Miller Review Hand Lectures Flashcards
Orthopaedic Hand Surgery
Epineurium
Surrounds group of fascicles
Perineurium
Extension of blood brain barrier around group of fascicles
Endoneurium
On each axon
Function of myelin
Increases conduction velocity via saltatory conduction over the nodes of Ranvier
Extrinsic nerve blood supply
Vasa nervosum
Timing of nerve repair
Immediate for clean/sharp laceration. Open wound with nerve rupture, wait 2-3 weeks for demarcation, then excise scar, then operate. If closed injury, wait 3-6 months to see if it recovers on its own.
Neurapraxia, axonotmesis, neurotmesis
Neurapraxia = stretch injury with conduction block at the axonal level, architecture still intact, recovers in 3-4 months. Axonotomesis = endoneurial level. Neurotmesis = epineurial level.
EMG findings in neurapraxia, axonotmesis and neurotmesis
Neurapraxia = no spontaneous activity, normal insertional activity. Axonotmesis/neurotmesis = increased insertional activity, fibrillations and sharp positive waves.
Timing for nerve repair/reconstruction
<18 months before motor endplate degradation
Indications for direct nerve repair
Acute laceration with no tension
Indications for conduit nerve repair
<2-3cm gap in a sensory only nerve
Indications for nerve allograft repair
3-5cm sensory only nerve
Indications for nerve autograft repair
>5cm defect or motor nerve
Principles of nerve repair
Debride back to healthy vesicles, avoid tension (<10% stretch)
Indications for grouped fascicular nerve repair
None, although you line up the original fascicles well, the risk of scar blocking conduction is too great
Best prognostic indicator for recovery after nerve injury
Better with younger age
Double Oberlin transfer
In brachial plexus injury, it could be 12 months before elbow flexion is restored, so transferring ulnar fascicles from FCU and median fascicles from FDS/FCR to motor branches of biceps and brachialis shortens the endplate reinnervation time significantly
Nerve transfer for hand intrinsic reanimation
AIN as it enters PQ is transferred to the motor branch of the ulnar nerve near the wrist
What do you see on EMG in severe carpal tunnel and motor endplate degeneration?
Positive sharp waves, fibrillations and fasciculations
Most sensitive test to determine sensory deficit in compressive neuropathy
2.83 Semes-Weinstein testing
Most sensitive physical exam test for carpal tunnel
Durkan’s > Phalen’s > Tinel’s
How far does an injured nerve grow per day
1mm/day, 1 inch/month
Nerve conduction changes in compressive neuropathy
Distal motor latency >4.5 m/sec and >3.5 m/sec for sensory latency
CTS-6
Score of 12 or greater has 80% chance of carpal tunnel syndrome, validated test used in lieu of EMG/NCS
Carpal tunnel pressure level that decreases nerve conduction
30mmHg, 0mmHg with wrist splinted in neutral
Most common variation of median nerve motor branch at carpal tunnel
50% extra-ligamentous
Most common complication in carpal tunnel release
Incomplete release
Sites of median nerve entrapment
Ligament of Struthers, 1% of population has a supracondylar process of the humerus. Lacertus fibrosis. Deep head of pronator teres. FDS arcade.
Pronator syndrome vs carpal tunnel syndrome
The palmar cutaneous branch of the median nerve will be numb in pronator syndrome, but not carpal tunnel syndrome, compression most often between heads of pronator. Tinel’s over proximal forearm, symptoms with resisted elbow flexion, resisted forearm pronation and resisted long finger PIP flexion.
Nerve syndrome associated with medial epicondylitis
Pronator syndrome
AIN syndrome
Motor only deficits in AIN and vague forearm pain. Can be compressed by PT, biceps bursa, FDS, FCR or Gantzer’s muscle (accessory FPL head)
Nerve compression syndrome associated with AIN syndrome
Parsonage-Turner syndrome
Ulnar nerve sites of compression
Arcade of Struthers, medial septum, medial head of triceps, Osborne’s ligament, FCU aponeurosis, deep flexor-pronator aponeurosis and anconeus epitrochlearis
Difference in exam in cubital tunnel vs. ulnar tunnel syndrome
Numbness in dorsal cutaneous branch of ulnar nerve seen in cubital tunnel, not in Guyon’s compression.
Wartenberg sign
Loss of adducting interosseous muscle and unopposed small finger drift due to radial nerve innervated EDM in ulnar nerve compression
Jeannes sign
MCP hyperextension w/key pinch due to adductor pollicus weakness in ulnar nerve compression
Masse sign
Loss of hypothenar musculature in ulnar nerve compression
Zones of ulnar tunnel syndrome
1) Proximal to nerve bifurcation from ganglion = sensory and motor deficits. 2) Deep motor branch from hamate fracture = motor symptoms only 3) Superficial sensory branch compressed from ulnar artery thrombosis = sensory loss only
Anatomy of the ulnar tunnel
Floor = transverse carpal ligament. Roof = volar carpal ligament. Radial = hamate. Ulnar = pisiform and ADM.
Treatment of ulnar tunnel syndrome
Can do ulnar tunnel release, release of carpal tunnel also provides relief of compression in ulnar tunnel
Sites of radial nerve compression
“FREAS” Fascial band at radial head, recurrent leash of Henry, ECRB leading edge, Arcade of Frohse at proximal supinator (most common) and distal supinator. Same sites of PIN syndrome.
Nerve compression syndrome associated with lateral epicondylitis
Radial tunnel syndrome
Wartenberg syndrome/cheiralgia paresthetica
SBRN compression
Adson test
Diminished radial artery pulse with inhalation due to sublclavian artery compression in thoracic outlet syndrome
Work-up for thoracic outlet syndrome
Non-specific paresthesias on exam that include MABC, lower plexus trunk signs with overhead activity, u/s 90% sensitive and specific, evaluate for Pancoast tumor
Branches off brachial plexus with contributions from every level
Radial and median nerve.
Pre-ganglionic lower C8-T1 root avulsion signs
Horner’s syndrome, ptosis, miosis, anhidrosis. Enapthalmos.
Treament of pre-ganglionic brachial plexus injuries
Typically reconstruction at 3 months, these do not regrow and are not amenable to repair
Treatment of post-ganglion brachial plexus injuries
If open wound and obvious injury, fix ASAP. If closed of LV GSW, observe 3-6 months, outcomes worse outcoes if later than 6 months and minimal reinnervation if you wait 1 year.
Nerve transfer options for brachial plexus root avulsions
Oberlin (FCU fascicle to musculocutaneous nerve for biceps), CN XI to SSN, branch to triceps to axillary nerve
Tendon transfer options for brachial plexus injury
Lower trap tendon transfer for external rotation
Free innervated gracilis muscle transfer
Used in late brachial plexus reconstruction
Associations with obstetric brachial plexopathy
High birth weight, large head and shoulder dystocia
Prognosis for full vs. incomplete recovery after obstetric brachial plexus palsy
Biceps/deltoid return by 2 months, expect full recovery. Biceps and deltoid return in 3-6 months, expect incomplete recovery. Surgery if no biceps function by 6 months.
Principles of tendon transfer
Adquate strength and excursion, no joint contracture, functional and expendable donors, transfer direction in line with pull, synergysm, one motor tendon unit to have one function
Tendon transfer feature that best correlates with amplitude
Fiber length, this is why you need good excursion of the tendon
At what point in muscle contraction is the force the greatest?
When muscle is at resting length
What is the concept of synergism in tendon transfers
Use a muscle that already has a similar function, for example, wrist extension and finger flexion are linked actions. Using a wrist extensor to transfer to re-establish finger flexion would be synergistic.
What factors do you want to match with tendon/muscle transfer?
Force, amplitude and direction
Tendon transfer to restore wrist extension in radial nerve injury
PT -> ECRB
Tendon transfer to restore finger extension in radial nerve injury
FCR, FCU or FDS to EDC
Tendon transfer to restore thumb extension in radial nerve injury
Palmaris, FDS or FCR to EPL
Brand tendon transfer for radial nerve injury
FCR -> EDC, PT -> ECRB, PL -> EPL
Tendon transfer for elderly patient with severe carpal tunnel syndrome and loss of opposition
Camitz: Palmaris longus to P1 of thumb to restore abduction, less so opposition
Transfer for pediatric patient with congenital absence of the thenars
Huber: ADM transfer to P1 of thumb to restore opposition
Size of fingertip injury you can heal by secondary intention
1 cm^2
Volar oblique fingertip injury with exposed bone in adult
Cross-finger flap, down side is flexion contracture
Volar oblique fingertip injury with exposed bone in child’s index or middle finger
Thenar flap, can’t do in adults because they get PIP contracture. Watch for thumb neurovascular bundle.
Volar oblique fingertip injury with exposed bone on thumb <1cm
Moberg
Benefit of digital island flap for fingertip injuries
Maintains sensory innervation, can be from same finger (homo) or different finger (hetero). May consider in index of thumb.
Treatment of transverse fingertip injury with exposed bone
V-Y, lateral V-Y (Kutler) or shortening and volar flap
Treatment for thumb fingertip injury with >1cm tissue loss and/or dorsal loss
1st dorsal MC artery kite flap
Age that can tolerate fingertip reattachment without revascularization
Up to 6 can tolerate composite graft
How does negative pressure improve wound healing
Decreased interstitial edema and bacterial load. Increased cell division and skin graft incorporation.
Benefits of full thickness skin graft
More durable, less contraction and better sensation.
How much length can you get from a z-plasty?
Depends on the angle, 50% lengthening at 45 degree angle, 75% lengthening at 60 degree angle and 25% lengthening at 30 degree angle
Percentage of hand with complete palmar arch
80% have connection between ulnar supplied deep arch and radial supplied superficial arch
Threshold for digital brachial index when evaluation for hand vascular disorders
>0.7 is normal
Baseball catcher presents with isolated numbness, cold intolerance, ischemic pain in the small and ring fingers. What studies do you want and what is the treatment?
He needs an arteriogram to evaluate for ulnar artery thrombosis and hypothenar hammer syndrome. Treatment is ligation if they have a complete arch and DBI >0.7 to limit showering of emboli. Reconstruct with reversed vein graft if DBI <0.7.
Management of small vessel occlusion in the digits in Buerger’s disease vs. other rheumatologic disorders
Buerger’s = tobacco cessation. Rheumatologic disease, calcium channel blocker and/or sympathectomy.
Digit most often affected by embolic disease
Ring finger, straight shot from the ulnar artery
Origins of embolic disease to the digits
Heart (check for murmur), sublclavian (TOS), ulnar artery aneurysms, IVDU
Raynaud’s disease
Not associated with underlying pathology, rarely progressive, often symmetric.
Raynaud’s phenomenon
Associated with underlying pathology like Sjogren’s, often one side more involved due to vaso-occlusion. Progressive.
Types of sympathectomy for Raynaud’s
Chemical (botox), thorascopic VATS and periarterial stripping of adventitia
Indications for digit replant
Thumb, multiple digits, wrist or proximal and children. Relative indication is distal to zone I.
Contraindications for digit replant
Zone II, segmental injury, prolonged ischemia, crush/avulsion, advanced age, multiple comorbidities, polytrauma
Digit ischemia time
No muscle = 12 hours warm ischemia, <24 hours cold ischemia.
Order of operations in digit replant
Bone, extensors, flexors, arteries, nerves, veins, fasciotomies
Causes of digital replant failure
1st 12 hours = arterial thrombus, After 12 hours = venous congestion, after 1 week = infection
Leeches excrete
Hirudin
Prophylaxis for leech therapy
CTX or cipro to cover aeromona hydrophilia
Most common procedure after successful digit replant
Tenolysis
Management of ring avulsion injury
Repair those with adequate circulation, repair and revascularize those with inadequate circulation with no tendon or bone injury.
Deformity associated with chronic mallet finger
Swan neck. Lateral bands migrate dorsally. After terminal extensor tendon is disrupted, more pull occurs through the central slip, extending the PIP and DIP remains flexed due to insufficient terminal tendon.
Deformity associated with chronic central slip rupture
Boutonniere. Lateral bands migrate volarly. After centeral slip is disrupted, the triangular ligament attenuates, more pull occurs through the terminal extensor tendon, extending the DIP and flexing the PIP.
Elson test
To diagnose a central slip disruption, flex the PIP and push against resistance. If DIP remains supple, no central slip injury, if DIP extends, the central slip is out and the lateral bands are activating.
Treatment for closed volar PIP dislocation with Boutonniere deformity
PIP figure 8 extension splint x 6 weeks
Non-op treatment for zone IV and V extensor tendon injury
Yoke splint