Module 2 Exam 1- Flashcards
Acute Respiratory Distress Syndrome (ARDS)
severe form of respiratory failure with rapidly progressing hypoxemia; causes alveolar injury and damage to endothelial lining; reduction of lung compliance, volume, and gas exchange; d/t pneumonia, sepsis, or multiorgan failure; most fatal in 24-48 hours
common causes of ARDS
aspiration of gastric contents, shock, sepsis
signs and symptoms of ARDS
hypoxia that doesn’t improve with O2 is major sign; tachypnea, increased dyspnea, tachycardia, HTN, restlessness, xray shows visible bilateral infiltrates and pulmonary edema (BNP distinguished from pulmonary edema)
prone positioning for ARDS
improves oxygenation, increases end-expiratory lung volume, improves bronchial drainage, improves functional residual capacity, improves gas exchange; use of rotoprone
high pressure alarm on ventilator
coughing, mucus plug, kinked tubing, decreased lung compliance (bucking the vent), atelectasis or bronchospasm
low pressure alarm ventilator
increase in lung compliance; leak in ventilator tube; leak in ET cuff
suctioning when pt intubated
not a routine thing; suctioned if - visible secretions in ET tube, onset of respiratory distress, suspected aspiration, increased airway pressure, adventitious breath sounds over trachea or bronchi, increased RR, sustained coughing, sudden decrease in O2 levels, or for sputum culture
sedation vacation
once a shift to observe neuro status
if cause of ventilator alarm cannot be determined…
ventilate manually until problem corrected
nursing management of epistaxis
pinch and lean forward for 5-10 minutes
obstructive sleep apnea
obstruction of upper airway causing decreased ventilation and gas exchange; intermittent breathing
clinical manifestations of obstructive sleep apnea
as described by parter- loud snoring/snorting, apneic events, gasping, recurrent waking, sleep disruption, choking; as reported by pt- excessive daytime sleepiness, workplace or car accidents, decreased cognitive functioning, retention and performance compromise
risk factors for obstructive sleep apnea
obesity, DMII, heart failure, pulmonary HTN, large neck circumference
diagnosis of obstructive sleep apnea
health history and sleep study
treating obstructive sleep apnea
lifestyle modifications, CPAP (constant positive airway pressure), surgery
polysomnography
overnight sleep study to monitor number, duration, and frequency of apneic episodes
nursing management of epistaxis
pinch soft portion of nose and lean forward 5-10 min; maintain adequate ABCs; vital signs (cardiac and pulse ox); teach pt to avoid nasal trauma, increase humidity if dry air, and seek health care attention if continues after 15 min
laryngectomy
removal oof whole or partial larynx/voice box
laryngectomy nursing intervention
preop teaching, reduce anxiety, maintain airway, control secretions, decide alternative communications d/t loss of communication following surgery, promote adequate nutrition and hydration, positive body image
complications with laryngectomy
respiratory distress, hemorrhage (leads to aspiration of blood), wound breakdown, aspiration, hoarse voice, edema
pulmonary embolism
clot in pulmonary artery or branch that occludes blood flow; can be fat, air, thrombus, amniotic, or septic; severity based on size; increased vascular resistance and pulmonary arterial pressureris
risk factors for developing ppulmonary embolism
virchows triad, DVT in legs arms or pelvis
virchows triad
venous stasis, hypercoagulative state (pregnant or COPD), endothelial damage (diabetes or HTN)
saddle PE
clots forms at biforkation of lungs blocking bloodflow to both lungs
clot busting drug for PE
altiplase
clot preventing drug for PE
warfarin
pulmonary embolism clinical manifestations
sudden dyspnea, pleuritic chest pain, anxiety/impending doom, tachycardia, cough, diaphoresis (profuse), hemoptysis, syncope, petechiae
diagnostic test for pulmonary embolism
elevated d-dimer, ekg, chest xray, ABG, spiral CT, VQ scan (ventilation/perfusion), pulmonary arteriogram (golden standard)