Midterm Flashcards

1
Q

Define and provide an example of dysphagia.

A

Dysphagia is an impairment in swallowing. It is often a comorbidity of other disorders. Dysphasia can contribute to malnutrition, aspiration, aspiration pneumonia, death, reduced quality of life, and longer hospital stays with a higher likelihood of readmission. Some disorders include stroke, brain injury, dementia, ALS, etc.

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2
Q

Describe the oral preparatory phase of swallowing.

A

Takes place in the oral cavity
Mastication of the bolus, mixing it with saliva and dividing the food for transport
Mastication occurs in a rotary motion with the tongue moving laterally to push the food around
Information is gained about bolus size, consistency, taste, pressure and temperature to help guide this phase
Saliva from the three salivary glands (Parotid, submandibular, sublingual) containing amylase to digest starch and mucous for lubrication, lubricates and dilutes the bolus to a consistency suitable for swallowing

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3
Q

What are the motor actions of the oral prep phase?

A

Labial seal
Buccal and facial tone
Adaquate and appropriate tongue movement
Lowering the velum, raising the posterior tongue
When necessary, mastication of the material with lateral and rotary motion of the mandible

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4
Q

What are the three sensory components of the oral prep phase?

A

Taste
Temperature
Pressure of the bolus, which provides information in order to stimulate saliva production and determine the pressure required to manipulate the bolus

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5
Q

Describe the oral phase of swallowing.

A

Takes place in the oral cavity

  1. Tongue tip raises to the alveolar ridge
  2. Posterior tongue drops to open the back of the oral cavity and soft palate elevates
  3. Tongue surface moves upwards, gradually expanding the area of tongue-palate contact from anterior to posterior, squeezing the bolus back
  4. Swallow is triggered when the bolus hits the anterior faucial pillars
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6
Q

What are the steps of the pharyngeal phase of swallowing?

A
  1. Bolus hits anterior faucial pillars
  2. Suprahyoid muscles contract, causing the hyoid to move supriorly and anteriorly
  3. Airway protection
  4. Pharyngeal muscles contract, raising the pharynx
  5. Tongue base retracts to PPW
  6. Pharyngeal constrictors are activated in rostral-caudal direction, causing pharyngeal peristalsis
  7. Thyrohyoid contracts, causing the larynx to move towards the hyoid, creating a negative pressure below the bolus to suck it towards the esophagus
  8. Epiglottis inverts
  9. Layngeal and pharyngeal elevation pull cricoid away from PPW, opening the UES
  10. If labial and nasopharyngeal seal are intact, UES opening creates more “suction force” that combine with driving force of the tongue and pharyngeal stripping wave to enhance bolus efficiency
  11. Completed when soft palate returns to original position and larynx reopens
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7
Q

What can modify the pharyngeal phase of swallowing?

A

Can be modulated in part by how the bolus is transported in the preceding phase Timing of trigger can be impacted by texture, taste, and volume

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8
Q

Describe the esophageal phase of swallowing.

A

Relaxation of UES lasts 0.5-1.2 seconds, just long enough for bolus to pass
UES returns to contracted state to avoid any retrograde bolus entry into the hypopharynx
Esophageal peristalsis is then activated and the bolus is propelled to the LES and stomach
Wave travels inferiorly, squeezing the bolus through the esophagus
After hypopharyngeal pressure peaks, LES is triggered to relax and bolus is squeezed into the stomach
Any residue is cleared via secondary waves up to two hours after
Transit times vary with age, bolus size, and texture (8-13 seconds in healthy adults)

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9
Q

Describe the mechanisms of airway protection.

A

Vocal fold closure (true and false): ceases respiration and seals off airway to prevent aspiration
Epiglottic deflection: tip of the epiglottis to the arytenoids
Elevation of the aryepiglottic folds/larynx
Anterior hyolaryngeal excursion

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10
Q

Describe the mechanisms of UES opening.

A

Intrabolus pressure: via tongue and pharyngeal muscles
Build up of positive pressure int eh pharynx/negative pressure in the esophagus
Cricoid elevation

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11
Q

What are some indications to give an instrumental exam? Provide at least four.

A

Patient’s signs and symptoms are inconsistent with findings on the clinical examination
Need to confirm a suspected medical diagnosis and/or assist in the determination of a differential medical diagnosis
Confirmation and/or differential diagnosis of the dysphagia is needed
There is either nutritional or pulmonary compromise and a question of whether the oropharyngeal dysphagia is contributing to these conditions
The safely and efficacy of the swallow remains a concern
The patient is identified as a swallow rehabilitation candidate and specific information is needed to guide management and treatment

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12
Q

What are some methods of radiation safety?

A

Step to the sides and backwards, as clinician exposure is highest immediately in front of the patient
Take a position 6ft away from the source = should be a zero exposure location
Lead vest and collar for clinician with lead napkin for client
Dosimeters on collar and waist
Always have a plan going in to minimize exposure time, ensuring location and posture of client is good, etc.
Patients should cover reproductive organs

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13
Q

Name the different types of motor speech disorders and their sites of impairment.

A

Flaccid: Cranial nerves and brainstem, LMN (primarily LMN)
Spastic: cerebral cortex, white matter tracts, or bilateral UMN (Specifically pyramidal and extrapyramidal systems)
Ataxic: cerebellum
Hypokinetic: basal ganglia system (Typically parkinsons)
Hyperkinetic: basal ganglia system
Apraxia: left hemisphere, most likely broca’s area or primary motor cortex

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14
Q

Differentiate UMN and LMN based on strength, reflexes, atrophy, and presence/absence of fascinations.

A

Strength: reduced weakness (UMN), weakness (LMN)
Reflexes: hyperreflexia (UMN), hyporeflexia (LMN)
Atrophy: n/a (UMN), Yes (LMN)
Fasciculations: n/a (UMN), Yes (LMN)

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15
Q

What are some advantages and disadvantages of an electrolarynx? Provide at lease two for each.

A

Pros:
- Can use immediately post-surgery
- Easy to learn
- Communicate in noisy environments
- Can use at different locations in the neck
Cons:
- Sounds mechanical
- Expensive and needs to be charged or uses batteries
- Can breakdown
- Success can be impeded by hearing loss or hand tremors

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16
Q

What are some advantages and disadvantages of TEP speech?

A
Pros:
- Quick and easy to learn
- More natural and dynamic voice
- Hands free option
- Reversible
Cons:
- May not be a candidate due to surgery and effects of radiation
- Cleaned and care for daily
- May need more surgery (Secondary puncture vs. primary)
- Cost
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17
Q

What are some advantages and disadvantages of esophageal speech?

A
Pros:
- Natural sounding voice
- No electronic aids, prostheses, or hands to help with communication
- No cost
Cons:
- Tension and stress can impede speech
- Hard to learn (1-2 years for complete proficiency)
- Need to be highly motivated
- Poor voice dynamics
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18
Q

Provide 5 things to keep in mind when training on an electrolarynx.

A
  • Need a good seal between device and skin
  • Requires good or over-articulation
  • Speak slower but maintain normal prosody
  • Speak in shorter sentences
  • On/off timing important at start of sentence and at the end
  • Avoid stoma blast
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19
Q

Provide 5 things to keep in mind when training on an TEP.

A
  • Relax!
  • Occlude the stoma by prasing finger or thumb over it and gently pushing from the diaphgram
  • Maintain good seal on the stoma
  • Do not push from the upper chest, neck, or shoulders
  • Start with an ah sound or counting one to ten
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20
Q

List the key head and neck structures of the oral prep phase.

A

Obicularis Oris (CN VII) – closing the lips
Buccinator (CN VII) – counter force to the tongue to facilitate proper bolus control
Masseter (CN V) – elevates the mandible (closes the jaw)
Temporalis (CN V) - elevates and retracts the mandible (closes the jaw)
Medial pterygoid (CN V) – closes the jaw by raising the mandible against the maxilla
Lateral pterygoid (CN V) – assists in opening the mouth by drawing the condyle and articular disk forward
Tongue muscles (CN XII):
- Intrinsic: superior and inferior longitudinal, vertical, and transverse
- Extrinsic: genioglossus, styloglossus, hyoglossus, and palatoglossus (CN X)

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21
Q

List the key head and neck structures of the oral phase.

A

Levator veli palatine (CN X) – raises the soft palate to close off nasal cavity
Hyoglossus and styloglossus (CN XII) – posterior tongue depression
Superior pharyngeal constrictor (CN X) - forceful closure of the nasopharynx and constriction of the pharynx
Mylohyoid (CN V) – hyoid bone elevation
Stylohyoid (CN VII) – draws hyoid back and elevates the tongue
Geniohyoid (CN XII) – hyoid bone elevation

22
Q

List the key head and neck structures of the pharyngeal phase.

A

Laryngeal skeleton – hyoid, thyroid, arytenoids, cricoid, corniculates, and epiglottis
Lateral cricoarytenoid (CN X) – adducts vocal folds
Transverse arytenoid (CN X) – adducts vocal folds
Thyroarytenoid (CN X) – helps close off airway by narrowing inlet
Thyrohyoid (CN XII) – depresses hyoid, elevates larynx
Cricopharyngeus (CN IX) – contracts at rest to prevent reflus, UES
Muscles of the pharynx:
- Three sets of semicircular muscles (constriction) = Superior constrictor, Middle constrictor, Interior constrictor
- Two paired muscles (shortening and elevation) = Salpingopharyngeus (CN X) – elevates pharynx, Stylopharyngeus (CN IX) – elevates pharynx, (Palatopharyngeus)

23
Q

Describe FEES

A

Involves passing a flexible endoscope transnasally to obtrain a superior view of the pharynx and larynx
Evaluates structure and function of the upper aerodigestive tract, enabling visulatization of base of tongue, nasopharynx, hypopharynx, and larynx
Allows for assessment fo secretions and secretion management
Use to identify normal and abnormal anatomy, evaluate integrity of airway protection related to swallowing, and evaluate compensatory techniques
Can look at spillage before the swallow, residue after, penetration/aspiration, patient reaction to these, and the impact of maneuvers and compensation

24
Q

What is the protocol for FEES?

A

Can use topical anesthetic
Pass the scope transnasally
Look at velopharyngeal closure and the appearance of the hypopharynx and larynx at rest
Watch how they handle secretions and swallowing frequency
Test base of tongue (Pull back against resistance) and pharyngeal function (say strained, loud, or hi e, grunt, and gargle)
Look at laryngeal function
- Respiration: sniff, pant
- Phonation: say e, repeat he he he five times, or change pitch
- Airway protection: hold breath lightly, cough, or clear throat
Sensory testing: administer puff of air, touch pharynx or tongue with tip of the scope
Administer food and liquid, working up in consistency given, allowing patient to self-feed
- Do not intervene until issues are noted then limit the amount, remove self-feeding, change the consisteny, or give postural changes
Generally try three of each stimuli, beginning at liquid and altering volume, viscosity, and maneuver

25
Q

Describe VFSS

A

Dynamic x-ray video of the swallow, considered gold standard
Allows examination of structural anomalies, identify aspiration and patient response, identify post-swallow residue, observe swallow physiology and why aspiration and residue are occurring, suitability of interventions, and evaluate treatment outcomes

26
Q

What is the protocol for VFSS?

A

Generally begin in seated position and in lateral view
Begin with saliva swallow and work up (thin liquid with bolus hold, extremely thick liquid/puree, cookie with paste)
Invovles altering type, order, and volume of stimuli

27
Q

Compare FEES and VFSS in terms of swallowing stages observed.

A

FEES: Pharyngeal stage before and after the swallowing (inferences made about oral, what occurs during swallow, and esophageal stage)
VFSS: Oral, pharyngeal, and esophageal

28
Q

Compare FEES and VFSS in terms of where they can be performed.

A

FEES: Any location (mobile)
VFSS: radiosuite

29
Q

Compare FEES and VFSS in terms of who can’t receive it.

A

FEES: Very few. Craniofacial trauma, dementia, brain trauma, confused or comatose, movement disorders
VFSS: Pts unable to leave bed, room, or ward, or unable to position in upright position, ventilator, intensive care, uncooperative pts

30
Q

Compare FEES and VFSS in terms of best indicators for use.

A

FEES: Complaints of choking on food, suspicion of aspitation or penetration, need for diet consistency
VFSS: Complaints of oral stge prep problems, suspicion of aspiration or penetration, food sticking in throat

31
Q

Compare FEES and VFSS in terms of limitations

A

FEES: Some cannot tolerate nose insertion, white out period, may miss aspiration or penetration, does not address oral and esophageal
VFSS: Due to turning on and off of fluoro, prone to miss behaviours after swallow, unable to view laryngeal surface anatomy, barium changes food viscosity

32
Q

Define stuttering

A

A disorder of speech production in which the natural flow of speech is typically disrupted by involuntary repetitions of sounds, syllables, or words, sound prolongations, blocks and/or pauses.
Presents as a wide variety of both visible and hidden symptoms involving speech behaviours, feelings, beliefs, self-concepts, and social interactions

33
Q

What are the three core behaviours of stuttering?

A

Repetitions (Sounds and syllables)
Prolongations
Blocks (audible or silent)

34
Q

What are the two types of secondary stuttering behaviours?

A

Escape and avoidance

35
Q

Define escape behaviours.

A

Occurs when a speaker is stuttering and attempts to terminate the stutter and finish the word

36
Q

Define avoidance behaviours.

A

Learned when a speaker anticipates stuttering and recalls negative experiences they have had when stuttering

37
Q

Give an example to illustrate how feelings and behaviours influence stuttering in adults.

A

In their social lives, this might cause a fear of talking due to teasing or not being listened to. This can result in difficulty establishing friendships, avoidance tactics, beliefs that they are helpless victims to their stuttering and may actually result lead to increased disfluencies.

38
Q

What are the characteristics of advanced stuttering?

A

Longer, tense blocks, often with tremors of the lips, tongue, and jaw
Complex patterns of avoidance and escape behaviours
Emotions of fear, embarrassment, and shame are very strong, with client having negative feelings about themselves as a person who is helpless and inept when they stutter (self-concept may be pervasive).

39
Q

Compare how advanced stuttering might affect a child vs. an adult.

A
Child:
Less aware of disfluency
More likely to be explicitly bullied
Escape and avoidance behaviours are less ingrained
Fewer options re:interactions during ADLs
Maybe easier to work through treatment
Fewer options for children over 6
Possible less willing to engage
Adult:
Able to explain their stuttering
More aware of their disfluencies
Avoidance and escape behaviours are more habitual
More options re:interactions during ADLs
More difficult to work through treatment
More treatment options in adulthood
More willing to engage and more motivated
40
Q

Define an inorganic voice disorder.

A

Often referred to as a functional voice disorder, they result from improper function (ineffective, inefficient) or use of structurally and neurogenically sound system.
The most common disorder is MTD with some others including diplophonia, vocal fatigue, functional aphonia, or ventricular phonation

41
Q

Describe the anatomical characteristics of MTD.

A

Muscle tension dysphonia is characterized by excessive activity of the laryngeal and paralaryngeal muscles, with an increase in force or tension in that region during voicing.
Primary occurs in the absence of an underlying organic etiology, where it is considered the primary cause of the dysphonia
Secondary occurs in the presence of an underlying organic etiology
Some symtoms include pain in the larynx, neck, and surrounding areas, a globus sensation, chronic cough or irritation of the throat, and various perceptual symptoms

42
Q

List factors that contribute to MTD.

A
Upper respiratory infection
Heavy vocal demand
Reflux
Environmental irritants, smoke, allergies
Stress
Emotion (or physical) trauma
43
Q

What are some similarities between vocal nodules and polyps?

A

Both thought to occur as a result of vocal misuse or trauma and can be acute or chronic

44
Q

What are some differences between nodules and polyps?

A
Nodules: 
Common in children
Less likely to require phonosurgery
Usually bilateral
Often take time to develop (begin as inflammation)
Polyps: 
Rare in children
More likely to require phonosurgery
Usually unilateral
Can occur after a single phonotraumatic experience, such as yelling at a concert
45
Q

List the lesion site, defining characteristic, associated voice impairment, and any notes for flaccid dysarthria.

A

Lesion site: LMN
DC: Weakness
AVI: Breathiness
Notes: lesion can be directly on CNX

46
Q

List the lesion site, defining characteristic, associated voice impairment, and any notes for Spastic dysarthria.

A

Lesion site: Bilateral UMN
DC: effortful, slow movements
AVI: harsh voice quality, strain-strangled voice
Notes: hyper adduction during phonation

47
Q

List the lesion site, defining characteristic, associated voice impairment, and any notes for ataxic dysarthria.

A

Lesion site: cerebellum
DC: incoordination
AVI: n/a
Notes: articulation and prosody are affected

48
Q

List the lesion site, defining characteristic, associated voice impairment, and any notes for hypo kinetic dysarthria.

A

Lesion site: basal ganglia
DC: reduction
AVI: harsh, breathy voice
Notes: range and face of individual movements are reduced (prominent feature)

49
Q

List the lesion site, defining characteristic, associated voice impairment, and any notes for hyperkinetic dysarthria.

A

Lesion site: basal ganglia
DC: irregularity
AVI: harsh, strain-strangled
Notes: Abnormal movements when motor steadiness is expected

50
Q

List the lesion site, defining characteristic, associated voice impairment, and any notes for apraxia.

A

Lesion site: Left (dominant hemisphere)
DC: groping
AVI: n/a
Notes: artic and prosody are affected