MSK Flashcards
Stages of Fracture Healing
- Haematoma
- Inflammation –> Granulation tissue
- Cartilage callus
- Lamellar bone (primary
- Remodelling –> secondary bone
Factors that affect fracture healing
- Age
- malnourished
- local pathology
- neurological condiitons
- NSAIDs
- smoking
- DM
- Hormones - Cortisol, GH, Oestrogen, T3 &4, PTH
Describing a fracture
- Patient details
- Pattern of fracture
- anatomical location
- Intra/extra - articular
- Deformity (translation/ angulation)
- Soft tissue involvement
- Specific # classification
General priniciples of fractures management
4R's Resuscitation - & assess NV status Reduction Restriction Rehabilitation (PT, OT, home help)
Open fracture management
Analgesia: M+M
Assess: NV status, soft tissues, photograph
Antisepsis: wound swab, copious irrigation, cover with
betadine-soaked dressing.
Alignment: align # and splint
Anti-tetanus: check status (booster lasts 10yrs)
Abx - Fluclox 500mg IV/IM or Co-amox
Mx: debridement and fixation in theatre
Gustillo’s classification of open fractures
- Wound <1cm in length
- Wound ≥1cm c¯ minimal soft tissue damage
- Extensive soft tissue damage
a) periosteal stripping
b) requires free tissue flap
3) NV damage
Major complication of open fractures
Wound infections and gas gangrene (clostridium perfringes)
± shock and renal failure
Rx: debride, benpen + clindamycin
Methods of reduction in fractures
Manipulation / Closed reduction
Under LA, RA, GA
Traction to disimpact
Manipulation to align
Traction
Employed to overcome contraction of large
muscles: e.g. femoral #s
Skeletal traction vs. skin traction
Open reduction (and internal fixation) Accurate reduction vs. risks of surgery Intra-articular #s Open #s 2 #s in 1 limb Failed conservative Rx Bilat identical #s
General complications of fractures
Tissue Damage
Haemorrhage and shock
Infection
Muscle damage → rhabdomyolysis
Anaesthesia
Anaphylaxis
Damage to teeth
Aspiration
Prolonged Bed Rest Chest infection, UTI Pressure sores and muscle wasting DVT, PE ↓ BMD
Specific fracture complications
Immediate Neurovascular damage Visceral damage Early Compartment syn. Infection (worse if metal) Fat embolism → ARDS Late Problems - union AVN Growth disturbance Post-traumatic OA CRPS Myositis ossificans
Pathophysiology of compartment syndrome
Raised pressure within any enclosed facial space leading to localised tissue ischaemia
Oedema following # → ↑ compartment pressure → ↓
venous drainage → ↑ compartment pressure
If compartment pressure > capillary pressure →
ischaemia
Muscle infarction →
- Rhabdomyolysis and ATN
- Fibrosis → Volkman’s ischaemic contracture
Presentation of compartment syndrome
Pain > clinical findings (not relieved by analgesia)
Pain on passive muscle stretching
Warm, erythematous, swollen limb
↑ CRT and weak/absent peripheral pulses
shiny skin
tense compartment
+/- paraesthesiae
Prevention of compartment syndrome
avoid repeated manipulation
use open plaster initially
elevate leg
mobilise early
Rx of compartment syndrome
Elevate limb Remove all bandages and split/remove cast Get senior help Prophylactic Abx and Analegesia Fasciotomy
Problems with union
Delayed Union: union takes longer than expected
Non-union: # fails to unite - Hypertrophic Bone end is rounded, dense and sclerotic - Atrophic Bone looks osteopenic
Malunion: # healed in an imperfect position
Poor appearance and/or function
Causes of non/delayed union
Ischaemia: poor blood supply or AVN
Infection
↑ interfragmentary strain
Interposition of tissue between fragments
Intercurrent disease: e.g. malignancy or malnutrition
Management of delayed/ non-union
Optimise biology: infection, blood supply, bone
graft, BMPs
Optimise mechanics: ORIF
Features of AVN
Death of bone due to deficient blood supply.
Sites: femoral head, scaphoid, talus
Consequence: bone becomes soft and deformed
→ pain, stiffness and OA.
X-ray: sclerosis and deformity.
Features of myositis ossificans
Heterotopic ossification of muscle @ sites of
haematoma formation
→ restricted, painful movement
Commonly affects the elbow and quadriceps
Can be excised surgically
Definition of complex regional pain syndrome
Complex disorder of pain, sensory abnormalities,
abnormal blood flow, sweating and trophic changes in
superficial or deep tissues.
No evidence of nerve injury.
(type 2 - persistent pain following injury caused by nerve lesions)
Causes and presentation of CRPS
Causes
Injury: #s, carpal tunnel release, ops for Dupuytren’s
Zoster, MI, Idiopathic
Presentation
Wks – months after injury
NOT traumatised area that is affected: affects a
NEIGHBOURING area.
Lancing pain, hyperalgesia or allodynia
Vasomotor: hot and sweaty or cold and cyanosed
Skin: swollen or atrophic and shiny.
NM: weakness, hyper-reflexia, dystonia, contractures
Rx of CRPS
Usually self-limiting
Refer to pain team
Amitryptilline, gabapentin
Sympathetic nerve blocks can be tried.
Salter harris classification
- Straight across physis
- Above (metaphysis)
- Lower (epiphysis)
- Through (all 3)
- CRUSH
TYPE 2 most common
Risk factors for osteoporosis
SHATTERERED Steroids Hyper- para/thyroidism Alcohol and Cigarettes Thin (BMI<22) Testosterone low Early Menopause Renal / liver failure Erosive / inflame bone disease (e.g. RA, myeloma) Dietary Ca low / malabsorption, DM
Age