Midterm Flashcards

1
Q

Broad causes of dyspnea

A

Pulmonary, CV, Systemic (anemia), psychiatric (anxiety), etc

…effective history taking will delineate system at fault

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What os pleuritic chest pain?

A

Pleuritic chest pain is reproduced with maneuvers that cause motion between the pleura and the chest wall, such as coughing, laughing and taking a deep breath

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Side effects of COPD medications

A

Beta agonists: tremor, tachycardia

Anticholinergic muscarinic antagonists: dry mouth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Patient education for stable angina

A

Discuss dx with Pt so pt understands lifestyle modifications such as reduced energy expendature, especially early mornings and after meals

Lifestyle modifications: smoking cessation, weight loss, dietary adjust

Engage the patient in decision for Cardiology referral or cath

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

OSE for Angina

A

Sympathetic innervation: heart = T1-6, Lungs = T1-7

Parasympathetic innervation: Vagus

Chaman Points for Heart: Anterior 2nd ICS at sternal border and posterior transverse spaces btwn T2/3

OMT is not indicated for stable angina

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What type of symptom is dyspnea?

A

It is subjective and a self-reporting symptom

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

SOAP Note: OSE

A

OSE is an objective finding and must include 3out of 4 tart findings to meet guidelines for billing: tenderness (pt comment), asymmetry, restrictions in motion (not the same as orthopedic assessment), and tissue texture changes

List the somatic dysfunction region

Assessment: SD Thorax

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

CAD in men vs women

A

CAD is more common in men >50 and women >60

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Physical exam for Stable Angina: special considerations

A

Pulmonary evaluation

CV auscultation: use left lateral decubitis posttion to listen for S3, S4, or murmur associated with mitral regurg

PMI for size and shift of apex, listen for bruits, UE/LE pulses and perfusion

Evaluate for edema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Labs for Angina DDX

A

Labs are centered around causes of atherosclerosis:

Fasting glucose and lipid panel

renal functions (BUN, Cr, Na, K, CO2, Cl)

***So…order a BMP with lipid panel

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

When is a patient with stable angina at greater risk for coronary event?

A

When a patient is unable to exercise for less than 6 minutes, the pt is at high risk for coronary events. Strong consideration for interventional cardiac cath and recannalization of vessels with >50% occlusion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the most common etiologies for dyspnea?

A

Cardiac and pulmonary (~85%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Diagnostic Data for COPD

A

6-minute walk with pulseOx to assess O2 sat with activity and replicate dyspnea; repeat auscultation at the end of the walk, listening for expiratory wheezing not present at rest

Peak Flow Assessment: COPD diagnosis when FEV1/expected FEV1 <70%

PFT (spirometry) and CXR can be used in addendum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

American Thoracic Society definition of dyspnea

A

A subjective experience of breathing discomfort that consists of qualitatively distinct sensations that vary in intensity. The experience derives from interactions among multiple physiological, psychological, social and environmental factors and may induce secondary physiological and behavioral responses.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

When does Stable Angina progress to Unstable?

A

When angina occurs at rest or without provocation and takes 2-3 nitro doses to abate. Unstable angina is the beginning of ACS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

ECG in stable angina

A

Likely normal in the absence of sx. Findings may include changes consistent with previous MI (Q-waves), repolarization abnormalities (ST seg and T wave changes), LVH or rhythm abnormalities

If the baseline ECG is abnormal (eg LBBB) then radionucleotide perfusion images, cardiac megnetic resonance (CMR) stress testing or PET imaging may be necessary to ID regioal ischemia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Angina “equivalents”

A

dyspnea, nausea, fatigue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Dyspnea special considerations

A

Acute vs chronic; made worse or better with…; pleuritic vs nonpleuritic; risk factors (tobacco, HTN, COPD, DM, etc.); ROS: constitutional, edema, palps, recent travel or illness, orthopnea, etc

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Sympathetic innervation of Head and Neck

A

T1-5

includes upper esophagus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Sympathetic innervation of the Lungs

A

T1-7

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

5 model goals of OMT with COPD

A

Biomechanical: improve thoracic cage compliance & skeletal motion

Neurological: Normalize autonomic tone

Resp/Circ: Maximize efficiency of the diaphragm & enhance lymph return

Metabolic/Energetic/Immune: enhance self-regultion/healing mechanisms

Behavioral: Improve psychosocial components of health

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Sympathetic innervation of the upper GI

A

T5-10

Includes the lower esophagus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

COPD/Dyspnea OSE

A

Sympathetic innervation of the heart: T1-6; Sympathetic innervation of the lungs: T1-7; Parasympathetic innervation: Vagus; Chapman’s points for lungs: Ant 2nd, 3rd, 4th ICS along sternal border, posterior-lateral T2 spinus process, intertransverse spaces between T2/3, T3/4, and T4/5

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Parasympatheic innervation by the Vagus nerve (OA, AA)

A

Heart, lungs, esophagus, upper GI, small intestine, kidneys, ascending and transverse colon, upper ureter

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Sympathetic innervation of the ureters
Upper: T10-11 Lower: T12 - L2
26
Parasympathetic innervation by Sacral Plexus
S2-4 Colon, rectum, repro organs, bladder, pelvis, lower ureter
27
Sympathetic innervation of the genitourinary tract (including the bladder)
T10-L2
28
Objective signs of dyspnea
Tachypnea, accessory muscle use, tripoding...visible signs of increased work of breathing
29
Long-term mgmt and preventative measures for Stable Angina
Secondary prevention: assess for other cv sx (claudication), screen for thyroid dysfunction, anemia, etc. (things that may increase cardiac work load and cause sx to recur Tertiary prevention: Cardiac rehab (increase exercise tolerance)!, smoking cessation, tx of lipid disorders
30
Sympathetic innervation of the ascending and transverse colon
T10 - L2
31
Alternative Dyspnea DDX with "normal" Pulse-Ox
Anemia: Hb will be saturated but Hct will be low therefore a pt may have a 95% pulse Ox but still be hypoxic. Severe anemia can cause fatigue and dyspnea as well as cardiac sx of HF and angina. Physical findings include pallor, especially of the conjunctiva and bounding pulses. Obtain CBC to evaluate for anemia
32
Describe end-stage COPD
End-stage COPD is associated with cachexia, weight loss, bitemporal wasting and diffuse loss of subcutaneous adipose tissue. Pts will qualify for hospice at this stage
33
COPD Physical Exam
Palpation of chest for tenderness and access lung expansion; Percuss to compare sides- in a ladder pattern; auscultate- also in a ladder pattern - 2 anterior and at least 4 posterior while pt takes deep breaths through an open mouth; special tests- tactile fremitus and transmitted voice sounds as well as rib motion. \*\*Additional assessment of the upper airway is also recommended
34
Long-term COPD management (Primary, Secondary, and Tertiary preventative measures)
_Primary prevention_: annual flu vaccine, Pneumococcal Vaccine (PCV13) followed by PPSV23 at least 1 yr later, TDaP (Bordatella pertussis) _Secondary prevention_: avoidance of dust, fumes, industrial exposure by using respiratiors or masks _Tertiary prevention_- smoking cessation/abstenence, pulm rehab
35
Diagnostic Data flow Chart for Dyspnea
Hx and PE plus walking PulseOx, peak flow assess. No DX, move to phase 1: Phase 1 - CXR, Spirometry, ECG, CBC, BMP. No DX, move to phase 2: Phase 2 - Chest CT, Lung Vol, DLCO, neuromuscular function testing, echocardiogram, stress testing. No DX, move to phase 3: Phase 3 - Consider Cardiopulm exercise test and subspecialty referral
36
Effect of cigarettes on COPD
Accelerated decline in FEV1 in a dose-dependent response Only 15% is explained by pack-years, suggesting environmental and genetic components
37
Short-term treatment of COPD
Pt education: provide written material as well as discuss diagnosis, smoking cessation/avoidance and idetify other triggers s/a season change, air quality, ect; when to seek medical attention Medications: inhaled and PO: short-acting inhaled bronchodilators for rescue-Beta agonists (albuterol) and anticholinergic muscarinic antags (ipratropium) to improve FEV1; long-acting bronchodilators if symptoms persist - B agonists (salmeterol), anticholinergic muscarinic antags (tiotropium), LAMA\>LABA for sx improvement OMT (5 models) Pulmonology referral for refractory or complicated cases
38
Common physical exam findings in COPD
Barrel chest, limited rib motion, limited lung expansion with limited exhalation. Percussion may reveal **generalized hyperresonance** d/t hyperinflation. Decreased breath sounds, wheezing and prolonged expirations are common. Transmitted voice sounds and fremitus are _decreased_ d/t **hyperinflation**
39
Angina on stress test
On a treadmill stress test, ST Depressions identified during increased cardiac workload, may also reproduce sx of dyspnea
40
Medications and side effects for Stable Angina
ASA: bleeding, bruising, avoid NSAIDS (GI bleed) Nitrates: HA, hypotension, syncope, reflex tachycardia B Blockers: fatigue, depression,, bradycardia, heart block, bronchospasm, postural hypotension Dihydropiridine: HA, LE edema, fatigue, flushing, reflex tach Non-dihydropiridine: bradycardia, heart conduction defect, low EF, constipation
41
Describe presenting the patient
Brief, generally ~ 5 min Provide enough info to tell audience how you came to your conclusion Must be in chronologic order Must follow SOAP note format Accurately review events that lead to Pt making the appt ID risk factors and/or other underlying medical conditions that may affect dx Generate an assessment and plan (commit!)
42
SOAP note physical exam...what do you document?
EVERYTHING you did regardless of outcome! Use correct grading/scales, clearly specify location/direction, document positive and negative findings
43
Complications of Stable Angina
Prgression to unstable angina --\> ACS --\> MI CVA PVD
44
Sympathetic innervation of the small intestines and ascending colon
T9-11
45
How would a patient describe angina?
Heavy, squeezing, pressure, tightness or choking..rarely described as actual pain.
46
Sympathetic innervation of the descending and sigmoid colon
T12 - L2
47
Conclusions drawn from cardiac stress tests
Overall sensitivity is ~75% A negative result does not exclude CAD, although it makes the likelyhood of three-vessel or left main CAD extremely unlikely Contraindications to cardiac stress testing includes unstable angina w/in 48 hours, unstable ECG rhythm, acute myocarditis, sever aortic stenosis, uncontrolled heart failure, severe pulmonary HTN, and active infective endocarditis
48
Symptoms of stable angina
Generally begins on exertion Levine sign Discomfort, tight, squeezing, heavy pressure but rarely "pain" May radiate to neck, jaw, back, shoulder, ulnar surface of arm. Trapezius area is generally spared Stable angina gets better with rest and 1-2 Nitro
49
Sympathetic innervation of the extermeties
Upper: T2-7 Lower: T11- L2
50
Chest X-ray findings with COPD
Typically include flattening of the diaphragm, increased AP diameter which is best appreciated by increased space between sternum and mediastinum on a lateral film
51
Sympathetic innervation of the Adrenal glands
T5-10
52
Sympathetic innervation of the Heart
T1-6
53
Complications of COPD
Progressive hypoxia (supplemental O2) Exacerbations increase as FEV1 decreases, driven by increased airway inflammation (\>50% are bacterial, 30% viral) Respiratory failure during an exacerbation: tx with O2 or bipap, possibly intubation/mechanical vent
54
What is the prupose of admission orders?
To outline an initial treatment plan for pts entering a medical facility for specialized care _as a means to communicate_
55
What is an observation admission?
When a patient needs hospital care but expected to stay only on e night to observe progress. Can transition to an inpatient admission if pt meets criteria
56
Admissions orders: ADC VANDALISMM
Admit, Diagnosis, Condition Vitals, Activity, Nursing, Diet, Allergies, Labs and Diagnostics, IV fluids, Specalists/Consults, Medication, Monitoring
57
What is the three piece framework for admissions orders?
1. _Conditions for admission:_ admit, diagnosis, condition, allergies, meds 2. _Diagnostic procedures_: Vitals, activity, Nursing, Diet, Labs, IV fluids, Specalists/consults, Meds 3. _Safety_: Medications, Monitoring
58
What does the patient Admit specify?
1. The service 2. The attending 3. Particular Unit 4. Need for telemetry
59
What are criteria for admissions to ICU?
1. Requiring, or likely to require, advanced respiratory support 2. Patients requiring support of two or more organ systems 3. Patients with chronic impairment of ONE or more orgen systemswho also require support for an acute, reversable failure of another organ 4. When the PT needs care that cannot be addressed on another unit d/t medication intensity, need for close monitoring, high risk of declinen in condition
60
How are diagnosies communicated in admissions orders?
List the primary DX...it may only be a working diagnosis, but this DX is the biggest concern you have for the Pt (do not list S/SX as a diagnosis) List in order of priority if you mention multiple and break the into two groups: first the new/acute problem; second the chronic problems \*Listing chronic problems can be helpful in allowing those processing the orders to know the chronic conditions that lead to or are contributing to the admission
61
What is critical condition on admission orders?
Patients typically are going to ICU as they are potentially unstable or have been unstable prior to the admission and affectively stablilized enough to be transitioned to a medical unit that is appropriate for their diagnosis and care
62
What info is needed for Vitals in patient admission orders?
1. frequency...usually every 4-8 hours, but can be special instructions such as every 2 hours for 8 hours, then move to every 4 hours 2. parameters for notifying the physician
63
What are Nursing orders on patient admissions?
Orders specific for nursing functions, usually not needed any longer as nurses now develop care plans for pts which include nursing orders. Examples: deep breathing exercises every 2 hours Routine oral care Routine hygiene
64
What could be listed in Specalists/Consultations in admissions orders?
Medical Specialists Nutrition Phys/Occ Therapy WOund CAre Metabolic support Palliative Care/Hospice
65
What did Dr. Tyler say was the most important aspect of the admissions orders?
Medications - Medication reconciliation - ID all meds and doses - Delete/Add - Specific to admission \*\*a patient must have a diagnosis for each medication, cannot state it is something the pt already takes (if the pt takes a PPI every day, even OTC, a diagnosis of GERD needs to be made or the med d/c'd)
66
Body Water compartments and IV fluid distrobution
ECW = 20% (plasma and interstitial) Colloids remain in plasma, NS/LR stay in ECW ICW = 40% 5DW/Dex Saline spread through ECW and ICW Minerals, proteins, Fat, glycogen = 40%
67
The "5 R's" to fuid replacement
Resuscitation Replacement-Redistribute Routine Maint. Oral/Enteral Maint.
68
Tonicity of IV fluids
Isotonic: NS, LR, D5W (+/-) Hypotonic: 1/2NS. D5W (+/-) Hypertonic: 3%NaCl, D10W, D5 1/2NS, D5NS, D5LR
69
When apporaching fluid needs of the pt, how much does the average person need in fluids and electrolytes?
25-30 ml/kg water per day and about 1 mmol/kg of Na and K
70
Holliday-Segar Method of pediatric fluid replacement
First 10kg = 100ml/kg in 24\* second 10kg = 50ml/kg in 24\* remaining kg = 20ml/kg in 24\* \*\*24kg kid = 1000mL + 500ml + 80ml = 1580 ml/24 hours = 66 ml/hr -With volume loss, compare normal weight to lost weight: 1 kg = 1 L; Replace 1/2 the fluid vol in 8 hours and the rest over the next 16 hours with monitoring
71
Resuscition fluid in adults
With severe volume depletion, 1-2 L of isotonic saline are given bolus, then replacement is continued at a rapid rate until cinical signs improve
72
When is Lactated Rigner's used?
LR is favored by surgical specalties - LR has a tendency to increase the chance of emboli formation in the midst of PRBC transfusion - In thoery, the Ca in LR could overwhelm the chelating capacities of the citrate in stored blood, resulting in clot formation \*\*Avoid use in Rhabdo! , NS is the better choice
73
What must be watched with NS resuscitation?
Thsi isotonic solution can induce a metabolic acidosis with aggressive resuscitation
74
Is D5W really isotonic?
It is considered isotonic, but may cause significant electrlyte shifts d/t hypotonicity after the initial response phase