Module 6 - Derm I & II Flashcards

1
Q

Danger signs of moles *

A

ABCDE
asymmetry
borders
color
diameter
evolving
itching bleeding crusting is bad

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2
Q

melanoma

A

goes down to dermis
add more

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3
Q

primary prevention
secondary
tertiary

A

sunscreen, long sleeves
skin exams
biopsy

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4
Q

BCC vs SCC

A

BCC
- most common skin ca
- shiny, pearly, papular, nodule
- umbilicated center + telangiectasia
- NOSE

SCC
- more common in immunosuppressed
- hyperkeratotic lesion with crusting and ulceration
- more aggressive growing
- ear

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5
Q

Contact Dermatitis Treatment *

A

DO
- take lukewarm showers (not hot)
- lotion after showers (syran)
- drinks lots of water

Rx: med-high potency topical steroids
- If > 20% BSA PO steroids

  • antihistamines not helpful
  • biopsy if not responsive to treatment
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6
Q

Poison Ivy *

A
  • wash area ASAP
    astringents (domeboro), aclamine lotion, oatmeal baths
  • helps soon itching or dry weeping rash
  • dont bother with pre lotions
  • cool compress or OTC topicals (IVY DRY)
  • DO NOT USE topical diphenhydramine, antibiotics, caine topicals
  • oral antihistamines are NOT helpful
  • OTC mild hydrocortisone
  • med-high potency top steroids for mod-sev

oral steroids if >20% BSA
- prednisone taper 2-3 weeks
- dont use methyl prednisone

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7
Q

Eczema Treatment *

A

AVOID:
- triggers (wools, soaps, detergents)
- temperature changes
- sweating
- low humidity in winter
- foods: eggs, peanuts, soy, milk
- stressful stimulants
- allergens and dust
- excessive handwashing

TREATMENT:
emollients can be used for dryness
- 1st line: low potency topical steroid
(1-2.5% hydrocortisone)
- intermediate cases
(triamcinolone)
- severe cases
(betamethasone)

Oral antihistamines ARE effective in controlling and preventing pruritic

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8
Q

Long term use of steroids *

A
  • skin atrophy such as striae
  • telangiectasias
  • systemic effects
    ( growth restriction in children, risk of glaucoma)
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9
Q

Topical Drugs for Atopic Dermatitis *

A

Atopic Dermatitis = Eczema

Corticosteroids
- inhibit release of inflammatory cytokines
- use smallest amount to limit exposure and s/e

TREAT: thin layer once or twice a day in affected areas

PREVENT: thin layer once or twice weekly

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10
Q

Topical corticosteroids comparison and potency from readings *

A

occlusive dressings can significantly increase absorption - but means increased s/e. (also including skin irritation and folliculitis). Dont use occlusive dressings with betamethasone and fluticasone because of high potency.

Total dose for high potency should not exceed 50g a week bc of possible adrenal suppression. Clobetasol may cause adrenal suppression - dont discontinue abruptly but instead switch to lower potency agent. Sx of decreased adrenal suppression: weakness, lb loss, hypotension, GI distress.

Applying to wet skin may IMPROVE effectiveness. Can use different products on different parts of the body.

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11
Q

Very high potency *
High potency
Moderate
Low

A

Very high potency: scalp, palms, soles, resistant thick lesions
High potency: palms, soles, scalp
Moderate: extensive area of skin
Low: elderly children face folds genitals extensive area of skin

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12
Q

Pityriasis Rosea *

A

Begins as rash (harolds patch) and spreads like christmas tree
- spring and fall, favors adolescents and young adults

Harold patch: salmon, oval, scaly (trunk, UE)

  • heals after 2-4 weeks, gone by 6-14 weeks
  • idiopathic cause
  • NOT CONTAGIOUS or STD

systemic symptoms sometimes
No treatment necessary

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13
Q

Tinea vs. Nummular Eczema *

A

tinea = ring worm

tinea: central flaring
NE: dry, flaky lesion throughout

tinea capitis: needs systemic therapy
tinea ungium (nail): needs systemic therapy

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14
Q

Antifungals *

A
  • apply to lesion + 2 inches surrounding area
  • continue for 1-2 weeks after lesion resolves
  • itching resolves with antifungal treatment but if extra therapy for itching needed: sarna sensitive
  • avoid topical steroids !
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