Midterm Flashcards

1
Q

Epidermis is how thick

A

.06-.6 mm

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2
Q

What cells form the epidermis?

A

Keratinized stratified Squamos

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3
Q

Pigmentation comes from number of ______________. Have __________ for dark skin and ____________ for light skin.

A

Melanocytes
Fewer
More

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4
Q

Basil layer

Strauss baselie

A

Deepest responsible for kerantocytes

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5
Q

Stratum spinosum

A

Keratin layers

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6
Q

Stratum granulosum

A

Flattened keratinocytes

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7
Q

Stratum lucid

A

Clear layer of dead keratinocytes

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8
Q

Stratum corneum

A

Horny layer

Entirely layer of dead kerantocytes

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9
Q

Darker the skin more_____________

A

Cohesive

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10
Q

Dermis contains

A

Epidermal appendages
Rede ridges
Dermal papillae

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11
Q

Epidermal apendages include

A

Hair follicle
Sebaceous glands
Sudorferous glands
Nails

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12
Q

Rede ridges

A

Epithelial extension project into dermis

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13
Q

Dermal papillae

A

Interdigdating with rede ridges

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14
Q

Dermis is made up of

A

Collagen
Blood vessels
Lymph n
Epidermal appendages

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15
Q

Function of dermis

A

Sensation
Nutrient
Strength skin
Contribute to healing

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16
Q

What are the two layers of the dermis

A

Papillary

Reticular

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17
Q

Papillary layer is

A

Blister, surface of skin

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18
Q

Reticular layer

A

Structural support

Type one collagen, vasculature, nerves, hair, elastic fibers

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19
Q

Subcutaneous or hypodermis layer

A

Fatty layer

Highly vascularized

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20
Q

Pear shape adipose

A

Weight in hips
Subcutaneous fat
Vascularized, padding, release energy

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21
Q

Apple shape weight

A

Weight in the middle
Visceral fat
DM, heart condition, surround internal organs

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22
Q

Function of the skin

A
Barrier to infection
Regulate heat loss
Sensation 
Excretion 
Biochemical 
Produce vitamin d
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23
Q

Papillary layer of dermis

A

Blister, split the surface

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24
Q

Reticular layer of dermis

A

Structural support

Type one collagen, vasculature, nerve, hair, elastic fiber

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25
Adipoctyes action
Appetite control, regulate bp, coagulation, mm
26
Leptins action
Control the CnS, | Inc energy consumption
27
Skin phototype
Smallest amount of radiation predicts skins vulnerability to UV light
28
Skin color depends on
Number of melanocytes
29
Variation in skin tone dependent on
Amount of melanin
30
Skin color function
Protect from UV light, protect from photo damage, absorb and deflect Rays
31
Erosion ( abrasion) stage 1
Heal with epithelization
32
Ulcer
Below the epidethelium into the dermis
33
Stage 2
Partial thickness
34
Stage 3
Full thickness need a skin graft to heal
35
Function of the ground substance (gag and water)
Nutrition to CT Barrier against infection Fluid fiber distance of collage, reticular and elastin
36
Damage to the CT leads to
Change in scar tissue Decrease water and gags Dec critical fiber distance Dec amount of collagen
37
Phase one healing-inflammation
Hemostasis- control bleeding with platelets Phagocytosis- marcoohages cells PMN Angiogenesis- mast cells
38
Phase two - proliferative
Angioblast Fibroblast- secrete tropocollagen, synthesize gag, replaced by scar tissue Myofibroblast- wound contraction Keratinocytes
39
Phase 3 remodeling
Reorient along the stress line
40
Clinical implications for healing through the three stages
``` Encourage gag re synthesis Prevent cross linking Promote appropriate movement Rice Appropriate mvmt Strength and function ```
41
Sources of burns
``` Flame Hot liquids Contact Electricity Hot gas Chemicals Friction Expose to heat and cold Radiation ```
42
Superifical first degree burn
Only epidermis Avascularity No blister 2-5 days
43
Superifical partial thickness second degree
Epidermis into upper layer of dermis Epidermis completely destroyed Papillary layer involved causing blisters to form Heal 5-21 days
44
Deep partial thickness deep second degree
``` Destruction of epidermis and see damage to dermal layer Appendages ruined or destroyed Red tan white dull looking Eschar or coagulation may occur 21-35 days ```
45
Full thickness burn third degree
All epidermal and dermal layers destroyed Coagulation of necrosis cells, loss of blood vessels, massive edema, cellular inflammation No pain no blanch
46
Electrical burn fourth degree
Destruction of epidermis down to bone Entrance and exit wounds Ice berg effect
47
Volts__________, amps___________ nerves
Burn, destroy
48
Zone of coagulation
Direct intense heat | Skin death,Eschar
49
Zone of statsis
Injured cells die within one to two days | Blood supply compromises
50
Zone of hyperemia
Redness around the other two zones
51
Breakdown exceeds the production of scars
Scars are less bulky
52
Production exceeds breakdown
Hypertrophic scars
53
Keloid scar
Large firm skin extend beyond boundaries
54
Superifical thick skin graft types
Meshed- cover burn with less skin, infected or irregular wound Sheet- cosmetic, durable, contraction limited
55
Signs of inhalation burn
``` Facial burns Singed nose hairs Tinged sputum Close space injury Bronchoscope Hoarseness ```
56
Rehab for inhalation injury
Clear airway Good oxygen stats Monitor SOB Promote mobilization of secretions
57
Treatment plan for burns
Prevent loss of rom Restore lost function and rom Return to pre burn status
58
Patient positioning for burns
Control edema Prevent pressure ulcers Maintain soft tissue elongation
59
Exposed tendons called for what type of rom?
AROM
60
Acute PT for a burn patient
CPT Positioning splinting A and prom Education
61
Subacute PT for burns
``` CPT Positioning A and prom Education Hep Strength and amb ```
62
Out PT for burns
``` CPT Positioning A and prom Transfer Gait strength Scar management Education hep ```
63
Dyschromia
Increase in melanin or unequal distribution | After inflammation
64
Hypo pigmentation
Localized widespread loss of melanin in skin
65
For arterial insufficiency need to work on
Aerobic and strength training
66
For venous insufficiency need to work on
External support Aerobic activity Strengthening
67
For DM need to work on
Aerobic Strength Diet Weight control
68
Goals of wound care
Full closure Prep for surgical closure Manage wound deterioration Maintain non healing stable ulcers
69
Surgical debridement involve
Non seclective Remove both viable and non viable tissue Wide excision to viable tissue with sharp sterile Removal of bone or necrotic tissue
70
Sharp debridement
Selective Remove no viable tissue Inc risk of infection
71
Sharp debridement contraindications
ABI
72
Sharp debridement indications
Extensive necrosis Advanced cellulitis Thick Eschar Callous formation
73
To stop excessive bleeding
``` Pressure for ten minutes Elevate Ca aliginate Xiyloccune Vasoconstriction Nitrate Catherize ```
74
Mechanical debridement
``` Scrubbing non selective Wet to dry Whirlpool Forced irrigation Pulsed lavage ```
75
Systemic effects of whirlpool
``` Inc hr, rr Sedation Mm relax Analgesia Change in temp ```
76
Enzyme debridement
Selective | Liquefy necrotic tissue
77
Autolytic debridement
Most selective | Use mositure retaining dressing to allow body's own macrophages neutrophils and phagocytes
78
Maggot debridement
Selective | Disinfect and promote growth of fibroblasts
79
Ideal dressing
Keep moist allow gaseous exchanges, thermal insulate, impreamble to microgranism, non adherent or traumatic
80
Non occlusive dressing
No air or drainage Allow drainage to evaporate Acute surgical wounds, primary adhesion, infected, highly drainage
81
Semi occlusive dressing
Allow gaseous and vapor exchange | Autolytic debridement, scant, moderate draining
82
Occlusive dressing
Keep everything out Automatic debridement, minimum drainage, perineal wound, primary shallow wound, 2nd deep cavity wound, 2nd skin protection
83
Moderate to high absorptive
``` Gauze Alignates Hydro fibers Foam Filter ```
84
Low to no absorptive
Hydro colloids Transparent films Impregnated gauze
85
Hydrating dressing
Hydrogel
86
Misallenous dressing
Composits, contact layer, negative wound therapy
87
Inflammation cells
Macrophages + | Neutrophils -
88
Fibroblast cells
Fibroblast +
89
Remodel cells
Myofibroblast + | Epidermal -
90
Wet -> dry dressing
Mod to high absorptive Mechanical non selective debridement of necrotic wound Wound fillers Readily available
91
Wet -> moist
Semipermeable occlusive filter over gauze over wound bed Prevent drying out of wound Mechanical selective debridement. Put directly on wound
92
Wet-> wet
``` Help take gauze that is packed into wound out Wound fillers Put into wound Put on to keep from drying out Semipermeable or occlusive film dressing ```
93
Wound packing
With gauze to allow absorption of moisture and for undermining Aide in autoltyic debridement Semipermeable to mositure vapor
94
Films
``` Thin membrane coated with layer of acrylic adhesive No absorb Moisture vapor oxygen premeable Autoltyic debridement. Secondary dressing ```
95
Hydrogels
``` Group of polymers contains water in gel base Provide moisture to the bed Absorb exudate Aids in autoltyic debridement Relieves pain Necrotic arterial wound On dry wound ```
96
Alginates or hydro fiber
``` From Ca and Na salts Maintain wound moisture Highly absorptive Doesn't adhere to wound Secondary dressing Hemostitc -aliginate No min drainage ```
97
Foams
Mod to high absorptive Maintain wound mositure Aids in autoltyic debridement. May have adhesive border
98
Hydro colloids
``` Combo of foams with adhesives Maintain mositure of wound bed Totally occlusive Aids in autoltyic debridement No secondary dressing No drainage held ```
99
Enzymes
Eat necrotic tissue | Cover with something else
100
Bolster dressing
Additive layer to help maintain moisture and pressure | High drainage first absorb by gauze
101
Non sticks
Silicon, petroleum gauze | Good to allow fluid to seek through won't stick to wound
102
Antibacterial dressing
``` Silver based - inactivated bacterial DNA Iodine based - good against bacteria, mycobacterium, fungus, Protozoa, virus - skin prep or sough infected Topical antiseptics -used only on acute wounds. Bad for healthy tissue Topical antibiotics - short periods, clinical wounds infection Honey based - debridement of necrosis tissue - inhibit bacterial growth ```
103
Wound stimulating dressing
Collagen dressing - stimulate fibroblasts Hypotonic saline - gauze impregnate with sodium and chloride Hypergranulation tissue and necrotic tissue - draws fluid out of cells by setting up an osmotic gradient