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NM 703 Primary Care > Module 5 Dermatology > Flashcards

Module 5 Dermatology Flashcards

1
Q

Patho of Acne Rosacea

A
  • Cause is unknown

* Possibly linked to immune-mediated Inflammation

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2
Q

Complications of Acne Rosacea

A

•Ocular Rosacea

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3
Q

Risk factors for Acne Rosacea

A
  • Age
  • Fair skinned
  • Female
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4
Q

Subjective data for Acne Rosacea

A
  • Facial flushing
  • “I always look red”
  • “I have bumps and fluid filled bumps on my face”
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5
Q

Objective Data for Acne Rosacea

A
  • Facial erythema
  • inflammatory papules and pustules
  • watery and irritated eyes
  • No Comedones noted
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6
Q

Differentials for Acne Rosacea

A
  • Adult acne vulgaris
  • Photodermatitis
  • Seborrheic dermatitis
  • Contact dermatitis
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7
Q

Testing for Acne Rosacea

A

None

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8
Q

Management of Acne Rosacea

A 
•Topical or oral antibiotics
–Metronidazole 1% or 0.75% gel or lotion BID for 3-4 months
–Azelaic acid 15% gel or cream BID fo2 months 
–Plexion Cleanser
–Tetracycline  250-500mg BID
–Doxycycline 100-200mg/day
–Minocycline 50-100mg/day
•Nonpharmacologic 
–Skin care
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9
Q

Patient education for Acne Rosacea

A
  • Long-haul
  • Patience and understanding of the treatment
  • Routine follow up necessary
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10
Q

Referral for Acne Rosacea

A

Dermatologist. Ophthalmologist, Mental Health Provider

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11
Q

What is Acne Vulgaris

A

a condition of the pilosebaceous follicles

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12
Q

Complications of acne vulgaris

A 
•Keloids
•Medication adverse effects
–Renal impairment
–DVTs
–Hyperkalemia
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13
Q

Risk factors for Acne Vulgaris

A
  • Family history of acne
  • Allergies
  • Certain medications can cause acne
  • Hormonal factors
  • Seasonal factors like less sunlight
  • Facial products
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14
Q

Subjective/Objective data for Acne Vulgaris

A
  • Variety of lesions including comedones, papules, pustules, and nodules on the face, chest, back, and shoulders
  • “Pimples”
  • Red bumps
  • Black bumps
  • White bumps
  • Pain
  • Scarring
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15
Q

Differential Diagnoses for Acne Vulgaris

A
  • Closed comedones acne, milia and suspicious hyperplasia
  • Open, comedonal acne, dilated pore of Weiner, and Favre-Racouchot syndrome
  • Inflammatory acne, rosacea, and perioral dermatitis
  • Fungal, flat warts, molluscum contagiosum, folliculitis
  • Tuberous sclerosis
  • Facial angiofibroma
  • Adnexal tumors
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16
Q

Testing for Acne Vulgris

A

May be done to find cause

  • Total testosterone
  • DHEAS
  • Androstenedione
  • Luteinizing hormones
  • Follicle stimulating hormone
  • Lipid profile
  • Glucose tolerance testing
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17
Q

Management of Acne Vulgaris

A

1st:cleansers->topicals->t.antib->po antib

•Nonpharmacologic
–Mild cleansers
•Pharmacologic
1st–Retin-A, Differin, Tazorac, azelaic acid, benzoyl peroxide, salicylic acid
–Topical antibiotics (not recommended in mono-therapy)
–Oral antibiotics
–Hormone therapy: combined OC-prog only makes it worse
–Retinoid therapy

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18
Q

Patient education for Acne Vulgaris

A
  • Adherence is key
  • Treatment is a longhaul
  • Follow-up visits are necessary
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19
Q

Referral for Acne Vulgaris

A

Dermatologist, Mental Health Professional

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20
Q

What is Cellulitis?

A

Bacterial infection most often caused by

Streptococcus or Group A B-hemolytic streptococci

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21
Q

Complications of Cellulitis

A
  • Severe systemic infection
  • Osteomyelitis
  • Periorbital cellulitis
  • Death
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22
Q

Risk factors for Cellulitis

A
  • Immunocompromised at greater risk

* Obesity

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23
Q

Subjective data for Cellulitis

A
  • Redness
  • Pain
  • Drainage
  • Swelling
  • Feverish
  • Warmth at the site
  • Chills
  • Malaise
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24
Q

Objective data of Cellulitis

A 
• Unilateral (most often a
limb)
• Inflamed
• Red
• Hot
• Swollen
• May or may not have open
sore visible
• Fever
• Tender to touch
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25
Q

Differential diagnosis of Cellulitis

A
  • Deep vein thrombosis
  • Osteomyelitis
  • Thrombophlebitis
  • Neoplastic disease
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26
Q

Testing for Cellulitis

A
  • CBC with differential
  • Creatinine
  • Bicarbonate
  • Creatinine phosphokinase
  • Purulent focus culture
  • Gram stain
  • Radiographs or ultrasound
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27
Q

Management of Cellulitis

A
  • Systemic antibiotics (PCN, Amox. Augmentin preferred x 5d unless allergic)
  • Rest, elevate, compression, educate
  • Refer if I&D needed or other (p.278)
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28
Q

Patient education for Cellulitis

A
  • Complete entire course of antibiotics
  • Notify the office if symptoms are worsening
  • Keep area dry clean and elevated
  • Educate on the prevention of skin infections
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29
Q

Referral for Cellulitis

A

• Immediate referral for severe complications

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30
Q

What is Eczematous Dermatitis

A

pruritic inflammatory skin disorder which has exacerbations and remissions

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31
Q

Subjective data with Eczema

A
  • Dry patches on skin
  • Crusting and oozing from skin sites
  • Severe itching
  • “An itch that rashes”
  • Thickened area on skins
  • Symptoms on upper extremities
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32
Q

Objective data of Eczema

A
  • Pattern of rash / itch of inner folds
  • Linear excoriations
  • Boarders are undefined
  • Rash in folds, wrists, dorsa of feet, face, and neck
  • Fissures on palms of hands
  • Well-demarcated area of erythema, scaling, or crusting at the site of exposure
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33
Q

Differentials for Dermatitis

A
  • Mycosis fungoides
  • Immunodeficiency (especially with severe itching and with recurrent infection)
  • Scabies
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34
Q

Dermatitis Testing

A
  • Primarily none

* Could do KOH, skin biopsy, skin patch testing (contact dermatitis)

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35
Q

Management of Eczema

A 
Management:
•Nonpharmacologic 
–Elimination diet 
–Phototherapy 
–Avoid: rubbing alcohol , Scratching, Goal is management of pruritis, Triggers

•Pharmacologic
–Antihistamines
–Emollient
–Topical corticosteroids
• Hydrocortisone 1% apply to affected area twice daily x2 weeks
•Triamcinolone for acute persistent flare ups

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36
Q

Treatment of Allergic Dermatitis

A

Steroids for allergy & education to avoid allergen (ex. poison ivy)

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37
Q

Treatment of Irritant Dermatitis

A

Avoid the irritant, protect the skin with gloves, petroleum or other hypoallergenic barrier, avoid detergents and use alcohol- based cleansers that do not remove natural barriers and preserve skin integrity

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38
Q

Complications of Oral Herpes

A
  • Complications are rare but can occur in those who are already immunocompromised
  • Possible complications include aseptic meningitis, urinary retention, cutaneous dissemination, bacterial superinfection, arrhythmia multiform, and spontaneous abortion
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39
Q

Risk for Oral Herpes

A

Female
Black

  • Contact with a person who has an active lesions or sections
  • Contact with a person how has the know virus
  • Open cuts and sores
  • Immunocompromised
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40
Q

Subjective data of Oral Herpes

A 
• Primary, latent and
recurrent
– Prodrome of burning
– Blister on the mouth
– pain at site
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41
Q

Objective data of Oral Herpes

A

• Primary, Latent, Recurrent
– Single or multiple vesicles
– Tender to touch

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42
Q

Differentials of Oral Herpes

A
  • Erythema multiforme
  • Impetigo
  • Varicella
  • Herpes Zoster
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43
Q

Diagnostics of Oral Herpes

A 
• Thorough history and physical
• Lab conformation for new cases
– Cutaneous herpes simplex viral culture
• Serologic testing
– Does not differentiate between type one and type two so used with caution
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44
Q

Treatment of Oral Herpes

A 
• Initial episode
– Acyclovir 400mg PO 3 times daily for 7-10 days
– Valacyclovir 1gm PO daily for 7-10 days
– Famciclovir
• Recurrent episode
– Acyclovir 400mg PO BID x5 days
– Valacyclovir 500 PO BID for 3 days
– Famciclovir
• Suppression
– Acyclovir 400mg PO BID
– Valacyclovir 500mg or 1gm PO daily
– Famciclovir
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45
Q

Patient education for Oral Herpes

A
  • Start treatment at onset of prodromes for recurrent
  • Avoid contact when you have an active lesion
  • Understand that you can transmit even if you have no lesion or when using suppressive therapy
  • Explain the risks of neonatal transmission during pregnancy to both female and male
  • Lip balm with sunscreen when exposed to UV light to avoid
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46
Q

Referral for Oral Herpes

A

• Obstitrican/MFM if patient is pregnant

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47
Q

Patho of Herpes Zoster

A
  • Virus that lies dormant after initial infection
  • Virus replicates
  • Penetrates the epidermis
  • Replication is multifactorial
  • Can spread by contact and air and live for hours to two days
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48
Q

Complications of Herpes Zoster

A
  • Blindness
  • Motor paralysis
  • Facial palsy
  • Pneumonia
  • Hepatitis
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49
Q

Risk factors for Herpes Zoster

A
  • Age
  • Immunosuppressed
  • Previous Varicella infection
  • Pregnancy
  • Very young
  • Unvaccinated
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50
Q

Subjective data of Herpes Zoster

A
  • Painful itching, then eruption of blisters
  • Burning, stabbing, aching sensation
  • Feverish
  • One-sided rash on the trunk of body
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51
Q

Objective data of Herpes Zoster

A
  • Unilateral rash
  • Low-grade fever
  • Erythematous and maculopapular clusters of clear vesicles
  • Tender to touch
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52
Q

Differentials for Herpes Zoster

A
  • Allergic dermatitis
  • Dermatitis herpetiformis
  • Contact Dermatitis
  • Coxsackievirus
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53
Q

Testing for Herpes Zoster

A
  • PCR analysis
  • Tzanck preparation
  • DFA test
  • Viral culture
  • Alkaline phosphatase, bilirubin, C-reactive protein
  • WBC
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54
Q

Treatment of Herpes Zoster

A

• Antiviral within 72 hours of onset
– Valcyclovir 1,000 mg orally three times daily for seven days
– Famciclovir 500 mg orally three times daily for seven days
– Acyclovir 800 mg orally five times daily for 7 to 10 days
• Pain management
– NSAIDS
– Gabapentin
– Pregabalin
– Amitriptyline
– Acetaminophen

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55
Q

Patient education for Herpes Zoster

A
  • Notify early of symptoms
  • Use moist dressings, pain management, and bedrest to relieve discomfort
  • Susceptible individuals while you are infectious
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56
Q

Referral for Herpes Zoster

A

If on face

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57
Q

What is Intertrigo

A

Bacterial or fungal disorder that occure in persistent skin to skin contact, friction, moisture, warmth and inadequate ventilaition

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58
Q

Complications of Intertrigo

A
  • Unusual to have complications with topicals
  • With systemic antifungal’s complications would be hepatotoxicity
  • Note that oral ketoconazole is no longer approved as a treatment due to this reason
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59
Q

Subjective data for Intertrigo

A 
• Moist glistening
plaques, pustules
• Odor
• Discharging
• Pain, fever
60
Q

Objective data for Intertrigo

A 
• Red patches
• Moist glistening
plaques, pustules
• Odorous
• Discharging
• Tender to touch
• Warm to touch
61
Q

Diagnostics of Intertrigo

A
  • KOH microscopy

* Liver function test

62
Q

Treatment of Intertrigo

A 
• Removal of the infecting organism
• Exudate the lesions treated with drying agents
– Aluminum sulfate soaks
• Topical anti-fungal
• Oral anti-fungal
63
Q

Patient education of Intertrigo

A
  • Caution patients on over-the-counter steroid creams
  • Absorbent powders help reduce moisture and prevent reinfection
  • Take the antifungals for the duration as directed to prevent recurrence
64
Q

Patho of Skin Cancer

A
  • Pathogenesis is multifactorial
  • Heavy sun exposure: Causes immunosuppressio UVR is a carcinogen and promotes tumor growth
  • Normal aging process: Thinner, frail skin, susceptible to insult
65
Q

Complications of Skin Cancer

A

Disfigurement, death

66
Q

Risk factors for Skin Cancer

A
  • Heavy sun exposure with no UV protection -Aging
  • Family hx
  • Prior hx of other cancer
67
Q

Subjective data for Skin Cancer

A
  • Open sores that do not heal, burn, itch, or crust over -Change in the size or shape of a “skin spot,” lesions, or mole
  • Change in color
  • A pearly looking skin spot
68
Q

Testing for Skin Cancer

A

Skin biopsy:Shave, Punch or Excisional

69
Q

Patient education for Skin Cancer

A
  • Yearly skin exams
  • Limit sun exposure
  • Use protective clothing
  • Use sunscreen (SPF >15)
  • Seek prompt care within 4-6 weeks for nonhealing wound
70
Q

What is Melanoma (Skin Cancer). Referral?

A

Malignant melanoma is the most fatal, one person dies per hour

Derm or surgeon

71
Q

Most common form of Cancer

A

Basal Cell Carcinoma

72
Q

Subjective of Basal Cell Carcinoma

A

Pearly / Shiny, visible vessels – Normal skin color or slightly pigmented

73
Q

Treatment of Basal Cell Carcinoma

A

Electrodessication and curettage

74
Q

Objective of Squamous Cell Carcinoma

A

-Scaly / Scabby, bleeds easily, sore that does not heal, “volcano shape” -Bleeds easily

75
Q

Treatment of Squamous Cell Carcinoma

A

Total excision

76
Q

Objective of Actinic Keratosis

A

Key: precursor for squamous cells carcinoma

A rough, scaly patch on the skin caused by years of sun exposure

77
Q

Patho of Lice

A
  • Wingless insects and survive by feeding on human blood-adhere to the shaft of the hair and fibers of clothing
  • Belong to the Anoplura family
  • Feed several times per day
  • They lay 5 to 10 eggs per day
  • Their louse pierces the skin and injects its saliva into human blood
78
Q

Complications of Lice

A
  • Sleep disturbances

* Impetigo from scratching

79
Q

Risk factors for Lice

A
  • Can affect anyone
  • More common in school age
  • Sharing combs/brushes
  • Sharing hats
  • Sharing bed-linens
80
Q

Subjective data for Lice

A
  • Itching

* “Something crawling”

81
Q

Objective data for Lice

A
  • Nits and louse on clothing or body of the individual
  • Lice on the back of the head, neck, and behind the ears
  • Linear excoriations on the body
  • Hyper pigmentation with linchenification
82
Q

Differentials for Lice

A
  • Seborrheic dermatitis •Scabies
  • Eczema
  • Insect bites
  • Psoriasis
83
Q

Testing for Lice

A

Wood lamp if indicated

84
Q

Treatment of Lice

A

-First line:
•Nix
•Permethrin 5%- Apply to dry hair and rinse after 10 minutes
•Ivermectin lotion 0.5%

85
Q

Patient Education for Lice

A
  • Use a Nit comb
  • Comb through 2 times
  • Hygiene practices
  • Discard infested clothing and linen
86
Q

Patho of Scabies

A
  • Mite-Causes poorly defined pruritic eruption
  • Female mite is responsible for the infestations
  • Burrows into the stratum corneum, laying two to three eggs per day
  • The eggs reach maturity in 28 to 30 days and then start a new cycle
87
Q

Risk factors for Scabies

A
  • Highly contagious •Common in overcrowded and low socioeconomic environments
  • Prolonged skin-to-skin contact like sexual activity
88
Q

Subjective data for Scabies

A

•Intense itching at night

89
Q

Objective data for Scabies

A
  • Intraepidermal burrows
  • Small Papules
  • Burrows located on the wrist, genitalia, breast, buttock, webs of fingers
90
Q

Differentials for Scabies

A
  • Seborrhic dermatitis
  • Insect bites
  • Impetigo
91
Q

Testing for scabies

A

Scraping of a burrow and microscopic identification of mites, eggs, or feces

92
Q

Treatment of Scabies

A

First line:
•Permethrin 5% cream
•Repeat in one week

93
Q

Patient education for Scabies

A
  • Identify and treat household contacts
  • Wash all clothing and bedding in hot water
  • Remind patients that symptoms can last for two weeks after treatment
94
Q

Patho of Pruritis

A

-Itchy skin that is uncomfortable and you have an irritating sensation that makes you want to scratch.
•Itchy skin is often caused by dry skin. It’s common in older adults, as skin tends to become drier with age
•Multi-factorial

95
Q

Complications of Pruritis

A 
•Chronic pruritis
•Interruption of daily activates
•Decreased sleep
•Onset of anxiety or 
depression
•Skin injury
•Scarring
96
Q

Risk factors for pruritis

A
  • Age
  • Other skin conditions
  • Internal disease
  • Nerve disorders
  • Psychiatric conditions
  • Irritation and allergic reactions
97
Q

Subjective data for Pruritis

A
  • Redness
  • Scratch marks
  • Dry, cracked skin
  • Constant sensation to scratch
98
Q

Objective data for Pruritis

A
  • Redness
  • Scratch marks
  • Bumps, spots or blisters
  • Dry, cracked skin
  • Leathery or scaly patches
  • Excoriation
99
Q

Differentials for Pruritis

A
  • Parasitic infection
  • Liver disease
  • Fungal infection
  • Viral allergy
  • Dermatitis
100
Q

Testing for Pruritis

A

CBC, TSH, LFT, Chest X-ray

101
Q

Treatment of Pruritis

A 
•Nonpharmacologic
–Remove causative agent if possible
–Use creams, lotions, or gels that are soothing
–Avoid stress
–OTC allergy medication
–Cool compresses
–Avoid scratching
•Pharmacologic
–Corticosteroid creams and ointments
•Triamcinolone 0.25% to 0.1% apply to wet skin
–Other creams and ointments
–Oral medications
–Light therapy (phototherapy)
102
Q

Patient teaching for Pruritis

A
  • Mild cleanser
  • Increase hydration
  • Adherence to daily Moisturizing
103
Q

Referral for Pruritis

A

•Dermatologist, Mental Health Provider

104
Q

Patho of Psoriasis

A
  • Chronic, inflammatory, autoimmune disorder
  • Characterized by dermal hyperproliferation
  • Genetic association
105
Q

Complications of Psoriasis

A 
• Infection
• Guttate psoriasis
• Erythrodermic
psoriasis
• Pustular psoriasis
106
Q

Risk factors for Psoriasis

A

Family hx

Monozygote twin

107
Q

Subjective data for Psoriasis

A 
• Scale like patches
• Areas bleeding easily
• Patches on elbows,
knees, scalp, genitals,
intergluteal folds
• “Raindrop” plaques
108
Q

Objective data for Psoriasis

A 
• Well circumscribed
erythematous
maculopapular lesions
covered in silvery white
scale (Image, p. 320)
• Pitting of nails [p.318]
• Well demarcated lesions
• Patches on elbows, knees,
scalp, genitals,
intergluteal folds
109
Q

Differential for Psoriasis

A
  • Seborrhea
  • Atopic dermatitis
  • Pityriasis rosea
  • Gout
110
Q

Testing for Psoriasis

A

• None, diagnosis is based
on presentation
• *You could do a biopsy

111
Q

Treatment of Psoriasis

A
  • Reduce epidermal proliferation and decrease inflammation
  • Topical corticosteroids
  • Ointments are preferred
  • Occlusion with clear plastic wrap can increase efficacy of therapy
112
Q

Patient education for Psoriasis

A
  • Educate about chronic nature and need for long term control to prevent sequelae
  • Adherence to prescribed medication
  • Avoid injury to skin
  • Avoid certain medications
113
Q

Referral of Psoriasis

A
  • Dermatologist if unresolved

* Rheumatologist

114
Q

Patho of Tinea Pedis (Athletes foot)

A

Superficial infection-Usually dermatophytes or yeast
• There are typically three major sources that are responsible for the transmission of dermatophytes
• Human to human
• Animal to human
• Soil to human or soil to animal
• Three weeks before symptoms appear

115
Q

Risk factors for Tinea Pedia (athletes foot)

A
  • Post-puberty
  • Age
  • Gender
  • Immunity status
116
Q

Subjective data for Tinea Pedis

A
  • Itching
  • Foot odor
  • Skin appears to be tearing
  • Skin is red
117
Q

Objective data for Tinea Pedis

A
  • Skin is erythematous
  • Scaling eruption between the toes and the souls, and sides of the feet
  • Ulcerations and inflammation
118
Q

Differential for Tinea Pedia

A
  • Atopic Dermatitis
  • Contact Dermatitis
  • Folliculitis
  • Psoriasis
119
Q

Patho of Tinea Ungunium/Onchomycosis

A

Fungus in nails, fingers or toes

120
Q

Subjective and Objective data for Tinea Unguium/Onchomycosis

A
  • Yellowish-brown discoloration
  • Greenish tinge on nail
  • thicker nail the normal
121
Q

Patho of Tinea Corporis

A

Fungus in skin-worse after sun exposure

122
Q

Subjective data of Tinea Corporis

A
  • Red patches
  • Worse after sun exposure
  • Mildly itchy
  • Looks like scales
123
Q

Objective data for Tinea Corporis

A
  • Appears as tissue thin coating of fungus on skin
  • Scaly
  • Regions often have slightly elevated borders
124
Q

Patho of Urticaria

A
  • Red, itchy welts that results from a skin reaction
  • Symptom not disease
  • Immediate hypersensitivity to an allergen or antigen that appears as itchy lesions on the skin
  • Vascular reaction in upper dermis of skin – Type 1 IgE mediated
  • Allergen
  • Nervous reaction
125
Q

Risk factors for Urticaria

A

None

126
Q

Subjective data for Urticaria

A
  • Acute onset of raised red areas
  • Pruritus (itch) before lesions appear
  • Swelling
  • Triggered by heat, stress, exercise
  • Cleared up within minutes
127
Q

Objective data for Urticaria

A

•Confluent circular erythematous rash

128
Q

Differentials for Urticaria

A

•Dermatitis

129
Q

Testing for Urticaria

A

None

130
Q

Treatment of Urticaria

A 
•First-line H1 blocker
–Loratadine (Claritin)
–Fexofenadine (Allegra)
–Cetirizine (Zyrtec)
–Desloratadine (Clarinex)
•H2 blockers
–Cimetidine (Tagamet)
–Famotidine (Pepcid)
•Anti-inflammatory meds
131
Q

Patient education for Urticaria

A

•Benign symptom

132
Q

Patho of Verruca (non-genital warts)

A

Caused by HPV

• Virus contacts broken skin and enters the epidermal epithelial cells and replicates.

133
Q

Complications of Verruca

A
  • Pain
  • Decreased activity
  • Altered gait or deformity (plantar warts)
  • Scarring
  • Damage of nails with treatment
  • Nerve damage is rare but can occur if treatment is vigorous in areas of superficial nerves
134
Q

Risk factors for Verruca

A
  • Decreased immunity
  • Chronic wet hands/feet
  • Family
  • Showers
135
Q

Subjective data for Verruca

A
  • May be asymptomatic
  • May describe painful area
  • “I have a wart/warts”
136
Q

Objective data for Verruca

A
  • Projections (filiform)
  • Round domes (common)
  • Flat (plantar)
  • Single of groups
  • May form plaques (mosaic warts)
137
Q

Differentials for Verruca

A
  • Carcinoma/Melanoma
  • Actinic Keratosis
  • Secondary syphilis
  • Molluscum contagiosum
  • Foreign body
  • Face - perioral dermatitis
  • Hands/feet - periungual warts, actinic Keratosis
  • Psoriasis
  • Lichen planus
138
Q

Testing for Verruca

A

Inspection

139
Q

Treatment for Verruca

A

• Topical Application
– Salicylic acid & duct tape
– Imiquimod 3.75% or 5% cream (Aldara)
– Podofilox .5% twice daily for 3 days, off for 4 days, repeat
– Sinecatechins 15% apply three times daily for up to 16 weeks.
– TCA or BCA must be applied by the health care provider
• Cryotherapy
• Laser Ablation
• Surgical

**There is no Cure-Rule out melanoma first

140
Q

Patient education for Verruca

A
  • Requires commitment
  • May be painful
  • Soak and debride
  • Warts are benign
  • May take years to completely resolve
  • Side effects of the medication
141
Q

Referral for Verruca

A
  • Dermatology - large warts or cosmetically sensitive areas

* Podiatry - plantar warts

142
Q

Which is the most occlusive vehicle?

A

Ointments

143
Q

Which is the least occlusive vehicle?

A

Solution

144
Q

Which vehicles should be used for dry conditions?

A

Ointments, cream, lotion

145
Q

Which vehicle should be used for wet conditions?

A

Solution, gel

146
Q

Order the vehicles from least potentiating to most potentiating?

A

solution, gel, lotion, cream, ointment

NM 703 Primary Care (8 decks)

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