MOD2 Flashcards

1
Q

Paraphilic disorders

A

disorders of sexual behaviour

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2
Q

Behavioral accounts

A

This explains patients behaviors and does not diagnose
learning mechanisms (classical and operant) are not disordered but are normal
learned behavior is maladaptive

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3
Q

limitations of classical conditioning

A

Elimination of problem association is not sufficient
Poor or lack of generalization (extension of effect beyond specific instances present during initial learning)
Significant individual differences
non-uniform distribution of fetish objects . . . pink, black, smooth, silky, shiny

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4
Q

operant conditioning interventions

A

masturbatory satiation . . . continued masturbation during the refractory period, concurrent with fantasizing
verbal satiation . . . verbalizing fantasies while withholding from masturbation
orgasmic reconditioning . . . switching fantasy content following arousal; evidence of efficacy is weak

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5
Q

Features of depression

A
  • affect
    sadness, loneliness, emptiness, irritability
  • motivation
    loss of interest, yearning for escape, paralysis of the will
  • cognition
    negative self-concept, pessimism, guilt, negative interpretations of experience
  • behavior
    reduced activity
  • physical
    retarded movement, fatigue, weight change, appetite, sleep, sex drive
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6
Q

Components of psyche

A

ID - embodies innate drives and instincts
Ego - works to mediate ID and superego
Superego - moral values (sense of conscious)

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7
Q

Normal discharge of energy

A

Healthy mechanisms (sex, sport, pursuit of desires)
Unconscious mechanisms (express needs of unconscious in indirect way; uncontrollably)

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8
Q

Introjection

A

integration of identities of self with loved (lost) one
direction of feelings (anger) onto self
low self-esteem, depressed mood, helplessness

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9
Q

regression

A

to the oral stage of development (18–24 mths)
dependence; new dependencies, to elicit support

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10
Q

Beck and limitations of the psychoanalytic account

A

They ascribe purpose to symptoms (there is a reason someone is sleeping less)
They defy validation (cant measure directly)
They are not disorder specific
They may explain limited aspects of disorde

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11
Q

Soultion to the limitations of the psychoanalytic account

A

consider basic themes in the thinking (cognitive content) reported by depressed patients

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12
Q

The cognitive triad

A

The self: viewed as deficient, inadequate, understandable, worthless, defective
The world: experiences construed as defeat, deprivation, burdensome
The future: suffering will continue indefinitely
depressed affect and motivational changes are direct consequences of the primary triad

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13
Q

Thinking errors of depression (cognitive model)

A

patients tend to make logical errors in reasoning
Arbitrary inference: jump to conclusions
Selective abstraction: attend to negative and ignore other aspects of situations
Over generalizations: draw sweeping conclusions from minimal events
Dichotomous thinking: thinking in all or none terms

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14
Q

strengths of the cognitive model

A

Captures the fundamental problem of basing self worth or the sense of true value in personal experience
Intuitive appeal; clinical utility, effectiveness of intervention based on cognitive model.

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15
Q

Predictive validity

A

correlation between a score and a criterion outcome

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16
Q

Content validity

A

the items represent the target universe

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17
Q

Face validity

A

the extent to which a test is subjectively viewed as covering the concept it purports to measure

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18
Q

Concrete validity

A

concerns how well a set of indicators represent or reflect a concept that is not directly measurable

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19
Q

Principles for demonstrating construct validity

A
  1. Defined the construct implicitly, by a network of ideas about the construct. Constructs may be nearly descriptive . . . or very conceptual.
  2. These ideas must lead to testable hypotheses, predicted relationships among observable phenomena.
  3. The ideas (in 1) and hypotheses (in 2) must be explicit so evidence can be properly evaluated.
  4. Construct validity is not represented in a single number. Many types of evidence are relevant: content validity, predictive and concurrent validity (with diverse criteria), test-retest reliability, internal consistency (as well as more advanced psychometric properties)
  5. When a prediction fails, one or both of the following may be at fault: the test . . . modify the test — the network . . . modify the construct
  6. Evaluating the construct validity of a test is essentially no different to developing and testing theories.
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20
Q

Personality disorder in DSM5

A

An enduring pattern of inner experience and behavior deviating from expectations of an individual’s culture and:

21
Q

Odd Eccentric Cluster

A

Paranoid, Schizoid, and Schizotypal Dramatic

22
Q

Flamboyant Cluster

A

Antisocial, Borderline, Histrionic, and Narcissistic

23
Q

Anxious Fearful Cluster

A

Avoidant, Dependent, and Obsessive-Compulsive

24
Q

Schizotaxia

A

problem with the brain that could result in schizophrenia

25
Q

A peak is evidence of

A

discontinuity

26
Q

Results from studies show that schizophrenia is

A

discontinuous as there are peaks and they are at the proposed 10%

27
Q

misery and distress

A

dimensional

28
Q

Somatic signs of depression

A

taxonic

29
Q

worry

A

dimensional

30
Q

borderline personality

A

dimensional

31
Q

bulimia nervosa

A

dimensional

32
Q

Cognitive-Rigidity Diathesis-Stress Model (single process theory)

A

Cognitive rigid individuals when placed under naturally occurring conditions of high life stress are cognitively unprepared to develop effective alternative solutions necessary to cope with the stressors in their environment, as a result of their inability to engage in effective problem solving they are assumed to become helpless under such circumstances and this helplessness places the individual at heightened risk for suicidal behavior.

33
Q

Ideation-to-Action Frameworks (multiple process theories)

A

These frameworks distinguish between suicidality in terms of ideation (suicidal thinking) and the behavior (the actions)
Distinguish different causal pathways for these two things

34
Q

Interpersonal theory of suicide

A

Two key processes that affect suicide ideation/thinking
Thwarted belonging
Perceived burdensomeness

Key process for suicidal behavior
Acquired capability e.g. lower fear, elevated pain tolerance

35
Q

3 step theory

A

Pain + hopelessness = ideation
Connectedness > pain then mild ideation else severe
Capacity for suicide

36
Q

Reductionism

A

Mistaking low level as a cause

37
Q

study of group of people with spider phobia

A

Post treatment; behavior in brain was normalized

38
Q

Clinical impressions of schizophrenia

A

Ambivalence
Associative disturbances
Affective disturbances
Preference for fantasy over reality

39
Q

Formal thought disorder

A

impaired capacity to sustain coherent discourse, and occurs in the patient’s written or spoken language

40
Q

Study showed direct and indirect semantic priming is … in schizophrenia + TD

A

increased

41
Q

Rates of reoccurrence bipolar

A

¼ within 6 mths; 90% within 10 yrs)

42
Q

following a single depressive episode, …% to …% of cases transition to bipolar disorder

A

5 - 10

43
Q

Theory: Kindling and Sensitisation (Post)
— recurrence of episodes is the norm

A

Kindling — stimulation that is insufficient to produce overt behavioral effects, when supplied intermittently, can lead to the development of those effects
Sensitisation — increasing behavioral responses to intermittent stimuli

In anticipation of a stressor, the body kicks in with compensatory mania that helps lift mood out of depression, but in the absence of depression mania occurs

44
Q

4 components of kindling and sensitisation

A

Real loss: comes before stressor or loss (= UCS)
symbolic components of the stressor or loss (= CS)
characteristics:
Quality
Intensity
periodicity (temporal cycle)
(perceived) degree of control
history and environmental context

Real reaction: depression (= UCR)

Mechanism leading to increasing recurrence:
symbolic components of a previous trigger of a depressive response . . . become capable of producing the behavioral, physiological and biochemical alterations usually associated with an affective episode”
trigger = anticipated stressors, imagined losses
patients become sensitized to the recurrence of episodes
episodes are more easily triggered
episodes may eventually occur spontaneously

Mechanism leading to emergence of manic behavior:
subjects with a history of depressive responses may display [conditioned] depression-compensatory responses (i.e. mania) when confronted with cues that would ordinarily elicit a depressive reaction

45
Q

Kennedy et al. (1983)

A

argued the likelihood of bipolar disorder occurring is related to the prior major life stressor.

46
Q

Leverich and Post (2006)

A

patients with abuse history had an earlier age of onset of illness and went longer without treatment for bipolar

47
Q

Blueuler (1911)

A

Four core features of schizophrenia: Ambivalence, Associative disturbances, Affective disturbances, Preference for fantasy over reality

48
Q

Schotte & Clim, 1982

A

The level of self-reported suicidal ideation/intent is greatest when stress is high and problem solving is poor.
Findings were that
- Stress, depression and hopelessness are important predictors of ideation
- No evidence regarding cognitive rigidity
- In isolation, problem-solving not related to ideation