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Integumentary (Exam3) > Module 7: Alopecia (Weeks 8 & 9) > Flashcards

Module 7: Alopecia (Weeks 8 & 9) Flashcards

1
Q

List the causes of acquired canine alopecia:

A
  • Pattern Baldness
  • Color dilution Alopecia
  • Black Hair Follicular Dysplasia
  • Seasonal Flank Alopecia
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2
Q

Hair Follicles
Most omnivores and herbivores have simple follicles which means …

A

each infundibulum contains 1 hair shaft

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3
Q

Hair Follicles
Carnivores (dogs & cats) and rabbits, have compound follicles which means …

A

infundibulum contains multiple hair shafts
- Some hair follicles in the compound follicle have a large diameter – Primary or guard hairs – but the majority have a small diameter – secondary or undercoated hairs

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4
Q

Hair Follicles
Human & Sheep depending on body location have …

A

simple or compound follicles
- Human: head mostly compound, body simple

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5
Q

Hair Cycle
Hair follicles show intermittent activity:
- Is the period of active growth => _____________
- Is the transitional phase => _____________
- Is the resting phase of the hair cycle => _____________

A
  • Anagen
  • Catagen
  • Telogen
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6
Q

Factors Influencing Hair Cycle
The Relative duration of the cycle varies with:

A
  • Age
  • Region of the body
  • Breed
  • Sex
    (Can be modified by a variety of extrinsic and intrinsic factors)
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7
Q

Factors Influencing Hair Cycle:
- Photoperiod
- Ambient temperature
- Hormones (thyroidal, gonadal, adrenal, pituitary and pineal)
- General state of health

A

Extrinsic Factors

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8
Q

Factors Influencing Hair Cycle:
- Growth factors and cytokines

A

Intrinsic factors

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9
Q

Definition:
Is a partial or complete lack of hair in areas where it is normally present

A

Alopecia
Classification:
- Congenital
- Acquired
- Auto-inflicted
- Infectious
- Inflammatory Reactions
- Miscellaneous
- Follicular dysplasia
- Endocrinopathies

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10
Q

Clinical Approach: Alopecia
History and general physical examination directed to detect abnormalities in other organs
List Clinical Signs:

A
  • PU/PD
  • Pendulous abdomen
  • Testicular asymmetry or cryptorchidism
  • Vulvar enlargement
  • Changes in activity level
  • Weight gain
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11
Q

Clinical Approach
(T/F) If pruritus is present, the cause of pruritus should be investigated first

A

True
- If absent: Pattern of hair loss, Presence of inflammation, Presence of lesions
- Skin scraping, skin cytology, dermatophyte culture

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12
Q

Clinical Approach: Breeds
Alopecia X

A

Pomeranians

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13
Q

Clinical Approach: Breeds
Pattern baldness

A
  • Dachsunds
  • Boston Terriers
  • Chihuahuas
  • Whippets
  • Manchester Terriers
  • Greyhounds
  • Italian Greyhounds
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14
Q

Clinical Approach
List some of the causes for failure to regrow hair after clipping:

A
  • Hypothyroidism
  • Hyperadrenocorticism
  • Alopecia X
  • Post clipping alopecia
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15
Q

List Non-Inflammatory Alopecia:

A
  • Pattern Baldness
    • Miniaturization of the hair follicle
  • Follicular Dysplasias
    • Color Dilution Alopecia
    • Black hair follicular dysplasia
    • Seasonal flank Alopecia
  • Endocrine dermatopathies
    • Sertoli cell Tumors
    • Hypothyroidism
    • Cushing’s disease
    • Hair cycle arrest (Alopecia X)
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16
Q

Pattern Baldness:
What are the Four Syndromes?

A
  1. Most Common: alopecia pinnae, ventral neck, ventrum, caudomedial thighs
    (alopecia remains restricted to these areas)
  2. Pinnal alopecia of Dachshunds: Complete pinnal alopecia by 8-9 year old
  3. Caudal thigh alopecia of greyhounds
    - Bald thigh syndrome in sighthounds
    - Possible causes have been proposed
    - environmental, trauma, stress
  4. American Water Spaniels - most likely a follicular dysplasia (50% of cases)
    • Alopecia neck, caudal thighs and trunk
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17
Q
  • Dachshunds
  • Boston Terriers
  • Chihuahuas
  • Whippets
  • Manchester terriers
  • Greyhounds
  • Italian Greyhounds
    These are the most common breeds with …
A

Pattern Baldness

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18
Q

History - Early onset, breed predisposition
Rule out:
- Hair follicle dysplasia
- Sex hormone dermatoses
- Hypothyroidism
- Hyperadrenocorticism
- Alopecia areata (autoimmune)
Histopathologic examination - Biopsy samples from affected and normal skin

A

Pattern Baldness

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19
Q

Pattern Baldness: Treatment

A
  • Oral melatonin: good response in approximately 50% of the cases
  • Doses: 3-6 mg PO q 8-12 h
    - Give for 2-3 months before evaluating response
  • Low level laser or phototherapy
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20
Q
  • “Color mutant alopecia,” “Blue dog disease”
  • Inherited autosomal recessive disorder – abnormalities in melanin transfer and storage
A

Color Dilution Alopecia
- Dogs with color-diluted coats (blue or fawn) are affected e.g. Dobermans, Italian greyhounds, chihuahuas, labradors

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21
Q
  • Hypotrichosis to alopecia affecting diluted areas
  • Clinical signs start between 6 months and 3 years
  • The rate of hair loss is variable
  • Prone to develop bacterial folliculitis
    These are clinical signs for …
A

Color Dilution Alopecia

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22
Q
  • Breed and coat color are suggestive
  • Trichogram: shows hairs with large melanin clumps, causing distortion and fracture of the hair shaft
  • Histopathologic examination to confirm diagnosis
    These are the …
A

Diagnosis for Color Dilution Alopecia

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23
Q
  • Seen in bi- or tricolored breeds and solid-colored (black) breeds
  • Coat changes are noted as early as 4 weeks
  • Progressive hair loss until all black hairs are lost
A

Black Hair Follicular Dysplasia

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24
Q
  • Only the black coat affected is very suggestive
  • Rule out demodicosis and dermatophytosis
    - do skin scrapes
  • Histopathology
    - Melanin clumping
    These are the …
A

Diagnosis for Black Hair Follicular Dysplasia

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25
Q

Black Hair Follicular Dysplasia: Treatment

A
  • No effective treatment
  • Treat secondary infections if present
  • Do not use harsh shampoos, be gentle when grooming
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26
Q
  • Episodes of recurrent truncal hair loss
  • High incidence in some breeds suggests genetic influence
  • The seasonal nature and annual recurrence suggest the influence of photoperiod
    - Onset of alopecia occurs mostly between late autumn and early spring
A

Seasonal Flank Alopecia

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27
Q

List the Breeds more commonly affected by Seasonal Flank Alopecia:

A
  • Boxer
  • English bulldog
  • French bulldog
  • Miniture schnauzer
  • Airedale terrier
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28
Q
  • Ruling out another clinical differential diagnosis
    - Hypothyroidism
    - Demodicosis
    - Sex hormone imbalance
    - Alopecia X
  • Histopathology: multiple biopsy samples from
    the affected area(s)
    These are the …
A

Diagnosis for Seasonal Flank Alopecia

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29
Q

How would you manage Seasonal Flank Alopecia?

A
  • Difficult to evaluate response to treatment due to spontaneous hair regrowth
  • Oral Melatonin: 3-6 mg q 8 to 12 h for 1-2 months. Start before or shortly after the onset of alopecia
  • Cosmetic problem
  • Low level laser or phototherapy
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30
Q

What can help to confirm diagnosis of Seasonal Flank Alopecia?

A
  • Biopsy: mutiple samples from the affected and non-affected areas
  • Histopathology: dystrophic, atrophic and keartin-filled hair follicles with finger-like projections into the dermis
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31
Q

Alopecia X

A

Alopecia X

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32
Q

Term:
Has been recently adopted because the nature of this condition is currently unknown

A

Alopecia X
- “adrenal sex hormone imbalance”
- Hormonal imbalance or change in receptor sensitivity at the hair follicle level are suspected

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33
Q

List the breeds mostly associated with Alopecia X:

A

Pomeranians, Keeshond, Chow Chows, Samoyed, miniture poodles

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34
Q

What is the typical signalment for Alopecia x?

A
  • M/F, intact or neutered
  • Middle age to older
  • Specific to some breeds
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35
Q

List the clinical signs foe Alopecia X:

A
  • Bilaterally symmetric alopecia originating in frictional areas
  • Primary hairs are lost first followed by a variable loss of secondary hairs– “puppy-like coat”
  • Rump, perineum, caudal thighs, neck, tail
  • end-stage disease may result in total alopecia of the trunk, neck and proximal legs
  • Head and distal legs are usually spared
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36
Q

How do you rule out endocrinopathies?

A
  • ACTH stimulation test
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37
Q

Histopathology:
- Areas of most advanced hair loss

A
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38
Q

(T/F) Melatonin can be used for Alopecia X

A

True, one study showed hair regrow in 14/23 pomeranians with Alopecia X after 4 months

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39
Q

How long will it take to see improvements using Melatonin?

A

3 months

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40
Q

Alopecia X - Treatment:
- Blocks the excessive production of cortisol by reversibly inhibiting the action of the enzyme 3-beta hydroxysteriod dehydrogenase

A

Trilostane

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41
Q

(T/F) Microneedling also works as a treatment for Alopecia X

A

True

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42
Q

low level laser “phototherapy”

A
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43
Q

Alopecia - Hormonal

A
  • Sertoli Cell Tumor
  • Hypothyroidism
  • Hyperadrenocortisicism
  • Alopecia X
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44
Q

What does Sertoli cell tumors normally cause?

A

Male feminization Syndrome:
- Middle aged to older dogs
- Functional Sertolli cell tumors are most common in cryptorchid

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45
Q

List the breed predisposition to Sertoli cell tumors:

A
  • Boxers, Shelties, Collies, Weimaraners, Cairn terrier, Pekingese
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46
Q

List the clinical signs for Sertoli cell tumors:

A
  • Linear preputial dermatosis
  • Pendulous prepuce
  • Gynecomastia
  • Enlarged nipples
  • Prostate is often enlarged and it can be infected
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47
Q

How do we manage?

A
  • Castartion usually curative
  • Clinical response is seen within 3 months
  • Remission followed by relapse indicates functional metastases
  • Cisplatin can be tried for cases associates with metastases
  • Dogs with estrogen-induced bone marrow suppression have a poor prognosis
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48
Q

Hypothyroidism

A

Hypothyroidism

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49
Q
  • May develop because of a defect in any part of the hypothalamic - pituitary - thyroid axis
    - Congenital - uncommon (dwarfism)
    - Acquired - more common
    - Primary hypothyroidism - most cases
    - Secondary hypothyroidism - inadequate production of TSH from the pituitary gland < 5% of cases
A

Hypothyroidism

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50
Q

What is the most common endocrine disorder in dogs?

A

Primary Hypothyroidism
- lymphocytic thyroiditis or idiopathic atrophy
- Progressive destruction of the thyroid gland - over 1-3 years
- 75% gland must be destroyed before clinical signs develop

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51
Q

Signalment for primary hypothyroidism:

A
  • 7-year-old (middle age)
  • Spayed females and neutered male dogs are at increased risk
  • Golden retrievers, dobermans, great danes, shelties, and other breeds predisposed to hypothyroidism tend to develop the disease earlier (2-3 years)
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52
Q

List the dermatologic manifestations of hypothyroidism:

A
  • Thinning hair coat
  • Symmetrical non-inflammatory alopecia
  • Hyperpigmentation
  • Seborrhea
    • abnormal keratinization
      • greasy or dry
      • generalized
  • “rat tail”
  • Secondary infections
  • Myxedema (less often)
    • Accumulation of glycosaminoglycans
  • Failure to regrow hair after clipping
    ** occur in about 70% of dogs **
53
Q

What promotes the initiation of the anagen phase of the hair cycle?

A

Thyroid Hormones

54
Q

List the Neuromuscular abnormalities of Hypothyroidism:

A
  • Weakness
  • Facial nerve paralysis
  • Vestibular disease
    - its confused with ear infections
  • Seizures may occur secondary to serve hyperlipidemia
  • Megaesophagus and laryngeal paralysis have been reported but not proven
  • Insulin resistance
55
Q

What is the most severe form of hypothyroidism?

A

Myxedema coma
- impaired mental status
- treatment w/ intravenous I-thyroxine is recommended

56
Q

List finds of Diagnosis-Routine lab work:

A
  • Hypercholesterolemia
    - 75% of cases
  • Mild nonregenerative anemia
  • Hyponatremia
  • Elevated ALP, CK?
57
Q

Autoantibodies to T4 or T3:
- False elevation of affected hormone
- elevation may be marked (into hyperthyroid range) or may bring T4 into the normal range
- Does not affect fT4d

A

interfere with assays for T4 or T3

58
Q

Which are thyroid-sensitive teats?

A

T4 and fT4
(TSH (elevated) is less sensitive)

59
Q

(T/F) A combination of low T4 or fT4 and elevated TSH is very accurate for the diagnosis of primary hypothyroidism

A

True

60
Q

Treatment for hypothyroidism:

A

Levothyroxine
- Overdose signs: anxiety, panting, PD/PU/PP, diarrhea, pruritus

61
Q

Hyperadrenocorticism

A

Hyperadrenocorticism

62
Q

What is another endocrine disease common in dogs?

A

Hyperadrenocorticism
- Excess cortisol production by the adrenal glands
- Iatrogenic due to administration of glucocorticoids

63
Q

Naturally occurring:
(T/F) Most cases of hyperadrenocorticism are due to Adrenal tumors

A

False, it is due to PDH (80-85%)
- There are rare reports of dogs having both

64
Q
  • Pituitary microadenoma (80-90%) less than 1 cm in diameter
  • Pituitary macroadenomas
A

Pituitary dependent hyperadrenocorticism

65
Q

(T/F) Macroadenomas are much more likely to cause neurologic signs

A

True

66
Q

(T/F) Adenocarcinomas are very common

A

False, adenocarcinomas are RARE

67
Q
  • Increased ACTH production from the pituitary gland
A

Pituitary Adenomas

68
Q
  • Adenomas or adenocarcinomas affecting one (common) or both (rare) adrenal glands
A

Adrenal Tumors

69
Q
  • Thin skin and hyperkeratosis
  • Atrophic hair follicles and sebaceous gland
  • Fragile blood vessels (decrease collagen)
  • Delayed wound healing
  • Increase susceptibility to infections
  • Calcinosis cutis
    • calcium deposits
    • firm white papules
    • Treatment: Minocycline
A

Glucocorticoids and the skin

70
Q

List the signalment:

A
  • Middle-aged to older dogs (>6 years old) -> rare in young dogs
  • Any breed can be affected
  • 75% of dogs with PDH are < 20 kg
  • No sex predilection
71
Q

List the cutaneous signs:

A
  • Thin
  • Hypotonic
  • Easy bruising
  • Comedones
    • skin scrape
  • Poor healing
  • Hyperpigmentation
72
Q
  • Free catch urine sample
  • High Sensitivity
  • Low specificity
  • High negative predictive value
A

Urine cortisol:Creatinine ratio

73
Q
  • evaluates adrenocortical reverse
  • it can differentiate spontaneously from iatrogenic Cushing’s
  • Iatrogenic
    • minimal to no increase in cortisol levels
A

ACTH Stim test

74
Q
  • Good Screening test
  • it can be abnormal with non-adrenal illness
A

LDDST
- high dose dexamethasone suppression test
- endogenous ACTH
- Abdominal US
- CT/MRI

75
Q
  • Mitotane (Lysodren)
  • Trilostane (Vetoryl)
  • Surgery
  • Radiation
    This is the treatment for …
A

PDH

76
Q
  • Trilostane
  • Mitotane
  • Surgery
    This is the treatment for …
A

AT

77
Q

Module 7 MiniModule 2: Dermatophytosis

A

Module 7 MiniModule 2: Dermatophytosis

78
Q

(T/F) Dermatophytosis is zoonotic

A

True

79
Q

(T/F) Dogs are asymptomatic carriers of dermatophytosis

A

False, Cats are asymptomatic

80
Q

What is the most common dermatophyte in dogs and cats?

A

Microsporum canis
_ remember that it is in dogs AND cats

81
Q

List th ecommon dermatophytes:

A
  • Microsporum canis
    - cats
    - dogs
  • microsporum gypseum
    - cats
    - dogs
  • Microsporum persicolor
    - dogs
  • Trichophyton menragrophytes
    - cats - uncommon
    - dogs
82
Q

Dermatophyte Infection:
What is the infective form?

A

Arthrospore
- formed by segmentation and fragmentation of fungal hyphae

83
Q

Term:
Growing hair

A

Anagen

84
Q

Describe the infection of dermatophytes:

A

Arthrospore -> Invades anagen hair follicle -> Folliculitis (bacteria can also cause folliculitis & demodex)

85
Q

Stages of development:
- Adherence of arthroconidia to corneocytes (skin cells)

A

Stage 1

86
Q

Stages of development:
- Penetrate the stratum corneum (top layer)

A

Stage 2

87
Q

Stages of development:
- Dermatophyte invasion of keratinized structures

A

Stage 3

88
Q

Term:
is an inflammatory, pus-filled sore (abscess) that sometimes oozes

A

Kerion = Nodular Dermatophytosis

89
Q

What is the most common dog breed affected by dermatophytosis?

A

Yorkies

90
Q
  • Single or multiple erythematous, alopecic, dome-shaped, exudative nodules
  • Nodules can fistulate, ulcerate and drain serous to purulent debris with tissue grains
  • More superficial
A

Nodular Dermatophytosis = Kerion

91
Q

Feline Clinical Signs:

A
  • Alopecia
  • Erythema
  • Scale/crusting dermatitis
  • Papules - “miliary dermatitis”
  • Paronychia or nychodystrophy of single digit (claws affected)
    - Uncommon
  • Kerion - Uncommon
  • Draining nodules - Pseudomycetoma - Persian cats
92
Q
  • Subcutaneous nodules (one or multiple)
  • reported in Persian cats
  • Dorsum or tail base
  • Treatment:
    • Systemic therapy
    • +/- Surgical excision
A

Pseudomycetoma

93
Q

List the Primary Differential Diagnoses of Dermatophytosis :

A
  • Demodicosis
  • Staphylococcal folliculitis
  • Autoimmune - Pemphigus complex
    - crusting pattern
94
Q

Term:
a parasitic skin disease with medical and veterinary importance caused by the Demodex mites

A

Demodicosis

95
Q

(T/F) No one test is identified as “Gold standard” for dermatophytosis

A

True

96
Q

Diagnosis for dermatophytosis is confirmed by a number of tests, including:

A
  • Wood’s lamp
    - only 50% of M canis fluoresce an apple green color
    - remember that there are many different types of dermatophytes and not all fluoresce
    - False positives: crusts or medications can fluoresce
    - MAIN POINT: if hairs fluoresce, sample these for fungal culture (looking for hair shaft to light up)
  • Trichogram
  • Fungal culture (DTM)
  • Biopsy for histopathology
  • PCR
97
Q

What is Biopsy recommended for?

A
  • Nodular form (kerion, pseudomycetoma)
  • Suspected onychomycosis
  • Special stains may be needed for histopathologic diagnosis (Gomori methenamine silver or periodic acid-Schiff stains)
  • Can identify the presence of spores or hyphae; cannot identify species
98
Q

(T/F) PCR is going to detect viable and non-viable DNA

A

True

99
Q

Fast and accurate
- > 95% sensitivity, 99% specificity

A

PCR
- don’t use to check if treatment is working

100
Q

(T/F) Localized treatment is recommended for dermatophytosis

A

False, it is not recommended because spores are on the entire coat, not just where the lesion is
- lime sulfur dip (2%, malodor, patient cannot lick), miconazole/chlorhexidine shampoo

101
Q

(T/F) Systemic therapy for dermatophytosis can be used in patients with confirmed cases

A

True
- until you have 2 negative fungal cultures

102
Q

The “-zole” are going to prevent …

A

Ergosterol synthesis
- most commonly used: Itraconazole

103
Q
  • inhibits squalene epoxidase
A

Terbinafine
- tolerate it better

104
Q

(T/F) You cannot use Ketoconazole in cats

A

True, they will vomit everywhere and high-risk hepatotoxicity

105
Q

Module 7 MiniModule 3: Demodicosis

A

Module 7 MiniModule 3: Demodicosis

106
Q
  • Very common
  • Inflammatory parasitic disease
  • Characterized by the presence of larger-than-normal numbers of demodectic mites
A

Canine Demodicosis
- Demodex canis
- Demodex cornei
- Demodex injai
- Demodectic mange, Follicular mange, red mange

107
Q

What is the “in-between” size mite?

A

Demodex canis
- Hair follicle, sebaceous glands

108
Q

What is the smallest mite?

A

Demodex cornei
- Surface

109
Q

What is the largest mite?

A

Demodex injai
- Hair follicles, sebaceous glands
- longest of the mites

110
Q
  • Entire life cycle on skin (18-35 days)
  • Reside in hair follicles, sebaceous glands
  • Feeds on cells, sebum, and epidermal debris
  • Normal resident of dog’s skin and ear canal
  • Transmission occurs form the bitch to nursing puppies during first 2-3 days of life
A

Demodex canis

111
Q

IF most dogs harbor the mite as part of their normal skin fauna, why do some dogs develop the generalized disease?

A
  • Not completely understood
  • Possible role of mite-specific immunoincompetence
112
Q

List the Types of Canine Demodicosis:

A
  • Clinical Disease
    • Localized
    • Generalized (squamous, greasy, pustular, pododemodicosis)
      • Juvenile-onset
      • Adult-onset
  • Course and prognosis vastly different
  • Non-pruritic unless secondary infection
113
Q

Generalized Demodicosis:
- Scaly, alopecic patches
- Coalesce
- Erythema, crusting, follicular plugging, comedones
- Hyperpigmentation

A

Squamous

114
Q

Generalized Demodicosis:
- +/- alopecia
- Terriers
- D. injai
- Greasy skin may persist after resolution of demodex

A

Dorsal greasy skin

115
Q

Generalized Demodicosis:
- Very severe form (most)
- May be life-threatening
- Deep Gram-negative infection
- Alopecia, papules, pustules
- Furuncles, draining tracts, cellulitis, edema, ulcers, erosions, nodules
- Depressed, lethargic, febrile, septicemia
- Lymphadenopathy

A

Pustular

116
Q

Generalized Demodicosis:
- Concurrent with other lesions
- Only manifestation
- Difficult to treat

A

Pododemodicosis

117
Q
  • Concurrent disease
    • Glucocorticoid administration
    • Hyperadrenocorticism
    • Chemotherapy Rx
    • Hypothyroidism
    • Neoplasia
  • Diagnose and treat (if possible)
  • Idiopathic
  • Less likely to cure
A

Adult-onset

118
Q
  • Rare
  • Demodex cati: long, slender follicular mite, morphologically comparable to Demodex canis (180-210 microns long)
  • Demodex gatoi: short, with broad, blunted abdomen. Dwells superficially in the stratum corneum (80-90 microns long)
  • Demodex felis: (113-175 microns long) medium
A

Feline Demodicosis

119
Q

List the clinical signs for D. gatoi:

A
  • Pruritic, however, may be asymptomatic
  • Contagious
  • Any age
  • Alopecia, scaling, crusted lesions
  • Erythema, hyperpigmentation, stubby hairs
  • Ventral alopecia
120
Q

List the clinical signs for D. felis:

A
  • +/- Pruritus
  • Cats lose large clumps of hair, hairs easily epilating - smooth shiny non-inflammatory areas of alopecia
  • Non-blanching erythema
  • Most had URI - but were in shelter
121
Q

What is the best test for diagnosis of Demodex canis?

A

Deep skin scraping
- negative rules out

122
Q

Canine Demodicosis:
In addition to diet, heartworm test, fecal floatation (juvenile-onset generalized demodicosis) what else do you want to do to diagnose Adult-onset generalized demodicosis?

A
  • CBC
  • Biochemical profile
  • Urinalysis
  • Thyroid test
123
Q

What is the ONLY FDA approved product to treat demodicosis?

A

Amitraz: Mitaban
- Broad-spectrum miticide
- Side effects
- Alpha-adrenergic agonist
- Antidote
- rinse dog
- Yohimbine IV

124
Q

What is an FDA product NOT approved for the treatment of demodicosis?

A

Ivermectin
- Toxicity
- Antidote IV lipid emulsion
- Don’t use in hearing breeds
- P-glycoprotein
- test for mutation

125
Q
  • FDA product NOT approved for Demodex treatment
  • Heartworm negative
  • Expensive
A

Milbemycin Oxime

126
Q
  • Moxidectin + imidacloprid
  • Juvenile dogs with mild disease
  • Weekly administration
A

Advantage Multi, Bayer

127
Q

(T/F) 90% of cases of localized demodicosis will resolve on their own, so no treatment is necessary

A

True

128
Q
  • This is a superficial-dwelling mite
  • This mite can cause pruritus
  • This mite is contagious to other cats
A

Demodex gatoi (short)

129
Q

You have a client with a dog with AD that also has demodicosis. What treatment is safe to use for the dog’s pruritus?

A

Cytopoint

Integumentary (Exam3) (3 decks)

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