Midterm 1 Flashcards

1
Q

Development Definition

A

change in the individuals level of functioning

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2
Q

quantitative change

A

change in number or amount
- measurable change

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3
Q

qualitative change

A

change in structure or organization as marked by the emergence of a new behaviour

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4
Q

heredity

A

set qualities fixed at birth: characteristics and traits

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5
Q

can heredity be modified?, How?

A

yes, by environment

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6
Q

maturation

A

biological changes, height, weight, etc

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7
Q

stability

A

state in which characteristics and abilities stay the same or function similarly across lifespan

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8
Q

aging

A

process occurring with passage of time leading to loss of function

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9
Q

behaviour

A

physical observable actions

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10
Q

affect

A

emotional experience

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11
Q

cognition

A

thinking abilities

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12
Q

motor development

A

study of change in movement behaviour

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13
Q

motor learning

A

relatively permanent gains in motor skill capability (from practice or experience) - long term change

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14
Q

motor control

A

neural, physical, and behavioural aspects of movement

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15
Q

2 Sequences of growth

A
  1. cephalocaudal development
  2. proximodistal development
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16
Q

cephalocaudal development

A

growing proceeds from head to feet

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17
Q

proximodistal development

A

growing proceeds from centre of body to periphery
- trunk to shoulders to hands

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18
Q

genotype

A

blueprint of information in DNA

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19
Q

phenotype

A

observable traits

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20
Q

twins studies determined

A

genes matter for motor skills
- influence of genes declines over time (in comparison to environmental influenes

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21
Q

CNS

A

spinal cord and brain
- take info from environment

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22
Q

PNS

A

nerve fibers that connect the body to CNS
- autonomic system
- controls voluntary movements like walking

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23
Q

Brainstem

A
  • reflexes
  • breathing
  • heartbeat and rhythm
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24
Q

medulla

A
  • in brainstem
  • sensory signals from SC and motor signals from brain
  • respiration, BP, HR
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25
Q

Pons

A
  • in brain stem
  • coordination and posture
  • involuntary influences
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26
Q

midbrain

A
  • in brainstem
  • reflex movements from visual and auditory stimulation
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27
Q

thalamus

A
  • in brainstem
  • important integration center
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28
Q

cerebral cortex contains

A
  • frontal lobe
  • parietal lobe
  • occipital lobe
  • temporal lobe
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29
Q

motor cortex contains

A
  • primary motor cortex
  • premotor cortex
  • supplementary motor area
  • posterior parietal cortex
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30
Q

primary motor cortex

A
  • actual execution of movements
  • speed and force of actions
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31
Q

premotor cortex

A
  • working memory
  • plan and guide movement
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32
Q

supplementary motor area

A
  • preparation of movement
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33
Q

posterior parietal cortex

A
  • planned movements, spatial reasoning, attention
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34
Q

basal ganglia

A

integrate sensory motor centres
- unconscious behaviour
- planning and coordinating movements
- fundamental body movements

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35
Q

cerebellum

A

responsible for balance and the timing of movements

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36
Q

nerve fiber types

A
  • afferent fibers
  • efferent fibers
  • cranial nerves
  • spinal nerves
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37
Q

afferent fibers:

A

transmit sensory info to SC and brain

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38
Q

efferent fibers

A

transmit motor impulses from CNS to periphery

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39
Q

cranial nerves

A

transmit info with the brain

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40
Q

spinal nerves

A

transmit info within SC

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41
Q

neuron

A

Specialized cells that send and receive signals

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42
Q

3 types of neurons

A
  • sensory neurons: signals to SC, brain, afferent
  • motor neurons: signals from CNS to muscles
  • interneurons: info through CNS
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43
Q

nature contributes to

A

pre-wiring

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44
Q

environment

A

fine tunes connections

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45
Q

brain development sequence of events

A
  • cell proliferation
  • migration: move where needed
  • integration and differentitation
  • myelination
  • cell death
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46
Q

when does the brain growth spurt/ critical period of neural development happen

A

3rd trimester to 4th year of life

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47
Q

what happens with the development of dendrites

A

thick ones develop prenatally and are responsible for brain growth from birth to age 2

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48
Q

synapotgenesis

A

building synapses
- transmission between axon of neuron and dendrite of another neuron

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49
Q

where does the most neuron growth happen after birth

A

hippocampus
- neurogenesis: influenced by PA!

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50
Q

roles of myelination

A
  • visual pathways
  • touch
  • motor control
  • higher brain functions
  • memory
51
Q

when does memory develop

A

well into the 30s

52
Q

when does touch develop until

A

2 years old

53
Q

link of myelination to what types of movement

A

neurogenic behaviours : neural connection to muscle
reflexes : connections of sensory input and motor response
voluntary movement control

54
Q

when does cell pruning and death happen around, and why does it happen

A

10 years of age
- use it or lose it

55
Q

how much neutrons may the brain lose in pruning and cell death

A

40-75% of neutrons they originally made

56
Q

windows of opportunity for 1. gross motor skills 2. fine motor skills 3. un\used connections

A
  1. prenatal to age 5
  2. improves till age 9
  3. fade away around age 10
57
Q

brain growth sequence

A
  1. midbrain: reflexes, automatic processes
  2. cerebrum: complex thinking and functioning
  3. cerebellum: coordination and balance
58
Q

bone structure compenents

A
  • extracellular matrix
    organic (collagen) and inorganic components

bone cells

59
Q

what are the types of bone cells

A

osteocytes
osteoblasts
osteoclasts
osteogenic cells

60
Q

inorganic component allows for

A

hardness and structure

61
Q

osteoclasts job

A

carve for calcium, protein, other ions for bone respiration

62
Q

bone remodelling is a

A

lifelong process

63
Q

compact bone

A

80% of bone mass
(cortical)
- dense outer region
- allows bone to resist bending and torsion

64
Q

spongy bone

A

20% of bone mass (trabecular)
deep to compact bone, mesh like trabeculae
allows for bone to resist forces in many directions

65
Q

bone marrow

A

found within medullary cavity and within spongy bone
- red marrow
- yellow marrow

66
Q

red marrow

A

site of hematopoiesis (formation of blood cellular components)
- cranium, vertebral body, ribs, sternum, allium, proximal epiphyses of humerus and femur

67
Q

yellow marrow

A

found in long bones of adults
- adipose tissue

68
Q

bone remodelling: formation & resorption

A
  • balanced activity of osteoclasts and osteoblasts
    modulated by hormones
    modulated by physical stress
  • use it or lose it policy
69
Q

what hormones modulate bone remodelling

A
  • parathyroid hormones
  • calcitonin
  • estrogen
    -testosterone
  • growth hromone
  • cortisol
70
Q

peak bone density determined by

A
  • genetics (60-80%)
  • hormones (estrogen and testosterone)
  • physical activity
  • nutrition (calcium, vitamind D, protein)
71
Q

predictors of bone loss

A
  • hormone changes
  • estrogen, testosterone
  • physical inactivity
  • chronic inflammation
  • inadequate nutrient intake or absorption
  • smoking
  • high alcohol intake
72
Q

when is peak bone density in females

A

around 15 years old
- stays ok till menopause (around 52). then rapid decline

73
Q

when is peak bone density in males

A

16-22 years old

74
Q

osteoporosis

A

loss of bone mass and density, loss of bony matrix and mineralization
- cortical bone becomes porous and thin
- trabecular bone loss and thinning

75
Q

Bone mass

A

amount of bone tissue in the skeleton

76
Q

bone density

A

mineral mass per unit of volume of bone

77
Q

signs of osteoporosis

A

shrinking, losing height, loose geometry

78
Q

two forms of osteoporosis

A
  1. primary : age related: increased risk among older females
  2. secondary: due to another disorder or issue
    - conditions associated with bone loss
    - prolonged use of drugs
    - alcoholism
79
Q

risk factors for osteoporosis

A
  • genetics
  • age
  • sex
  • menopause (estrogen deficiency)
  • sedentary life
  • nutrition deficits or malabsorption
  • cigarette smoking
  • prolonged glutoccorticoid use
80
Q

what is the ratio of osteoporosis risk for females vs males

A

females= 1:3
males= 1:5

81
Q

how does low estrogen cause osteoporosis and resorption

A

low estrogen stimulates increased osteoclasts activity, ramping up the RANK-L pathway and pro inflammatory cytokines
DIRECT IMPACT: increased osteoclast, decreased osteoblasts activity
INDIRECT IMPACT: increased cytokine activity (stimulates OC), increased OC activity via RANK receptor pathway

82
Q

how does chronic inflammation effect peak bone density level

A
  • pro inflammatory cytokines increase OC activity
83
Q

age related decline causes

84
Q

when does plasticity happen

A
  • durning normal development
  • new skills
  • after injury healing damaged areas
85
Q

what is plasticity

A

brain and body ability to adapt to change and reorganize in response to experiences and environmental stimuli

86
Q

when does the brain reach nearly 90% of its adult size

87
Q

when does the brain reach its full size

88
Q

what is the growth sequence of brain

A

1.midbrain first
2. cerebrum second
3. cerebellum last

89
Q

neuron loss physiology

A
  • dendrites shrink
  • axons loose density
  • psychomotor slowing
  • decline in valance
  • cerebellum loss of cells
  • slower nerve signals
90
Q

physical anthropology

A

provides information and scientific procedures related to the study of biological growth and development

91
Q

anthropometry

A

branch of science concerned with biological growth and body measurement

92
Q

what happens in the germinal period- prenatal development

A

cell division and implantation

93
Q

what happens in the embryonic period- prenatal development

A

foundation for Motor skills and cognitive functions
- teratogen exposure may lead to congenital disorders

94
Q

what happens during the fetal period- prenatal development

A

growth and maturation of organs and tissue

95
Q

low birth weight/pre-maturity effects

A

(less than 5lbs)
- caused by premature birth or fetal growth restriction
*not automatically bad, can catch up
but can have health risks; especially less than 1lb
- motor dysfunction and delay risk

96
Q

what are hormones

A

chemical messengers that tell the body what to do and responsible for major growth and development in puberty

97
Q

what is involved in hormonal control

A
  • hypothalamus : body control centre
  • pituitary gland : master gland

supporting glands:
- gonads
- thyroid gland
- adrenal gland

98
Q

hormonal sequence

A
  1. hypothalamus
  2. pituitary gland
  3. thyroid, gonads, adrenal : produce growth hormones
  4. produce thyroxine, testosterone and estrogen, and androgens
99
Q

testosterone

A
  • levels increase 10x in puberty for males, 2x for females
  • builds muscle
  • strengthens bones
  • closes growth plates
100
Q

estrogen

A
  • increases 10x in puberty for females, 2x in males
  • growth spurt
  • bone development
  • tells body to store fat
  • closes growth plates earlier than boys
101
Q

when is growth hormone therapy prescribed

A
  • to children 2-2.5 standard deviations below mean height for age (SGA)
102
Q

why is assessing brain growth important

A

we can spot hydrocephalus (fluid build up in brain)

103
Q

when does height reach a steady state

A

Men: 18-30
Women: 16.5-30

104
Q

when does height decline with age

A

after our 30s

105
Q

what physical changes happen to bones during growth

A
  • bones grow longer, thicker
  • long bones go through modeling respiration
    (extra bone tissue breaks down to maintain shape> middle stays slim and ends grow bigger
  • short bones develop out of the core bone in centre and enlarge
106
Q

difference between female and male bone growth

A
  • females 20% advance skeletal maturity at birth
  • greater size in males bones post puberty
107
Q

adulthood : body weight change

A

20x weight than at birth
- declines later in life due to bone and muscle tissue loss

108
Q

what does a DXA scan measure

A
  • body fat
  • muscle and bone density
109
Q

hyperplasia body fat cells growth

A

rapid growth before birth and into first year of life

110
Q

hypertrophy of body fat cells

A

dependent on food intake, can lead to hypertrophy in tandem

111
Q

advanced aging effects

A
  • heigh decreases with age
  • weight increases steadily from 20s-60 years old
  • weight declines after 60 years old
    fat storage:
  • males store fat in belly
  • females store fat in hips, thighs, breasts
  • lean body mass decreases with age due to bone and muscle mass decline
112
Q

how much of fat storage patterns do genetics effect

A

patterns in storage inherited by 50% genetic

113
Q

maturity can be estimated by

A
  • morphological age
  • dental age
  • sexual age
  • Skeletal age
114
Q

sexual age determined by

A
  • sex characteristics
  • menstruation in females
115
Q

skeletal age determined by

A
  • cartilage change over time (ossification)
116
Q

maturity variations are measured by

A

comparing skeletal age and chronological age
- 20% difference from average or 1-3years classifies an early/late maturer

117
Q

secular trends

A

growth and maturity rates that change throughout generations
- better living conditions

118
Q

postnatal development influences

A
  • nutrition (balanced< for repair of body tissues)
  • physical activity (no effect on body physique but can contribute to positive development)
119
Q

cardiorespiratory structures to have memorized

A
  • heart
  • air flow
120
Q

heart growth

A
  • grows rapidly (big growth spurt in later years)
121
Q

heart grows in proportion to

A

heart blood volume

122
Q

heart growth sex differences

A

relatively the same until puberty
- at puberty *male heart 15% larger then female

123
Q

lung growth - prenatal development

A
  1. lung buds
  2. bronchial
  3. sensory/motor and nerves
  4. capillaries (at 20 weeks)
  5. alveolar-capillary interface: gas exchange
124
Q

lung growth- postnatal development

A

-at birth 20 million alveoli present
- rapid growth after birth
year 1: 3x lung capacity
year 8: same alveoli as adult
after age 8: alveoli growth bigger : chest wall grows