Midterm Theory Flashcards

1
Q

A chronic, systemic, autoimmune disorder causing symmetrical, erosive synovitis of the joints. Eyes, lungs and the cardiovascular system may be affected

A

RA

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2
Q

What joints are most often affected in RA?

A
  • Fingers (DIPs)
  • Hands
  • Wrists
  • Knees
  • Feet
  • Upper cervical (C1/C2)

Viral pathogens, and antibodies launch a full scale attack on these health tissues bilaterally

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3
Q

Who is most commonly affected by RA?

A
  • Women are 2.5/3 times more affected than men (3/1)

- 20-40 most frequently

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4
Q

What are Ci’s to tx and assessment for RA?

A
  • No testing during flare ups
  • AF ROM and PR ROM tested in pain free ranges only
  • No PF testing in severe R.A
  • AR testing in pain free ranges only
  • All relevant treatment precautions in the event of neuropathies, pulmonary and cardiovascular involvement
  • Do not work directly on or distal to inflamed joint
  • Do not traction C-spine or perform joint play techniques if there is C-spine involvement
  • Do not friction ligaments capsules or tendons in severe progressive R.A or of the client is taking anti-inflammatories
  • Modify hydrotherapy and techniques in the case of joint replacement or flare up
  • Avoid vigorous techniques or overly long treatments as they will fatigue the client make them susceptible to a flare up
  • Avoid heavy applications during flare ups. Use cool cloths, arm or foot baths
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5
Q

What is the difference between RA and OA?

A
RA
Autoimmune and bilateral
Could lead to OA
Have Burchard's: PIPs
Swan Neck: DIPs

OA
Degenerative and not bilateral
Bones/cartilage. Wear and tear with age
Hands, hips, knees, L4/L5, C4/C7 - upper thoracic

Both affect the joints

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6
Q

What are CI’s to tx and assessment for clients with OA?

A

1) Do not mobilize hypermobile joints!

2) Do not mobilize joints with osteoformation

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7
Q

What is the recommended remedial exercise program for clients with OA? How does it differ from RA?

ADD MORE

A

OA
Increased ??
Walking/swimming - weight bearing exercise to decreased impact

RA:
Swimming, pain to stop pain?

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8
Q

Disorder of carbohydrate, protein and fat metabolism. Resulting from imbalance b/t insulin availability and insulin needed

A

Diabetes

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9
Q

What are different types of Diabetes?

A

1) Diabetes Mellitus Type 1: Insulin dependent
2) Diabetes Mellitus Type 2: Non insulin dependent
3) Gestational Diabetes: Glucose intolerance during pregnancy

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10
Q

What are the 3 P’s of Diabetes?

A

Polyuria: Excessive urination
Polydipsia: Excessive Thirst
Polyphagia: Excessive hunger

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11
Q

Prior to treating a client with insulin dependent diabetes, what is important to know?

A

Injection sites

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12
Q

What are common insulin injection sites?

A

Tummy, Thigh, Upper Arm

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13
Q

Progressive condition of axial skeleton presenting as pain and stiffness of the spine leading to bony ankylosis of the SI joint

A

Ankylosing Spondylitis

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14
Q

What are the 2 types of AS?

ADD ANSWER

A

1) Marie-Stumpell

2) Bechtrew

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15
Q

How can you alter your diet when you suffer from Gout?

A

No: Red meat, organ meat, anchovies, sardines, seafood, lentils, mushrooms, peads, asparagus, alcohol

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16
Q

hat are the extra-articular symptoms of clients with AS?

A

1) Pain in the SI & Lumbar
2) Muscle stiffness or pain after prolonged rest, interrupted sleep due to pain, fatigue, fever, weight loss
3) Misdiagnosed with muscles strain or spasm, loss of ROM in spine, lordosis, kyphotic spine, osteoporosis of the spine

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17
Q

What is Scleroderma?

A

Autoimmune disease of connective tissue characterized by thickening (fibrous) of the skin, fascia, tendon sheaths of the body

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18
Q

What is CREST?

A

C: Calcinosis
Formation of tiny calcium deposits

R: Raynaud Phenomenon
Spasms of the small articular, supplying the finger, toes, lungs

E: Esophageal Dysmotility
Poor functioning muscles of the lower ⅔ of the esophagus

S: Sclerodactyly
Localized thickening of the skin over the fingers and toes

T: Telangiectasia
Tiny red blotches, caused by dilated capillaries that appear on the face

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19
Q

This hormone produced by the pancreas. It regulates carbohydrate and fat metabolism. It takes up glucose and stores it as glycogen

A

Insulin

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20
Q

What is the most common complication associated with diabetes?

A

Blindness is most common Chronic Complication of Diabetes

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21
Q

What are the complications associated with diabetes?

A

1) Peripheral Neuropathies:
such as paresthesia in the legs and feet, muscle weakness, foot drop, carpal tunnel syndrome and impotence in men

2) Vasomotor Reflex Defects:
lead to dizziness and syncope (fainting) when moving from supine to standing

3) Impaired Innervation to the Bladder:
can lead to urinary stasis and the development of bladder infections and renal complications

4) Nephropathies:
diabetics have increased susceptibility to pyelonephritis (kidney inflammation) and papillary or glomerulosclerosis (scarring of tiny vessels in kidney)

5) Retinopathies:
Diabetes is the leading cause of acquired blindness. There is an increased risk of cataracts and glaucoma, microaneurysm formation (spots on retina), hemorrhage, scarring and retinal detachment.

6) Vascular Complications:
Increased risk for coronary artery disease, cerebrovascular disease, peripheral vascular disease

7) Diabetic Foot Ulcers:
Poor circulation and tissue health results in ulceration, infections, gangrene and eventually amputation. Common sites for damage are the back of the heel, plantar metatarsal area or great toe. Feet should be inspected daily for blisters, open sores, fungal infections. Feet should be kept warm, clean and dry. Nails should be trimmed carefully to avoid cuts.

8) Periarthritis and Adhesive Capsulitis:
result in limited joint mobility particularly in the hands (Dupuytren’s Contracture)

9) Reflex Sympathetic Dystrophy (RSD):
a condition characterized by pain, hyperesthesia (sensitivity to stimuli/touch), hyperhidrosis, tenderness and swelling of the hands and feet

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22
Q

What is the cause and contributing factors of Diabetes? (For both types)

A

1) Absolute insulin deficiency or impaired insulin production from the pancreas
2) Defective insulin receptors on cells
3) Genetic predisposition which causes an auto-immune response to the pancreatic B-cells which produce insulin
4) Environmental factors in a genetically susceptible person (viruses and chemical toxins)

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23
Q

What are the symptoms associated with Diabetes Type 1?

A

1) Polyuria (increase urine output)
2) Glycosuria/ ketonuria (glucose and ketones in urine)
3) Polydipsia (increase thirst)
4) Polyphagia (increase in hunger)
5) Unusual weight loss with normal eating and activity
6) Extreme fatigue
7) Irritability
8) Sweet smelling breath
9) Nausea and vomiting
10) Blood sugar levels fluctuate often including hyperglycemia and hypoglycemia, either can have potentially serious consequences – ketoacidosis
11) Often difficult to stabilize this type of diabetes mellitus

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24
Q

What are the symptoms associated with Diabetes Type 2?

A

1) Same as IDDM +
2) Frequent infections (esp. Skin, gums, bladder)
3) Slow wound healing, cuts and bruises
4) Tingling and numbness in hands and feet
5) Blurred vision
6) Fairly stable and easy to control

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25
Q

What are the contraindications for treatment of Diabetes?

A

1) Keep juice, fruit on hand for sugar
2) No massage on injection sites
3) When in doubt administer sugar only not insulin
4) Monitor cuts, ulcers. Ensure areas is clean - prone to infection
5) Peripheral Neuropathies cause altered sensation - adjust hydro as they may not feel hot or cold
6) Try to arrange appt just after insulin injection
7) No deep on areas where sensation changes

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26
Q

What is gestational diabetes?

A

Glucose intolerance during pregnancy

-They are also at a higher risk for developing diabetes 5-10 years after delivery

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27
Q

What are the risk factors of gestational diabetes?

A

1) Women with a family history of diabetes
2) History of stillbirth or spontaneous abortion
3) Presence of fetal anomalies in previous birth
4) Previous history of having large babies
5) Obesity of mother
6) Advanced maternal age
7) Five or more pregnancies

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28
Q

Understand the rationale on treating on or around injection sites?

A

New injection sites should be avoided during massage as excess insulin may be retained in the tissue and can be released during massage on-site. The tissue may also be tender.

Old injection sites need to be located as repeated scarring and tissue breakdown can occur in these areas. Extra caution is needed in massaging these areas as the tissue may be fragile. Common injection sites are the abdomen, thighs and biceps

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29
Q

Chronic, degenerative progressive deterioration of a joint characterized by a loss of articulate cartilage and reactive changes at the margins of joints and subchondral bone
Wear and tear of cartilage, unilateral

A

Osteoarthritis

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30
Q

Be able to describe the process of degeneration at the joint?

A

Inflammation of the joint capsule → fibrosis → capsular restriction → facet irritation → gradual degeneration due to altered biomechanics → OA

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31
Q

Define primary osteoarthritis?

A
  • Idiopathic

- Aging, Wear and Tear, Genetic Factors

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32
Q

Define secondary osteoarthritis?

A
  • Caused by trauma, pathology
  • Inflammation: Infection in joint
  • Bone disease
  • Bleeding Disorders
  • Neurological Disorders
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33
Q

What are the symptoms that are associated with the Early stages of osteoarthritis?

A

1) Few visible changes, capsule/ligaments thickening
2) Swelling is low grade not visible
3) Slight decrease ROM
4) End-feel normal/springy
5) Surrounding muscle guarding
6) Pain after normal use, relieved by rest
7) Morning stiffness last 30 mins

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34
Q

What are the symptoms that are associated with the late stages of osteoarthritis?

A

1) Visible palpable change in joint and shape, Osteophytes
2) Swelling low grade but chronic
3) Progressive loss ROM (PR ROM)
4) End feel premature and bony
5) Surrounding muscle spasm
6) Pain during rest
7) Morning stiffness last longer than 30 mins

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35
Q

What are the common locations of osteoarthritis?

A

1) Hips, Knees, SI, Facet joint, L5-S1
2) Small joints hands and feet
3) Lumbar, Cervical spine

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36
Q

Know the treatment approaches for clients with the various stages of osteoarthritis.

A

1) Correct predisposing factors
Correct muscle imbalance through stretch, eliminate TP

2) Educate client on proper body use
Lifting objects, stretching before activity, taking breaks, posture

3) Encourage Activity
Move through pain in EARLY OA

4) Rest and cool hydro
During FLARE UP

5) Massage
GSM, PR ROM, Low grade joint play, tissue health, etc

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37
Q

What is the relationship between osteoarthritis and facet irritation

A

1) OA, FJI and Herination may all be connected or lead to one another
2) If client has FJI due to faulty posture and the FJI becomes chronic, then degenerative changes occur causing OA of FJ leading to instability of the spine which can lead to DDD due to increase mechanical stress on IVD which may result in Disc Herniation

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38
Q

Define Disc prolapse?

A

Tearing of both the inner and outer layers of the annulus fibrosis causing the nucleus pulposus to bulge directly into the intervertebral space.

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39
Q

Define Disc Herniation?

A
  • A bulging of the nucleus pulposus against the outer layers of the annulus fibrosis into the intervertebral space. – - Tearing of the inner annulus fibrosis layers leads to a structural weakening, but the outer layers remain intact.
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40
Q

What are the causes of DDD?

A

1) Postural or occupational deviation leading to excessive rotation
2) Fixation of the spine will lead to degenerative changes especially above and below the sites of fixation. Joints above and below a fixation will make up for the loss of movement and become hypermobile causing excessive wear and tear on the spine.
3) Direct trauma such as a fall or whiplash or lift and twist injuries

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41
Q

What are the CI’s for DDD? (Same with OA)

A

1) As disease progresses ROM will diminish due to osteophyte formation, and bone rubs against bone
2) Don’t mobilize hypermobile joints (above and below fixations of spine)
3) Don’t mobilize joints with osteophytes, possible to break of and become loose body
4) No hydro during flare up
5) No temp extremes if joint has been placed with pins, plates, etc
6) No Friction around joint that has been treated with Corticosteroid
7) Don’t remove protective muscle guarding around hypermobile joint
8) Modify hydro if client has altered sensation along nerve root

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42
Q

In RA what are all the classes for an RA diagnosis?

A
  1. Morning Stiffness- in and around ,lasting at least 1 hour before maximal improvement
  2. Arthritis of 3 or more joints- Simultaneous soft-tissue swelling or fluid (not bony overgrowth alone) observed in 3 of the following 14 joints: left or right PIP’s, MCP’s, wrists, elbows, knees, ankles, and MTP joints
  3. Arthritis of hand joints- at least one area swollen in a wrist, MCP, or PIP joint (DIP’s are rarely involved)
  4. Symmetric Arthiritis- simultaneous involvement of the same joint areas on both sides of the body (bilateral involvement of the PIP’s, MCP’s, or MTP’s is acceptable without absolute symmetry).
  5. Rheumatoid Nodules- subcutaneous nodules over bony prominences, extensor surfaces, or surrounding joints.
  6. Serum rheumatoid factor- abnormal amounts of serum rheumatoid factor.
  7. Radiographic changes- hand and wrist radiographs showing erosion and bony decalcification localized in or adjacent to involved joints. (O.A of a joint alone does not qualify)
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43
Q

In RA what 4 do you need present for an RA diagnosis?

A

1) Morning Stiffness
2) Arthritis of 3 or more joints
3) Arthritis of Hand joints
4) Symmetric Arthritis

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44
Q

What are the causes and contributors to RA?

A

1) Idiopathic
2) Autoimmune disease, and occurs bilaterally
3) Most have RF factor in blood
4) 60% found to have genetic marker HLA
5) Emotional trauma/stress exacerbate attacks
6) Ratio 3/1 for women

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45
Q

What is swan neck deformity and boutonniere deformity?

A

1) Swan Neck: Deformity of MCPs

2) Boutonniere: Deformity of PIPs

46
Q

What medical treatment aims are there for RA?

A

1) Slow progression to prevent RA: rest, education
2) RA foundation support groups
3) Physical/emotional rest
4) Reduce weight on inflamed knees
5) Limits activities like housework, gardening
6) Swimming, Walking encouraged to reduce joint stress
7) Aspirin, NSAID’s, gold salts, DMARD’s
8) Surgery last resort

47
Q

Who is AS most commonly found in?

A

Most frequently seen in North American, rarely in Asians and African Americans

48
Q

What is spinal fusion of ossification of ligaments and IVD?

A

Ankylosing Spondylitis

49
Q

2 Types of AS?

A

1) Marie-Stumpell

2) Bechtrew

50
Q

What is the cause of Ankylosing Spondylitis?

A
  • Idiopathic

- Presence of mononuclear cells in acutely affected tissue suggest autoimmune response

51
Q

Describe the progression and presentation of Ankylosing Spondylitis?

A

1) Insidious onset, mild pain, stiffness in lumbar area
2) Sometimes, present acute onset, severe pain in SI and lumbar spine
3) Pain local in SI and lumbar may radiate to hips, down back and thigh
4) Pain at rest, lying in bed
5) Morning stiffness
6) Interrupted sleep due to pain
7) Fatigue
8) Ease back pain developing forward flexed posture
9) Lost of ROM in all lumbar movement

52
Q

What is the medical treatment for clients with Ankylosing Spondylitis?

A

1) Controlling pain, maintain mobility, supress inflammation
2) Encouraged do exercise regime, maintain healthy weight
3) AS Support group
4) Education on sleep positions
5) Aspirin, NSAIDs prescribed
6) Surgery

53
Q

Abnormally high amounts of uric acid in the blood (hyperuricemia). Not all people with hyperuricemia develop gouty arthritis

A

Gout

54
Q

This disease is chemical causing

A

Gout

55
Q

What is the pathology behind Gout?

A

Uric acid is a metabolite (a waste product from the normal metabolism of purine). About 2/3 of uric acid is filtered through the kidneys where it is either reabsorbed or excreted. The other third is eliminated through the GI tract

56
Q

What are the risk factors for Gout?

A

1) A diet high in purine containing foods. This includes red meat, organ meat, anchovies, sardines, fowl, lentils, mushrooms, peas, asparagus, spinach, alcohol (especially fortified alcohol such as sherry or port).
2) Obesity and/or sudden weight gain
3) Moderate to high alcohol intake
4) High blood pressure
5) Diabetes
6) Renal pathologies
7) Heredity

57
Q

What are your treatment aims in an attack or between flare ups of Gout.

A

1) Treatment may proceed as normal with a focus on encouraging circulation and elimination of metabolic waste. Steam baths or saunas may aid in elimination.
2) Encouraged increased fluid intake.
3) Chronic and severe gout may come with other cardiovascular or renal complications as well as severe joint degeneration and tophi deposits. All precautions for these respective complications must be taken into account when treating.

58
Q

What is the medical treatment for clients with Gout?

A

1) A small amount of synovial fluid is extracted from the joint and tested for the presence of calcium pyrophosphate dihydrate (CPPD) is the main diagnostic test. The diagnosis is often supported by x-rays revealing a linear calcification in fibrocartilage.
2) Treatment for pseudo gout is often the same as gout with the administration of colchicine (both abortively and preemptively prescribed) and in severe cases of acute synovial effusion, drainage and corticosteroid injections are often performed.

59
Q

Disease is unknown, girls more affected than boys, 75% expected to recover

A

Juvenile RA

60
Q

3 types of Juvenile RA?

A

1) Pauciarticular
Arthritis of 4 or fewer joints
Most common in knees

2) Polyarticular
More than 4 joints
Most common in hands, feet and TMJ

3) Still’s Disease
Affects the whole body
High fever, pink rash, anemia

61
Q

An autoimmune disease characterized by chronic inflammation affecting any organ of the body

A

Systemic Lupus Erythematosus (SLE)

62
Q

What is the cause of Systemic Lupus Erythematosus?

A

1) Idiopathic and Hereditary from Autoimmune disease
2) Connective tissues become inflamed by Lupus
3) Severe head/personality changes due to inflammation of connective tissue in CNS
4) DLE Lupus is the most Mildest form

63
Q

3 types of Systemic Lupus Erythematosus?

A

1) Discoid Lupus (DLE)
Mildest form
Limited to skin

2) Subacute Cutaneous Lupus (SCLE)
Lesions resembling psoriasis and are found on sun exposed areas
Affects face, chest, upper back/skin

3) Systemic Lupus (SLE)
Inflammation of almost every organ in body
Fever, malaise, joint, muscle, poor circulation (Raynauds)

64
Q

This form of Lupus is the Mildest form

A

Discoid Lupus (DLE)

65
Q

This Lupus: Lesions resembling psoriasis and are found on sun exposed areas
Affects face, chest, upper back/skin

A

Subacute Cutaneous Lupus (SCLE)

66
Q

This Lupus: Inflammation of almost every organ in body

Fever, malaise, joint, muscle, poor circulation (Raynauds)

A

Systemic Lupus (SLE)

67
Q

What are the diagnostic criteria of SLE?

A

1) Non-erosive polyarthralgia (joint pain) in 2 or more joints with join effusion
2) Mala (butterfly rash on face)
3) Discoid (flat) Lesions on sun exposed areas
4) Photosensitivity (symptoms increase with sun exposure)
5) Renal disorders such as Proteinuria, nephritis, glomerulonephritis (inflammation of tiny filters in the kidney known as glomeruli)
6) Chest pain related to pleuritis or pericarditis
7) Blood disorders such as anemia, increased bleeding or thrombosis
8) Neurological disorders such as seizures, psychosis or coma
9) Ulcerations of the lining of the mouth, nose or throat
10) Cellular immune defects such as low red, white or platelet cells
11) Immunologic Disorders (anti-DNA antibodies, false positive for Syphilis)

68
Q

An autoimmune disease of connective tissue characterized by thickening (fibrosing) of the skin, fascia, tendon sheaths and organs of the body
Idiopathic and Heredity in autoimmune disease

A

Scleroderma

69
Q

What are the aims of treatment for Scleroderma?

During Flare up

A

Massage may not be tolerable, avoid areas of inflammation

70
Q

What are the aims of treatment for Scleroderma?

Between Flare up

A

MT and underwater massage are recommended by MD’s, physiotherapy

71
Q

Overall Aims for Scleroderma?

A

1) Decrease pain and edema
2) Maintain circulation
3) Reduce stiffness of affected joints
4) Gently maintain mobility of affected joints
5) Focus on stress reduction
6) Position for comfort and/or systemic complications such as hypertension/congestive heart failure
7) Shorten tx to 30min
8) Full diaphragmatic breathing should be encouraged
9) Decrease pain and edema with drainage techniques such as unidirectional effleurage, running vibrations, stroking towards lymph nodes, 8 soldiers technique, pump and scoop techniques
10) Decrease mm spasms with indirect techniques such as GTO, O&I, mm approximation
11) Swedish techniques should include holdings, light stroking, soothing vibrations, light muscle squeezing and circular kneading
12) Hydrotherapy of the affected joints should be cool (watch cool/cold applications to fingers and toes to avoid Raynauds flare up)
13) Gentle isometric contractions and ROM to affected joints (usually 1-2 times) for affected joints may be performed during the treatment and as self care

72
Q

Causes of OA?

A

1) When the space between the joint decreases and therefore leads to a rubbing of the joint, this will cause bone to lay down (ossification) and forms osteophytes
2) The joint capsule deteriorates causing an instability in the joint and will most always have a capsular pattern associated with it
3) Disrupted by postural changes, activity related occupations, or autoimmune diseases
4) OA may also be due to an increase in the lordotic curve of the lumbar spine

73
Q

CI’s of RA?

A
  • 1) No testing during flare ups
  • 2) ROM testing in pain free ranges only
  • 3) All relevant treatment precautions in the event of neuropathies, pulmonary and cardiovascular involvement
  • 4) Do not work directly on or distal to inflamed joint
  • 5) Do not traction C-spine or perform joint play techniques if there is C-spine involvement
  • 6) Do not friction ligaments capsules or tendons in severe progressive R.A or of the client is taking anti-inflammatories
  • 7) Modify hydrotherapy and techniques in the case of joint replacement or flare up
  • 8) Avoid vigorous techniques or overly long treatments as they will fatigue the client make them susceptible to a flare up
  • 9) Avoid heavy applications during flare ups. Use cool cloths, arm or foot baths
74
Q

CI’s of AS?

A

1) If fusion has not taken place: prone position should have NO abdominal pillow supine should have NO knee or neck pillow however a small towel roll may be used to support knees and neck
2) Joint mobilizations to the affected but not fused joints. Mobilizations should be low grade and designed to reduce pain and maintain mobility
3) Fascial work and PNF techniques are appropriate as well as trigger point therapy if the client is not in severe pain from a flare-up
4) • Frictions should not be performed on sites of fibrosis and/or fusion. This fibrosis/ossification process cannot be reversed or diminished by friction therapy and May in fact cause a local flare up.
5) • If fusion has taken place: The client will likely not be able to lie in prone. Position client in sidelying. When in supine the client should now be well supported under the knees and neck.

75
Q

CI’s of SLE (Lupus)?

A
  • Do NOT over treat client, limit treatment time and don’t be too aggressive
  • Modify hydrotherapy if Raynaud’s or other sensory deficits are present
  • Clients may have to be positioned in right side-lying (left side up) or semi-reclined if they have cardiac complications
  • Clients may be most comfortable in semi-reclined if pleuritis or pericarditis is present. This reduces the pressure of the viscera pushing up against the lungs and heart.
  • Segmental massage to limbs may be performed first if client has cardiac complications.
  • Do NOT encourage Full Deep Breath or coughing if plueritis or percarditis is present, as this will increase pain
  • Do NOT perform frictions if client is on anti-inflammatories
  • Do NOT massage a feverish client
  • Do NOT apply passive forced over pressure to unstable joints
76
Q

CI’s to Gout?

A

1) Acute attack no local treatment
2) Do not use heavy applications of cold hydro such as a cold pack
3) Do not attempt to cover the foot with a blanket, sheet or sock. Allow the foot to remain exposed. DO NOT bump it
4) If treating the back in prone, allow the affected foot drop off the table to avoid pressure on the toe. Do not bump it during treatment.

77
Q

CI’s to Scleroderma?

A
  • Excessively long or overly aggressive treatments may trigger a flare-up. Limit treatment times to no more than 45 min in length
  • Avoid hot hydrotherapy during flare up
  • Avoid cold hydrotherapy on hands, feet or ears if Raynauds is present
  • Avoid massage or joint play directly in inflamed joints during flare up
  • Modify client position for hypertension, congestive cardiac failure or GERD(gastroesophageal reflux disease) (semi reclined)
  • Modify treatment for hypertension or congestive cardiac failure (start with hands or feet and perform segmental massage)
78
Q

What is also called morphea scleroderma?

A

Localized Scleroderma

79
Q

Define localized scleroderma?

A
  • Onset primarily in children and young (usually female) adults.
  • Does not have visceral or serologic manifestations
  • They may be bordered by a purple areola (circular area of different pigmentation)
  • Affects limbs and face
  • Skin, subcutaneous tissue, muscle and sometimes bone is replaced with fibrotic connective tissue that appear as white or pink oval patches, bands or lines
  • Mauskopf (mouse headed appearance occurs as the lips pucker causing difficulty opening mouth)
80
Q

Define Limited Systemic Sclerosis?

A
  • Onset usually in 30’s and 40s
  • 1st symptom is Raynauds Phenomenon
  • Subsequent symptoms include skin edema and tightening of the hands, face and feet
  • Often develop pulmonary hypertension and esophageal disorders causing dysphagia, possible development of trigeminal neuralgia
  • People with this form may have overlapping autoimmune conditions such as R.A or Lupus
81
Q

What disease is often referred to as CREST?

A

Limited Systemic Sclerosis (LSS)

82
Q

Who does RA affect more common?

A

Women ratio 3/1

83
Q

Does OA or RA have more morning stiffness?

A

RA has more morning stiffness than OA

84
Q

What is Mala rash associated with?

A

Lupus

85
Q

Is RA unilateral or bilateral

A

Bilateral

86
Q

What is most affected in Polyarticular JRA? How many joints?

A
  • hands, feet and TMJ are often affected

- 4 or more joints

87
Q

How many joints are affected in Periarticular?

A

Less than 4

88
Q

What is the wear and tear of OA?

A

Cartilage

89
Q

How would you accommodated for diabetes in your treatment room?

A

Have juice packs in case sugar drops

90
Q

What health question is most important for diabetes?

A

When was last injection and the injection sites

91
Q

Which joint is most effected in JRA: Pauciarticular?

A

Knee

92
Q

Are RA and OA the only diseases that affect the joint?

A

Yes

93
Q

CI of Arthritis in CS/Bone spurs?

A

No traction of CS

94
Q

How long does morning stiffness last in OA?

A

30 mins

95
Q

How long does morning stiffness last in RA?

A

1 Hour

96
Q

• Ketones (Glycosuria) in urine:

A

Type 1 Diabetes

97
Q

What would you modify if client has Neuropathy?

A

Hydro and pressure

98
Q

Which Type of diabetes is monitored with diet and exercise?

A

Type 2

99
Q

Would you use hyrdo on acute inflamed joints?

A

No

100
Q

Remission of any inflammatory disease what is CI’d?

A

Heat is STILL CI’d : Will cause a flareup

101
Q

Goals of AS in early stage

A

help maintain mobility in thoracic and lumbar – control pain

102
Q

Main aim of Lupus?

A

reduce muscle and joint pain and increase ROM

103
Q

AS, Spine fusion is what?

A

ossification of the ligaments and IVD

104
Q

What tissue is inflamed by Lupus?

A

Connective tissues

105
Q

What area does Gout occur to most?

A

Big toe, uinlateral

106
Q

What would cause severe head/personality changes in Lupus

A

Cause is inflammation of connective tissue in CNS

107
Q

Is there chest expansion in AS?

A

No, it collapses in because you are bent over

108
Q

Localized Scleroderma affects:

A

Skin, fascia, muscle. NOT LUNGS

109
Q

1st thing affected with Limited Scleroderma (LSS)

A

Skin

110
Q

•Onset for Limited Scleroderma

A

30’s-40s

111
Q

Onset for Localized Scleroderma

A

Children, usually female

112
Q

Cause of Scleroderma

A

Idiopathic and Heredity from Auto Immune disease