Motor System Impairments Flashcards

1
Q

Motor System Impairments - what are the primary motor system impairments?

A
  • Motor Weakness [paresis]
  • Muscle tone abnormalities
  • Coordination problems

Motor Weakness:

  • strength = generate sufficient force for posture/movement
  • weakness = inability to generate normal levels of force
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Motor System Impairments - describe some neural factors that influence motor weakness:

A
  1. Decreased number of motor units (MUs) recruited
  2. Predominantly type 1 MUs recruited
  3. Decreased MU firing rate [remember, firing rate influences Ca and thus muscle force]
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Motor Weakness: Paresis

A

‘Paresis’ results from decreased MU recruitment; difficultly in recruiting or modulating output of motor neurones

Results from lesion/s within descending motor pathways= decreased descending drive

Because of decreased descending drive, including serotonergic/noradrenergic drive to SC

  • cant recruit high threshold motor units [remember PICs]
  • difficulty modulating firing rate when increasing force

More apparent in distal muscles

Produces peripheral changes in muscle, exacerbates problems

Incorrectly used to believe:

  • ST training would cause problems with muscle tone
  • no reason to examine, evaluate or treat weakness

[motor weakness seen more in distal segments - distal segments more affected]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Motor Weakness: Treatments:

A

Biofeedback e.g. Sensory amplitude electrical stimulation over muscles (10s on/10s off)

Functional electrical stimulation [FES]

  • applied to wrist extensors for 30min 3x/day to strengthen extensors
  • combined with task training of upper extremity [for lifting weight] for 15 min 2x/day to improve hand function
  • applied to perineal nerve in hemiplegic patients with foot drop for tib ant muscle during gait.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Muscle Tone Abnormalities:

A

Muscles tone is characterised by a muscle’s resistance to passive stretch
A certain level of muscle tone is normal

Flaccidity[Spasticity>Rigidity]- hypertonicity

Damage to descending tracts results in increased alpha motor neurone excitability that causes:

  • increased muscle tone [enhanced tonic stretch reflex activity; velocity dependent]
  • exaggerated tendon jerks [hyper excitability of physical stretch reflexes]

Clinical Term - spasticity
Results in hyperactive stretch reflex, abnormal limb postures, excessive coactivation, clonus and stereotyped movement synergies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Muscle Tone Abnormalities: Rigidity

A

Another form of hypertonicity
Increased resistance to passive limb movement, not velocity dependent
Predominantly affects flexor muscles of trunk and limbs
Problems with gait, speech, posture, eating

{may result from disinhibition of basal ganglia}

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Muscle Tone Abnormalities: Hypotonicity

A

Decreased stiffness in muscle lengthening
Flaccidity is a total loss of muscle tone
Exhibited by many patients, e.g. Spinocerebellar lesions, Down syndrome.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Muscle Tone Abnormalities: Spasticity

What does altering a patients position do?

A

Mild spasticity treated with therapeutic ex, splinting and orthotics
Severe spasticity treated with chemodenervation [e.g. Botox], surgery and medications
Ex treatments can be combined with muscle stimulation [high or low freq]

Altered position:
Supine facilitates extensor tone
Prone facilitates flexor tone
Side-lying inhibits effect of asymmetrical tonic neck reflex; facilitates bilateral symmetrical activation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Coordination Problems: Name 2 activation and sequencing problems

A

Results from disruption of activation, sequencing, timing and scaling of muscle activity

  1. Flexion synergy of upper extremity in stroke:
    Scapular retraction
    Shoulder abduction & external rotation
    Elbow flexion
    Forearm supination
    Wrist & finger flexion
2. Extension synergy of lower extremity:
Hip extension, addiction & internal rotation
Knee extension 
Ankle plantar flexion & inversion
Toe plantar flexion
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Coordination Problems: Timing Problems

A

Initiating problems, slowed movement time, and problems terminating movement. [initiation problems - Reaction time]

Neuromuscular:

  • inadequate force generation
  • rate of force generation
  • insufficient ROM to allow movement
  • motivation to move
  • abnormal postural control

Cognitive:

  • cant recognise command or signal to move
  • difficulty recalling and selecting movement plan
  • difficulty assembling and initiating plan
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Coordination Problems: Name some problems patients have terminating movement:

A
  • inability to stop movement
  • inability to change direction
  • inability to control appropriate forces of the agonist muscle at end of ROM
  • inadequate timing and force generation of antagonist muscle
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Coordination Problems: Name the 3 scaling force problems

A
  • Dysmetria = problems judging distance or ROM, cant scale forces appropriately, patients with cerebellar lesions
  • Hypermetria = overestimation of force or ROM
  • Hypometria = underestimation of force or ROM
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Coordination Problems Helped by?

A

Repetition and practice of functional task specific movement [therapist provides correction feedback]
Weight bearing activity for coordination in lower extremities [e.g. Yoga]
Repetition of timed coordination tasks [e.g. Tai Chi]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Ataxia - definition?

Why is the Cerebellum important ?

A

Lack of movement coordination, motor dysfunction condition with neurological basis, often involves cerebellum.

Cerebellum important for:

  • Planning of a movement
  • Control of posture & equilibrium
  • Control of smooth limb movement, coordination [compares descending with ascending info]
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Ataxia - Cerebellar

May exhibit all of the below:

A
  • Vestibulo cerebellar dysfunction = postural instability, wide stance
  • Spino cerebellar dysfunction = ‘drunken sailor’, gait, gaze nystagmus [stare or gaze at object]
  • Cerebrospinal cerebellar dysfunction = intention, tremor, slurred speech, unpredicatable and inconsistent hand writing.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Define Sensory Ataxia:

A

Loss of proprioception info
Stomping gait, postural instability
Remember, visual input is crucial for sensory feedback [Romberg’s positive]

17
Q

Define Vestibular Ataxia:

A

Dysfunction of the vestibular system
Acute/unilateral: vertigo, nausea and vomiting
Slow-onset, chronic bilateral: may only show dysequilibrium

Mild form - dystaxia

18
Q

Ataxia - Treatment

A

Re-educate coordination, postural control, gaze stability [eye and head training exercises], correct use of sensory input
Stretching & strengthening
Avoid fatiguing exercises if treating patient with hereditary Friedrich’s ataxia, but ex still mandated.

19
Q

Tendinosus

A

Damage to [usually chornic] or degeneration of tendon

20
Q

Athetosis

A

Slow, writing movements

21
Q

Chorea

A

Irregular, rapid, uncontrolled movements

22
Q

Myoclonus

A

Brief twitches or spasms