Module 4: The ABCDE assessment Flashcards

1
Q

Most (50–80%) of patients show signs of deterioration prior to arrest. What are common sings of deterioration?

A

Hypoxia or hypotension

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2
Q

Airways

A
  1. Look for signs of airways obstruction
    - See-saw respirations
    - No breath sounds (complete) vs diminished/noisy breath sounds (partial)
  2. Treat as a medical emergency
    - Simple methods (suction, Mcgills forceps, basic manoeuvres, adjuncts)
    - Tracheal intubation failing this
  3. High concentration oxygen
    - At flow rate 15 L min-1
    - Use self-inflating bag if tracheal tube in situ
    - Sats aim acute RF 94-98%, if risk hypercapnic RF 88-92%
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3
Q

Breathing

A
  1. Treat life-threatening conditions immediately (e.g. acute severe asthma, tension pneumo, pulmonary oedema)
  2. Assess: RR, depth & pattern, sats, chest expansion, breath sounds, percuss, auscultate, tracheal position, palpate chest wall
  3. Give oxygen to all critically ill patient
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4
Q

What are you assessing for when listening to breath sounds from a short distance away?

A

Rattling = airway secretions

Stridor/wheeze = partial (!but significant) obstruction

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5
Q

What are you assessing for when percussing?

A

Dullness = consolidation or pleural fluid
Hyper-resonance = pneumothorax

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6
Q

What are you assessing for when auscultating?

A

Bronchial breath sounds = lung consolidation with patent airways

Absent/reduced breath sounds = pneumothorax, pleural fluid, lung consolidation causing complete obstruction

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7
Q

Where and why do you check tracheal deviation?

A

Suprasternal notch

Towards affected side (loss of volume on that side)
- Pneumonectomy, atelectasis

Away from affected side (increase in pressure or volume on that side)
- Tension pneumothorax, large pleural effusion, upper mediastinal mass e.g. MNG

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8
Q

Feel the chest wall to detect?

A

Crepitus = the crackling/popping noise from air under the skin) =surgical emphysema, pneumothorax, ruptured airway/oesophagus

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9
Q

True/false: all critically ill patients should be given oxygen.

A

True.

In the COPD subgroup: still give oxygen but aim for a lower partial pressure and oxygen saturation. High concentrations of O2 may depress their breathing (i.e. risk of hypercapnic or ‘T2’ respiratory failure).

Give oxygen via a Venturi 28% mask (4 L min-1) or Venturi 24% mask (4 L min-1) initially then reassess.

Aim for SpO2 88-92%.

They may have an oxygen alert card, or ABGs from previous exacerbations, to guide you.

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10
Q

If the patient’s depth or rate of breathing is judged to be inadequate, or absent, you should?

A

Use bag-mask or pocket-mask ventilation to improve oxygenation and ventilation.
In cooperative patients without airway obstruction, consider NIV.
In patients with acute exacerbation COPD, NIV is useful and can prevent needing tracheal intubation & invasive ventilation.

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11
Q

Circulation

A

1.Hypovolaemia should be considered as the primary cause of shock u.p.o.
2. Assess hand colour & temp, CRT (press for 5s on a fingertip at heart level), peripheral & central pulse (rate, quality, regularity, equality), BP, HS
3. Give IVF to all patients with fast HR and cool peripheries
4. Exclude haemorrhage in surgical patients (overt/hidden)
5. Insert one/more 14 or 16 G cannulae (short & wide-bore = enables highest flow)
6. Take bloods, give fluids
7. Reassess HR and BP every 5 mins, aiming for the patient’s normal BP or, if unknown, aim >100 mmHg systolic. Repeat fluid challenge if no improvement.

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12
Q

Why may CRT be prolonged i.e. >2s?

A

Poor peripheral perfusion
Cool surroundings
Old age

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13
Q

How would you interpret pulse quality?

A

Bounding – sepsis
Thready –poor CO

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14
Q

How would you interpret BP?

A

Low dBP –arterial vasodilation –anaphylaxis, sepsis

Narrow pulse pressure (35-45 mmHg) –arterial vasoconstriction –cardiogenic shock, hypovolaemia –may occur with rapid tachyarrhythmia

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15
Q

What bloods are you taking? What fluids are you giving?

A

Bloods: routine haematological, biochemical, coagulation, microbial, cross-matching (before IVF)

Fluid: 500 mL crystalloid (Hartmann’s or NaCl) over <15 min. 250 mL if know cardiac failure or trauma.

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16
Q

What would signs of HF be? What should you do?

A

Dyspnoea, raised HR, raised JVP, pulmonary crackles on auscultation, a 3rd HS

Decrease/stop the fluid infusion)
Alternative means to increase tissue perfusion – inotropes, vasopressors

17
Q

If the patient has primary chest pain and suspected ACS?

A

Record a 12-lead ECG early
Immediate general treatment for ACS:
1. 300 mg aspirin oral (crushed/chewed) asap
2. GTN spray
3. Oxygen only if SpO2 <94% on RA
4. Morphine or diamorphine, titrated IV to avoid sedation/respiratory depression

18
Q

Disability

A
  1. Examine pupils
  2. Rapid consciousness assessment: AVPU or GCS
  3. Blood glucose, rapid finger-prick (ABG or VBG in sick/periarrest patients)
  4. Lateral position for unconscious patients if airway not protected
19
Q

Management of hypoglycaemia, <4.0 mmol L-1 in unconscious patient?

A

50 mL of 10% glucose IV

Give further doses until consciousness regained, up to 250 mL 10%.
Repeat BMs.

20
Q

Exposure

A
  1. Fully expose the patient, respecting dignity and minimising heat loss
  2. Abdomen
  3. Calves