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N202 Exam 3 Unit 2 > MI 18 Questions > Flashcards

MI 18 Questions Flashcards

1
Q

HR*SV

A

CO

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2
Q

CO*BMI

A

CI

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3
Q

Length of Cardiac Fiber & amy of bld in ventricle during diastole –> Force of contraction

A

Starlings Law

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4
Q

Normal Ejection Fracture

A

60-70%

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5
Q

Leading cause of morbidity and mortality

A

MI

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6
Q

Transmural Necrosis

A

4-6h

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7
Q

Subendocardial Necrosis

A

w/in minutes

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8
Q

Q wave

A

Zone of infarction

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9
Q

ST Elevation

A

Injury

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10
Q

T wave inversion

A

Ischemia

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11
Q

Location of Infarct determined by

A

EKG

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12
Q

Leads V1-V4

A

Anterior (LAD)

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13
Q

Leads II, III, aVf

A

Inferior (RCA)

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14
Q

Leads V5-V6, I, aVl

A

Lateral, Circumflex

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15
Q

Healing process post MI begins

A

in 24h

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16
Q

Post MI tissue is vulnerable

A

10-14d

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17
Q

Most Indicative Lab

Dx Acute MI

A

Troponin (protein)

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18
Q

Determine extent and tx of MI

A

Serial EKG
ST-elevation (1mm+/-)–> (Fireman’s hat)
Appear w/in few hrs

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19
Q

ST elevation returns to normal post MI

A

~2weeks

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20
Q

Normal Q

A

<1mm

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21
Q

Q wave changes (post MI) appear..

A

hours to days after MI..

Does not return to norm.

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22
Q

Primary goal of Tx (MI) acute phase

A

RELIEVE PAIN***

then control arrhythmias & stop progression

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23
Q

Greatest S/S of Ischemia

A

pain

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24
Q

Most common SE of MI

A

arrhythmias–> constant monitoring

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25
Q

First line anti arrhythmic

A

Amiodarone

26
Q

Decrease REMODELING of ventricle, ventricular dilation, CHF & mortality

A

ACE Inhibs

27
Q

Increase in Temp post MI

A

Indicates myocardial necrosis process

28
Q

PTCA w/in

A

90mins of MI

29
Q

ONLY FOR WITNESSED V-FIB ARREST W/ ROSC

A

Therapeutic Hypothermia

–>improved neurological o/c

30
Q

Hypothermia Protocol Temp

A

32-34C for 18-24h
Monitored w/ bladder probe
Ice packs and cooling blankets

31
Q

Cause sudden death 2h post AMI

32
Q

Sinus Brady post AMI

A

40% inferior wall

No Tx unless S/S

33
Q

Most common reperfusion rhythm
tolerated well
no Tx

A

Accelerated Idioventricular Rhythm

34
Q

hypersensitivity to necrotic products
Pericarditis w/ pleural effusions
10d-3mo Post MI

A

Dressler’s Syndrome

35
Q

Pleuritic pain w/ breathing
Friction Rub
Left Pleural Effusion
Fever

A

Dressler’s Syndrome

36
Q

Ventricular Septal Rupture

A

Medical Emergency

Blood shunted from LV–>RV

37
Q

New, Loud systolic murmur
Progressive dyspnea
Tachycardia
Pulmonary Congestion

A

Ventricular Septal Rupture
Cardiac cath/ Sx in OR–>patch
Nipride (afterload reducers)
Lasix (decrease preload)

38
Q

5-6d post MI
Sudden neck vein distention
Decreased BP (hypotention)
PEA

A

Cardiac Rupture

Rapid, Typically death

39
Q

1st line antiarrhythmic

A

Amiodarone

40
Q

For PVC’s & Vtach give

41
Q

decrease infarct sz, pre/after load, HR/ contractility

42
Q

decrease remodeling of ventricle

A

ACE

SE: Cough

43
Q

Therapeutic Hypothermia only for

A

witnessed V-fob arrest with ROSC

44
Q

Tx PVC

A

IV Lido, Pronestyl, Cordarone

45
Q

Most common re-perfusion rhythm

A

Accelerated Idioventricular Rhythm (No Tx)

46
Q

Common cause of Acute Pulmonary Edema

A

Left Sided HF

47
Q

Paroxysmal Nocturnal Dyspnea

Coughing w/ frothy blood tinged sputum

A

Acute Pulmonary Edema

48
Q

Digoxin

A

Positive Inotropic Agent

Cardiac glycoside

49
Q

Dobutamine (Dobutrex)

A

B-adrenergc agonists

+ Inotropic Agent

50
Q

SE of Dobutamine

A

Increased Ventricular Irritability

Increased O2 demand by myocardium

51
Q

Dobutamine –>

A

increased contractility and renal, mesenteric, coronary, & cerebral blood flow

52
Q

Venous Arterial Smooth Muscle Vasodilator

53
Q 
Cyanide Toxicity 
N/V
Confusion 
Tinnitus 
Hypotension
54
Q

Biventricular Pacing

Resynchronization

A

only when asynchronous btwn ventricles

55
Q

loss of 40% fund myocardium –>

A

Cardiogenic Shock

56
Q

Dopamine

A

Vasopressor
Increases Contractility, CO, BP
Use central line-vessicant

57
Q

SE Dopamine

A

HA, Palpitations, Tachycardia

Tissue Sloughing

58
Q

Dobutamine Decreases

59
Q

Dobutamine Increases

A

CO, SV
(minimal HR & BP)
Preferred when no hypotension

60
Q

Inocor decreases

A

preload and afterload

relaxes vascular smooth muscle

61
Q

3 purposes of Intra-Aortic Ballon Pump

A

Relieves the LV workload
Forces blood into Coronary arteries
Decreases Afterload

N202 Exam 3 Unit 2 (3 decks)

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