Mix Qs Flashcards
Pathology in pronator Teres Syndrome
Entrapment of the Median nerve at the pronator teres and beneath the FDS
Grocery bag neuropathy
Carrying bag with arm flexed
Pronator Teres Syndrome
Describe severity of carpal tunnel syndrome As to: Mild Moderate Severe
Mild-prolongation of SNAPS w/ slight attenuation
Moderate- both SNAPS are slowed with prolonged median latency
Severe- moderate + absent SNAPs and prolongd or absent CMAPs + fibrillation potentials in thenar muscles
Anterior Interosseus Syndrome. Is it purely motor? Purely sensory? Or mixed?
Purely Motor
Abnormal ‘ok sign’ with weakness of FPL and FDP
Anterior Interosseus Syndrome
Possible location of radial nerve compression/neuropathies
- Axilla
- Spiral groove
- Posterior Interosseous nerve compression
- Wrist (Wartenberg syndrome or handcuff neuropathy
Saturday night palsy or honeymooner palsy
Nerve affected
Location of compression
Radial nerve at the spiral groove
Associated with Monteggia fracture or fall with outstretched hand presenting with weakness of MCP and wrist extension with preserved IP extension
Radial Neuropathy with posterior interosseus nerve compression
Muscles spared in posterior interosseous syndrome
Brachioradialis and ECRL
Types of ulnar nerve compression at the Guyon’s canal at the wrist with presenting symptoms
Type 1 hypothenar and deep ulnar branch Type 2 deep ulnar branch-hypothenar spared Type 3 superficial ulnar sensory branch Painless wasting of hand muscles Intrinsic muscle atrophy
Possible locations of entrapment neuropathies of the median nerve
- At the wrist-Carpal Tunnel Syndrome
- At the forearm:
Pronator Teres Syndrome
Anterior interosseous syndrome (kiloh-nevin
syndrome)
Beneath the ligament of Struthers
Pathology of posterior interosseous syndrome?
Entrapment of the radial nerve with a fibrous band at the origin of the supinator muscle or otherwise known as the arcade of Frohse
What nerve and and the location of the entrapment in Wartenberg Syndrome
Radial nerve at the wrist. No motor weakness
Diagnostic Criteria for ALS
EL ESCORIAL WITH AWAJI MODIFICATION
A. Presence of lower motor neuron degeneration by clinical, electrophysiologic or neuropathologic examination
B. Presence of upper motor neuron degeneration by clinical exam
C. Progressive spread of sympt9ms or signs within a region or to other regions
With
Absence of EDX or pathologic evidence of other disease process
Neuroimaging evidence of other disease
Diagnostic categories of ALS
Clinically definite- clinical or EDX evidence by the presence of LMN and UMN signs in the bulbar region and atleast two spinal regions or the presence of LMN and UMN signs in 3 spinal regions
Clinically probable-UMN and LMN signs in atleast 2 regions with some UMN signs rostral to the LMN signs
Clinically possible- UMN and LMN found only in one region or UMN signs found in two or more regions or LMN signs found rostral to UMN signs
Medical treatment that slow progression of ALS
Riluzole- inhibits the presynaptic release of glutamate-> glutamate toxicity is thought to contribute to neuronal death in ALS
When to initiate Mech Vent Support in Patients with GBS?
When Vital capacity falls below 20ml/kg
What are the 3 types of Diabetic neuropathy?
- Demyelinating, symmetric, distal sensorimotor polyneuropathy
- Demyelinating asymmetric proximal polyneuropathy or diabetic amyotrophy
- Focal neuropathy-median, ulnar and peroneal nerves commmonly affected
CSF analysis findings in patients with GBS
Albuminocytologic dissociation or elevated CSF protein but with normal WBC level
3 subtypes of GBS
AIDP-mc
AMAN
ASMAN
Miller-Fisher
Triad of Miller-Fisher Variant of GBS
Ataxia
Areflexia
Opthalmoplegia
Dose of IVIG treatment for GBS
0.4/kg daily for 5 consecutive days
Most common organism responsible for GBS
Campylobacter Jejuni
Triad of Wernicke Syndrome
Ataxia, dementia, opthalmoplegia