Mahler comps swallow tx Flashcards
To learn specific swallow postures, compensatory maneurvers and treatments by site of lesion
consequences of swallow disorders
Dehydration
malnutrition
aspiration pneumonia
asphyxia
cough reflex critical points
- Triggered by penetration of food into larynx
- may signal neurogenic dysphagia
- No cough m/b silent dysphagia
cough reflex reduction causes
- disruption of sensation and / or motor response
also TIM: Tracheostomy, Intubataion, Medications
Diseas3e most likely to be associated with dysphagia and why?
Brain stem vs cortical or sucortical. Nucleus tratus solitaris in brainstem
common causess of dysphagia? Why
STROKE: because stroke interrupts the corticobulbar pathways that connect cortical and brain stem centers for swallowing
brainstem strokes cause dysphagia how?
disruption of
- corticobulbar tracts
- Nucleus tractus solitaris
- swallow centers either side of medulla and pons
brain stem stroke dysphagia symptoms: by oral, pharyngeal, laryngeal and VF
- Oral: reduced strength of lips, tongue, jaw (ipsilataeral)
- Pharyngeal swallow: delay, unilateral paresis or reduced contractions
- Larynx: reduced elevation, closure and/or CP opening
- vocl fold paresis: unilateral
Left CVA dysphagia symptoms
- Langauge: readingl, writing, understanding symbols
- . oral:CONTRALATERAL weakness of lips and tongue, drooling, residue, delayed OTT
- delayed pharyngeal swallow
- SWALLOW APRAXIA: delay in initiation of swallow, lingual groping, lack of recognition of bolus as to be swallowed
Right CVA dysphagia symptoms
LOSS OF INTELLECTUAL CONTROL OVER SWALLOWING.
Frontal: lack of attention, insight, reasoning and judgement, impulsivity
2. IF PARIETAL: perceptual deficits and lack of tontopic orientation, hemineglect: loss of visualiztion of spatial relationships of left-side space
3. contralateral weakness of lips, tongue, delayed OTT, residue
4. delayed pharyngeal swallow
Right CVA patients need feeding oversight why?
reduced orientation, percption, attention, impulsivity, errors in judgment, impulsivity, loss of intellectual control over swallow
Wnen do SLP’s evaluate Right CVA patients?
as soon as they wake up, first consult
TBI/Head Injury dysphagia symptoms?
Cognitive probs of attention, impulsivity, poor memory, reasoning, judgement., and (parietal) orientation
- Oral: reduced tongue control, delayed or prolongued OTT
- Pharyngeal swallow delay or absent
- silent laryngeal penetration and aspiration (w/o cough due to intubation or trecheaostomy)
Dementia deficits and dysphagia
- Cognition: decreased awareness
- Reduced initiation or oral preparatory movements: nonpurposful bolus manipulation and loss of control
- Poor BOT retraction
- delayed pharyngeal swallow
- reduced larngeal elevation
PD and dysphagia key points 2 key points and rationale
- with PD, deficits in all stages of swallow
- 95% of PD have dysphagia and OFten not aware of swallow deficits which show in MBS
Rationale: basal ganglia influence sensory components in trigeminal system and cause abnormalities in sensorimotor responses throughout swallow
PD and oral stage defictis
1, rationale: reduced elevation of middle and posterior tongue during speech and swallow (due to rigifidy not weakness) =
2. limited tongue and mandibular excursion
3. Lingual hesitation, peicemeal deglutition (aka rocking)
4. poor posterior bolus formation
5 = overall excessive mastication and OTT
PD and pharyngeal stage deficitis: rationale and symptoms
RAtionale: the pharyngeal contrractions of striated muscle of pharynx are often delayed, therefore laryngeal
1. lowed laryngeal elevation –> slowed laryngeal closure
PD and spiration
prevalence of aspiration w/o cough in patients w/o dysphagic complaints
Oncologic conditions and dysphagia key points
- cognition and language generally intact
2. tx depends on amt and location of ablative surgery, reconstruction, scar tissue and remaining functional structures
Oncologic conditions and pretreatment counseling?
- Query patient’s understanding of swallow
- explain role of SLP
- Discuss outcomes of various medical/surgical interventions
Oncologic conditions and typical oral phase disorders (3)
- prolonged OTT
- Increased oral residue secondary to decreased tongue strength
- Reduced ability to propel bolus into pharyx (BOT)
3.
Oncologic conditions and typical pharyngeal phase disorders
- reduced BOT retraction
2 reduced laryngeal elevation and vestibule closure - reduced bolus clearance = residue in valleculae or pyriform sinuses
Oncologic conditions and late effects of radio therapy?
1, impaired neural transmission
- impaired muscled contractions
- fewer, smaller muscle fibers
- muscle replaced with fibrotic tissue due to reduced blood supply
- restricted mouth opening (trismus)
- bone loss (necrosis)
- altered or reduced taste
Oncologic conditions and early effects of radiation?
pain, soreness
ulcers and bleeding
mucositis (inflammation)
xerostomia
Oncologic conditions and dysphagia symptoms?
- aspiration after swallow due to reduced pharyngeal contraction
- reduced bolus formation and transport
- aspiration before the swallow due to delayed phayngeal swallow (residue left in pyriform sinuses and vallecutae inspirated before next phayngeal initiation