Midterm Flashcards

1
Q

Etiologies of Addiction

A

Personal responsibility/moral– person is of blame,root of stigma

Agent–blame substance

Genetic/biological

Social learning

Sociocultural

Disease**

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is addiction?

A

a chronic disease of brain reward, reflected in pathologically seeking out substance
* Organ= brain, defect in reward system (midbrain, prefrontal cortex–>fight vs. flight system, logic system)–> bx: inability to stop using substances, despite harmful consequences–> impact: continued SU, cravings (response when drug cut off), impulsive decisions

Frequency, compulsion, persistence despite adverse consequences

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

neuron

A

signal processor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

synaptic cleft

A

space b/w neurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

axon

A

sender

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

dendrite

A

receiver

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

reuptake

A

recycle/reuse

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

neurotransmitter

A

signal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

dopamine

A

pleasure/feel good

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

prefrontal cortex

A

logic/thinking

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

midbrain

A

fight vs flight

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

neuroflooding

A

“being high”–lots of neurotransmitters in the synaptic cleft

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

dependence

A

brain is functioning only when the substance is present; you become dependent on the presence of a drug–body adapts, requiring more to maintain a certain effect (tolerance), eliciting drug/mental symptoms if use stops

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

tolerance

A

requiring more for same effect

why? our bodies seek BALANCE, to level off neurotransmitter levels, we kill off endogenous neurotransmitters, receptor down regulation, or deactivate receptors b/c we come to expect external source of substance in high doses

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

withdrawals

A

lack of needed neurotransmitters

why? body became dependent on substance, when you no longer provide body w/ external source, your body adapted by down regulating receptors or killing endogenous ones, balance is OFF–> withdrawal symptoms (explains anhedonia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

anhedonia

A

inability to feel pleasure

Why? you have fewer receptors than you originally did, you may have lower endogenous levels of dopamine being released–>not producing same effects as your brain pre-substance use

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

hypofrontality

A

inability to use prefrontal cortex

stress–> activation of fight vs. flight–> deactivation of prefrontal cortex–> seeking out dopamine to help deal with stress–> cravings–> SU

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

cravings

A

natural response d/t cutting off a substance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

9 areas of treatment planning (HELMMSSDF)

A

housing, ed/vocational, legal, medical, mental health, social/leisure, SU, D/C, family

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

problem statement

A

should reflect 9 problem domains; written in bx terms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

goal

A

pt’s words, strengths-based, presence vs. absence, compliments problem definition

22
Q

objective

A

what will the pt do to accomplish their goal, SMART, action terms

23
Q

Intervention

A

compliment objective, therapists portion, must include frequency

24
Q

ASAM

A

numbers represent benchmarks a continuum, meaning pts move up or down in terms of intensity

25
6 dimensions of ASAM
-acute intoxication/withdrawal/SU hx -health (physical) -psych/mental health -readiness to change -relapse, continued use -recovery/living situation
26
Order of treatment
referral--> screening--> assessment--> diagnosis--> decide
27
screening
detect possible presence of problem and need for further evaluation
28
OARS= motivational interviewing
O: open ended questions A: affirm R: reflective listening S: summarize
29
RULE= motivational interviewing principles
R: resist telling clients what to do U: understand motivations L: listen with empathy E: empower
30
SMART= treatment plan goal setting
S= specific M= measurable A= attainable R= realistic T= time-frame
31
FRAMES= SF
F= feedback R= responsibility for change A= advice M= menu of options E= empathic counseling style S= self-efficacy
32
ATL
Ask, Tell, Listen
33
precontemplation
(pros> cons): no intention to change bx, not even on mind
34
contemplation
(pros= cons): I’m thinking about change
35
preparation
(pros of use< cons): I am intending to change
36
action
(pros of abstinence> cons): I am successfully changing bx
37
maintenance
(pros of abstinence> cons): I am remaining free from bx, >6 mos
38
termination
end of cycle, no temptations, 100% confident in all previous situations; years of maintenance
39
Narcan mechanism of action
reverses overdose by... OPIOID RECEPTOR ANTAGONIST-->binds to opioid receptor and reverses/blocks effects of other opioids (higher affinity compared to opioids)
40
Suboxone mechanism of action
* Buprenorphine= partial agonist-->producing fraction of rxn as other opioids, weaker rxn-->no high, but no withdrawals, block other opioids from attaching * Naloxone= antagonist-->block reaction-->used in suboxone to deter intravenous use-->if person tries to inject suboxone intravenously, naloxone will block opioid receptors--> immediate withdrawal symptoms w/o high
41
semi-synthetic opiod
modification of an opiate (natural); ex: morphine heroin
42
synthetic opioid
chemically derived in lab; not derived from poppy
43
opioid
(all-encompassing natural+ synthetic+ semi-synthetic)
44
opiate
(derived from poppy)
45
opioid mechanism of action
o Opioid receptor agonists o Bind to opioid receptors--impact on Ca channels-- release of dopamine-- euphoria+ other effects o Half life= .6-150 hours
46
opiate exames
opium, codeine, morphine
47
semi-synthetic opioid examples
heroin hydrocodone oxycodone oxymorphone buprenorphrine
48
synthetic opioid examples
methadone fentanyl tramadol
49
meds can address (opioid)
withdrawals, cravings, aversion therapy (intravenous), psych disorders, "maintenance", overdoses (narcan)
50
DSM mild, moderate, severe
2-3= mild 4-5= moderate 6+= severe
51
screening tool examples
CAGE, CRAFFT, S2BI, TAPS, UA