Midterm Flashcards

1
Q

What is classified by activity?

A

white spot, brown spot , initial, incipient, and active caries lesion (progressive)

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2
Q

white spot

A

noncavitated caries/carious lesion, with enough mineral loss to change the appearance of enamel (loss of translucency). Not all white spot lesions are incipient, some may have been there for a long time and could involve the enamel and dentin

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3
Q

active caries lesion

A

progressive develops fast, will not cause recession

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4
Q

arrested or inactive lesion

A

lesion not undergoing net mineral loss

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5
Q

brown spot

A

noncavitated caries/carious lesion where net mineral loss and acquisition of pigments lead to loss of translucency and a brown discoloration

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6
Q

How do you classify an active caries lesion?

A

visual appearance, tactile feeling, and potential for plaque accumulation:

likely active if enamel is whitish/yellowish opaque and chalky (with loss of luster); feels rough when the tip of the probe is moved gently across the surface; lesion is in a plaque stagnation area. In dentin, lesion is likely active when the dentin is soft or leathery on gently probing.

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7
Q

How do you evaluate a carious lesion?

A

Three Step Evaluation Process:

  1. In a plaque stagnation area?
  2. Visual appearance?
  3. Gentle Tactile feeling with ball ended probe

more predictors indicating activity then lesion is active

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8
Q

rampant caries

A

multiple active carious lesions in the same patient; may also involve unusual teeth surfaces, ex. mandibular incisors

sometimes classified by their causality

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9
Q

early childhood caries

A

the presence of one or more decayed (non-cavitated or cavitated lesions), missing (due to caries) or filled tooth surfaces in any primary tooth in a preschool-age child between birth and 71 months of age

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10
Q

Bottle caries or nursing caries

A

caused by the frequent and long-term exposure of a child’s teeth to liquids containing sugars

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11
Q

What are the 3 different plaque hypothesis?

A
  1. specific plaque hypothesis
  2. non-specific plaque hypothesis
  3. ecological plaque hypothesis
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12
Q

Specific plaque hypothesis

A

proposed that only a few organisms out of the diverse collection in the plaque flora were actively involved in the disease. Preventive measures targeting specific bacteria (e.g. immunization) would be a logical consequence of this hypothesis.

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13
Q

nonspecific hypothesis

A

considered the carious process to be caused by the overall activity of the total plaque microflora. A consequence of this approach is that all plaque should be disturbed by mechanical plaque control (toothbrushing).

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14
Q

ecological plaque hypothesis

A

proposes that the organisms associated with disease may be present at sound sites. Demineralization will result from a shift in the balance of these resident microflora driven by a change in the local environment. Frequent sugar intake (or decreased sugar clearance if salivary secretion is low) encourages the growth of acidogenic and aciduric species, thus predisposing a site to caries. The consequence of this hypothesis is that both mechanical cleaning and some restriction of sugar intake are important in controlling caries progression.

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15
Q

Primary Prevention

A

Includes those measures that prevent the development of the clinical signs of caries in the absence of disease, that is, prevent the initiation of the disease.

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16
Q

Secondary Prevention

A

Centers on the prompt and efficacious treatment of disease at an early stage and includes measures that arrest and/or reverse the caries process after initiation of clinical signs.

17
Q

Tertiary Prevention

A

Involves measures that remove irreversibly damaged tooth tissue and replace it in such a way as to prevent further progress of the caries process. (Note: some secondary and tertiary preventive options involve a ‘‘hybrid’’ interaction
of nonoperative and operative procedures).

18
Q

Dental fluorosis

A

too much fluoride

19
Q

Dental erosion

A

can be dietary, occupational, intrinsic;

physical result of a pathologic, chronic, localized, loss of dental hard tissue chemically etched away from the tooth surface by acid and/or chelation without bacterial involvement.

20
Q

dental erosion (simple term)

A

acid hits tooth, gets rid of enamel

21
Q

abrasion

A

loss of tooth due to a foreign element, brushing too hard, piercing

22
Q

demastication

A

wearing away of tooth substance during food chewing , influenced by abrasiveness of individual food

23
Q

attrition

A

loss of tooth due to opposing teeth

24
Q

abfraction

A

loss of tooth structure due to occlusion due to bite

25
What causes fluorosis
caused by too much fluoride ingestion during tooth development; can occur in primary and permanent teeth; in severe cases tooth enamel can be damaged; intrinsic, permanent stain, usually white but can be dark brown or orange even w/ very low fluoride intake from water a certain level of dental fluorosis will be found