Meckel's diverticulum & intestinal obstruction Flashcards
What is the cause of Meckel’s diverticulum?
What is Meckel’s diverticulum - where?
- Caused by a remnant of the embryological vitelloinstestinal duct (2% of population have it)
- Only 2% of people with Meckel’s develop symptoms
- The diverticulum is 2cm long, on the antimesenteric border of the bowel, 20 inches from the ileocecal valve
- It may be lined by gastric acid secreting epithelium, or heterotropic pancreatic tissue
What are the possible patholigical effects of Meckel’s diverticulum?
-
Caecal volvulus
- part of the colon twists on its mesentery, resulting in acute, subacute, or chronic colonic obstruction
- If tethered to the umbilicus, the diverticulum may act as the apex of a volvulus to present like a volvulus with obstruction
-
Intussusception
- Part of the intestine folds into the section next to it
- Often gangrenous by the point of operation
-
Appendicitis
- Diverticulum becomes inflamed, presenting identical to appendicitis (sometimes also with umbilical cellulitis)
-
Peptic ulceration
- Pain around umbilicus that is related to mealtimes, due to ulceration of the gastric acid secreting epithelium
- A sinus tract may also exist between the diverticulum and the umbilicus (patent viteloointestinal duct)
Describe the signs and symptoms in a patient with intestinal obstruction
Symptoms:
- Vomiting
- Undigested food suggests gastric outlet obstruction
- Bilous vomiting suggests upper small bowel obstruction
- Faeculent vomiting (thicker/foul-smelling) suggests more distal small bowel obstruction
- Pain
- Colicky abdominal pain in early obstruction
- Pain may be absent in long-standing obstruction
- Constipation
- May not be absolute (no passage of wind) in proximal obstruction
Signs:
- Distention
- Tinkling bowel sounds
- Dehydration
- Central resonance to percussion, dull flanks
- Scars: previous surgery causing adhesions
- Palpable mass (causing obstruction)
- NO abdominal tenderness (unless strangulation)
List the common causes of small bowel obstruction
- Adhesions (80%) - extramural
- Hernias - extramural
- Crohn’s disease - intramural
- Intussusception (part of the intestine folds into the section next to) - intramural
List the common causes of large bowel obstruction
- Carcinoma of the colon - intramural
- Diverticular disease - intramural
- Sigmoid volvulus - extramural
- Constipation (forgein body or faecal impaction ect) - intraluminal
What are the complications of bowel obstruction?
- The bowel wall becomes oedematous (tissue with excess interstitial fluid) and distends
- Bacteria proliferate in the obstructed bowel
- As the bowel distends, vessels become stretched and the blood supply is compromised, leading to strangulation (→ ischaemia & necrosis)
- Eventually the bowel will perforate
- Symptoms develop more gradually in large bowel obstruction due to the capacity (even greater if the ileo-caecal valve is incompetent)
Interpret these AXRs
Bowel obstruction on AXR; (1) Small bowel obstruction, showing valvulae conniventes crossing a dilated, centrally-located bowel; (2) Large bowel obstruction, with peripherally located dilated bowel segments
Small bowel - >3cm = dilated. The valvulae conniventes run the whole way across the small bowel
Large bowel - >5cm = dilated. The haustra do not run the whole way across the large bowel.
What investigations should be done for a patient with suspected small intestinal obstruction?
- Bloods: FBC, U&Es, Amylase, LFTs
- ABG
- Urinalysis
- Supine AXR: distened proximal bowel, absent gas distally
- Erect CXR: fluid levels in small bowel obstruction, air under diaphragm if perforation
- Contrast enema: differentitates obstruction and pseudo-obstruction, can identify the level of obstruction and ileo-caecal competency (gastrograffin may also have therapeutic effect)
- CT scan: can indicate level of obstruction, but cannot always give the diagnosis
Compare and contrast small to large bowel obstruction
Vomiting:
- Absent/faeculant in LBO
- Bilous in SBO
Constipation:
- Absolute in LBO
- May not be absolute in SBO
Progression:
- More rapid in SBO
The best differentitator is plain film radiograph of the abdomen