Movement Disorders Flashcards
Quadriplegia/Tetraplegia
whole body involved in varying degrees
Diplegia
- Whole body involved but LE> UE symmetrical or asymmetrical distribution
- Good head control and min/mod involvement of UE
Hemiplegia
One side (face, UE, LE) involved
Monoplegia
one extremity involved
Paraplegia
- Used more for spinal cord
- Involvement of trunk & LE to varying degrees
Name some Objective Muscle Tone Grading Scales
- Modified Ashworth Scale
- Tardieu Scale
- Wilson Howle Assessment of Motor Tone
What are the structures involved in spasticity?
- Motor cortex or white matter projections to/from corticosensorimotor areas of brain, pyramidal system, corticospinal tracts
True or False: Spasticity would be characterized by an increase in muscle tone of muscle groups
True
Patients would present with hypertonicity
With spasticity when may tension increase?
- Increased tension when lengthen (spastic catch/clasp-knife response)
- Increased with rate/velocity & position change
True or False: A patient with spasticity will present with firm/tense on palpation, and increased DTR/MSR
True
In a patient with spasticity PROM will be?
Decreased with atypical firm end feel
A patient with spasticity will have (BLANK) co- contraction and (BLANK) reciprocal inhibition
- Increased
- Decreased
T/F: Spasticity presents with distribution of either more flexors or extensors and usually asymmetrical
True
Why may a patient with spasticity have disuse atrophy?
Because they are only working through a small range activity
What structures are involved when a patient has rigidity?
Basal ganglia
What is rigidity?
- Increased muscle tone of muscle groups that is continuous or sometimes intermittent
- Sustained muscle contraction even when relaxed
- Increased tension uniformly & immediately as lengthens
What will occur in a patient with rigidity with a change of rate/velocity and position?
- No change or increased DTR/MSR
- No change
How will PROM present as with a patient with rigidity? And how is the is distributions of flexors & extensor?
- Decreased PROM with atypical firm end feel
- More equal distribution of flexors & extensors & symmetrical
A patient with rigidity will have (BLANK) co contraction & (BLANK) reciprocal inhibition
- Increased
- Decreased
T/F: A patient with rigidity can present with disuse atrophy
True
What is lead pipe rigidity?
Constant resistance to movement throughout ROM
What is cogwheel rigidity?
Alternative episodes of resistance & relaxation through ROM
What is the broad definition of akinesia/hypokinesia/bradykinesia grouped together?
Disinclination of patient to use affected body part or do natural movement of those parts, no/slow imitation & execution of movement
What is the position of decorticate posture?
- UE flex/ LE extend
What is the position of decerebrate posture?
- UE extend/ LE extend
What is the position of opisthotonic?
Extreme extension positioning
What are the involved structures in a patient with hypotonicity/ atonia?
UMN system, cerebellum, unknown sites
A patient with hypotonicity/atonia will present with:
- (BLANK) muscle tone
- (BLANK) tension with lengthened
- (BLANK) DTR/MTR
- (BLANK) change with rate/velocity & position
- Decreased
- Decreased
- Decreased
- Possible change
A patient with hypotonicity/ atonia will present as (BLANK) on palpation?
Soft/ mushy
T/F: a patient with hypotonicity will have atrophy, weakness, decreased endurance & some tonic reflexes
True