Minimally Invasive Dentistry W5/6 Flashcards

1
Q

Why do Pit and Fissure Caries occur?

A

Plaque are cariogenic microbiota accumulate in pits and fissures. Can’t reach these areas with toothbrush.

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2
Q

What are Pit and Fissure Sealants, and what are their four functions?

A

Low viscosity, plastic material that can flow into, conform and occlude pit and fissure system. Prevents dental caries.

  1. Physical barrier- against food etc
  2. Eliminate environment- against strep mutant and cariogenic bacteria.
  3. Cleansibility- smooth surface, easy to clean for self-cleansing
  4. Chemically releasing fluoride- mixed with GICS can act as fluoride reservoir
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3
Q

How do you prep the tooth for Pit and Fissure Sealants?

A

Remove the pellicle/plaque via pumice, probe, bristles, prophy paste and air abrasion

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4
Q

What are Glass Ionomer based Pit and Fissure Sealants?

A

Transitional sealant where moisture control isn’t possible. Have a low retention rate.
Are mixed with fluoride and made with glass and acrylic.

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5
Q

What are the general steps in a pit and fissure sealant?

A
  1. Pumice or prophy paste rinse and dry
  2. Acid etch- 15 seconds of 35% phosphoric acid etch- rinse and dry
  3. Place sealant material without trapping air
  4. 20 second light cure for each sealant
  5. Check the set, edges and occlusion
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6
Q

What is minimally invasive dentistry and the 4 steps associated?

A

First occurrence, early detection, earliest cure of damaged/defective tooth on a microlevel. Followed by minimally invasive treatment to repair irreversible damage.
Recognise, rejuvenate, restore/repair and review

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7
Q

What are the three types of non-carious damage to teeth?

A

Tooth wear, developmental defects and trauma

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8
Q

Identify the 4 types of tooth wear

A

Attrition- Tooth to tooth contact
Abrasion- Tooth to non-tooth contact (friction)
Abfraction- cervical V shaped enamel-dentine defects (idiopathic- no history of abrasion) due to masticatory stress.
Erosion- chemical process acid attack

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9
Q

What are the two sources of acid erosion?

A
  • Intrinsic acid- gastric acid. Voluntary (induced vomitting) or involuntary
  • Extrinsic acid- dietary or environmental fumes
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10
Q

What are the three types of mechanical wear to teeth?

A

Attrition: tooth to tooth contact

Abrasion: tooth to non-tooth contact

Abfraction: cervical V shaped wear of enamel-dentin due to masticatory stress

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11
Q

What are examples of hereditary and non-hereditary developmental defects?

A
  • Hereditary- affects all teeth: ameleogenisis imperfecta or dentinogenesis imperfecta
  • Non-hereditary- hypoplasia (enamel defect) or hypo mineralisation (lack of mineral content in enamel)
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12
Q

Define caries

A

Preventable, chronic and biofilm mediated disease modulated by diet.

A multifactorial oral disease caused by imbalance of oral flora (biofilm) and high fermentable carbs on the tooth surface over time.

Influenced by a shift in the equilibrium of protective vs pathological factors

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13
Q

What five ways can caries be described by?

A
  1. Location- pit and fissure, root, or smooth surface
  2. Direction of Progression
  3. Zone- enamel, CEJ or dentine
  4. Extent - incipient vs cavitated
  5. Progression rate- acute or arrested
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14
Q

What are the four factors necessary for caries to occur?

A
  1. Susceptible host
  2. Cariogenic bacteria
  3. Fermentable carbs
  4. Time
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15
Q

What is the net demineralisation for dental hard tissue damage to occur?

A

Critical pH of 5.5

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16
Q

What is plaque, and what are the three sites where plaque forms?

A

Firmly adherent microbial biofilm attached to surface of teeth and is the prime agent of dental caries.

Margins, fissures and interproximal areas

17
Q

What is the difference between rampant caries and arrested caries?

A
  • Rampant: acute, accelerating caries progression- multiple carious lesions i.e. high carb diet, drugs
  • Arrested: chronic, inactive lesions- enamel white spot lesions
18
Q

Can arrested dentine caries be reversed? Why?

A

no- because destruction of collagen matrix has began

19
Q

Identify enamel caries vs CEJ caries?

A
  • Enamel: subsurface demin causes White Spot Lesions- which are initially smooth and opaque.
  • CEJ: lesions extend laterally and then penetrate dentinal tubules
20
Q

What are white spot lesions?

A

Subsurface demin that causes Enamel prism porosities

21
Q

Once caries is in dentine, how is it recognised visually?

What are the three types of caries in dentine?

A

Tooth goes greyish colour.

  • Soft: outer carious dentin soft and infected
  • Firm: intertubular dentine
  • Hard: tertiary, sclerotic dentine
22
Q

Give an example of caries classification system used

A

ICDAS

23
Q

What are the visual methods of detecting caries?

A

Using mirror and probe, loupes , caries detecting dyes

Then X-rays

24
Q

What are some factors that increase risk in caries?

A

Diet, medical history, saliva, dental work, plaque control

25
Q

What are the non-invasive and prevention treatment modalities for caries?

A
  • Fluoride
  • CPP-ACP
  • Xylitol
  • Dental ozone
26
Q

When would you use dental ozone and what type of modality is it?

A

Primary root caries, pit and fissure carious lesions.

Non-invasive, natural, uses 2200ppm to kill 99% of microbial bacteria and oxidises caries and speeds up remineralisation

27
Q

Engine and turbine rotary instruments are used in operative treatment and are invasive. What is an example modality?

A

air abrasion, hard tissue laser, chemo-mechanical removal.

28
Q

What is Stephan’s Curve?

A
  • Depicts the sudden decrease in plaque pH after glucose rise.
  • Returns to normal after 30-60 minutes.
  • Hard tissues demineralise below critical 5.5 pH
  • Mature plaque metabolises and uses fermentable carbs = acidic environment