Maternity study cards Flashcards

Class

1
Q

Intimate Partner Violence is the most common form of violence against women worldwide

A
  1. Battering occurs in repeated cycles
  2. Violence and abuse include one or more of the following : Physical, Sexual, Emotional, Economic or Psychological elements.
  3. Most women abused prior to pregnancy, will be abused during pregnancy and abuse may escalate during pregnancy.
  4. Pregnant adolescents are abused at higher rates than adult women and should be considered at high risk.
  5. During Pregnancy, women should be screened for abuse at every prenatal visit and when she presents in the emergency room or Labor and delivery.
  6. Any reports of abuse or suspected IPV must be documented.
  7. Any IPV which could constitute a felony must be reported to appropriate authorities.
  8. Involvement of minors or elders must be reported to CPS.
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2
Q

When abuse is confirmed:

What to do?
What to say?

A

1.Provide her with resources such as the phone number of the Battered women’s shelter, hotline, safety plan
2.If the patient reveals she is experiencing IPV, make sure she has a safe place to go.
3.Offer to let her use a facility phone if she wants to make these calls.
4.If she is staying with the abusing partner, make sure she has a safety plan:
bag packed with necessities for an overnight stay
cash or check book
identification or legal documents required for identification
Extra set of car keys
Can hide the bag or leave it with a neighbor
5.Let the victim know that nobody deserves to be treated that way but avoid speaking negatively about the abuser (can cause victim to get defensive)
I am afraid for your safety and for the safety of your baby/children
I believe you
Abuse is progressive and will only get worse
You are not alone
I am here for you
You deserve better than this
You deserve to be treated with respect

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3
Q

What are the serious complications which can occur with the use of combined (Estrogen and Progestin) contraceptive Methods?

A

ACHES= A: Abdominal Pain
C: Chest Pain and/or shortness of Breath
H: Headache Which are sudden, persistent, increased BP or Stroke
E: Eye problems, blurred vision
S: Severe leg pain, Blood clots, Thromboembolic process

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4
Q

What are some factors affecting the effectiveness of Contraceptives?
(Perry, page 105, Box 5.7)

A
  1. Frequency of Intercourse (Condoms, withdrawal, barrier methods, natural family planning methods)
  2. Motivation to prevent Pregnancy
  3. Clear understanding of Method and use of method.
  4. Adherence to method
  5. Liklihood of pregnancy for the individual user
  6. Consistent use of the method
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5
Q

Can exclusive breastfeeding be a reliable form of birth control?

A

No, there is no reliable way to tell when ovulation has returned. Many women do not have a menstrual cycle while they are breastfeeding.

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6
Q

What are some ways that the nurse can provide Family centered Care?

A
  1. Remember that women and their support people are PARTNERS in their healthcare and not just passive recipients
  2. Communicate. Keep them in the loop. If you know you will need to weigh the baby between midnight and 2am, tell them ahead of time.
  3. Acknowledge the support people and family members when you enter the room to provide care.
  4. Include the primary support person.
  5. Ask mom “Who is going to be helping the most with the new baby?”
  6. Identify some ways we can include the support person?
  7. Include the support person in the teaching.
  8. Encourage them to provide hands-on care and provide encouragement
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7
Q

How does a nurse provide culturally sensitive care?

A
  1. Cultural relativism vs Ethnocentrism
  2. Culture determines viewpoint. Yours and theirs.
  3. Affirm the value and uniqueness of every culture
  4. Recognize that the behavior of others may be based upon different assumptions and logic than those held by the nurse.
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8
Q

Sexually transmitted infections include all of these

A
Bacterial
 Chlamydia
Gonorrhea
Syphilis
Group B Strep (Box 4.2) pg.71 Perry

Protozoa
Trichomoniasis

Viruses
Human Papilloma Virus
HIV
Herpes Simplex virus type 1 and 2
Viral Hepatitis A and B
Zika Virus
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9
Q

What tool do we use to screen women for depression during and after Pregnancy?

A

Edinburgh Postpartum Depression Scale

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10
Q

What are the risk factors for Postpartum Depression?

A
  1. History of anxiety or depression
  2. Previous history of Postpartum Depression
  3. Younger age
  4. Unintended Pregnancy
  5. History of Premenstrual Dysphoric Disorder
  6. Family history of mood disorders
  7. Unmarried
  8. Marital Discord
  9. Substance abuse
  10. Lack of social support
  11. Low self-esteem
  12. Complications of Pregnancy and Birth
  13. Women who are victims of intimate Partner violence.
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11
Q

What is the difference between Postpartum depression, Postpartum Blues and Postpartum Psychosis?

A

Signs of Postpartum Blues (Should go away in a few days to a week)
Sad, anxious or feeling overwhelmed
Crying spells
Loss of appetite
Difficulty Sleeping
Signs of Postpartum Depression (Can begin any time in the first year)
Same signs as Postpartum Blues but last longer and are more severe
Thoughts of harming baby or self
Not having any interest in the baby
Not being able to care for baby or self
Anxiety
Loss of enjoyment
Signs of Postpartum Psychosis
Seeing or hearing things that are not there
Feelings of confusion
Rapid Mood swings
Trying to harm yourself or your baby.

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12
Q

What is Universal newborn screening?

A

Blood spot screening: Ideal time of collection is 24 - 48 hours of age. Some screening tests are not valid if the specimen is collected before 24 hours of age, so another screen will be needed in order to complete the blood spot screening process. Early discharge guidance: • Blood spot screening should be performed prior to discharge, regardless of age. • Arrange to collect a subsequent blood spot screen between the optimal time of 24 and 48 hours of life. This additional screen should be collected no later than 48 hours given the severity of the disorders.

Hearing screening: Ideal timing is after 12 hours of age and before discharge. Hearing screening is valid at any age, so standard recommendations for follow-up and documentation/communication of results should be followed.

Pulse oximetry screening: Ideal timing is 24 - 48 hours of age. Early screening may not accurately measure a newborn’s circulation as ductal closure may not have occurred yet, which increases the likelihood of both false positive and false negative results. Early discharge guidance: • Pulse oximetry screening should be performed prior to discharge, regardless of age. • Alert newborn’s primary care provider that the newborn was discharged early and oxygen saturation levels should be assessed at the first well-child check.
PG 539 Perry

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13
Q

What is NAS and how do we screen for it?

A

Evidence-based standardized protocols are needed to provide the best treatment for the mother-infant dyad and improve outcomes for infants neonatal abstinence syndrome (NAS) and mothers with substance use disorders.
Nurses must increase their awareness, establish trust, and support mothers who frequently feel judged and stigmatized because of drug use.
An infant’s recovery from NAS depends on positive attachment to a mother who is able to respond effectively to her infant’s needs.
To improve outcomes for infants with NAS and the mothers of these infants, a broader healthcare perspective is needed that recognizes that NAS isn’t an acute medical condition but requires a multidisciplinary team care approach.
Diagnosis
Diagnosing an infant with NAS requires an accurate history of the mother’s drug use (including the last drug used and the time of consumption) and evidence of withdrawal. A scoring system can be used to aid diagnosis. Points are assigned based on the severity of each sign, and the total score helps determine the treatment plan. No strong evidence exists that one scoring tool is superior to another; however, research shows that a standard approach to diagnosis and treatment using a scoring tool improves outcomes and is recommended by the American Academy of Pediatrics.

Finnegan NAS scoring system
The Finnegan NAS scoring system, developed in 1975, is the seminal and most common tool used to guide pharmacologic NAS treatment. Nurses evaluate infants every 1 to 4 hours, based on their age, and score them on the presence and severity of common withdrawal signs, including central nervous system, metabolic vasomotor, respiratory, and GI disturbances. Pharmacologic treatment is recommended for any infant who receives a score ≥ 8 on three consecutive evaluations.

The Finnegan tool is long and complex, but a simplified short form recently was developed as a more efficient option. The short form allows for rapid assessment with limited items for scoring; however, although simpler to use, limited evidence exists to validate its use.

Other assessment tools to guide pharmacologic NAS treatment include the 11-item Lipsitz Neonatal Drug-Withdrawal Scoring Sys­tem, the seven-item Neonatal Narcotic With­drawal Index, the seven-item Neonatal With­drawal Inventory, and the 19-item MOTHER NAS Scale. These tools haven’t been as widely adopted as the Finnegan tool.

Eat, Sleep, Console scoring tool
The new Eat, Sleep, Console (ESC) NAS assessment scoring method uses regular assessments of the infant’s ability to eat, sleep, and be consoled to determine the need for pharmacologic treatment. (See Eat, Sleep, Console.) Several studies of the ESC approach report decreased lengths of stay, less unnecessary exposure to pharmacologic treatments, and lowered care costs.

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14
Q

What are the symptoms of NAS?

A
Irritability
Seizures
Hyperactivity
High-pitched cry
Tremors
Exaggerated Moro reflex
Hypertonicity of muscles
Poor feeding
Diarrhea
Dehydration
Vomiting
Uncoordinated sucking
Gastric residuals
Diaphoresis
Fever
Mottled skin
Tachypnea
Nasal flaring
Nasal stuffiness
Disrupted sleep patterns
Excoriations
Temperature instability
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15
Q

What is Meconium Aspiration Syndrome?

What are the risk factors for Meconium Aspiration syndrome?

A
Meconium Aspiration Syndrome is a condition in a newborn that causes respiratory distress when meconium is aspirated into the lungs.
Risk Factors:
Thick/moderate fresh MEC stained fluid
Late preterm and term babies increased risk after 38 weeks, 
SGA/IUGR babies,
Post-term babies,
Post-dates, 
SGA, 
Placental insufficiency, 
Cord compression, 
Fetal distress 
Mother is obese  
Breech presentation.
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16
Q

What is Meconium aspiration syndrome?

What are the risks involved in MAS?

A

It is a common cause of neonatal respiratory pathology characterized by in utero or perinatal aspiration of meconium-stained amniotic fluid that causes respiratory distress. It is a respiratory difficulty found in term and preterm infants with clinical features include tachypnea, nasal flaring, and grunting and chest retractions.

Can cause the following:
Respiratory distress, cold stress, weight loss, jaundice, and infection.
Airway obstruction, interference with surfactant, chemical pneumonitis (bile acids, etc.), pulmonary HTN.

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17
Q

What is shoulder dystocia?
What are the risk Factors?
What are nursing responsibilities?

A

Defined as a delivery in which additional maneuvers are required to deliver the fetus after normal gentle downward traction has failed
Shoulder dystocia occurs when the fetal anterior shoulder impacts against the maternal symphysis
Shoulder dystocia occurs with equal frequency in primigravid and multigravid women

More common in infants born to women with diabetes
The single most common risk factor for shoulder dystocia is the use of a vacuum extractor or forceps during delivery
However, most cases occur in fetuses of normal birth weight and are unanticipated, limiting the clinical usefulness of risk-factor identification
Maternal
Abnormal pelvic anatomy
Gestational diabetes
Post-dates pregnancy
Previous shoulder dystocia
Short stature

Fetal
Suspected macrosomia
Labor related
Assisted vaginal delivery (forceps or vacuum)
Prolonged active phase of first-stage labor
Prolonged second-stage labor

Nursing responsibilities
H- Call for additional HELP/assistance
E – Evaluate for episotomy
L – Legs (McRobert’s Maneuver)
P – Pressure (suprapubic)
E – Enter the vagina
R – Remove the posterior arm
R – Roll the patient
To hands and knees
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18
Q

Laura is a 25 year old currently pregnant. Her history indicates that she has three living children. Her first child was born at 38 weeks gestation. Second pregnancy was a boy born at 34 weeks gestation. She has a three year old child who was born at 35 weeks gestation. She had an abortion at 6 weeks gestation and a 24 week stillborn.
What is her gravidity and parity using the GTPAL system?

A

G6 T1 P3 A1 L3
Six pregnancies total including current pregnancy.
One term infant
Three preterm including the 24 week stillborn
One Abortion at 6 weeks
three living children.

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19
Q

Elizabeth is pregnant at 8 weeks per her LMP. Her history includes a delivery at 25 weeks, a delivery at 40 weeks, a delivery at 37 weeks and a stillborn at 18 weeks.
What is her Gravida and Para?

A

G5 P3
Currently pregnant
Three pregnancies that delivered after 20 weeks
One pregnancy that ended prior to 20 weeks. (Does not count in the Para but counts in the Gravida)

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20
Q

What does the Postpartum nurse teach parents about safe sleep for baby?

A

Always place baby on his or her back to sleep, for naps and at night.
Share your room with baby.
Keep baby close to your bed, on a separate surface designed for infants.
Use a firm and flat sleep surface, such as a mattress in a safety-approved crib* , covered by a fitted sheet with no other bedding or soft items in the sleep area.
Breastfeeding reduces the risk of SIDS Babies who are breastfed or are fed expressed breastmilk are at lower risk for SIDS compared with babies who were never fed breastmilk. According to research, the longer you exclusively breastfeed your baby (meaning not supplementing with formula), the lower his or her risk of SIDS. If you bring baby into your bed for feeding, remove all soft items and bedding from the area. When finished, put baby back in a separate sleep area made for infants.* If you fall asleep while feeding baby in your bed, place him or her back in the separate sleep area as soon as you wake up.*A crib, bassinet, portable crib, or play yard that follows the safety standards of the Consumer Product Safety Commission (CPSC) is recommended. For information on crib safety, contact the CPSC at 1-800-638-2772 or http://www.cpsc.gov.

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21
Q

Why is Vitamin K given to babies after birth?

How is Vitamin K administered and when is it given?

A

Vitamin K actually comes from the German word “koagulation,” the process by which blood clots that we know of as coagulation.

According to the CDC, all babies are born with a deficiency in vitamin K, and they cannot easily make more. Vitamin K does not cross the placenta and only small amounts are in breastmilk or formula; about 90% of the vitamin K adults consume comes from leafy green vegetables. There is no dietary option that offers sufficient vitamin K for newborns.

Your newborn is at risk of internal bleeding because of vitamin K deficiency, which can cause motor skill delays and even death. Giving the vitamin K shot at birth ensures that all babies are protected. The CDC reports that “newborns who do not get a vitamin K shot are 81 times more likely to develop severe bleeding than those who get the shot.”

Vitamin K is given 1-2 hours after birth by IM injection.

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22
Q

Under what circumstances would a Pregnant woman receive Rhogam?

A

The problem comes if the mom is Rh- and the baby is Rh+. And it most likely won’t be a problem in a first pregnancy. But it can become a problem in later pregnancies.

If mom’s Rh- blood comes into contact with the baby’s Rh+, the mom’s body automatically makes antibodies against the Rh positive factor. The Mom’s body senses something foreign and is trying to protect itself by making these antibodies. When our bodies make antibodies, their purpose is to attack and destroy. Usually this is helpful when our bodies do this for things like colds and flu’s but in this case it can have adverse effects on further pregnancies.

The mom and baby don’t share a blood system during pregnancy, but sometimes the mom’s blood and baby’s blood could mix. This could happen during certain invasive medical tests, during delivery, during a miscarriage or an abortion.

If a mom’s Rh- blood mixes with a baby’s Rh+ blood during any of those situations, the mom’s body will create Rh antibodies. Then in future pregnancies, if Mom has another Rh+ baby, the antibodies in the mom’s body will cross the placenta and attack the new baby’s Rh+ blood. The baby’s blood won’t have enough oxygen, and the developing baby will suffer serious illness or even death.

So how does the RhIg (Rhogam) shot solve the problem?
First, your doctor will do a blood test to see if you are Rh positive or negative and if you have developed Rh antibodies. Then, if you are Rh- and have not developed antibodies, your doctor will give you a shot of RhIg (Rhogam). This Rhogam shot prevents the mom from making antibodies against the Rh factor, so the mom’s body won’t attack the blood of any future babies.

If you have a miscarriage or abortion, make sure to ask your medical provider whether you are Rh positive or negative and if you need the Rhogam shot.

If you are Rh- and your body has already made antibodies against the Rh factor, a Rhogam shot will not work. In that case, your doctor will closely monitor any future pregnancies and keep a close eye on your baby’s development.

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23
Q

What are the major risk factors for Postpartum Hemorrhage?

A
Prolonged labor
Augmented Labor
Rapid, Precipitous labor
History of a Postpartum Hemorrhage in a previous delivery
Episiotomy or perineal tear
Preeclampsia
Placenta Previa
Placental abruption
Uterine over distention
Multiple pregnancy
Polyhydramnios
Grand Multiparity >5 pregnancies
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24
Q

What are the 4 T’s of Postpartum Hemorrhage?

A
Tone
Tissue
Thrombin
Trauma
The leading cause of Postpartum hemorrhage is Uterine Atony!
25
Q

As the nurse caring for a patient 1-2 hours Postpartum, you notice a boggy uterus, increased bleeding with large clots.
What is your priority after you call for help?

A

Vigorous Uterine Massage.
Insure that the Patient has an empty bladder as a full bladder can displace the uterus and prevent it from becoming firm.
Administer medications to increase uterine tone per the provider. Oxytocin, Misoprostol, Methergine, Hemabate as ordered.
Vital signs
Administer IV fluids as ordered for volume replacement..
Administer Blood products as ordered.
Support the patient and the family and keep them informed.
Administer Oxygen if needed.

26
Q

What is TORCH and what is it’s significance in Pregnancy

A

What is the TORCH test
TORCH is an acronym commonly used in the evaluation of stillbirth, IUGR or neonatal failure to thrive
T = Toxoplasmosis
O = Other (includes HIV, Syphilis etc)
R = Rubella
C = Cytomegalovirus
H = Herpes (simplex and varicella)
Typically serum IgG and IgM for these infections repeated after 3w
Best regarded as a screening test only
In Summary Rubella Cytomegalovirus Herpes simplex Varicella Zika Virus
90% risk of fetal damage in the 1st trimester
Cytomegalovirus
A common cause of congenital deafness & mental retardation but there is little we can do about it
Herpes simplex
Common but most genital Herpes in pregnancy is secondary and there is only a 1-3% risk of vertical infection
Varicella
The greatest risk is to the mother (10% severe infection) whilst the overall risk of FVS is 2%
Zika Virus
Risk of microcephaly UNKOWN

27
Q

What are the risks associated with Rubella during pregnancy?

Is it recommended that women get vaccinated for Rubella during pregnancy?

A
Rubella in a Pregnant Woman
Before 12 weeks pregnancy up to 90% risk of:
Miscarriage
Cardiac anomalies
Nerve deafness
Cataracts or retinopathy
Mental retardation
Often multiple defects
Fetus somewhat safer in the 2nd trimester but…
Sensineural deafness is the most common problem
In the second half of pregnancy
Hepatosplenomegaly
Failure to thrive, osteitis
Diabetes, hypothyroidism and GH deficiency
Progressive pan encephalitis
= The Congenital Rubella Syndrome

Test all women who are pregnant (or plan to conceive) for immunity
Avoid infection if pregnant
Spread by nasopharyngeal droplets +/- 7 days from the time of rash
Retest any pregnant woman who is exposed to the virus
Immunize women postpartum if low titer or non immune
Immunization of a pregnant woman with the live virus is not recommended but is also not associated with teratogenesis

28
Q

What is SIDS?

What are the risk Factors for SIDS?

A

Sudden unexplained infant death (SUID) is the sudden and unexpected
death of an infant due to natural or unnatural causes. Sudden infant death
syndrome (SIDS) is one of several causes of SUID. However, SIDS, unlike the
other SUID causes, is a diagnosis of exclusion. Although most conditions
or diseases usually are diagnosed by the presence of specific symptoms,
SIDS is a diagnosis that should be given only after all other possible causes
of sudden, unexplained death have been ruled out through a careful case
investigation, which includes a thorough examination of the death scene, a
complete autopsy, and a review of the infant’s medical history (Willinger, James
In the United States, SIDS is the most common cause of death in infants aged one month to
one year and the third leading cause of infant mortality, after congenital anomalies and short
gestation/low birth weight. SIDS currently accounts for about 2,300 deaths per year
Modifiable risk factors for SIDS include
• Stomach and side sleeping positions.
• Overheating.
• Soft sleep surfaces.
• Loose bedding.
• Inappropriate sleep surfaces (such as a sofa or water bed).
• Sharing the same sleep surface (such as a bed) with an individual other than a
parent or sharing the same sleep surface with an individual who is overly tired or
under the influence of alcohol or drugs.
• Maternal and secondhand smoking.

29
Q

What is an APGAR score?

How is it determined?

A

What is APGAR? an easy and quick assessment tool used to assess the status of a newborn baby after birth.

APGAR is a mnemonic that stands for:

Appearance (skin color)

Pulse (heart rate)

Grimace (reflex irritability)

Activity (muscle tone)

Respiration (effort)

*APGAR scoring is performed at 1 minute and 5 minutes after birth and may be reassessed at 10 minutes (5 minutes later) after birth, if the score is 6 or less.

Each category is scored 0-2 and added up for a score 0-10.
Appearance:

0: pallor or blue all over
1: body pink but extremities blue (hands and feet)…acrocyanosis
2: Body and extremities all pink
Pulse:

0: absent
1: <100 bpm
2: >100 bpm
Grimace:

0: not response to stimulation
1: grimace to stimulation (no cry)
2: cry and active movement to stimulation
Activity:

0: none, flaccid
1: some flexion of arms and legs,
2: arms and leg flexed
Respiratory:

0: absent
1: weak cry, irregular
2: strong cry
Interventions based on the APGAR Score:

Score 7-10: no interventions, baby doing good just needs routine post-delivery care
Score 4-6: some resuscitation assistance required. Oxygen, suction…. stimulate the baby, rub baby’s back
Score 0-3: need full resuscitation

30
Q

You’re assessing the 1 minute APGAR on a newborn. You note the heart rate to be 140 bpm. The baby is crying loudly and vigorously when dried and stimulated. The body appears pink. The feet and hands have a blue color. Arms and legs are moving. The Newborn’s respirations are 60. What is the APGAR score at 1 Minute?

A
Appearance  1
Pulse              2
Grimace          2
Activity            2
Respirations    2
 Total =9
31
Q

How do we determine the difference between Morning Sickness and Hyperemesis Gravidarum?

A

Hyperemesis gravidarum is a complication of pregnancy caused by high serum levels of β-hCG that is characterized by severe nausea and vomiting such that weight loss and dehydration occurs. Signs and symptoms may include vomiting several times a day and feeling faint. It is more severe than morning sickness. Symptoms usually get better after the 20th week of pregnancy, but may last the entire pregnancy. The condition is also seen in patients with a hydatidiform mole and in multiple gestations. Treatment is with thiamine supplementation, antiemetics, and steroids for severe, intractable hyperemesis.

32
Q

What are the causes of Respiratory Distress Syndrome in Newborns?
What are the signs of Respiratory Distress Syndrome?

A

The term respiratory distress syndrome is most often applied to a severe lung disorder in the neonate which is primarily related to lung immaturity.
It is responsible for more infants death and neurological complications.
Respiratory distress syndrome is a syndrome of premature neonates that is characterized by progressive and sometimes fatal respiratory failure resulting from atelectasis and immaturity of lungs. It was formerly known as Hyaline membrane disease.
Respiratory Distress in the Newborn – Clinical Presentation Cyanosis Grunting Retractions Tachypnea Nasal flaring
Extreme: Apnea, Shock, Bradycardia

33
Q

What is the treatment for Respiratory Distress syndrome?

A

Neonatal RDS occurs in infants whose lungs have not yet fully developed
• The disease is mainly caused by a lack of a slippery substance in the lungs called surfactant
• Neonatal RDS can also be due to genetic problems with lung development or
-Infection
Treatment of RDS
Treatment • Approach Considerations
– Corticosteroids
– Surfactant Replacement Therapy
– Oxygenation and CPAP – Vapotherm
– Assisted Ventilation
– High-Frequency Ventilation
– Nitric Oxide
– Supportive Therapy
– Parent and Family Support
-Placement of an Endotracheal tube (breathing tube, also called an ET) into windpipe
• Mechanical breathing machine (to do the work of breathing for the baby)
• supplemental oxygen (extra amounts of oxygen)
• continuous positive airway pressure (CPAP) - a mechanical breathing machine that pushes a continuous flow of air or oxygen to the airways to help keep tiny air passages in the lungs open
• Extracorporeal membrane oxygenation (ECMO)

34
Q

What information do we provide to parents regarding Car seat safety?

A

In 2019, 608 child passengers age 12 and younger died in motor vehicle crashes,1 and more than 91,000 were injured.2 Of the children 12 and younger who died in a crash (for whom restraint use was known), 38% were not buckled up. Parents and caregivers can make a lifesaving difference by checking whether their children are properly buckled on every trip.
Child restraint laws require children riding in vehicles to use approved restraint devices such as car seats, booster seats, or seat belts that are appropriate for their age, height, and weight.
Rear-facing car seat: Birth until age 2–4.
Infants and toddlers should be buckled in a rear-facing car seat with a harness, in the back seat, until they reach the maximum weight or height limit of their car seat. This offers the best possible protection. Check the car seat manual and labels on the car seat for weight and height limits. Never place a rear-facing car seat in the front seat. Front passenger air bags can injure or kill young children in a crash.
Buckle children in the middle of the back seat.
Properly buckle children in the middle seating position of the back seat when possible, because it is the safest position in the vehicle.18
If the middle seating position contains only a lap belt, older children using booster seats or seat belts should sit in a different seating position in the back seat where they can use both a lap and shoulder belt.
Most rear-facing and forward-facing car seats can be safely installed in the middle seating position using the lap belt only, so younger children using these types of seats can be safely positioned in the middle.
Remove bulky coats or snowsuits before strapping baby in the car seat
Harness straps should be at or below shoulder level
Adjust car seat angle so that baby’s head does not flop forward
Place blanket over the straps.
Perform a pinch test. Tighten the harness straps snugly. To make sure it’s tight enough, try this classic car seat safety trick: If you can pinch the harness strap at your child’s shoulders, the straps are too loose.

35
Q

What are Mongolian Spots?

A

The Mongolian spot is a development condition exclusively involving the skin. The blue color is caused by melanocytes.
Melanocytes are melanin- containing cells, that are deep under the skin.
Mostly seen on Asians, African Americans , Hispanics, and Native Americans.
There’s really nothing you can do about Mongolian spots you just have to wait until they disappear usually by at the age of 2.
Fewer than 5% of children with Mongolian spots have visible marks by the time they’re adults.

36
Q

What are the indications for use of Magnesium Sulfate in Pregnancy?

A

Magnesium sulfate, or mag for short, is used in pregnancy to prevent seizures due to worsening preeclampsia, to slow preterm labor, and to prevent injuries to a preterm baby’s brain.

37
Q

What are the signs and symptoms of Magnesium Sulfate Toxicity?
What is the Antidote for Magnesium Sulfate Toxicity?

A

One of the initial magnesium sulfate toxicity symptoms is a decrease in the reflexes of the person. This is due to the blocking of the transmission in the neuromuscular receptors. The blood pressure will also decrease at this point. Other symptoms that may be present are nausea, vomiting and flushing of the skin. Further increase in magnesium level may lead to facial paresthesia and muscle weakness.

When the patient’s serum magnesium level reaches 10mEq/L, it may cause a paralysis of the respiratory system and bradycardia or a decrease in heart rate. There are instances where patient with this serum magnesium level experiences complete heart block and myocardial infarction.
Antidote for Mag Toxicity= Calcium Gluconate
Monitoring a patient on MgSO4:

Clinical parameters
Presence of patellar reflex
Urine output (at least 100ml in last 4 hours)
Respiratory rate (>12/min)
If any of the 3 parameters is not fulfilled, the MgSO4 dose is withheld/delayed
10ml of 10% Calcium gluconate used as antidote for toxicity

38
Q

What are some advantages of Breastfeeding for Baby and Mom?

A

The breastfeeding benefits of colostrum
For the first 2-4 days of your baby’s life, your breasts will discharge colostrum, a yellowish liquid rich in proteins. These important proteins are vital to the growth of a healthy immune system. The protein is simply digested and consumed by the body, particularly by the quickly growing brain. Colostrum gives factors that better maturation of the gut and good metabolism. Colostrum is the most excellent and well-designed food for your baby in the first few days of life. Breastfeeding benefits for Mothers Health Breastfeeding gives health advantages to mothers ahead of emotional pleasure. Breastfeeding benefits research show that women who have breastfed activity decreased rates of breast cancer and ovarian cancer next in life. Some researchers have discovered that breastfeeding may decrease the risk of forming type 2 diabetes, rheumatoid arthritis, and cardiovascular disease, including high blood pressure and high cholesterol. Exclusive breastfeeding benefits by month prevents the return of the mother’s menstrual period. There are fewer tours to the doctor, and less cash is wasted on medications Breastfeeding strengthens mother-baby bonding Hormones discharged during breastfeeding generate emotions of warmness and peace in the mother. Mothers who breastfeed manage to lose weight and gain their pre-pregnancy body more efficiently than mothers who bottle feed. Mothers who breastfeed recover from childbirth faster and simply. The hormone oxytocin, released while breastfeeding, acts to restore the uterus to its normal size more speedily and can decrease postpartum bleeding. Breastfeeding benefits for the baby Excellent nutrition There is an improved immunity to infections, and therefore fewer occurrences of sickness and hospitalization Baby is less likely to produce allergies Baby experiences fewer abdomen upsets and costiveness Breastfed newborns tend to have fewer cavities Breastfeeding promotes the proper development of the baby’s jaw and teeth. Breastfed infants tend to have higher IQs due to proper brain development early in life Babies benefit emotionally because they are held more Breastfeeding improves mother-baby bonding In the long term, breastfed babies have a lowered risk of starvation, obesity, and heart attack compared to formula-fed babies. Reduced risk of allergies and lactose intolerance. Breast milk is hygienic Baby has few diaper rashes and less thrush

39
Q

What are some risks for Infants of Diabetic mothers?

A

If diabetes is not well controlled during pregnancy, the baby is exposed to high blood sugar levels. This can affect the baby and mother during pregnancy, at the time of birth, and after birth.

Infants of diabetic mothers (IDM) are often larger than other babies, especially if diabetes is not well-controlled. This may make vaginal birth harder and may increase the risk for nerve injuries and other trauma during birth. Also, cesarean births are more likely.

An IDM is more likely to have periods of low blood sugar (hypoglycemia) shortly after birth, and during first few days of life. This is because the baby has been used to getting more sugar than needed from the mother. They have a higher insulin level than needed after birth. Insulin lowers the blood sugar. It can take days for babies’ insulin levels to adjust after birth.

IDMs are more likely to have:
Hypoglycemia
Breathing difficulty due to less mature lungs
High red blood cell count (polycythemia)
High bilirubin level (newborn jaundice)
Thickening of the heart muscle between the large chambers (ventricles)
If diabetes is not well-controlled, chances of miscarriage or stillborn child are higher.

An IDM has a higher risk of birth defects if the mother has pre-existing diabetes that is not well controlled from the very beginning.

40
Q

What are Neural Tube defects?
What supplements can be taken to Prevent Neural Tube defects?
What screening blood test detects pregnancies at increased risk for NTD?

A

Neural tube defects (also called NTDs) are birth defects of the brain and spinal cord. Birth defects are health conditions that are present at birth. They change the shape or function of one or more parts of the body. Birth defects can cause problems in overall health, how the body develops or how the body works.

A baby’s neural tube normally starts out as a tiny, flat ribbon that turns into a tube by the end of the first month of pregnancy. If the tube doesn’t close completely, an NTD can happen. NTDs can cause serious problems for babies, including death.

NTDs happen in about 3,000 pregnancies each year in the United States. Hispanic women are more likely than non-Hispanic women to have a baby with an NTD.

The two most common NTDs are spina bifida and anencephaly. Spina bifida affects about 1,500 babies a year in the United States. If your baby has spina bifida, the tiny bones of the spine don’t close completely, and part of the spinal cord pokes through the spine. Children with spina bifida may have paralyzed legs (not able to move) and problems controlling their bladder and bowel (going to the bathroom). Milder forms of spina bifida may cause fewer problems for children.

Anencephaly is one of the most severe NTDs. It affects about 1,000 babies each year in the United States. Anencephaly is caused when the upper part of the neural tube that forms the brain doesn’t close completely. Babies with this condition are missing major parts of the brain, skull and scalp. They do not survive long after birth, usually for just a few hours. Girls are 3 times more likely than boys to have anencephaly.

Taking folic acid before and during early pregnancy can help prevent NTDs in your baby. Folic acid is a B vitamin that every cell in your body needs for normal growth and development.
Second Trimester Screening (The Quad Screen)

The obvious difference between the quad screen and the 1st trimester screen is the timing. Another key difference is that along with screening for Down syndrome and trisomy 18 it will also screen for neural tube defects (like spina bifida).

The quad screen is just a blood draw. This test will be looking at four proteins that are made by the pregnancy (AFP, hCG, UE3, DIA). The protein levels will be measured and used to calculate the risk assessment (factors such as maternal age, ethnicity, ect. are again contributory).

The detection rate for Down syndrome is approximately 80% with a false positive rate of 4-5%. The detection rate for trisomy 18 is 80% with a false positive rate of <0.5%. Lastly, the AFP protein is used to screen for open neural tube defects with a detection rate of 80%.

What causes NTDs?
We’re not exactly sure what causes NTDs. Experts think some may be caused by genetics. This means an NTD can be passed from parents to children through genes. Genes are part of your body’s cells that stores instructions for the way your body grows, looks and works.

NTDs also may be caused by things in your environment, which includes where you live, where you work, the kinds of foods you eat or have access to and how you like to spend your time. Some things in your environment can be harmful to a pregnancy, like certain drugs, cigarette smoke, air pollution and lead.

Are you at risk for having a baby with an NTD?
Any woman can have a baby with an NTD. But there are things that may make you more likely than other women to have a baby with an NTD. These are called risk factors.

Your risk for having a baby with an NTD is higher if:

You’ve had a baby with an NTD. If you’ve had a baby with an NTD, there’s a 2- to 3-percent chance of having a baby with an NTD in another pregnancy. A genetic counselor can help you understand your risk for having a baby with an NTD. This is someone who is trained to help you understand medical conditions that run in families, and how they can affect your health and your baby’s health.
You or your partner has an NTD, your partner has a child with an NTD or someone in either of your families has an NTD. This means you have a family history of NTDs. Use the March of Dimes Family Health History Form to keep track of health conditions, including NTDs, that may run in your family. Fill it out and share it with your health care provider or genetic counselor.
Other risk factors for NTDs include:

Taking certain anti-seizure medicines. Talk to your health care provider before you get pregnant about how the medicine may affect your pregnancy.
Obesity. Some studies show that being obese increases your risk for having a baby with an NTD. If you’re obese, you have an excess amount of body fat and your body mass index (BMI) is 30 or higher. To find out your BMI, go to cdc.gov/bmi. Talk to your provider about getting to a healthy weight before pregnancy.
Diabetes. If your diabetes is uncontrolled, you may be at increased risk for having a baby with an NTD. Eating healthy foods and being active every day can help you keep your diabetes under control.
Using opioids in the first 2 months of pregnancy. Opioids are highly addictive drugs. Your provider may prescribe an opioid to you as a painkiller if you’ve been injured or had surgery. Common prescription opioids include codeine, hydrocodone and oxycodone. These often are sold and used illegally. If you take any opioid during pregnancy, it can cause serious problems for your baby, like premature birth and drug withdrawal called neonatal abstinence syndrome (also called NAS). If you’re pregnant and taking any drug or medicine that may be an opioid, tell your health care provider right away.
A high body temperature early in pregnancy. This may be caused by a fever or by spending a lot of time in a hot tub or sauna. If you’re pregnant, stay out of hot tubs and saunas. If you do use them, limit the time to less than 10 minutes.
How can you help prevent NTDs in your baby?
Taking folic acid before and during early pregnancy can help prevent NTDs in your baby. NTDs happen in the first month of pregnancy, before you may know you’re pregnant. This is why it’s important to have enough folic acid in your body before you get pregnant.

41
Q

What are the risk factors for Preterm labor?

A

Preterm birth is when a baby is born too early, before 37 weeks of pregnancy have been completed. In 2020, preterm birth affected 1 of every 10 infants born in the United States. The preterm birth rate declined 1% in 2020, from 10.2% in 2019 to 10.1% in 2020. However, racial and ethnic differences in preterm birth rates remain. In 2020, the rate of preterm birth among African-American women (14.4%) was about 50 percent higher than the rate of preterm birth among white or Hispanic women (9.1% and 9.8% respectively).

A developing baby goes through important growth throughout pregnancy including in the final months and weeks. For example, the brain, lungs, and liver need the final weeks of pregnancy to fully develop. Read Your Baby Grows Throughout Your Entire Pregnancy Babies born too early (especially before 32 weeks) have higher rates of death and disability. In 2019 preterm birth and low birth weight accounted for about 17% of infant deaths (deaths before 1 year of age). Babies who survive may have

Breathing problems
Feeding difficulties
Cerebral palsy
Developmental delay
Vision problems
Hearing problems

Many times we do not know what causes a woman to deliver early, but several known factors may increase the likelihood that a woman could deliver early. This list below gives some examples of these factors by medical and pregnancy conditions, behavioral factors, and social, personal, and economic characteristics.

Social, Personal, and Economic Characteristics
Teens and women over age 35
Black race
Women with low income

Pregnancy and Medical Conditions
Prior preterm birth
Infection
Carrying more than 1 baby (twins, triplets, or more)

Behavioral
Tobacco use
Substance use
Stress

42
Q

How is Preterm labor managed?

A

Management of Preterm Labor
Preventing preterm birth is a major challenge for modern obstetric medicine.6 Spontaneous preterm labor often occurs without warning or previous indication, but the goal of preterm labor management is to prolong pregnancy as safely as possible to permit fetal development and maturation.3

Corticosteroids: One of the most useful tools in the obstetric armamentarium for reducing morbidity and mortality related to preterm birth, corticosteroids are the best-documented beneficial agents for use in preterm labor.1 The use of corticosteroids is indicated to prevent preterm birth in women between 26 and 34 weeks’ gestation who are at risk for preterm birth owing to preterm labor, PPROM, or severe preeclampsia or other medical conditions that necessitate preterm delivery.2

In combination with delivery in a facility with a level III NICU, corticosteroids consistently improve outcomes of preterm birth.6 Corticosteroids administered as late as 12 to 24 hours before preterm birth reduce the incidence of neonatal complications after preterm labor, including respiratory distress syndrome (RDS), intraventricular hemorrhage, and necrotizing enterocolitis.1-3,8 Administered prior to 35 weeks’ gestation, corticosteroids activate the hypothalamic-pituitary-adrenal axis to enhance functional maturity in the fetus, specifically lung maturity.9

The American College of Obstetricians and Gynecologists (ACOG) recommends either betamethasone or dexamethasone to promote fetal lung maturity.1,2 Intramuscular administration of betamethasone 12 mg every 24 hours for two doses or dexamethasone 6 mg every 12 hours for four doses is indicated for women at risk for preterm birth between 26 and 34 weeks’ gestation.1,3 Repeat doses of corticosteroids are not beneficial for improving outcomes following preterm birth.3

Antibiotics: Preterm labor is often associated with infections and inflammation, and subclinical infection is associated with PPROM.1,10 However, most studies have not found that prophylactic antibiotic treatment confers a substantial benefit in preventing preterm labor or birth. Additionally, antibiotics do not reduce the occurrence of neonatal RDS or sepsis.1
Antibiotics do not affect preterm labor in patients with intact membranes.6 Among women with PPROM, antibiotics may reduce the risk of preterm birth within 48 hours of administration, but little evidence of benefit exists for other outcomes. Specifically, antibiotic administration may delay delivery after PPROM, which provides time for corticosteroid administration.2,10 Amoxicillin-clavulanate should be avoided in women who are at risk for preterm birth because of the increased chance of neonatal necrotizing enterocolitis.10 The ACOG does not support the use of antibiotics for pregnancy prolongation in women with intact membranes.11

Bacterial vaginosis, an overgrowth of anaerobic bacteria, is associated with an increased risk of preterm birth; treatment of the vaginosis reduces the risk.2 Symptomatic bacterial vaginosis is often treated with oral clindamycin 300 mg twice daily for 7 days, metronidazole 500 mg twice daily for 7 days, or metronidazole 250 mg three times daily for 7 days.6

Antibiotics are appropriate during preterm labor as prophylaxis against group B streptococcus (GBS) sepsis.1 Current guidelines consider IV administration of one dose of ampicillin 2 g followed by ampicillin 1 g every 6 hours for 48 hours to be adequate GBS prophylaxis in women who have tested positive for GBS, had a previous infant with GBS disease, had GBS bacteriuria during the current pregnancy, or whose GBS status is unknown.12

Tocolytics: Tocolytic drugs inhibit uterine contractions.1,2 Four main classes of tocolytics, with varying degrees of safety and effectiveness, are used: beta-adrenergic agonists, magnesium sulfate, calcium channel blockers, and nonsteroidal anti-inflammatory drugs (NSAIDs).1,3 General contraindications to tocolysis include severe preeclampsia, maternal instability, placental abruption, intrauterine infection, lethal congenital or chromosomal abnormalities, advanced cervical dilation, fetal compromise or distress, and placental insufficiency.1,6 The choice of tocolytic should be based on maternal condition, potential adverse effects, gestational age, andcost.1,13 Once treatment is initiated, the patient’s response to tocolysis, including adverse effects, should be continuously monitored.13 Prolonged use of tocolytic agents is not recommended.1

Despite widespread use, tocolysis lacks robust evidence for the prevention of preterm birth. Most tocolytic drugs prolong gestation for 2 to 7 days.1,2,6,14 This delay in preterm birth allows sufficient time for the administration of corticosteroids and maternal transfer to a hospital with an appropriate NICU, if such interventions are warranted.1,4,6,13 Tocolytic agents potentiate the effects of corticosteroids, and the concomitant use of tocolytics and corticosteroids is widespread.6,9 Tocolytic therapy offers no benefit in neonatal RDS or mortality.14 TABLE 1 provides a summary of common tocolytics and their doses, administration, and adverse effects.1,2,6,15
Magnesium sulfate, one of the most common obstetric drugs in the U.S., is used primarily for seizure prophylaxis in preeclampsia.16 Despite its lack of proven efficacy, magnesium sulfate is also the most commonly used tocolytic agent in the U.S.4,13 Magnesium sulfate can cause maternal lethargy, drowsiness, double vision, nausea, and vomiting.1 More serious maternal adverse effects include pulmonary edema, hypotension, muscle paralysis, tetany, cardiac arrest, and respiratory depression.3 Magnesium sulfate can cause fetal toxicity at high doses.3,16

While most tocolytics do not show a benefit in neonatal outcomes, magnesium sulfate has gained attention for its use in preventing cerebral palsy. Several studies have shown that, among women at risk for preterm birth, low-dose administration of magnesium sulfate reduced the risk of cerebral palsy among surviving neonates.16-18 The neuroprotective effect of magnesium sulfate results from a reduction in vascular instability and prevention of hypoxic and amino acid damage.17 The ACOG supports the administration of magnesium sulfate prior to anticipated preterm birth to reduce the risk of cerebral palsy.15 The recommended dose of magnesium sulfate is an IV bolus of 4 g to 6 g followed by 2 g to 3 g per hour.1

Terbutaline, a beta-adrenergic agonist, is a potent cardiovascular (CV) stimulant that is associated with an increased risk of pulmonary edema and maternal and fetal CV abnormalities. A bronchodilator, it is approved to prevent and treat bronchospasms associated with asthma, bronchitis, and emphysema.19 The ACOG does not support the use of terbutaline to prevent preterm labor but states that, when it is used as a tocolytic, terbutaline should be administered as a subcutaneous 0.25-mg dose every 20 minutes to 3 hours.1 As a continuous infusion for tocolysis, terbutaline is initiated at a rate of 2.5 mcg/min to 10 mcg/min, and the rate can be increased gradually every 10 to 20 minutes to a maximum of 17.5 mcg/min to 30 mcg/min.20

Terbutaline is not FDA approved as a tocolytic agent. In February 2011, the FDA released a safety announcement advising that terbutaline be used for tocolysis no longer than 48 to 72 hours owing to the risk of serious maternal CV problems that could lead to death. The FDA required that a boxed warning and contraindication be added to the labeling of both injectable and oral terbutaline. The FDA acknowledgedthat clinicians may continue to use terbutaline on an off-label basis to prevent preterm birth in urgent situations, but warned that the drug should never be used in the outpatient setting. The FDA’s statement was based on postmarketing surveillance data that included 16 reports of maternal death and 12 cases of maternal CV events following terbutaline administration since the drug’s approval in 1976.19

Nifedipine, a calcium channel blocker, reduces the risk of preterm delivery within 7 days of treatment prior to 34 weeks’ gestation. Compared with beta-agonists, nifedipine also lowers the risk of RDS, necrotizing enterocolitis, intraventricular hemorrhage, neonatal jaundice, and NICU admission.21 Nifedipine poses few maternal or fetal risks, but its use with magnesium sulfate has led to CV collapse in some pregnant women. For tocolysis, the ACOG recommends a loading dose of nifedipine 30 mg administered orally followed by 10 mg to 20 mg every 4 to 6 hours.1

NSAIDs act as tocolytic agents by blocking the inflammatory process that triggers labor.2 NSAIDs have few maternal adverse effects, but they are associated with oligohydramnios and premature closure of the ductus arteriosis in the fetus.1,2 Also, neonates of mothers treated with NSAIDs may experience intraventricular hemorrhage, necrotizing enterocolitis, and bronchopulmonary dysplasia.2 These adverse effects are more common when NSAIDs are used after 32 weeks’ gestation, and NSAIDs are generally reserved for tocolysis earlier in pregnancy.2,14

The most commonly used NSAID for tocolysis is indomethacin. According to practice guidelines, a loading dose of 50 mg rectally or 50 mg to 100 mg orally followed by 25 to 50 mg orally every 4 to 6 hours for 48 hours is used for tocolysis.1,6 Ketorolac, also used as a tocolytic, is administered intramuscularly as a 60-mg loading dose followed by 30 mg every 6 hours for 48 hours. As a tocolytic, sulindac is administered at a dose of 200 mg orally every 12 hours for 48 hours.1
Although several interventions for managing preterm labor and preventing preterm birth are available, most are not universally effective, offering benefits in only a specific group of women or presenting safety concerns for the mother or fetus.2 Large-scale research is needed to conclusively define the best choices for managing preterm labor and preventing preterm birth.3

43
Q

Provider orders a 4 gram Mag Sulfate bolus IV to be given over 20 minutes
Pharmacy sent—50 grams of Mag Sulfate in 1000 mL. Program the pump appropriately.

A

4gms/20 min * 50gms/1000ml * 60min/1hr

240 mL/hr

44
Q

Patient is ordered PCN G 5 million units IV over 30 minutes

Pharmacy sent a bag with 5 million units in 250 mL. How do you set the IV pump in mL/hr

A

500 ml/hr

45
Q

What is Transient Tachypnea of the Newborn?
What are the risk factors?
How is it treated?

A

Transient tachypnea of newborn (TTN) results from failure of the newborn to effectively clear the fetal lung fluid soon after birth. TTN represents the most common etiology of respiratory distress in term gestation newborns and sometimes requires admission to the neonatal intensive care unit. TTN can lead to maternal-infant separation, the need for respiratory support, extended unnecessary exposure to antibiotics and prolonged hospital stays.
Caused by ineffective absorption of fetal lung fluid
Fetal Lung Fluid Clearance to review normal physiology
Failure or delay in clearance of intra-alveolar fluid in patients with TTN is due to:
Lack of ENaC expression or activity
Lack of active labor and its associated hormonal changes
Ineffective lung distention and lack of alveolar air interface
Immaturity of ENaC
Especially relevant in the late preterm infant
Clinical presentation is reflection of decreased lung compliance associated with pulmonary edema and ineffective fetal lung fluid clearance
Tachypnea (most consistent finding)
Increased work of breathing
Mild degrees of hypoxia
Timing:
Onset: Present very early after birth
Duration: 2- 3 days
Management
Supportive
Resolves by 72 hours of age
Does not require antibiotics for treatment
However, TTN is a diagnosis of exclusion and it is hard to differentiate TTN from other etiologies such as infectious early on. As a result, many patients are placed on antibiotics that are discontinued after 48 hours if cultures are negative and the diagnosis of TTN is more evident.

46
Q

What Medications are used to treat Postpartum depression?

A

Treatment
While optimal treatment strategies are still being researched, effective management of PPD includes both nonpharmacologic and pharmacologic interventions. Women with mild-to-moderate symptoms of PPD should try nonpharmacologic treatments, such as psychosocial therapy. In addition to being effective, these mechanisms are desirable for women who want help but wish to avoid using medications while they are breast-feeding. Interpersonal therapy (IPT), a type of psychotherapy, focuses on a woman’s relationships and the role transitions that occur after the birth of a child. A study conducted in 2000 found that 12 weeks of IPT were more effective than no treatment at improving social adjustment and decreasing PPD symptoms. Cognitive-behavioral therapy helps a woman identify negative thoughts, beliefs, and behaviors that are inaccurate or dysfunctional and learn to replace them with more positive, realistic thought processes. Other nonpharmacologic treatments for PPD are group therapy, nondirective counseling, light therapy, partner-support counseling, and peer support groups.
Due to their effectiveness, low incidence of side effects, safety in the event of overdose, and once-daily dosage formulations, selective serotonin reuptake inhibitors (SSRIs) are considered first-line agents for the treatment of PPD.3,16,28 Although the side-effect profile and overdose severity of the tricyclic antidepressants (TCAs) are greater than those of the SSRIs, TCAs have been shown to improve the symptoms of PPD also.
A major factor to consider in deciding whether to treat PPD with antidepressants is whether the patient is breast-feeding. It is necessary to inform patients that all antidepressants are excreted into breast milk and that studies have found detectable drug concentrations in the plasma of some breast-fed infants.10,35-38 It is important to note, however, that these infants were found to tolerate exposure without harm and that instances of toxicity were extremely rare.

Pharmacologic intervention is warranted when a woman’s PPD symptoms become moderate-to-severe or when the woman does not respond to nonpharmacologic therapy alone
Screening all women for the signs and symptoms of depression in the postpartum period is the most effective way to identify and treat the disorder before it progresses. Providers should screen for PPD at a woman’s first postpartum follow-up visit and again at six weeks.6 The Edinburgh Postnatal Depression Scale (EPDS) is a 10-item questionnaire designed to identify PPD; with 100% sensitivity and 95% specificity in multiple studies, this tool is utilized by physicians worldwide.10,18,19 Data on the success of the EPDS are limited. Other screening tools are the Postpartum Depression Screening Scale and the Beck Depression Inventory.20

47
Q

What tool is used to screen for Hyperbilirubinemia?

A

Bhutani’s Nomogram
The serum bilirubin was obtained before discharge, and the zone in which the value fell predicted the likelihood of a subsequent bilirubin level exceeding the 95th percentile. Risk is determined based upon Bilirubin level and the newborn’s hours of age.

Neonatal jaundice affects up to 84% of term newborns1 and is the most common cause of hospital readmission in the neonatal period.2 Severe hyperbilirubinemia (total serum bilirubin [TSB] level of more than 20 mg per dL [342.1 μmol per L]) occurs in less than 2% of term infants and can lead to kernicterus (i.e., chronic bilirubin encephalopathy) and permanent neurodevelopmental delay.2 Therefore, it is important to systematically evaluate all infants for hyperbilirubinemia.

48
Q

What are the risk factors for Placental Abruption?

A

Etiology cause of placental abruption
preeclampsia or chronic hypertension (11% to 65%)
advanced parity / age
maternal smoking
thrombophilias
cocaine abuse
preterm ruptured membranes - chorioamnionitis
external maternal trauma
uterine myoma
prior abruption
Very high risk of recurrent abruption in a subsequent pregnancy (10%).

49
Q

What role does Prostaglandin E1 Play in Cyanotic Congenital Heart Defects Such as Tetralogy of Fallot?

A

PROSTAGLANDIN E1 (PGE1) Maintains patency of ductus arteriosus
Mechanism of action is not well delineated but causes vasodilation of smooth muscle in the ductus
Therapeutic effects
Increases either systemic or pulmonary blood flow
For lesions dependent on PDA for pulmonary blood flow, improves O2 saturation
For lesions dependent on PDA for systemic blood flow, improves blood pressure and systemic perfusion

50
Q

What is a TET Spell?

Perry pg. 1234

A

Cyanotic (Tet) spells Acute hypoxemic attacks represent a true emergency and initial treatment is crucial to long term outcome. Usually, the underlying diagnosis is tetralogy of Fallot. In a Tet spell, an acute increase in obstruction to pulmonary blood flow (either in heart or in pulmonary circulation) results in increase in right-to-left shunting through an intracardiac septal defect. Alternatively, if systemic perfusion is reduced, as with hypovolemia or the development of a tachyarrhythmia, right-to-left shunting will also increase and a cyanotic spell develop.
7. Clinical presentation and diagnosis They are characterized by:
Period of uncontrollable crying / panic,
Rapid and deep breathing (hyperpnoea),
Deepening of cyanosis,
Decreased intensity of heart murmur,
Limpness, convulsions and rarely, death.
common in the early morning, shortly after the patient awakens,.
Prolonged agitation and crying are also cited as precipitants,
Also, noxious stimuli, such as phlebotomy or a bee sting, or any circumstance which leads to enhanced catecholamine output can precipitate a spell in a susceptible child.
A decrease in systemic vascular resistance (SVR) during exercise, bathing, or fever potentiates a right-to-left shunt and precipitates hypoxemia
In such cases(tet spells), the absence of a heart murmur is a worrisome indicator that pulmonary blood flow is severely compromised.

51
Q

What is the cause of Late Decelerations in the Fetal Heart Rate?

A

Late Deceleration
The fetal heart rate response to uterine contractions can be an index of either uterine perfusion or placental function.
A late deceleration is a smooth, gradual, symmetrical decrease in fetal heart rate beginning at or after the contraction peak and returning to baseline only after the contraction has ended.
A gradual decrease is defined as 30 seconds or more from the onset of the deceleration to the nadir.
It is indicative of:
Uteroplacental insufficiency.

52
Q

What constitutes a category III FHR tracing?

A

Three-Tier Fetal Heart Rate Interpretation System [41]

Category I : Normal.

The fetal heart rate tracing shows ALL of the following:

Baseline FHR 110-160 BPM, moderate FHR variability, accelerations may be present or absent, no late or variable decelerations, may have early decelerations.

Strongly predictive of normal acid-base status at the time of observation. Routine care.

Category II : Indeterminate.

The fetal heart rate tracing shows ANY of the following:

Tachycardia, bradycardia without absent variability, minimal variability, absent variability without recurrent decelerations, marked variability, absence of accelerations after stimulation, recurrent variable decelerations with minimal or moderate variability, prolonged deceleration > 2minute but less than 10 minutes, recurrent late decelerations with moderate variability, variable decelerations with other characteristics such as slow return to baseline, and “overshoot”.

Not predictive of abnormal fetal acid-base status, but requires continued surveillance and reevaluation.

Category III: Abnormal.

The fetal heart rate tracing shows EITHER of the following:

Sinusoidal pattern OR absent variability with recurrent late decelerations, recurrent variable decelerations, or bradycardia.

Predictive of abnormal fetal-acid base status at the time of observation. Depending on the clinical situation, efforts to expeditiously resolve the underlying cause of the abnormal fetal heart rate pattern should be made.

53
Q

What are the symptoms of Heart Failure in Neonates?

A
Symptoms
Incessant cry
Feeding difficulty
Excessive sweating
Frequent chest infection
Failure to thrive
Respiratory distress
Wheeze, rales
Hepatomegaly
Signs of shock
cardiomegaly
 CLINICAL MANIFESTATIONS…
Tachycardia
Venous congestion
Right-sided
Hepatomegaly
Ascites
Pleural effusion
Edema
Jugular venous distension
Left-sided
Tachypnea
Retractions
Nasal flaring or grunting
Rales
Pulmonary edema
54
Q

What constitutes a reactive Nonstress test?

What does a reactive NST indicate?

A

Reactivity of an NST.
A Nonstress test is considered reactive when two or more fetal heart
rate accelerations peak at least
15 beats per minute above the baseline and last 15 seconds within a 20 minute period.
What do the NST results mean? A reactive non-stress result indicates that blood flow (and oxygen) to the fetus is adequate.

55
Q

What is the cause of Variable Decelerations in the Fetal heart rate?

A

Variable deceleration Cord Compression
Early deceleration Head compression (Normal)
Accelerations in FHR OK
Late Decelerations Placental Insufficiency

56
Q

What is DIC?

A

Disseminated Intravascular Coagulation (DIC) is a syndrome characterized by altered activation and control of blood coagulation pathways causing accumulation of fibrin in the micro-vasculature, consumption of platelets and coagulation factors, which may result in organ failure and abnormal bleeding. Pregnancy is known to be a hypercoagulable state and complications such as placental abruption, pre-eclampsia and HELLP syndrome are some of the conditions associated to the onset of DIC.

57
Q

What is Neonatal sepsis?
What are the risk factors?
What are the symptoms?

A

What is Neonatal Sepsis?
Neonatal Sepsis refers to an infection of the newborn, specifically bacterial blood stream infections (BSI). Some of the systematic infections commonly seen in Neonatal Sepsis or Sepsis in Infants are meningitis, septicemia, pneumonia, arthritis, pyelonephritis, osteomyelitis, and gastroenteritis. All of these conditions have one symptom in common and that is fever in the newborn baby. Neonatal Sepsis or Sepsis in Infants is also known by the name of sepsis neonatorum. Hemodynamic compromise caused due to Neonatal Sepsis or Sepsis in Infants is difficult to measure and taken care of as symptoms usually do not emerge until it is too late and death of the infant becomes imminent.
Early-onset Neonatal Sepsis or Sepsis in Infants is inherited from the mother meaning that the offending bacteria or virus was present in the mother and had been passed to the baby causing this condition. The bacteria may be present in the birth canal and the baby may get it at the time of birth while passing through the birth canal.
Group B Streptococcus (GBS): is a leading cause of life threatening perinatal infections. 10 – 30% of women are colonized with GBS in the vaginal or rectal area. Most are asymptomatic or may include UTI, Chorioamnionitis
Fetal & neonatal effects: early onset GBS within 7 days of birth, usually 48 hrs. 1–2 % will develop early onset GBS, sepsis, pneumonia and meningitis. late onset is after the first week and meningitis is most common manifestation. Permanent neurological consequences may be seen in up to 50% of those who survive
Risk Factors include infection of the placental tissues and amniotic fluid, and rupture of membranes > 18 hours before birth.
Low socioeconomic status
Poor prenatal care
Poor nutrition
Maternal drug use
Maternal Fever
Chorioamnionitis
Maternal Urinary Tract infection
Male
Low Birth weight
Asphyxia
Symptoms:
Increased crankiness or irritability
Decreased tone or floppiness of the tone
Body temperature alterations
Reduced body movements
Diarrhea
Reduced sucking
Low blood sugar
Difficulty in feeding along with disinterest and vomiting
Fever over 100.4 degree Fahrenheit
Lethargy
Difficulty in breathing or breathing too fast
Jaundice
Skin colour change usually becoming pale, blue or patchy
Decreased urination
Swollen belly area
Bradycardia
Seizures
Bulging or swelling of the soft spot on the infant’s head.

58
Q

Pre eclampsia with Severe features is defined as

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Preeclampsia
BP elevation after 20 weeks of gestation with proteinuria (≥ 300 mg/24 hr) and/or one or more of the severe features of preeclampsia.
Features of Severe Preeclampsia
Hypertension: systolic >160 and/or diastolic >110.
Thrombocytopenia (platelet count < 100 x 109/L).
Impaired liver function (elevated liver transaminases to twice the normal values).
Renal insufficiency (serum creatinine > 1.1 mg/dL, or doubling of serum creatinine in the absence of other renal disease).
Pulmonary edema.
Cerebral or visual disturbances (new-onset).
Epigastric pain unresponsive to medication and not accounted for by alternative diagnoses.
Massive proteinuria ( > 5 g/24 hours) and fetal growth restriction (FGR) are NO longer considered as indicators of severe preeclampsia.

59
Q

As the Nursery Nurse, you are caring for a baby with a myelomeningocele. What are your priorities in this child’s care?

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NURSING MANAGEMENT:

  1. Preventing injury to the sac.
  2. Preventing skin break down
  3. Prevention of Infection
  4. Providing adequate nutrition
  5. Preventing UTI
  6. Parent education and support to reduce parent’s anxiety