MSK Disorders Flashcards

Osteomyelitis, Osteoporosis, Osteoarthritis, Rheumatoid Arthritis, Arthroplasty etc...

1
Q

What is the infection of bone caused by ,most common organism, Staphylococcus aureus.

A

Osteomyelitis

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2
Q

Inflammation occurs, followed by ischemia, bone tissue becomes necrotic - often resulting in bone abscess.

A

Etiology of Osteomyelitis

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3
Q

S/S of Osteomyelitis are…?

A

ACUTE- less than 4 weeks, Fever, Inflammation (redness, heat, swelling & pain)

CHRONIC- more than 4 weeks, Ulceration, Drainage, Localized Pain

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4
Q

What are the Diagnostic Testing for Osteomyelitis?

A
  • Blood test for Leukocytosis (increased WBC’s)
  • Elevated ESR ( Inflammation marker)
  • Bone Biopsy for infection
  • Blood Culture
  • MRI, X-ray, CT scan
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5
Q

What is the Nursing Care for Osteomyelitis?

A
  • Long term antibiotics
  • Sterile Dressing Changes
  • Hand Hygiene
  • Pain Management
  • Pain Control (bone pain)
  • May result in amputation

… infection in one part of the body may migrate into another bone in the body

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6
Q

Metabolic bone disorder with bone density loss, resulting in fragile bones which are more likely to fracture.

A

OSTEOPOROSIS

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7
Q

What is the Etiology of Osteoporosis?

A

• Peak bone density is reached @ 30-35yrs old. After peak yrs, bone breakdown exceeds the rate of bone build up, resulting in irreversible bone loss that increase with age.

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8
Q

What common cause results in bone loss in women?

A

In postmenopausal women, the absence of estrogen appears to slow down the absorption of calcium, resulting in bone loss

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9
Q
  • Is the most common & is not associated with another disease
A

PRIMARY

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10
Q
  • Results from an associated medical condition or procedure
A

SECONDARY

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11
Q

Risk factors for Osteoporosis have both non-modifiable & modifiable risk factors……what are the non-modifiable risk factors??

A
  • Gender ( female) • Aging. • Caucasian/Asian
  • Petite (small/ short) body build
  • Postmenopausal status. • Low testosterone
  • History of/ Family History of Osteoporosis or Fractures.
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12
Q

Risk factors for Osteoporosis have both non-modifiable & modifiable risk factors……what are the modifiable risk factors???

A
  • Smoking. • Excessive Alcohol use.
    • Anorexia nervosa • Nutrition. • Low Ca+ and Vit D.
    • Excessive Caffeine, protein or sodium consumption
  • Sedentary lifestyle (not exercising enough/ constantly sitting)
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13
Q

How to prevent Osteoporosis?

A

Adequate Ca+ & vit D, weight bearing exercises, muscle- strengthening exercises, advoid alcohol and smocking.

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14
Q

S/S of Osteoporosis?

A
  • Often undetected until a fracture occurs. • pain
  • decrease in hight • Spinal Deformities.
  • ADL’s may become limited. •
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15
Q

What Diagnosis Testing are use to detect Osteoporosis?

A
  • DEXA (bone density test). • X-ray
    • Calcium & Vit D may be decreased.
  • Increased🔺 Phosphorus
  • APL may be elevated w/ severe bone loss.
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16
Q

What treatments are used with Osteoporosis?

A

Control risk factors to prevent bone loss

Supplements

Antiresorptive medications

Anabolic meds

Diet change

Exercise

Fall prevention

17
Q

What nursing care INTERVENTIONS are pit in place for OSTEOPOROSIS?

A

Education on risk factors, pain reliefs & medication teaching. Increase fluids with meds.fall prevention (free of clutter on the floor ex: throw rugs, use non skid socks or shoes, using assistant devices.

18
Q

Metabolic bone disease, increased breakdown and formation of bone, results in abnormally formed- weak bones causing deformities (punched out areas of bones),pain fractures and osteoarthritis.

A

PAGAT’S DISEASE

19
Q

Etiology of pagat’s disease ?

A
  • Mainly effects men
  • X-rays shows punched out areas, bone scan shows how widespread it is , increased ALP due to Osteoblasts activity
  • PAIN IS THE MAJOR SYMPTOM, #1 ISSUE SEVERE PAIN
20
Q

Treatments for pagat’s disease?

A

NO CURE, but NSAID’s , bisphosphonates reduce bone resorption, calcitonin decrease bone loss

Encourage exercise as tolerated, joint replacement may be recommend.

21
Q

Nursing care for pagat’s disease?

A

Provide pain relief, supportive system care, and education and teaching.

22
Q

Systemic connective tissue disorder from the build-up of uric acid, more common in males. Uric acid is the waste product resulting from the breakdown of proteins (PURINES).

A

GOUT

23
Q

Etiology of gout?

A

Urate crystals are formed and deposited in the joints causing severe inflammation.(mainly BIG TOE).
Urate deposits can develop in the urinary system causing renal calculi (kidney stones) and under the skin cause tophi.

24
Q

What are the acute and chronic S/S of Gout?

A

Acute gout- one or more severely inflamed joints, attack usually being in the great toe, joint is edematois, red, hot, and extremely painfully.

Chronic gout- may not be obvious symptoms, renal stones may develop.

25
Q

What’s the Diagnostic Testing for Gout?

A

Elevated uric acid and joint fluid analysis to identify uric acid crystals.

26
Q

What are the theraputic interventions for Gout?

A

Acute attack medications-

° NSAIDs

° steroids

° colchicine > gout with diarrhea

° Indocin _ NSAIDs for treatment of acute and chronic gout.

° DAILY MEDS: febuxostate and allopurinol (zyloprine) to decrease acid production

° HERBAL REMEDY: Cherry Juice

27
Q

What nursing interventions/ teaching for Gout?

A

Analgesics meds NSAID’S (ASA)

Diuretics

Avoid alcohol consumption

Increase fluids to prevent renal calculi

**ADVOID HIGH PURINES (PROTEINS) FOODS, ex: ORGAN MEAT, SHELLFISH, ANCHOVIES, SARDINES, HARRIN (FISH)

28
Q

Occurs when the articular cartilage and bone ends of joints deteriorate due to the wear of aging. Joint space narrows, bone spurs develop, joint may become inflamed.

A

OSTEOARTHRITIS

29
Q

What are the risk factors of OA?

A

Severe risk factors include:

  • Age
  • Obesity
  • Activities that create mechanical stress on synovial joints.
30
Q

S/S of OA?

A
  • Joint pain or stiffness
  • Problems with ADL’s
  • Bony nodes on the joints of their fingers (HEBERDEN’S AND BOUCHARDS)
  • PAIN WITH ACTIVITY
31
Q

DX testing for OA?

A

• X-ray • CT. • MRI. • Analysis of synovial fluid

32
Q

Theraputic measures for OA?

A
  • No Cure, goal is to maintain optimum independence and pain management
  • Synvisc injection treats osteoarthritic knees,the medication acts like healthy synovial fluid,helps with pain and mobility
  • NSAID’s • Topical Creams.
  • Rest and exercise (balanced). • Diet. • Surgery
  • Heat and Cold Therapy. • Alternative therapies
33
Q

Chronic, progressive, systemic inflammatory disease, Destroys synovial joints and other connective tissues including amor organs

A

RHEUMATOID ARTHRITIS (RA)

33
Q

Chronic, progressive, systemic inflammatory disease, Destroys synovial joints and other connective tissues including amor organs

A

RHEUMATOID ARTHRITIS (RA)

34
Q

What’s the Etiology of RA?

A

Inflammatory cells and chemicals cause synovitis occurs 🔻

Synovium thickens and fluid accumulates (as inflammation progresses) 🔻

Destructive pannus erodes joint cartilage, destroying joint bone 🔻

Pannus is covered into bony tissue resulting in loss of mobility 🔻

Any connective tissue can be affected by RA

PAIN INCREASES WITH REST AND DECREASE WITH ACTIVITY

35
Q

S/S of RA?

A

EARLY: -bilateral, symmetrical joint inflammation

  • Red,.warm, swollen, stiff, painful joints
  • stiffness after resting
  • activity 🔻 pain & stiffness
  • low grade fever, weakness, fatigue, Anorexia, systemic s/s

LATE:- joint deformity. - Secondary Osteoporosis

**Pain 🔻 with activity

36
Q

DX testing for RA ?

A

Increased WBC’S and platelets by blood test

Other findings include….
Rheumatoid factor (RF) - is present with RA
-decreased RBC’s
-Decreased C4 component
- increased ESR (inflammation marker)
- Positive + for Antinuclear antibody (ANA) & C-Reactive protein ( CRP)
- X-RAY, MRI, BONE SCAN
- ARTHROCENTESIS* (removal of fluid from joint)