Module 2: Cardiovascular Exam Flashcards
What make up the atrioventricular (AV) valves & what is their purpose?
•Tricuspid
• mitral (bicuspid)
One way blood flow that separates atrium from the ventricles
What make up the semilunar valves & what is their purpose?
• pulmonic
• aortic
One way blood flow that separates pulmonary trunk& atrium
What is cardiac output & what factors affect this?
Volume of blood ejected from ventricles over 1 minute
Affected by:
• HR
• stroke volume
What is stroke volume & what does it depend on?
The amount (volume) of blood ejected w/ each heartbeat influenced by:
•Preload
• myocardial contractility
• after load
What is pre-load & at what part of the cardiac cycle does it occur?
Amount of blood in ventricles at the end of diastole
What are some factors that increase pre-load?
Any increased venous return to the heart such as:
• inspiration
• exercise
• dilated right ventricle in heart failure
What are some factors that decrease pre-load?
Any decreased venous return to the heart such as:
• exhalation
• Decreased ventricular output
• pooling of blood in the periphery: capillary bed or venous system
How does inspiration increase pre-load in the ventricles?
Decreased thoracic pressure allows more blood back to the heart
How is myocardial contractility increased & what are some examples?
Stimulation by sympathetic nervous system
- fight or flight
> organs are mobilized & their functions during stress and arousal
Ex) exercise, stress, fear
What happens when there is decreased contractility and what can influence this?
Blood Flow or O2 delivery to myocardium is impaired
^ age = decreased elasticity of muscles
What is after-load & what happens when there is an increase in after-load?
Amount of pressure/vascular resistance that the heart has to contract against
Increased resistance in after-load = decreased cardiac output
- left ventricle has to work harder to get the blood out
What factors resist contractility of the heart & increase after-load in the ventricles?
Tone in walls of aorta
Tone in large arteries (ex: subclavian steal)
Volume of blood in aorta
What factors influence Ventricular function of the heart & how can this be detected on physical exam?
• volume overload
• pressure overload
PE: change in palpable pulses & heart sounds
What happens when there is a pathological increase in pre-load?
Ventricular dilitation → volume overload
What happens when there is a pathological increase in after -load?
Stiffness in blood vessels making it hard to pump blood out of ventricles → atherosclerosis
What is being evaluated on inspection of the precordium?
- evidence of trauma
- bony abnormalities
- Skin abnormalities
- Vascular congestion
- visible lifts or heaves
on inspection of the precordium, patient’s chest wall extrudes & looks like a pigeon. what is this bony abnormality called?
pectus carinatum.
on inspection of the precordium, patient’s chest wall has an inward depression & looks like a funnel. what is this bony abnormality called?
pectus excavatum
how/where would you palpate the precordium for lifts/heaves & what do they feel like? what can they indicate?
light palpation using fingertips -> R & L 2nd ICS, L 4th ICS, & 5th ICS MCL
> impulse will noticeably raise your hand from the chest wall
*indication that cardiac output is increased Examples: Hypertrophy, Heart failure -anemia -anxiety -HTN -fever