Module 4 Flashcards

1
Q

What is the etiology of acute otitis media?

A

bacterial infection of the middle ear- could be caused by structural deformities, previous upper resp infections, sinusitis
clogged eustachian tubes allows increased risk of bacterial infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what are the common bacteria that can cause otitis media?

A

s. pneumoniae
h influenzae
st. pyogenes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what are the clinical findings of acute otitis media?

A

otalgia
discharge, pressure, decreased hearing, fever
erythema, effusion or redness of the TM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what is the treatment for acute otitis media?

A

amoxicillin 1st line
amoxi/clav 2nd life if resistant to first line
referral to ENT if recurrent

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what is the etiology of chronic otitis media?

A

chronic otorrhea with no otalgia
TM perforation with hearing loss
recurrent otitis media

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what are the most common bacteria that cause chronic otits media

A

p .aeringunosa
proteus
s. aureus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what is the treatment for chronic otitis media?

A

topical drops ie cipro or tobramycin
oral cipro
surgery if needed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is the etiology of serous otitis media?

A

eustachian tube contanly blocked
blocked tube causes transudation (passing of fluid through membranes)
usually occrs after resp URI, allergi rhinitis
CARCINOMA IF UNILATERAL
effusion, fullness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is the treatment for serous otitis media?

A
ventilating tube (tymphanpanstomy) 
surgical repair
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Differentiate between acute, subacute, and chronic cough

A

acute >3 weeks, subacute 3-8 weeks, chronic >8 weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Describe the management of someone with chronic cough

A
consider causes (COPD, pertussis, GERD, bronchitis)
diagnostics- CXR, nasopharyngeal swab or sputum
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

chronic cough protocol

A
OTC therapy
office spirometry if available or refer
CXR 
CT sinuses
PFT/methycholine testing
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Describe the step wise approach to asthma treatment

A

step down or step up depending on usage
1. rescue inhaler SABA (ventolin) ALWAYS NEED THIS
2. inhaled corticosteroid (flovent)
3. ICS + LABA (advair)
may need to refer if these are not sufficient for asthma relief

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Describe the step wise approach to COPD treatment

A

step down or step up depending on usage and symptoms
1. SAMA or SABA or combo of the two for mild symptoms and relief
2. LABA or LAMA or combo
3. LABA + LAMA + ICS (only if needed, increased risk of side effects with ICS)
may need to refer if these are insuffieicnt for COPD relief

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Etiology vs asthma and COPD

A

asthma: reactive airways, genetic predisposition caused by eisonophils and increased inflammatory process
COPD: reactive airways that are narrowing due to preventable causes, causes chronic bronchitis and emphysema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Clinical symptoms of asthma vs. COPD

A

asthma: wheezing (hallmark sign), chest tightness, cough, infrequent and episodic, can be triggered by environment or activity
COPD: increased sputum production, cough, SOB, dypsnea, does not usually improve with bronchodilators

17
Q

Diagnostic evaluation of COPD and asthma

A

asthma: spirometry, reversible airway construction post dilator, increase in FEV after bronchodilator
COPD: spirometry, FEV <0.7 despite bronchodilator, reveals ongoing constriction despite treatment

18
Q

Describe an asthma vs. COPD action plan

A

asthma: education surrounding triggers and disease management (including use of bronchodilators as rescue inhaler)
COPD: smoking cessation, use of inhalers, identify triggers, vaccination history

19
Q

Red flags for red eye

A
increasing pain
photophobia
increased redness
decreased visual acuity
foreign body/ penetrating eye injury
20
Q

Describe the clinical manifestations and treatment for viral conjunctivitis

A

usually accompanied by a URTI
quick onset, red eye and clear discharge, often unilateral but spreads bilaterally
follicles to lower lids, pharyngo conjunctival fever, tender preauricular and anterior chain lymph nodes

treatment: self limiting, cool compresses, artification tears, contagious if still tearing

21
Q

Describe the clinical manifestations and treatment for bacterial conjucttivitis, what are the most common pathogens causing this condition

A

usually accompanied by a URTI
purulent discharge, may or may not have eye pain
could be caused by alternative pathogens such as Ct or GC, most commonly MRSA, staph, s. pneumoniae, h influenzae, m catarrahlis

treatment: self limited, try 10-14 days without treatment
no resolution can try topical macrolides ie) erythromycin, tobramycin

22
Q

describe the clinical manifestations and treatment for allergic conjunctivitis

A

caused by allergens
itching, tearing, redness, stringy discharge, benign

treatment: antihistamines (drops or systemic)

23
Q

What is the approach to epistaxis?

A

usually from the anterior nares due to trauma, humidity, cocaine or etoh use, anticoagulation
bleeding into pharynx is usually posterior bleeding
manage by applying pressure for 15 minutes
nasal decongestants sprays
cautery or silver nitrate or balloon

refer if bleeding is >15 minutes, or recurrent large volume epistaxis

bleeding stable? no sraing and vigourous activity for 14 hours, control diet (spicy foods can trigger epixtaxis)
maintain lubrication

24
Q

What are common pathogens that cause sinusitis and what are the common treatments?

A

commonly viral, bacterial, and fungal causes
baterial: s. pneumoniae, h influenzae, m catarrhalis
clinical manifestations: inflammation of paranasal sinuses, congestion, purulent discharge, headache, pressure, quick onset, facial pain, dental pain
maxially and ethmoid sinuses are the most common areas
sinuses become blocked, bacteria sits and inflammatory process occurs

treatment: NSAIDs for pain
decongestants (nasal sprays)
saline rinses
usually imrpves in 2 weeks iwth no systemic abx

consider abx -amoxi/clav if severe symptoms and >10 days or immunocompromised

25
Q

contraindications to ear syringing

A
altered mental state or uncooperative
previous surgeries or tympanoscope
choleastema (non cancerous skin growth) 
anticoagulation
otitis media/perforation