Module 1 Lesson 3 Flashcards

1
Q

What ensures atrial excitation and contraction are completed before ventricular events?

A

Electrical conduction system of the heart

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2
Q

What is the primary function of the sinoatrial (SA) node?

A

Acts as the heart’s pacemaker, initiating electrical impulses

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3
Q

What does an electrocardiogram (ECG) measure?

A

The electrical activity induced in body fluids by the cardiac impulse that reaches the body surface.

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4
Q

What are the two phases of the cardiac cycle?

A

Systole (contraction) and diastole (relaxation)

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5
Q

How long is the cardiac cycle at a heart rate of 75 beats per minute?

A

0.8 seconds

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6
Q

What happens to ventricular diastole when heart rate increases from 75 to 180 beats per minute?

A

Ventricular diastole duration is reduced by about 75%.

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7
Q

What are the two main factors that regulate cardiac output?

A

Heart rate and stroke volume

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8
Q

How does parasympathetic stimulation affect heart rate?

A

It decreases heart rate by slowing SA node activity and increasing AV node delay.

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9
Q

How does sympathetic stimulation affect heart rate?

A

It increases heart rate by accelerating SA node activity and conduction velocity at the AV node.

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10
Q

What neurotransmitter is involved in sympathetic stimulation of the heart?

A

Noradrenaline (Norepinephrine)

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11
Q

Why is it important that atrial contraction occurs before ventricular contraction?

A

It ensures the ventricles are properly filled with blood before pumping.

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12
Q

How does the cardiac conduction system allow for coordinated heartbeats?

A

The SA node generates impulses that spread rapidly through the atria, delay at the AV node, and then conduct quickly through the ventricles.

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13
Q

What is the significance of the AV node delay?

A

It ensures the ventricles contract after the atria have emptied their blood into them.

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14
Q

Why does heart rate increase when the heart is removed from the body?

A

The parasympathetic nervous system normally slows the heart rate; without it, the intrinsic SA node rate is higher.

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15
Q

Why does parasympathetic stimulation hyperpolarize the SA node?

A

It increases K⁺ permeability, making it harder for the membrane potential to reach the threshold for an action potential.

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16
Q

Why does sympathetic stimulation increase the rate of depolarization?

A

It decreases K⁺ permeability and increases inward Ca²⁺ currents, allowing faster reaching of the threshold potential.

17
Q

How does the cardiac cycle ensure one-way blood flow?

A

Pressure changes open and close heart valves at the right time.

18
Q

Why does stroke volume decrease at very high heart rates?

A

Less time for ventricular filling leads to lower end-diastolic volume.

19
Q

If a patient’s ECG shows no P waves, what might be happening?

A

The SA node is not firing, possibly due to sinoatrial block or atrial fibrillation.

20
Q

How would an AV node block affect the ECG?

A

It would cause prolonged PR intervals or dissociation between P waves and QRS complexes.

21
Q

If a patient has a resting heart rate of 50 beats per minute, what could be the cause?

A

Increased parasympathetic (vagal) activity, which slows the SA node.

22
Q

How does the body compensate for a decrease in stroke volume?

A

By increasing heart rate to maintain cardiac output.

23
Q

If a patient has excessive sympathetic stimulation, what ECG changes might be observed?

A

Increased heart rate (tachycardia) and possibly shorter PR and QT intervals

24
Q

How would a beta-blocker affect heart rate?

A

It would reduce sympathetic activity, leading to a slower heart rate.

25
Q

Why does the dicrotic notch appear in the aortic pressure curve?

A

It represents the brief backflow of blood that closes the aortic valve after ventricular systole.

26
Q

How does a pacemaker help patients with arrhythmias?

A

It delivers electrical impulses to regulate heart rate and rhythm.