Involve the inner one third to one half of the ventricular wall and occur more frequently in the presence of severely narrowed but still patent arteries