Midterm Flashcards
What is the gold standard ancillary study for RCTs?
MRI
What is the goal for RTW/P (returning to work/play)?
90% strength
100% flexibility
What is the MC cause of shoulder P?
Shoulder impingement syndrome
In the PE, what is more predictive than Pain for GH instability (and dislocation)?
Apprehension
When would you relocate a GH dislocation?
If vascular compromise is present
What is the MC type of injury to AC joint?
AC Sprain
What shoulder injury would have an observable step defect?
- RCT
- Bicipital tendonopathy
- Shoulder impingement syndrome
- Subscapularis tendonitis
- GH instab/dislocation
- GH labral tear
- Adhesive capsulitis
- Calcification tendonosis
- AC sprains
AC sprains
MC RCT muscle?
Supraspinatus
RTC disorder happens because of 2 things:
- Traumatic injury
2. Progressive degeneration
PE for RTCs
AROM limited in most directions — neutral rotation may be painless. ABD rotation is P.
SITS tenderness on palpation
P w/stretching
P w/resisted mm testing
Or those: Appley’s I & II Codman’s arm drop Lift off Napoleon and hug Neer’s test w/ resistance Empty can w/ resistance
Acute tx for RTC?
POLICE: Protection Optimal Loading Ice Compression Elevation
P-free pROM, aROM
IFC, TENS, US, G5
Mob/manip/massage
Isometric mm strength
Acupuncture
Two types of bicipital tendonitis
Primary - isolated inflam WITHOUT shoulder pathology
Secondary - inflamm WITH pathology
PE findings for bicipital tendinopathy
Speed’s test
Hyperextension test
Modified yergason/bicipital instab test
What group of people are most likely to get shoulder impingement syndrome?
Athletes 18-35 yo
Name the 2 types of shoulder impingement syndrome:
Primary - d/t pathological narrowing
Secondary - caused by GH instab d/t repetitive overuse/hyperelasticity
Which type of shoulder impingement syndrome is MC?
Secondary - caused by anterior GH instab d/t repetitive overuse
What are the 2 categories of risk factors for impingement?
Structural (AC degen, calcification, altered tendon vascular ivy)
Functional (instability, muscular imbalance between IR/AD and ER/AB)
Important ddx from impingement syndrome
Cervical sprain/strain d/t CAD possibly associated with seatbelt contact on GH
RA
Neer’s pathogenesis
Which stages of Neer’s are responsive to conservative management
1 and 2
3 is less responsive and requires multidisciplinary mgmt
At what degrees of ABD is there max reduction in GH space
80-120˚
What is positive on PE for impingement?
Impingement sign w/forward flexion
Painful arc 80-120˚
Painful active internal rotation
Tenderness
Modified Neer’s
Hawkins-Kennedy
Common mm that are too strong/tight in the shoulder (that may lead to impingement)
Pecs Lats Internal rotators Upper trap Deltoids
Common mm that are too weak/tight in the shoulder (that may lead to impingement)
External rotators Subscap Serratus ant Levator scapulae Middle and lower traps Rhomboids/abs
What is the acute Tx for impingement
Same as RTC
Except pt should be careful in flexion/ABD >90˚