Module 2: Cardiovascular Ax Flashcards
cardiac output assessment
- determined by stroke volume and heart rate
- stroke volume is determined by preload, afterload, and contractility
- each of these components exert influence over the others, impacting overall cardiac output
cardiac output
volume of blood ejected from the heart in one minute (L/min)
what will heart rate do as a compensatory mechanism for low CO and low PaO2
increase
what are the effects of an excessively increased HR?
- reduced ventricular filling time (leading to decreased preload and decreased contractility)
- increased cardiac demand
- reduced coronary artery filling time (coronary arteries fill and supply cardiac muscle with oxygen during diastole)
stroke volume
amount of blood ejected from the heart with each contraction (mL/beat)
how is stroke volume determined?
by the volume of blood in the heart at the end of diastole (the period of relaxation between beats) and the amount of blood that is expelled during systole (the period of contraction when the heart pumps blood)
what is stroke volume influenced by?
- preload
- afterload
- ventricular contractility
preload
the amount of stretch or tension in the ventricular wall of the heart at the end of diastole just before the contraction or systolic phase begins
what is preload influenced by?
- venous return (circulating blood volume and its ability to return to the heart)
- cardiac rhythm (which impacts atrial kick and filling time)
- ventricular ability (related to ventricular compliance as well as ability to contract).
assessments that provide info about preload
- S3 heart sounds (caused by the “sloshing” of excessive blood flowing from the left atrium into the left ventricle)
- fluid balance (ins/outs, daily weights, mucous membranes, skin turgor)
- crackles on lung auscultation**
- edema**
- POCUS
- echo
- fluid volume responsiveness
- pulse pressure variation
- stroke volume variation
- CVP
can you rely on crackles and edema as evidence of preload status?
NO:
- Crackles can result from pulmonary congestion caused by fluid overload and poor contractility but can also signify consolidation from pneumonia.
- peripheral edema can take time to both accumulate and reabsorb. Fluid collected in the interstitial spaces is not a direct reflection of fluid currently circulating intravascularly
afterload
force, or the resistance, against which the ventricles must pump in order to eject blood
what is afterload determined by?
the tension or pressure that the ventricles must generate to overcome the resistance in the blood vessels and pump blood forward
what happens if increased resistance is encountered?
ventricles must contract more forcefully in order to maintain a normal stroke volume, increasing myocardial workload and oxygen demand
what if a ventricle is not able to generate sufficient force?
stroke volume will decrease, and the amount of blood leaving the heart will be reduced