Muskuloskeletal Flashcards

1
Q

Review the care of a child in a cast

what are the 5 P’s

A

Purpose: immobilization to promote healing and proper alignment

Neurovascular assessment

Education on cast:
-Do not put anything into the cast,

pain, pulse, pallor, parasthesia, paralysis (5 ps)

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2
Q

Acute Compartment Syndrome

Symptoms?

A

A serious and life-threatening complication that requires immediate action and may cause permanent damage to extremity.

Swelling or bleeding within a compartment of the musculoskeletal system.

Fascia does not stretch, therefore, increased pressure on the capillaries, nerves, and muscles in the compartment.

Blood flow to muscle and nerve cells is disrupted and results in ischemia.

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3
Q

Acute Compartment Syndrome

Symptoms? Biggest sign?

Management?

A

Extreme pain not controlled by medication!!!!!! BIGGEST SIGN

Swelling of extremity

Decrease or loss of pulses

Cool to touch

Extremity pale or cyanotic

Numbness or tingling

Management: Surgery (Fasciotomy) to reduce the pressure and restore circulation.

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4
Q

Discuss the child in traction including purpose and nursing management

A

Purpose: Maintains straight alignment of fractured bone.
Decreases pain and spasms

Do not remove, adjust, or add weights unless there is a provider order.

Allow weights to hang freely

Monitor skin for redness and signs of breakdown

Neurovascular assessment (5 P’s)

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5
Q

How to suspect abuse?
When do most inflicted fractures occur?
What fractures in infants?

A

Most inflicted fractures occur in infancy or early childhood.

Stated history not consistent with injury pattern or mechanism

Injury inconsistent with developmental stage

Discrepancy in caregiver’s story and/or conflicting caregiver stories

Presence of multiple fractures

Spiral fractures or rib fractures in infants
-Skeletal Survey done to detect other fractures
-Nurses are mandatory reporters!

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6
Q

What is Scoliosis?
Most cases are what?
When does it become noticeable?
What test to determine degree of curvature?

Treatment of severe and nonsevere scoliosis?

A

Abnormal lateral curvature of the spine.
Most cases idiopathic

Generally becomes noticeable after PRE ADOLESCENT GROWTH SPURT

Standing radiographs to determine degree of curvature

Severe Scoliosis:
Surgery with insertion of rods

Not severe- brace

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7
Q

Developmental Dysplasia of Hip (DDH)

A

Abnormal development of the hip joint during fetal growth or early infancy.

Genetic predisposition, intrauterine positioning, or hormonal factors,

The socket of the hip (acetabulum) is too shallow and the femoral head is not held tightly in place, so the hip joint is loose.

Hip instability can lead to dislocation of the hip joint, abnormal gait, and arthritis if not treated early.

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8
Q

Developmental dysplasia of the hip

Diagnostic Evaluation of DDH?

Infants and older children?

A

Infants:
-Ortolani and Barlow tests
-Galeazzi (Allis) sign
-Uneven thigh folds
-All newborns are evaluated for DDH using above procedures

Older children with limp, leg discrepancy
-Radiographic exam
Ultrasound < 4 months;
XRAY > 4 months)

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9
Q

Signs and symptoms of Developmental Dysplasia of Hip (DDH)

Ortoloni-Abducting?
B. Barlow-Adducting?
What is abnormal?
Galeazzi (Allis) Sign?
Harness and Cast?

A

A. Ortoloni-Abducting
B. Barlow-Adducting (push in on hip and then out)

**Abnormal: Hear a ‘click’ or ‘clunk’
with procedure

Galeazzi (Allis) Sign: Uneven Thigh Folds
Uneven Knee Height

Management:
Pavlik harness (0-6 mo)
Spica cast: (6-24 mo)

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10
Q

Legg-Calvé-Perthes Disease

Occurs in who?

A

ball-shaped head of the thighbone (femoral head) temporarily loses its blood supply

-Poor blood supply-> weakened bone-> gradually breaks apart and can potentially lose its round shape

Occurs in children 3 to 12 years of age (peak incidence 5-8)
Symptoms-Gradual onset, limping, hip pain

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11
Q

Legg-Calvé-Perthes

Diagnosis?
Treatment?

A

Diagnostic evaluation- MRI

Treatment- Rest (no weight) and NSAIDS

-Outcome- Body eventually restores blood supply to the ball and the ball heals (surgery rarely)

-Potential long-term complication of arthritis

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12
Q

Slipped Capital Femoral Epiphysis (SCFE)

Involves what growth plate?
Occurs at what age?
What factors cause it to occur?

A

Spontaneous displacement of the femoral head (epiphysis) from femoral neck.

Involves the femoral growth plate.

Occurs between 10-16 yrs old

Usually idiopathic, multifactorial
OBESITY
Puberty hormone changes,
Bone changes.

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13
Q

Slipped Capital Femoral Epiphysis (SCFE)

Symptoms?
Treatment?

A

Hip pain
Limping
Difficulty bearing weight on the affected leg

Treatment:
Surgery
Screw fixation is standard of care

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14
Q

Osteogenesis imperfecta (OI)
“Brittle Bone Disease”

What is it?
Symptoms? four

A

Genetic condition that causes fragile bones
affects connective tissue (>20 types OI)

Clinical features:
-Blue sclera
-Fracture early on in childhood
-Short stature
-Ligamentous laxity

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15
Q

Therapeutic Management of OI

A

Drugs- bisphosphonate therapy
-Pamidronate (Aredia)
-Promote bone density and prevent fractures

Nursing care management
-Caution with handling to prevent fractures
-PT/OT recommendations

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16
Q

Cerebral palsy (CP)

Etiology?
Symptoms are what?

A

PERMANENT MOVEMENT, BALANCE, AND POSTURE DISORDER that appear in infancy or early childhood.

Etiology: Abnormal development or damage to the brain that occurs before, during, or after delivery that affects both the neurological and musculoskeletal systems.

Symptoms are PERMANENT and NONPROGRESSIVE and vary from mild to severe defects.

17
Q

Risk Factors for Cerebral Palsy

A

Premature
Low birth weight
CNS malformation
Asphyxia during birth (lack of oxygen)
Intracranial hemorrhage after birth

18
Q

Cerebral Palsy (CP) symptoms

A

Delay in achieving developmental milestones

Difficulty swallowing or uncontrolled drooling

Poor sucking

Clenched fists beyond 3 months of age

Stiff or rigid limbs

Arching of back

19
Q

Types of Cerebral Palsy

Spastic?
Dyskinetic?
Ataxic?
Mixed?

Which one is most common?

A

Spastic: stiffness and tightness in muscles
(most common type).

Dyskinetic: involuntary, uncontrolled,
repetitive movements.

Ataxic: incoordination with decreased muscle tone

Mixed: Symptoms of Spastic, Dyskinetic, and Ataxic

20
Q

Therapeutic Management of cerebral palsy

Medications?
two

A
  1. Botox - reduce spasticity to improve mobility and pain control
  2. Baclofen ( Reduce Spasms-Implanted pump for intrathecal administration -(into spinal canal)
21
Q

neural tube defects

Attributed largely to?

Elavated what in pregnant women indicates neural tube defect?

A

congenital anomalies that occur due to the incomplete closure of the neural tube, which forms the brain and spinal cord.

Attributed largely to insufficient Maternal Folic Acid
-Recommendation mother takes 400mcg PO QD prenatally

Elevated Alpha-fetoprotein (AFP) in pregnant women indicates neural tube defect.

22
Q

Neural Tube Defects types

Spina Bifida Occulta?
Meningocele?
Myelomeningocele?

A

Spina Bifida Occulta:
-Dimple or hair noted at the base of the spine.
-No protrusion of meninges.
-Ultrasound to determine if there is any underlying problem.
-Treatment usually not needed.

Meningocele:
-protrusion of meninges, CSF, no nerves within a sac of skin.

Myelomeningocele:
-protrusion of meninges, CSF, and nerves within sac of skin.

23
Q

Therapeutic Management of neural tube defects

Surgical closure?
Nursing management?
Precausions?

A

Early surgical closure of the myelomeningocele sac within 24-48 hours

Nursing management
-Side lying or prone position to protect defect
-Prevent infection (cover w/ moist sterile dressing)
-LATEX precautions- all spina bifida patients
-Antibiotics

24
Q

Guillain-Barré Syndrome (GBS)

Ascending Paralysis!!

A

Rare autoimmune disorder where the body’s immune system attacks PNS

Etiology unknown

Usually preceded by viral illness that induces an abnormal autoimmune response targeting peripheral nerves.

25
Q

Therapeutic Management of GBS

A

Treatment is supportive
Respiratory support

Steroids, IV immunoglobulin, and plasmapheresis (plasma exchange).

Low-molecular-weight heparin, gabapentin, (neuropathic pain), analgesics

Risk of recurrence low

26
Q

Duchenne Muscular Dystrophy

What kind of disorder?
Characterized by what?
generally affects who and around what age?

A

Genetic disorder characterized by PROGRESSIVE muscle degeneration and weakness.

It primarily affects boys (x-linked), typically becoming evident around ages 3 to 5.

27
Q

Clinical signs and symptoms of Duchenne Muscular Dystrophy (DMD)

Affects what limbs?
Wheelchair bound by what age?
Average life expectancy?

A

Progressive muscle weakness*, wasting, and contractures

Affects proximal before distal limb muscles, & lower extremities before upper extremities

Waddling gait, frequent falls, Gower’s sign

Pts. usually wheelchair bound by ~12 years of age

Average expectancy 28 years

28
Q

What is Gower’s sign?

A

Child uses their hands to “walk” up their Thighs .gradually straightening their legs until fully upright.