Management of Tooth Wear Flashcards

1
Q

What are the 4 forms of Tooth wear?

A
  • Attrition
  • Abrasion
  • Abfraction
  • Erosion

  • Tooth wear can be multifactorial*
  • i.e. more than one type of tooth wear can occur simultaneously and can be exacerbated e.g. faster rates of attrition when erosion also present*
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2
Q

What is Attrition?

A

Attrition : The loss of tooth substance as a result of mastication, or of occlusal or proximal contact between the teeth.

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3
Q

What are the 2 classifications of Attrition?

A
  • Physiological attrition
  • Pathological attrition
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4
Q

What is Physiological attrition tooth wear ?

A
  • Happens in every individual with age, therefore more noticeable in older patients
  • Most commonly affects occlusal surfaces or incisal edges
  • Approximal wear occurs with mastication – contacts between adjacent teeth
  • Affects deciduous and permanent teeth, although deciduous teeth are more susceptible, as the enamel is thinner
  • Rate is reported to be higher in men than in women
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5
Q

What are clinical signs of Physiological Attrition?

A
  • Disappearance of incisor mamelons
  • Flattening of occlusal cusps
  • Exposed dentine may be dark brown in colour and lesions may be “cup-shaped”
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6
Q

What is Pathological attrition cause by?

A

Can be localised or generalised

Caused by a parafunction (abnormal habit e.g bruxism) or malocclusion

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7
Q

What is Bruxism?

A
  • A parafunction
  • Involves grinding and clenching the teeth
  • Cause is unclear, but thought to be associated with stress, or ‘occlusal interferences’
  • Can be a nocturnal habit
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8
Q

What are Signs and symptoms of bruxism?

A
  • Visible wear facets
  • Abnormal rate of attrition
  • Hypertrophy of masticatory muscles
  • Muscle tenderness
  • TMJ pain
  • Tooth mobility
  • Pulpal sensitivity to cold
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9
Q

How do you manage Bruxism?

A
  • Can lead to a reduced Occlusal-Vertical Dimension (OVD) – therefore, restorative considerations if attempting to ‘build-up’ the bite again
  • May need to remove occlusal interferences
  • Acrylic hard or soft splint may be worn to protect the teeth from further wear
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10
Q

What happens in dentine attration?

A

Dentine rate of attrition is higher than enamel, as it is less mineralised

Dentine attrition usually results in the faster formation of secondary/tertiary dentine, which prevents sensitivity

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11
Q

What is Abrasion?

A

Abrasion : Pathological wearing away of tooth structure that results from a repetitive mechanical process or habit

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12
Q

What are Signs of abrasion?

A

Most commonly seen on exposed root surfaces

Wear facets appear at the cervical margins

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13
Q

What are the 4 Categories of abrasion?

A
  1. Cervical abrasion
  2. Habitual abrasion
  3. Iatrogenic abrasion
  4. Industrial abrasion
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14
Q

What is Cervical abrasion and what is its most common cause?

A

Appearance of cervical grooves

Horizontal brushing technique most common cause

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15
Q

What is Horizontal Brushing?

A
  • Over-zealous toothbrushing technique
  • ‘Scrubbing’ – using excessive pressure
  • May be accelerated by abrasive dentifrice and/or a hard toothbrush
  • More likely if gingival recession already present (exposed dentine)
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16
Q

What is Habitual abrasion?

A
  • Clinical appearance depends upon cause
  • Usually localised to the area of the ‘habit’
  • Causes include:
  • Pipe smoking
  • Wind instruments
  • Opening pins with teeth or holding needles
  • Flossing with too much pressure
17
Q

What is Iatrogenic abrasion?

A

Opposing teeth grinding to accommodate restorations

Ceramic crowns

18
Q

What is Industrial abrasion?

A

Uncommon nowadays due to health and safety legislation

Workers exposed to abrasive particles in the atmosphere created during certain industrial processes (mining, sand blasting etc)

19
Q

What is Abrasion – diagnosis and treatment?

A

Diagnosis by assessing clinical picture, and by thorough history taking

Treatment is given mostly in the form of preventative advice, in order to limit further damage – find cause and remove

-If function or aesthetics affected, restorative treatment may be appropriate

20
Q

What is Abfraction?

A

Abfraction : the pathological loss of tooth substance caused by biomechanical loading forces…resulting in flexing and failure of enamel and dentine…

21
Q

What is seen on a tooth with Abfraction?

A
  • ‘V’ shaped notches at the cervical margin
  • Thought to be related to tooth ‘flexure’ at the cervical areas from occlusal loading
  • Leads to ‘micro-fractures’ in enamel
  • Cavitation occurs
  • Does not explain teeth not in occlusion
22
Q

What is Erosion?

A

Erosion : Progressive loss of hard dental tissue by an acidic chemical process without bacterial action

i.e. NOT related to caries

23
Q

What are signs of Erosion?

A
  • Can be seen on any surface
  • Appears smooth and polished or ‘glossy’
  • Eventually shallow depressions occur
  • ‘Proud’ restorations
  • Fractures of the incisal edges
  • Cervical area usually most severely affected
  • Surface may have “criss-cross” appearance
  • Hollows bear no relationship to occlusion
  • ‘cupping’ of lower molar cusps
24
Q

What are the 2 classifications of Erosion?

A

Extrinsic – source of acid outwith the body (diet, environment)

Intrinsic – source of acid from within the body (gastric juices)

25
Q

What can cause extrinsic erosion?

A
  • Frequent consumption of acidic foods and drinks
  • Habitually drinking carbonated drinks, fruit teas etc (swishing around the mouth)
  • Habitually sucking citrus fruits
  • Industrial hazard – acidic atmosphere
  • Swimming in heavily chlorinated water
26
Q

What can cause Instrinsic Erosion?

A

Reflux

  • Hiatus hernia
  • Gastric ulceration
  • Alcohol abuse/dependency
  • Gastro-oesophageal reflux (GORD/GERD)
  • Stress reflux

Vomiting

  • Eating Disorders (Bulimia Nervosa)
  • Pregnancy
27
Q

What is Bulimia nervosa?

A
  • Food binges, followed by self-induced vomiting
  • Unlike anorexia nervosa, patient maintains a normal body weight, but is secretive about eating habits
  • Results in palatal erosion of the maxillary teeth, and less commonly the occlusal surfaces of lower molars
28
Q

What are some Clinical signs of bulimia ?

A
  • Normal body weight
  • Erosion of palatal surfaces of upper teeth
  • Lesions on palate, fingers, oral mucosa, lips
  • Signs of malnutrition
29
Q

How do you manage Erosion?

A
  • Find and if possible, eliminate cause
  • If bulimia disclosed or suspected, encourage patient to seek help from GMP
  • Fluoride therapy
  • Maintain OH
  • ‘Spit don’t rinse’
  • Don’t brush immediately after acid has been in contact with the teeth – wait 30 mins • Restorations may be indicated
30
Q

When could we intervene?

A
  • Poor aesthetics
  • Loss of vitality as a result of NCTSL (non-carious tooth surface loss)
  • Loss of function
  • Sensitivity – N.B. not common unless erosion
  • The lesion is progressing - pulp at risk of becoming compromised
31
Q

How can Fluoride be use to help NCTSL?

A
  • Fluoride Varnish (Duraphat/Clinpro) applied to tooth in area of sensitivity (often cervical area)
  • High Strength Fluoride Toothpaste (HSFT) Duraphat 2800/5000
32
Q

How can Acrylic splints help NCTSL?

A
  • Used to break bruxism habit
  • Also to protect remaining dentition
  • Decision based on how much destruction is present, cause of destruction, how much more destruction is likely to occur
33
Q

When would you restore a tooth?

A
  • Very sensitive (not being controlled by any other means)
  • Patient concerned about aesthetics
  • Pulp becomes, or at risk of becoming compromised

NCTSL restorations notoriously fail because, underlying CAUSE of is not addressed