Mobilising principles Flashcards

1
Q

List factors which can contribute towards limited joint range

A

Degenerative changes, disease processes, trauma, immobility, muscle weakness, muscle spasm and pain

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2
Q

Give methods of joint mobilisation

A

Manual therapy techniques
Passive movements
Exercise interventions

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3
Q

List the principles of joint mobilisation which make movement in the desired plane easier, mobilising through range and to end range

A

Continuous movement (reduce inertia)
Increase Momentum (increase speed)
Increase Momentum (add mass)
Large amplitude, through range movement
Gravity-assisted movement
Auto-assisted movement
Reduce Friction
Mobilise to end-of range
End of range holds

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4
Q

Which law does the principle of ‘continuous movement’ follow?

A

Newton’s 1st Law
Reduces muscle work required to stop and start the movement.

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5
Q

Which law does the principle of ‘increased momentum’ follow?

A

Newton’s 2nd Law
Beware of using momentum to increase a range of movement which has a natural bony block (i.e. knee and elbow extension) as this could cause injury.

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6
Q

Which law does the principle of ‘large amplitude/through range movement’ follow?

A

Newton’s 2nd Law.
Increases sweep and squeeze of synovial fluid.

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7
Q

Which law does the principle of ‘gravity assisted movement’ follow?

A

Newton’s 2nd Law
Acceleration aided by gravitational force.

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8
Q

Which law does the principle of ‘auto-assisted movement’ follow?

A

Newton’s 2nd Law
Acceleration aided by own (healthy limb) force.

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9
Q

Which law does the principle of ‘reduced friction’ follow?

A

Newton’s 3rd Law
Reduces muscle work required to start or sustain the movement.

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10
Q

Which law does the principle of ‘end of range holds’ follow?

A

To effect neural (e.g. stretch tolerance) and viscoelastic changes in the connective tissue.

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11
Q

List key theraputic principles intended to increase patient compliance with exercises

A
  • Use of targets (especially to achieve end range)
  • Motivation (use of voice) & correction if required
  • Competition (with self or others)
  • Variety of exercise & individual approach (consider patient age & interests)
  • Instruction & demonstration
  • Explanation of potential benefits
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12
Q

Give the practical considerations behind mobilising exercise

A
  • Patients should be appropriately undressed to allow the Physiotherapist to visualise the affected joint
  • Consider the optimal starting position for each exercise from both patient comfort and exercise effectiveness perspectives
  • Include a localised light warm-up for the body area that is being treated e.g. ankle circling for 1-2 minutes prior to dorsiflexion mobilising
  • Use any available equipment to improve efficacy and to add interest for the patient
  • Always give the patient at least 2 exercises that are suitable for home use with clear instructions for frequency, reps and sets
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13
Q

To be effective what should the ROM achieved be for each joint?

A

In order to be effective, it is essential that a mobilising exercise programme is performed both through range and to end range and with sufficiently high repetition.

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14
Q

How many reps and sets should be perfomed for each mobilising exercise?

A

3 sets of 10-15 reps should be performed for each exercise

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15
Q

How frequently should the at home exercises be performed by the patient?

A

1/2 times daily

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16
Q

How long should static stretches be held at end of range?

A

30 secs

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17
Q

Describe Proprioceptive Neuromuscular Facilitation (PNF)

A

PNF is a series of techniques that use the following principles in order to effect a specific change in the function of the patient:
* All movement is governed by voluntary and sensory sources and utilises both voluntary and reflex movements.
* Proprioceptors increase and reinforce the demands of the voluntary system.
* Reflex reactions are stimulated to initiate and reinforce voluntary movement.

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18
Q

Describe the process of ‘contract-relax’ PNF stretching

A

This is a method of stretching with the goal of increasing passive range of movement. It is typically used to stretch two-joint muscles (e.g. hamstrings). Following a maximal resisted contraction of the antagonist at end range, the muscle is relaxed and passively moved further into range.
* Position the patient with the muscle to be stretched (antagonist) at end range (remember to consider both joints for two-joint muscles).
* Therapist to position themselves at a mechanical advantage and provide resistance as the patient maximally contracts the muscle to be stretched for at least 5 seconds.
* Instruct the patient to “relax” and as they do so, move the limb passively further into range, sustaining the passive stretch for at least 30 seconds.
* Without releasing the tension, repeat the technique until no more range is gained, finishing with a final 30s stretch.
* As this is a therapist-assisted technique, always teach the patient static stretching for the same muscle group as an accompanying home exercise.

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19
Q

Give examples of intra and extra capular stuctures which if dysfunctioned can reduce joint movement

A
  • Ligaments
  • Capsule
  • Cartilage
  • Tendons
  • Fascia
  • Synovium
  • Muscles
  • Skin
  • Bone
  • Subcutaneous tissue e.g. fat or bursa
  • Neurological Control

Reduced movement can lead to abnormalities in these structures
Mechanical stimulation is necessary to maintain function of these structures.

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20
Q

Briefly describe the composition of connective tissue

A

Made up of cells (e.g. fibroblasts/chondrocytes/chondroblasts/osteoblasts)
and the extracellular matrix.
The EC matrix is made up of fibres (reticulin/collagen/elastin) and ground substance (proteoglycans/GAGs/water)

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21
Q

Describe the turnover of collagen

A

Normally a balance between ongoing synthesis & degradation (enzyme-activated) of connective tissue matrix and ground substance mediated by fibroblasts. Turnover is relatively slow (especially compared with muscle) – collagen has a very long half-life in health, higher with injury or inflammation.

22
Q

Describe ‘tensile loading’ of collagen

A

Tensile loading from normal movement and function e.g. weight bearing stimulates synthesis of collagen which is laid down oriented parallel to the lines of stress. Collagen has very high tensile strength.

23
Q

Give factors that determine range of movement of a joint

A
  • The structure of the joint itself (arthrology)
  • Resistance within the joint itself
  • Normally incredibly low co-efficient of friction
  • The properties of each joint structure
  • Intra-articular e.g. cartilage
  • Peri-articular e.g. joint capsule
  • Extra-articular e.g. muscle-tendon units
  • How external forces are transmitted by the articular soft tissue
  • Function of the passive viscoelasticity- exhibit time-dependent strain. Elastic structures stretch and reform, viscous structures resist strain when stress is applied.
  • Elasticity – reformation of the substance after a stretch
  • Viscosity – strain increases with time (creep) Creep is increasing deformation under constant load
  • Varying concentrations of the following determine viscoelastic properties and hence the response to lengthening:
    Elastin
    Collagen
    Proteoglycans
    Water
  • Plasticity = deformation of a (solid) material undergoing non-reversible changes of shape in response to applied forces
  • Type of collagen also important.
24
Q

List mechanical properties of articular connective tissues

A

Dense connective tissue is very strong
* Organised structure
* Resistant to tensile stress = stiff
* Strongly resistant (rope-like) along lines of stress
* Tendon>Ligament>joint capsule strength
* Coiled coils. Example of MCL at the knee – strongly resists valgus stresses and under tension at end range knee extension

25
Q

Explain the properties of collagen fibres

A

Collagen fibres are incredibly strong. Their coiled coil structure is the same as rope. The crimp allows extension of the length without straining the fibrils. Rupture occurs at around 15% elongation in health connective tissues. Elasticity adds to the strength as no elasticity would result in a more brittle structure.

26
Q

Describe the effect of loading on connective tissue

A

If you hold the tissue at a constant load, a gradual deformation will occur without increasing the load further. If you constantly deform a tissue, there will be some stress relaxation over time. Studies suggest at least 30 mins of sustained loading is required to affect a permanent change in length of established connective tissue.

27
Q

Define ‘plastic deformation’

A

A permanent change in a tissue’s length..

28
Q

List causes of reduced range of movement

A

-Trauma: fractures, muscle sprains, strains
-Chronic disease: osteoathritis, rheumatoid arthrtis, neuromuscular disorder
-Immobility: nursing home, hospital, older persons

29
Q

List effects of reduced ROM

A
  • Function:
    UL - Reduced independence with functional tasks e.g. dressing oneself
    LL - Poor gait & functional mobility, falls risk
    Time off work
    Quality of life
  • Pain
  • Muscle weakness
  • Vicious circle of inactivity
    QoL e.g. family commitments, time off from hobbies, mental wellbeing.
  • Muscle weakness in the lost range.
  • E.g. 5 degrees off full knee extension causes limp
30
Q

Describe the cycle of immobility

A

Decreased ROM > decreased function > deconditioning > decreased motivation > decreased activity >

31
Q

Describe the physiological changes of immobility without injury for connective tissue

A

Immobility > stress deprivation > remodelling
Early changes:
-derceased collagen (increased degredation)
-decreased GAGs and water content
-decreased allignment of collagen fibres
-decreased crosslinks between collagen fibres (collagen is shorter, weaker and decreased resistance to tensile stress)

Late changes:
-fibroblast proliferation
-myofibroblasts
-dense, disorganised collagen fibrils
-adhesions
-fibrocartilage
-ossifications

Whilst the structures have shortened in the early phase, they are weaker and less resistant to tensile stress therefore opportunity to effect an improvement in ROM. Resistance to movement in the wrong direction => joint stiffness

32
Q

Describe the physiological changes of immobility in healing injured connective tissue

A

Similar process to immobilisation without injury but fibroblast and myofibroblast proliferation more significant therefore effects are compounded and more likely to result in adhesions, especially if joint completely immobilised e.g. fracture.
-Collagen fibres disorganised with some perpendicular fibres
-Weaker overall structure
-Weakly resistant in all directions
-Resists movement in multiple planes

33
Q

Are ‘stiff fibres’ desirbale to joint ROM?

A

Stiff fibres are strong fibres and are desirable (to a point). Injured or immobilised structures are actually much weaker but will resist in unhelpful directions. Net-like structure rather than rope-like structure.
Example = MCL at the knee. Normally resists valgus stress and end range extension. After immobility / injury anterior fibres would resist flexion and posterior fibres would resist extension earlier in range.

34
Q

Give examples of physiological changes of immobility to intra-articular structures

A

Cartilage:
* ↓ed water content
* ↓ed GAGs
* ↓ed nutrients from synovial fluid
* Thinning of extra-cellular matrix
* Fibro-fatty connective tissue proliferation
* Adherence of synoviocytes and protein deposits

Synovium:
* ↓ed number of synoviocytes
* ↓ed synovial fluid volume
* ↓ed synovial fluid movement
(loss of “sweep & squeeze”- due to less pressure changes within the joint especially when NWB.)
* ↓ed Lubricin (lubrication)
* Adhesions between micro-folds in synovial membrane

35
Q

Give examples of physiological changes of immobility to bone

A
  • ↑ resorption of both cancellous + compact bone (after approx. 8 weeks)
  • ↓ bone mineral density (BMD)
  • ↓ ability to withstand stress
  • increased risk of fracture with excessive force
36
Q

Give examples of physiological changes of immobility to muscle

A
  • Atrophy
  • Sarcomeres lost from end of myofibrils => shortening
  • Connective tissue proliferation => ↑ed collagen within endomysium, perimysium & epimysium => adaptive shortening

Important to note that effects on muscle are only one of the many effects of immobilisation on joints therefore treatment should focus on other structures and not solely muscle stretching. In health & early immobility (<4 weeks), the main limitation to joint range is the musculo-tendinous unit.
Effects more pronounced if bone or muscle received a trauma prior to immobilisation (as per connective tissue healing).

37
Q

Describe the window of opportunity/the timeline for intervention prior to immobilisation

A

Physiological changes that reduce ROM occur within 2 weeks of immobilisation.
Changes within 4 weeks are usually fully reversible, longer than 4 weeks may lead to permanent shortening.
↑ severity of injury → ↑ inflammatory response,↑ fibrous repair tissue,↑ likelihood of reduced flexibility of the injured tissue
Concept of window of opportunity- at 4 weeks for reversal of reduced ROM
Concept that preventing loss of range of movement is preferable to reversing it.
Irreversable changes occur after approximately 6-12 weeks after the onset of immobilisation/injury
Use example of POP immobilisation at onset then POP removal & immediate re-mobilisation of joint

38
Q

Give examples of mobilisation therapetic techniques

A
  • Active Exercise
    -Assisted Exercise
    -Gravity-assisted
    -Auto-assisted
    -Therapist-assisted
  • Passive Stretching
    -Therapist
    -Contract-relax
    -Self-administered
    -Splinting
  • Passive Movements
  • Physiological
  • Accessory
39
Q

What considerations must be given to mobilisation exercises

A

Efficacy?
Which structures?
Which mechanisms?
Practicality?
Frequency?
Intensity?
Duration?
Combination?
Passive physiological could be therapist or CPM machine.
Unlikely to gain new range with active movements alone – need some sort of assistance to overcome resistance at end of range given that muscle atrophy will have occurred in the lost range.
Auto assisted e.g. other arm/leg, body weight, using a strap

40
Q

What is through range mobilisation?

A

Exercises/techniques that take the tecniques through their full availble range of movement: active assisted, passive physiological.

41
Q

Give effects of through range mobilisation

A

-Stimulation of synoviocytes:
increased synovial fluid volume, increased synovial fluid movement (sweep and squeeze), increased lubricin

CAUSES increased synovial fluid to cartilage:
increased water content, increased GAGs, normalised EC matrix

-reduced friction
-reduced resistance to movement

42
Q

List the neural effects of end-range mobilisation

A

-Increased stretch tolerance
-Reduced antagonist muscle activity
-Pain modulation

Contract-relax utilises the neural effects to increase range of movement where it is limited by muscle.

43
Q

Give the structural effects of end-range mobilisation

A

-Stress collagen tissues:
Stimulates collagen synthesis
Collagen laid down parallel to lines of stress- strong in the ‘right’ direction
Creep and stress relaxation occur with sustained stretch
Plastic deforamtion may occur in ‘windown of opportunity’

44
Q

List the effects of static stretching

A

-Regular static stretching increases the max joint ROM
-Mechansim is debateable
-Increased stretch tolerance
-Little/no changes in tensile properties of muscle tissue
-Suitable for home exercises

45
Q

List the effects of dynamic stretching

A

-Similar effects to static stretching (increased max joint ROM, increased stretch tolerance)
-Not reccommended in patient population due to risk of reinjury

46
Q

GIve effects of propriceptive neuromuscular facilitation

A

-Contract/relax technique of the antagonist
-Maximal relaxion follows maximal contraction
-Increases the maximum joint ROM
-Increased stretch tolerance
-Little/no changes in tensile properties of muscle tissue
-Similar/more effective than static stretching

47
Q

Give features of a mobilising programme

A

-Through range
-End-rabge
-Sustained holds
-Frequent, needs to be done regularly by patient away from therapy sessions
-Easy to move into that plane of movement : patient in gravity-assisted/counterbalanced position

48
Q

Which rules/laws need to be considered in mobilisation programmes?

A

-Minimise inertia, continuous movements (newtons 1st law)
-Maximise momentum: large amplitude, speed up, long lever arm (newtons 2nd law)
-Minimise friction (newtons 3rd law)

49
Q

State newton’s first law of motion

A

Newton’s First Law of Motion states that an object in motion tends to stay in motion unless an external force acts upon it. Similarly, if the object is at rest, it will remain at rest unless an unbalanced force acts upon it. Newton’s First Law of Motion is also known as the Law of Inertia.

50
Q

State newtons second law of motion

A

Newton’s Second Law of Motion states that when a force acts on an object, it will cause the object to accelerate. The larger the mass of the object, the greater the force will need to be to cause it to accelerate. This Law may be written as force = mass x acceleration

51
Q

State newstons thrid law of motion

A

Newton’s Third Law of Motion states that for every action, there is an equal and opposite reaction. What this means is that pushing on an object causes that object to push back against you, the exact same amount, but in the opposite direction.