Mental Health Exam #2 Flashcards

1
Q

Define “personality”.

A

Personality: how we perceive and interact with the world

–Personality traits –> stylistic peculiarities that all people bring to social relationships

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2
Q

What are traits/characteristics of a personality disorder?

A

Personality disorder traits:

  • Difficulty accurately perceiving and interpreting the world around them
  • Difficulty with impulse control
  • Inappropriate emotional responses
  • Blaming (genuinely unaware that their personality traits are causing their problems)

Personality disorder characteristics:

  • Avoidance and fear of rejection
  • Blurring of boundaries between self and others (fusion)
  • Insensitivity to other’s needs
  • Demanding
  • Fault finding (“grievance collectors”)
  • Inability to trust
  • Lack individual accountability
  • Passive-aggressive
  • Tendency to evoke intense interpersonal conflict
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3
Q

What causes personality disorder (PD)?

A

No single cause exists for PD
PD is due to a combination of hereditary and temperamental traits, as well as environmental and developmental events
Personality traits are present from infancy
Disorder usually emerges in adolescence

Genetic factors

  • Research supports the dominant role of genetics
  • Identical twin studies

Neurobiological factors

  • Aggressive, impulse behaviors
  • Affective instability (ex: can be d/t stroke, TBI)

Psychologic influences

  • Childhood neglect is particularly damaging
  • Childhood trauma (excessively harsh and erratic discipline, alcoholic parent(s), etc.)
  • Abuse and chaotic home life are risk factors for borderline PDs (BPD) and antisocial PDs
  • Sexual abuse is a risk factor for BPD
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4
Q

What are the clusters of PDs?

A

Cluster A Disorders – odd

  • Seen as “odd” or eccentric
  • Have unusual beliefs
  • Avoid interpersonal relationship, often indifferent

Cluster B Disorders – emotional

  • Emotional reactivity
  • Poor impulse control
  • Manipulative
  • Unclear sense of identity

Cluster C Disorders – anxious

  • High anxiety and outward signs of fear
  • Internalized blame (even when not to blame)
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5
Q

What types of PDs fall under Cluster A?

A

Schizotypal Personality Disorder

  • Resembles schizophrenia, but with no psychosis
  • Odd, eccentric behavior and speech
  • Cognitive perceptual distortion (without psychosis)
  • May display magical thinking and rituals
  • Give-and-take conversations are difficult
  • Genuinely unhappy about lack of relationships
  • Social anxiety and unhappiness may increase over time

Paranoid Personality Disorder

  • Persistent, inappropriate suspicion and distrust of others
  • Present as hostile, irritable, injustice collectors
  • Jealous, lacking warmth
  • May appear businesslike and efficient (but generate fear and conflict in others)
  • Find malice in benign comments and behaviors (ideas of reference)

Schizoid Personality Disorder

  • Flat affect; appear indifferent to both praise and criticism
  • Unable to establish relationships
  • Restricted range of interpersonal emotions
  • Invest little energy in human relationships; conversely, may invest enormous energy in nonhuman interests (mathematics, astronomy, etc.) and often connect more with animals
  • Often creative, original thinkers
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6
Q

What types of PDs fall under Cluster B?

A

Antisocial Personality Disorder – “Mob bosses”

  • Persistent disregard for others
  • Persistent violation of others’ rights
  • Absence of remorse for hurting others (callousness)
  • Sense of entitlement
  • Deceitfulness
  • Impulsiveness; risky behaviors to “feel alive”

Borderline Personality Disorder – “Manipulators”

  • Unstable, intense relationships
  • Instability of affect, frequent mood changes
  • Emotional lability (shifting from anxiety to irritability, etc.)
  • Poor impulse control; self-destructive, suicide prone, often self-harm
  • Chronic depression
  • Projected identification
  • Splitting (pit staff against each other)

Narcissistic Personality Disorder – “Trump”

  • Grandiose sense of personal achievement
  • Sense of entitlement
  • Lack of empathy, exploiting others to meet own needs
  • Increasing attention seeking over time
  • Envious of others
  • Use of splitting, tantrums
  • Can be sadistic, with paranoid tendencies

Histrionic Personality Disorder – “Jessica Rabbit”

  • Manipulative, insensitive
  • Dramatic, rapidly shifting, charming, flamboyant; use sexually seductive behaviors
  • Need to become and remain the center of attention, love, and admiration
  • Constant, sudden emotional shifts and lability
  • Superficial, shallow, short-lived relationships
  • Lack insight into cause of relationship failures
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7
Q

What types of PDs fall under Cluster C?

A

Avoidant Personality Disorder

  • Feelings of low self-worth
  • Hypersensitive to criticism or rejection
  • Avoid situations requiring socialization, withdrawal
  • Fearful of disappointment or ridicule
  • Reluctant to express irritation or anger, even when justified
  • Social phobia

Obsessive-Compulsive Personality Disorder

  • Orderliness, stubbornness, attention to detail
  • Indecisiveness
  • Emotional constriction
  • Pervasive pattern of perfection and inflexibility
  • Perseveration (persistent pursuit of an action even in the face of repeated failures)
  • High achievers
  • Superficial, rigidly controlled intimacy
  • Stinginess

Dependent Personality Disorder

  • Belief in inability to survive if left alone
  • Excess need to be taken care of
  • Solicit caretaking through clinging and submission
  • Excessive submission
  • Intense fear of separation and being alone
  • Tolerant of poor, even abusive relationships
  • If relationship does end, the individual has an urgent need to get into another
  • Inability to make decisions without excessive reassurance
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8
Q

How do you assess patients with PDs?

A

Personality disorder assessment:

  • Assess suicidal/homicidal thoughts
  • Determine whether the patient has a medical disorder or another psychiatric disorder
  • View the assessment of personality function from ethnic, cultural, and social backgrounds
  • Ascertain recent and important losses
  • Evaluate for changes in personality in middle adulthood or later (may signal unrecognized substance use disorder)
  • Be aware of strong negative emotions that patient evoke
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9
Q

How do you manage PD behaviors?

A

Managing behaviors:

  • Assess the patient for a short period before labeling him or her as manipulative
  • Set limits on manipulative behaviors
  • –Arguing or begging
  • –Using flattery or seductiveness
  • –Instilling guilt and clinging
  • –Constantly seeking attention
  • –Pitting one person, staff member, or group against another
  • –Frequently disregarding the rules
  • –Constant engaging in power struggles
  • –Exhibiting angry, demanding behaviors
  • Behaviors should be objectively documented (time, date, and circumstances)
  • Provide clear boundaries and consequences
  • Enforce consequences
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10
Q

What should you avoid around people with personality disorders (PDs)?

A

Avoid:

  • Discussing yourself or other staff members with patients
  • Promising to keep a secret
  • Accepting gifts from patients
  • Doing special favors for patients
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11
Q

How should you communicate with someone who has a PD? What are the goals of this therapeutic relationship?

A

Nurses gently enhance their ability to be therapeutic when they use

  • Limit-setting
  • Trustworthiness
  • Dealing with manipulations
  • Authenticity with their own natural style

Goal of therapeutic relationship

  • Identify what precedes impulse acts
  • Explore effects on self and others
  • Recognize cues
  • Identify triggers
  • Discuss alternative behaviors
  • Teach coping skills
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12
Q

What are some barriers to a therapeutic relationship with someone who has a PD?

A

Creating a therapeutic relationship with some who has a PD is especially difficult

  • Suspiciousness, aloofness, and hostility will set up failure
  • Guarded and secretive style produces an atmosphere of combativeness
  • When patients blame or attack others, the nurse needs to understand the context of the complaints
  • Attacks spring from a feeling of being threatened
  • The more intense the complaints, the greater the fear of potential harm or loss
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13
Q

What types of therapies are used for PDs?

A

Psychotherapy
Cognitive behavioral therapy (CBT)
Dialectical behavior therapy (DBT)

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14
Q

What is the goal of DBT?

A
DBT = Dialectical Behavior Therapy
Goal: regulate emotions and achieve behavioral control (through developing distress tolerance skills and crisis interventions)
Decrease target behaviors, such as…
-Life-threatening suicidal behaviors
-Therapy-interfering behaviors
-Quality-of-life interfering behaviors
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15
Q

What is the goal of STEPPS?

A

STEPPS = Systems Training for Emotional Predictability and Problem Solving

  • Offers a 20-week manual-driven program
  • Reduces the intensity of the core aspect of BPD
  • Does NOT reduce hospital utilization and suicidal ideation
  • DOES reduce suicide attempts and visits to the emergency department
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16
Q

What medications are used to treat PDs?

A

There are no medications specifically for PD, but some meds can alleviate symptoms of PD
Benzodiazepines – NOT appropriate because of the potential of abuse and overdose; may only be used in emergency situations
-SSRIs – treats co-morbid depression and panic attacks
-Trazodone and venlafaxine – have low toxicity in overdose
-Carbamazepine – target impulsivity and self-harm
-Lithium, anticonvulsants – minimize aggression
-Atypical antipsychotics – help with psychotic features of BPD under stress

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17
Q

What factors make someone more likely to develop depression?

A
  • People with co-occurring chronic medical problems (HTN, backache, DM, heart problems, arthritis)
  • Women more likely (hormones)
  • Older adults (>65 yo.) more likely (often goes undiagnosed)
  • Accompanies other psychiatric disorders
  • Rate is growing in children and adolescents
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18
Q

What causes depression?

A

Biological Model

  • Biological theories – genetic factors (twin and adoption studies)
  • Biochemical factors – serotonin and norepinephrine are two major neurotransmitters involved in depression
  • Hormones – alterations in hormonal regulation

Diathesis Stress Model

  • Environmental
  • Interpersonal
  • Life events combined with biological predisposition (diathesis)
  • Psychologic stressors that trigger brain changes

Psychosocial theory

  • The stress-diathesis model of depression
  • Learned helplessness
  • Cultural considerations

Cognitive theory (Beck’s cognitive triad)

(1) Negative, self-deprecating view of self
(2) Pessimistic view of the world
(3) Belief that negative reinforcement will continue

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19
Q

What are the subtypes of depression?

A
Psychotic features
Melancholic features
Atypical features
Catatonic features
Postpartum onset
Seasonal affective disorder (SAD)
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20
Q

How do you assess patients with depression?

A

Depression assessment:

  • Self-assessment (RN) – unrealistic expectations of self, feeling what the patient is feeling
  • Assessment tools – MSE, psychosocial assessment
  • Assessment of suicidal potential
  • Key assessment findings (lack of self-care, flat affect, hopelessness, etc.)
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21
Q

How do you assess for suicidal potential?

A

Mood
Anhedonia (inability to experience pleasure)
Anergia (lack of energy)
Anxiety
Worthlessness, guilt, helplessness, hopelessness
Anger and irritability

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22
Q

How should you communicate with someone who has depression?

A

Communication guidelines:

  • Person with depression may speak and comprehend very slowly
  • Extreme depression – person may be mute
  • Nurses should not be uncomfortable with silence
  • Sitting with a patient in silence is a valuable intervention
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23
Q

What medications are used to treat depression?

A

For people with depression, but without psychotic features, a combination of specific psychotherapies (CBT, etc.) and antidepressant therapy may be superior to psychotherapy or psychopharmacologic treatment alone.

SSRIs = Selective Serotonin Reuptake Inhibitors
Fluoxetine (Prozac)
Sertraline (Zoloft)
Paroxetine (Paxil)

MOA: increase serotonin
Side effects: some anticholinergic effects (thought fewer than TCAs), N/V, serotonin syndrome risk (especially when administered with MAOIs)

TCAs = Tricyclic antidepressants
Amitriptyline (Elavil)
Nortriptyline (Pamelor)

MOA: increases norepinephrine
Side effects: anticholinergic effects, LETHAL if OD occurs

MAOIs = Monoamine Oxidase Inhibitors
Phenelzine (Nardil)
Tranylcypromine (Parnate)
EMSAM (selegiline transdermal system) – delivers MAOIs through skin

MOA: inhibits monoamine oxidase (which inactivates and degrades neurotransmitters)
Side effects: HTN crisis can occur if patient ingests tyramine foods (in some OTC meds (decongestants), cured meats, beer, wine, aged cheese, avocados, etc.)
Dietary restriction of tyramine must be maintained for 2 weeks after stopping MAOIs

Atypical antidepressants
Bupropion (Wellbutrin)
MOA: do not act on serotonin system; inhibit nicotinic acetylcholine receptors to reduce addictive effects
Good for nicotine withdrawal.

Mirtazapine (Remeron)
MOA: increase serotonin and norepinephrine
Combined with SSRIs to augment efficacy or counteract serotonergic side effects

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24
Q

What are the safety risks with antidepressants?

A

Antidepressants might contribute to suicidal behavior
No conclusive evidence supports that either new or old antidepressants precipitate suicide
FDA provides a “black box” warning
FDA recommends close observation for worsening depression or suicidal thoughts
Close observation is especially recommended in children, adolescents, and older adults starting antidepressant therapy.

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25
Q

What are some somatic therapies used for depression?

A

Electroconvulsive Therapy (ECT)
During ECT, the client is treated with pulses of electrical energy through electrodes applied to the scalp; the electrical stimulus is sufficient to cause a brief convulsion. General anesthesia and a skeletal muscle relaxant are administered to minimize the motor seizure and prevent musculoskeletal injury.
Indications for ECT
-Suicidal/homicidal
-Agitation or stupor is extreme
-Life-threatening illness that is a result of the refusal of foods or fluids
-History includes a poor drug response
-Standard medical treatment has no effect

Client teaching

  • NPO status is required for 6-8 hours prior to treatment except for sips of water with medications.
  • Anesthesia (eg, methohexital, propofol) and a muscle relaxant (eg, succinylcholine) will be administered; clients are unconscious and feel no pain during the procedure.
  • Driving is not permitted during the course of ECT treatment
  • Temporary memory loss and confusion in the immediate recovery period are common side effects of ECT

Other therapies

  • Vagus nerve stimulation
  • Rapid transcranial magnetic stimulation (rTMS)
  • Light therapy (for SAD)
  • St. John’s wort
  • S-adenosylmethionine (SAMe)
  • Peer support
  • Exercise
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26
Q

What are the subtypes of bipolar disorder?

A

Bipolar I disorder – manic and depressive episodes
Bipolar II disorder – primarily depressive episodes
Cyclothymic disorder – mood swings between mild depression and hypomania
Bipolar unspecified
Rapid-cycling bipolar disorder – switch at least 4x/year between mania and depression
Mania or hypomania with mixed features – mania: euphoric, irritability; hypomania: a less severe version of mania

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27
Q

What illnesses often co-occur with bipolar disorder?

A
Psychologic 
-Anxiety disorders
-Panic attacks
-Behavioral disorders
-Substance use
Note: substance and anxiety disorders worsen the prognosis and increase the risk of suicide 

Medical

  • Cardiovascular/cerebrovascular
  • Metabolic disorders
  • CNS system stimulation
  • Hypo/hyperthyroidism
  • Seizure disorders
  • HIV
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28
Q

What causes bipolar disorder?

A

Biological theories

  • Genetics
  • Neurobiological
  • Neuroendocrine
  • Neuroanatomical

Psychologic influences and cultural considerations

  • Stressful life events can trigger symptoms of borderline personality disorder (BPD)
  • Cultural difference and beliefs can vastly complicate the issue
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29
Q

What are the phases of mania?

A
  • Acute phase
  • Medical stabilization
  • Maintaining safety
  • Self-care needs

Continuation phase

  • Maintaining medication adherence
  • Psychoeducational teaching
  • Referrals

Maintenance phase
-Preventing relapse

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30
Q

How should you communicate with someone who has bipolar disorder?

A

Communication guidelines

  • Consistent limit setting is important to treatment
  • All team members need to communicate challenges
  • Patients with bipolar disorder are often ambivalent about treatment
  • Nonadherence to medication is a major cause of relapse
  • Therapeutic alliance is crucial
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31
Q

What communication strategies are effective with patients in acute mania?

A

Display a firm, calm approach
Express short, concise explanations or statements
Remain neutral
Maintain consistency
Conduct frequent staff meetings to agree on limit setting
Hear and act upon legitimate complaints
Firmly redirect energy

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32
Q

What is Milieu therapy (seclusion)?

A

Control during the acute phase of hyperactive behavior almost always includes immediate treatment with an antipsychotic medication. However, when a patient is dangerously out of control, seclusion or restraints may also be indicated.

Use of seclusion

  • Reduces overwhelming environmental stimuli
  • Protects a patient from harm to self or others
  • Prevents the destruction of property

Seclusion indications

  • Risk of harm to others or self
  • Patient is unable to control actions
  • Other measures (setting limits, beginning with verbal de-escalation, or using chemical restraints) have failed
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33
Q

What medications are used to treat bipolar disorder?

A
Lithium Carbonate (LiCO3) – Lithobid, Lithonate, Lithotabs
First-line agent for bipolar disorder 

MOA: Alters sodium transport in nerve and muscle cells and inhibits the release of norepinephrine and dopamine; (when sodium is gone, lithium takes over TOXICITY); does not inhibit the release of serotonin

Lithium blood levels

  • Therapeutic blood level (0.8 to 1.4 mEq/L)
  • Maintenance blood level (0.4 to 1.3 mEq/L)
  • Toxic blood level (1.5 to 2.0 mEq/L)

Contraindications: dehydration, kidney problems, N/V, sun exposure, exercise

Side effects (expected): hand tremors, weight gain, polyuria, thirst

Toxic side effects

  • Early signs (N/V, polyuria, slurred speech, muscle weakness)
  • Advanced signs (coarse hand tremors, mental confusion, ECG changes)
  • Severe signs (ataxia, blurred vision, seizures, stupor, coma, pulmonary complications, severe hypotension, death)

Anticonvulsant drugs – for rapid cycling and dysphoric mood; rough on liver

  • Divalproex (Depakote) – check Depakote levels
  • Carbamazepine (Tegretol)
  • Lamotrigine (Lamictal) - blood dyscrasias, Steven-Johnson rash

Anxiolytics – used in mania

  • Clonazepam (Klonopin)
  • Lorazepam (Ativan)

Atypical antipsychotics – used in mania; sedative properties

  • Olanzapine (Zyprexa)
  • Risperidone (Risperdal)
  • Aripiprazole (Abilify)
  • Ziprasidone (Geodon)
  • Quetiapine (Seroquel)
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34
Q

What is schizophrenia?

A

Schizophrenia is a devastating brain disease that targets young people in their teens and early twenties
Schizophrenia spectrum disorders are a group of psychotic disorders
Psychosis is not a diagnosis, but a symptom
Psychosis refers to a total inability to recognize reality (ex: delusion and hallucinations)

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35
Q

What illnesses often co-occur with schizophrenia?

A
Substance abuse disorders
Anxiety disorders
Depression
OCD
Panic disorders
Obesity (probably due to antipsychotic medications)  risk of DM and cardiovascular disease
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36
Q

What causes schizophrenia?

A

Combination of genetics and extreme non-genetic factors

Genetics:

  • Brain structure abnormalities (neuroanatomical)
  • Neurochemical (dopamine, serotonin, and glutamate abnormalities)

Non-genetic risk factors:

  • Prenatal stressors
  • Psychologic stressors – developmental, psychologic, physical, family stress
  • Environmental stressors – social adversity, chronic poverty, growing up in areas of crime, street drugs under 21 years
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37
Q

What are the phases of schizophrenia?

A

Prodromal – pre-psychotic phase (secluded, withdrawn)
Acute phase – positive and negative symptoms
Stabilization – decrease in severity
Maintenance phase – symptoms are in remission, with possible presence of milder, residual symptoms

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38
Q

What are the four types of schizophrenia symptoms?

A

Positive: + something to your life you are not supposed to have

  • Hallucinations
  • Delusions
  • Bizarre behavior
  • Catatonia

Negative: - something from your life you are supposed to have

  • Apathy
  • Lack of motivation
  • Anhedonia
  • Blunted or flat affect
  • Poverty of speech
  • Poverty of thought

Mood symptoms: (affective)

  • Depression
  • Anxiety
  • Demoralization
  • Dysphoria
  • Suicidality

Cognitive symptoms: (memory and thoughts)

  • Impaired memory
  • Inability to reason
  • Inability to solve problems
  • Lack of focus
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39
Q

What are alterations in thinking?

A

Impaired reality testing – absence of ability to correct errors in thinking

Delusions – false fixed beliefs not corrected by reasoning

  • Thought broadcasting (thinking everyone knows what you are thinking)
  • Thought insertion (thinking some put something into your head)
  • Thought withdrawal
  • Delusion of being controlled

Concrete thinking – impaired ability to think abstractly
Ex: “the grass isn’t always greener”

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40
Q

What are alterations in speech?

A
  • Associative looseness (no correlation between thoughts)
  • Neologisms (new words)
  • Clang association (things that sound alike, rhyme, start with the same letter, etc.)
  • Word salad (words that don’t go together)
  • Echolalia (pathologic repeating of another’s words) – seen in catatonia
  • Echopraxia (mimicking the movements of another) – seen in catatonia
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41
Q

What are alterations in perception?

A
  • Depersonalization (out-of-body experience)
  • Hallucinations (sees something not there)
  • Illusions (sees something real, but thinks it’s something else that is really not; ex: thinks IV tubing is a snake)
  • Command hallucination (telling them to do something; ex: hurt someone or self)
  • Derealization (feel like environment is changing around you; ex: one arm is bigger than the other)
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42
Q

What are alterations in behavior?

A
  • Bizarre behavior
  • Extreme motor agitation
  • Stereotyped behaviors (ex: former truck driver pretends they are driving)
  • Waxy flexibility (someone remains in one position for an abnormally long amount of time)
  • Stupor
  • Negativism (do the exact opposite of what you tell them to do)
  • Automated obedience (listens to you without thinking)

Loss of impulse control may result in agitated behaviors (ex: abruptly grabbing the TV remote from someone else and changing the channel)

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43
Q

How should you assess someone with schizophrenia?

A
Verify medical workup to rule out medical or substance-related psychosis (ex: PCP, LSD)
Then, assess for
-Drug and alcohol use
-Command hallucinations
-Belief system
-Comorbidity
-Medication regimen
-Family dynamics/support system
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44
Q

What are the outcomes identification for the phases of schizophrenia?

A

Phase I (Acute)

  • *Patient safety
  • Medical stabilization
  • Refrain from acting on delusions/hallucination

Phase II (Stabilization) and Phase III (Maintenance)

  • Medical adherence, understanding, and compliance
  • Continual recovery and functional improvement
  • Control and relapse prevention
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45
Q

What are the interventions for the phases of schizophrenia?

A

Phase I (Acute)

  • Psychopharmacologic treatment
  • Supportive/directive communication
  • Limit setting (milieu therapy, counseling)
  • Psychiatric, medical, and neurological evaluation

Phase II and Phase III

  • Family psychoeducation/community support
  • Health teaching
  • -Disease, medication management
  • -Cognitive and social skills
  • -Stress and anxiety control
  • Health promotion and maintenance
  • -Improve functional deficits
  • -Encourage nonthreatening activities
  • -Encourage family and social interaction
46
Q

How should you communicate with someone who has schizophrenia?

A

Communication guidelines

  • Lower the patient’s anxiety
  • Decrease defensive patterns
  • Encourage participation in therapeutic and social events
  • Raising feelings of self-worth
  • Increasing medication compliance
47
Q

What medications are used to treat schizophrenia?

A

Antipsychotic medications

  • Alleviate symptoms of schizophrenia but cannot cure underlying psychotic processes
  • Psychotic symptoms return with medication noncompliance
  • Antipsychotic drugs as effective in
  • -Acute exacerbations of schizophrenia
  • -Preventing a relapse
Conventional (First-Generation) Antipsychotics – target positive symptoms (+)
High potency 
-Trifluoperazine 
-Thiothixene (Navane)
-Fluphenazine (Prolixin)
-Haloperidol (Haldol)
-Pimozide (Orap)

Medium potency

  • Loxapine (Loxitane)
  • Molidone (Moban)
  • Perphenazine (Trilafon)

Low potency

  • Chlorpromazine (Thorazine)
  • Thioridazine (Mellaril)

Side effects: EPS, agranulocytosis, anticholinergic effects, orthostatic hypotension, lowered seizure threshold

Atypical (Second-Generation) Antipsychotics – target positive (+) and negative (-) symptoms

  • Aripiprazole (Abilify)
  • Clozapine (Clozaril) – agranulocytosis with this one!
  • Olanzapine (Zyprexa) – worst one for weight gain
  • Paliperidone (Invega)
  • Quetiapine (Seroquel)
  • Risperidone (Risperdal)
  • Ziprasidone (Geodon)

Side effects: metabolic syndrome (weight gain, dyslipidemia, altered glucose), risk of DM, HTN, atherosclerosis, and heart disease

48
Q

What are extrapyramidal side effects?

A

Dystonia (muscle stiffness)
Akathisia (restlessness)
Tardive dyskinesia (TD) – bizarre movements
Drug-induced Parkinsonism – shuffling gait, pill-rolling
*Neuroleptic Malignant Syndrome (NMS) – rare, but life-threatening; extremely high fever (>106F) and high BP

49
Q

How are extrapyramidal side effects treated?

A

Side effects often appear early in therapy and can be minimized with treatment

  • Lowering the dose
  • Prescribing antiparkinsonian drugs
  • -Trihexyphenidyl (artane)
  • -*Benztropine (Cogentin)
  • -Diphenhydramine (Benadryl)
  • -Biperiden (Akineton)
  • -Amantadine (Symmetrel)
50
Q

What are adjuncts to antipsychotic drug therapy?

A

Antidepressants – administered for severe depression
Lithium – reduce aggressive behavior
Benzodiazepines – improves positive and negative symptoms, decreases anxiety

51
Q

What are some other subtypes of psychotic disorders?

A

Schizotypal personality disorder
Schizophreniform disorder – disorder prior to schizophrenia dx
Brief psychotic disorder
Schizoaffective disorder – exhibit both schizophrenia and bipolar disorder
Delusional disorder
Shared psychotic disorder – ex: Waco and Jonestown
Substance/medication-induced psychotic disorder
Psychotic disorder associated with a known medical condition
Catatonic disorder (Catatonia)

52
Q

What are the six cognitive domains?

A
  1. Complex attention
  2. Executive functioning (decision-making)
  3. Learning and memory
  4. Language
  5. Social cognition
  6. Perceptual and motor ability (perceiving something + responding to it correctly)
53
Q

What are the three main categories of neurocognitive disorders?

A
  1. Delirium
  2. Mild neurocognitive disorder
  3. Major neurocognitive disorder
54
Q

How can you recognize delirium in a patient?

A

Essential features of delirium…

  • Cognitive disturbances (thinking, memory, disorientation, perception)
  • Attention disturbances (loss of focus, attention, confusion over situation and environment)
  • Sundown syndrome (increased confusion in the evening hours and early morning)
55
Q

What type of assessment do we do for delirium?

A

Initial presentation:

  • Acute onset
  • Sudden reduced clarity of the environment
  • Shifting attention, have to repeat questions
  • Fluctuating LOC
  • Autonomic hyperactivity (increased vital signs)
  • Hypervigilance (constantly alert, scanning the room)
  • Labile mood swings
  • Agitation and/or anger
  • Sundowning
56
Q

What are some risk factors for delirium?

A
  • Hospitalized patients
  • Older adults
  • Secondary to another medical condition
57
Q

What safety measures need to be implemented for patients with delirium?

A

Cognitive and perceptual disturbances:
-Can lead to illusions and hallucinations

Physical needs:
-Wandering, falling, nutrition/hydration

Mood and behavior:

  • Dramatic fluctuations
  • Labile (quick changing) moods can make patient impulsive
58
Q

How is delirium treated?

A

Medical management:
-Directed towards identifying and treating the underlying cause

Nursing implementation:

  • Directed towards patient safety
  • Always identify yourself
  • Keep distractions minimum
  • Communicate in simple and concrete phrases
  • Use reality-orientation aids (clocks, calendars)
  • Have patient wear eyeglasses or hearing aids
  • Maintain the same staff
  • Encourage family members to be supportive
  • Use of sitters
59
Q

What is a mild neurocognitive disorder?

A

Mild cognitive impairment (MCI):

  • Memory impairment is the main symptom (does not include dementia or age-related memory impairment)
  • Does not interfere with general cognitive functioning
  • Does not interfere with ADLs and socialization

Mild dementia:

  • More than one cognitive domain is affected
  • Interferes with navigation of daily life
60
Q

What is a major neurocognitive disorder?

A

Progressive impairment:

  • Cognitive functioning is severely affected (includes dementia/Alzheimer disease, primary type)
  • Global impairment of the intellect
  • No change in consciousness occurs
61
Q

What is the difference between primary and secondary dementias?

A
Primary:
-Irreversible
-Progressive
-Not secondary to any other diseases
Example: Alzheimer disease

Secondary:
-Result of some other medical condition
Example: AIDS-related dementia

62
Q

What are the two biggest risk factors for Alzheimer disease?

A
  • Age (older adults)

- Gender (females 2/3 more likely)

63
Q

What anatomical changes occur in AD? (Patho)

A
  1. Tau protein changes
  2. Neurofibrillary tangles
  3. Cortex shrinking
  4. Hippocampic degeneration
  5. Enlarged ventricles
  6. Beta-amyloid plaques
  7. Granulovascular degeneration
  8. Brain atrophy
64
Q

What are the four “defense behaviors” that occur with AD?

A
  1. Denial (cover up)
  2. Confabulation (making up answers in unconscious attempt to maintain self-esteem)
  3. Perseveration (repetition of phrases or behaviors)
  4. Avoidance of questions
65
Q

What are the cardinal symptoms of AD? *(think of the 4 A’s)

A
  1. Amnesia (memory impairment)
  2. Aphasia (loss of language)
  3. Apraxia (loss of motor control)
  4. Agnosia (loss of recognizing objects/people/places)
    - Disturbances in executive functioning (cannot make decisions)
66
Q

Describe the four stages of AD.

A

Stage 1: mild (forgetfulness, possible depression)

Stage 2: moderate (confusion, memory gaps, self-care gaps, apraxia, labile mood)

Stage 3: moderate to severe (unable to identify familiar objects or people, advanced agnosia or apraxia)

Stage 4: end-stage (agraphia, aka loss of communication, hyperorality, aka inappropriate items in mouth, bed bound)

67
Q

What nursing plans/interventions can be implemented for AD?

A
  • Gear the plan of care toward a person’s immediate needs
  • Identify level of functioning
  • Assess the caregivers’ needs
  • Provide realistic expectations
  • Identify community resources
  • Maintain good self-care (nurses)
68
Q

How should you communicate with someone who has AD?

A
  • Always identify yourself
  • Call the person by his/her name
  • Speak slowly, using short and simple words
  • Maintain face-to-face contact
  • Be near the patient when talking
  • Focus on one piece of information at a time
  • Encourage reminiscing about happy times
  • When patient is delusional, acknowledge the patient’s feelings and reinforce reality
  • Do not argue or refute delusions
  • Have patient wear eyeglasses or hearing aids
69
Q

What medications are used for AD?

A
  • No cure exists for AD
  • Prescription drugs currently approved by the FDA include cholinesterase inhibitors
Cholinesterase inhibitors: 
-Delay and prevent symptoms from becoming worse
-Are useful for mild to moderate AD
-Prevent the breakdown of acetylcholine, and stimulates nicotinic receptors to release more acetylcholine 
Examples:
--Donepezil (Aricept)
--Rivastigmine (Exelon)
--Galantamine (Razadyne)
--Memantine (Namenda)
Antidepressants:
-Treat the co-existing depression
Examples:
--SSRIs - well tolerated
--Mirtazepine (Remeron) - help with weight gain/sedation, given at night as sleep aide
70
Q

What community resources can be useful for AD patients and caregivers?

A
  • Transportation services
  • Supervision
  • Daycare centers
  • Support groups
  • Respite care
  • Meals on Wheels
  • Telephone helplines
  • Home health aides/nurses
  • The Alzheimer’s Safe Return program
71
Q

What is a SPMI?

A

SPMI = Severe and persistent mental illness

  • Mental disorders that persist over time and impair normal functioning
  • Affect adults
72
Q

What are some issues that those with SPMI face?

A
  • Medication side effects
  • Loss, hopelessness, depression
  • Co-occuring medical illness
  • Unemployment, poverty
  • Housing instability
  • Social isolation
  • Inadequate treatment
  • Substance use disorder
  • Victimization
  • Stigma
  • Anosognosia (do not know they are sick)
73
Q

What are some legal/ethical issues those with SPMI face?

A
  • Involuntary treatment requires a court order
  • Outpatient commitment
  • Criminalization of the mentally ill
  • Mental health courts (newly designed court programs, specific for SPMI persons)
  • Trans-institutionalization (shifting from one institution to another, or even onto the street)
  • De-institutionalization (moving from inpatient care into the community)
74
Q

How can you assess someone with a SPMI?

A
  • Risk to self or others
  • Depression, substance abuse, sleep impairment, impulsivity, delusions
  • Proper nutrition, clothing, self-care, medical care
  • Signs of nonadherence/relapse
  • Medical health problems (tumors, metabolic disorders, etc.)
75
Q

What interventions can be made for those with SPMI?

A
  • Pharmacologic, biological, and integrative therapies
  • Rehabilitation vs. recovery model
  • Evidence-based treatment
  • Programs of assertive community treatment (PACT, ACT) , treatment team is all in one place
  • Promotion of family support and partnerships (ex: NAMI)
  • Social skills training
  • Vocational rehabilitation
  • Teletherapy (offers services to veterans, rural locations)
76
Q

What is impulse control disorder?

A

-Involves a decreased ability to resist an impulse to perform a certain act
-Actions range from benign to harmful
-Tension builds until a particular action is taken
-Tension reduction reinforces future resistance
Examples:
–Intermittent explosive disorder
–Kleptomania
–Pyromania
–Gambling disorder
–Trichotillomania

77
Q

What pharmacologic interventions can help impulse control disorder?

A
  • SSRIs, bupropion, naltrexone - useful for kleptomania, trichotillomania, and gambling
  • Lithium and antipsychotics - useful for conduct disorders
  • Opioids - gambling
  • Anticonvulsants, lithium, SSRIs, propranolol - all reduce aggression and anxiety
78
Q

What nonpharmacologic treatments are used for impulse control disorder?

A
  • Hypnotherapy
  • CBT (habit reversal)
  • Biofeedback (changes physiologic response)
  • Behavioral conditioning (learned response for rewards or punishment)
  • Group psychotherapy
79
Q

What are the different kinds of sexual differences/disorders?

A

Gender dysphoria: once considered a disorder, now a lifestyle preference

Paraphilias and Paraphilic disorders
-Paraphilias = not perverse, no treatment needed (ex: foot fetish)
-Paraphilic disorders = can distress or harm (ex: pedophilia, beastiality)
Examples:
–Exhibitionistic disorder (sex in public)
–Fetish
–Frotteuristic disorder (grinding)
–Pedophilia
–Sexual masochism/sadism (BDSM)
-Transvestic fetishism (cross-dressing)
-Voyeuristic disorder (peeping tom)

80
Q

What is ADHD? How is it prevalent in our society?

A

ADHD = Attention Deficit Hyperactivity Disorder

  • Exhibits a persistent pattern of inattention, impaired ability to focus, and hyperactivity/impulsivity
  • Neurodevelopment disorder with multiple contributing factors
  • Heritability at about 60%
  • Underdiagnosed in adults

Adults who have been diagnoses with ADHD…

  • Are more likely to smoke, abuse alcohol or drugs
  • Greater risk of contracting an STD
  • More frequently change jobs
  • Have higher rates of depression
  • Have inhibited academic and socioeconomic achievement
81
Q

What medications are used for ADHD?

A

Stimulant medications: these medications can be abused, but are not generally addictive (only last in system for 6 hours at a time)

  • Methylphenidate (Ritalin)
  • Amphetamine variants (Adderall)

Other medications:

  • Antidepressants (buproprion)
  • Atomoxetine (Strattera)
82
Q

What are some examples of sleep-related disorders?

A
  • Insomnia: insufficient sleep
  • Hypersomnolence: excessive sleep
  • Narcolepsy; sudden urge to sleep
  • Obstructive sleep apnea/hypopnea: cessation and decrease in breathing during sleep
  • Circadian rhythm disorder: disregulation of internal sleep/wake cycle (ex: doctors, nurses, etc.)
  • Sleep arousal disorder: abnormal experiences during sleep, usually in response to a dream
  • Nightmare disorder: recurrence of highly realistic, upsetting nightmares
  • Restless leg syndrome: urge to move one’s leg in response to uncomfortable sensation
83
Q

What are some pharmacologic and nonpharmacologic treatments for sleep disorders?

A

Pharmacologic:

  • Methylphenidate, modafinil used for narcolepsy
  • Sedative hypnotics, benzos used for insomnia

Nonpharmacologic:

  • Continuous positive airway pressure (CPAP) for sleep apnea
  • CBT
  • Environmental management (dim lighting, calm environment, right temp)
  • Normalizing sleep patterns (routine)
84
Q

Define suicide and its concepts.

A

Suicide or completed suicide: the intentional ending of one’s life
Suicide attempts: life-threatening attempts that have not lead to death
Suicidal ideation: a person thinking of self-harm
-Always ASK
-Listen carefully to what is and is not said

85
Q

What is PAS? Where is it legal?

A

PAS = Physician-assisted suicide
Legal in…
-Oregon, Washington, Montana, and Vermont
-Netherlands, Belgium, Switzerland

86
Q

What are the guidelines for PAS?

A

Law established in 2009 that the patient must…

  • Not have any neuromedical and/or psychiatric conditions that may impair the ability to reason
  • Be diagnosed as having less than 6 months to live
  • Make a request orally and in writing
  • Have an agreement for PAS approved by two different physicians
  • Make the request again after a 15-day waiting period
87
Q

Why are suicide rates so high among veterans?

A

More deaths in active duty are by suicide than by combat.

  • Risky behavior among returning veterans (accidents, unintentional poisoning)
  • PTSD
  • TBI
88
Q

What medications may contribute to suicide?

A

Medications that may cause depressive symptoms include…

  • Antihypertensives
  • Benzodiazepines
  • Calcium channel blockers
  • Corticosteroids
  • Hormonal medications
  • Pain killers
  • Antidepressants (shortly after beginning therapy)
89
Q

What are some psychological theories behind suicide?

A

Sigmund Freud: murderous attack on self
Karl Menninger: revenge, depression, guilt
Edwin Shneidman: unbearable, psychologic pain - “there is no way out”, says self-destructive behaviors are subintentioned suicide (self-harm, hyperobesity, gambling, dangerous sexual behaviors, etc.)
Herbert Hendin: says it is malpractice not to prescribe medicine to those seriously depressed

90
Q

What are some risk factors for suicide?

A
  • Anxiety
  • Insomnia
  • Substance abuse
  • Poor critical thinking skills
  • Troubled emotional lives
  • Low threshold for emotional pain
91
Q

What are some neurobiological/societal/psychological theories behind suicide?

A

Neurobiological:

  • Strong association between suicide and low serotonin
  • Overactivity of nonadrenergic system (severe anxiety, agitation) increases risk
  • Hypothalamic-pituitary-adrenal (HPA) axis is associated with memory dysfunction, reasoning impairment, and suicidal ideation
  • Suicide “clusters” develop in some families

Societal/psychological:

  • Lack of a support system
  • Psychotic disorder (especially command hallucinations)
  • Feelings of worthlessness
92
Q

What two populations of people are most likely to attempt suicide and why?

A

Adolescents and young adults (14-24 years):

  • Substance abuse
  • Aggression
  • Depression
  • Social isolation
  • Family loss
  • Perception of failure (school, work, social)
  • Difficulty with sexual orientation
  • Unplanned pregnancy

Older adults (>65 years):

  • Social isolation (solitary living arrangements)
  • Lack of financial resources
  • Poor health
  • Most older adults who commit suicide have visited their PCP within a month before the suicide
93
Q

What behavioral cues may indicate someone is planning to attempt suicide?

A
  • Giving away prized possessions
  • Writing farewell notes
  • Making a will
  • Putting personal affairs in order
  • Failing to sleep for more than one night in a row (global insomnia)
  • Exhibiting a sudden improvement in mood
  • Neglecting personal hygiene
94
Q

What is the difference between overt and convert statements about suicide?

A

Overt statements: clear intent

  • “I can’t take it anymore.”
  • “Life isn’t worth living anymore.”
  • “I wish I were dead.”
  • Living is useless.”

Covert statements: hidden message
“It’s okay now. Everything will be fine.”
-“I won’t be a problem much longer.”
-“Nothing feels good and never will again.”
-“I want to give my body to medical science.”

95
Q

How do we assess for suicide?

A
  • Presence of a plan
  • Previous suicide attempt
  • History of mental illness (especially depression or substance abuse)
  • Impulse or aggressive behavior
  • Adverse life events
  • Isolation
  • Family history
  • Incarceration
  • Exposure to suicide from family, friends, peers, news, fiction
  • Chronic illness/pain
  • As a nurse, you ALWAYS ASK:
  • “Are you thinking of killing yourself?”
  • Assess the precipitating event
  • Assess the risk factors
  • Assess the protective factors
  • Assess the lethality of the plan (highly lethal = guns, hanging, carbon monoxide; lower risk = slashing wrists, ingesting pills)
96
Q

If a patient has a history of previous suicide attempts - what do you assess?

A

Intent - Is discoverability a high probability?
Lethality - Was the method used highly or less lethal?
Injury - Did the patient suffer physical harm?

97
Q

Define the mnemonic SAD PERSONS.

A

S - Sex (male)
A - Age (adolescents and older adults)
D - Depression

P - Previous suicide attempts
E - Excessive substance abuse
R - Rational thinking loss
S - Separated, widowed, divorced
O - Organized plan
N - No social support
S - States future intent
98
Q

What are some protective factors from suicide?

A
  • Family/community support
  • Effective clinical care for mental, physical disorders
  • Restricted access to highly lethal methods of suicide
  • Cultural/religious beliefs that discourage suicide
  • Acquisition of problem-solving
  • CBT
99
Q

What interventions are used during and after a crisis period?

A

During the crisis:

  • Provide a safe environment
  • Document the patient’s activity (every 15 minutes)
  • Maintain records of nurse/physician actions
  • Place patient on “suicide precautions/observation”
  • Construct a verbal or written no-suicide contract
  • Encourage the patient to talk about their feelings

After the crisis period:

  • Arrange to have patient stay with family or friends (or in hospital)
  • Remove weapons and pills
  • Encourage patient to talk freely, discuss alternatives, avoid decisions
  • Arrange for crisis counseling
  • Prescribe anxiolytics or antidepressants (only a 1-3 day supply should be given at a time)
100
Q

What happens if a patient completes suicide?

A
  • Post-trauma loss debriefing within 24-72 hours for the family
  • Address normal mourning feelings (guilt, stigma, isolation)
  • 45% of survivors report mental/emotional deterioration within 6 months

Nurses:

  • Psychologic postmortem assessment
  • Agency protocol analysis
  • Documentation completion
101
Q

What is the difference between anger, aggression, and violence?

A

Anger: a normal and not always logical human emotion

  • No judgment needs to be passed on it
  • Anger varies in intensity

Aggression: the act of initiating hostility

  • Often self-protective or protecting others
  • Arouses thoughts of attack

Violence: unjust, unwarranted, or unlawful display of verbal threats, intimidation, or physical force
-Does not always has its roots in anger, but the intent is to inflict harm

102
Q

What other disorders make someone more likely to develop violent behavior?

A
PTSD
Substance use disorders
Depression
Anxiety
*Psychosis
*Personality disorders
103
Q

What environmental influences make people more likely to be violent?

A
  • Childhood aggression (strongest predictor of adult violence)
  • Males (15-24 years)
  • Diagnosis of conduct disorder
  • Family history of violence
  • Low socioeconomic status
  • Subculture (video games, gangs, etc.)
104
Q

What are subjective and objective assessments of violent behavior?

A

Subjective:

  • H&P
  • Family and friends
  • Patient’s background
  • “Have you harmed someone before?”

Objective:

  • Irritable affect
  • Hyperactivity (clenched fists, pacing, etc.)
  • Increasing anxiety and tension
  • Raised voice
  • Stone silence
  • Possession of a weapon
  • Intense or avoidant eye contact
105
Q

What Milieu characteristics can be conducive to violence?

A
Loud
Overcrowded
Staff inexperience
Controlling staff
Poor limit setting
Staff inconsistency
106
Q

Explain the stages of the violence cycle.

A

Preassaultive stage: getting upset
-De-escalation approach (calm, speak softly, stand at a 45 degree angle, emphasize you are on the patient’s side, set limits)

Assaultive stage: SAFETY

  • Medication
  • Seclusion
  • Restraints
Postassaultive stage: reassess
-Seclusion and restraints should be assessed regularly
-Remove once patient is able to follow 
directions 
-Critical incident debriefing by staff
-Documentation
107
Q

What is the protocol for reintegration after restraint or seclusion use?

A

After restraints and seclusion:

  • Monitor every 15 minutes
  • Gradual reintegration (patient’s ability to handle increased stimulation)
  • Structured reintegration (taking off one restraint at a time, planned time-out periods)
108
Q

How should you respond to verbal abuse?

A
  • Leave immediately (let them know you will return when the situation is calmer)
  • If mid-procedure, discontinue conversation and eye contact, then leave when procedure is complete
  • Respond positively to, and reinforce, nonabusive communication
109
Q

What medications do we use for ACUTE aggression?

A

Benzodiazepines (lorazepam, etc.):
-First choice for acute aggressive episodes, rage

Second-generation antipsychotics (ziprasidone, olanzapine):
-For emergencies

110
Q

What medications do we use for CHRONIC aggression?

A

Anticonvulsants (carbamazepine) - labile mood, poor impulse control, dementia
Beta blockers (propranolol) - organically-based violence, ex: dementia
Antipsychotics - disorganized behavior
Buspirone
Clonidine - anxiety, agitation
Lithium- labile mood, impulsivity
SSRIs - anger attacks