M101 Initial Assessment Flashcards

1
Q

What is the order of the A – E Assessment of an acutely unwell patient?

A
Airway 
Breathing
Circulation
Disability 
Exposure
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2
Q

When is the A – E Assessment of an acutely unwell patient carried out in a different order?

A

military - patient usually bleeding out

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3
Q

What does a nasal cannulae provide?

A

small and unpredictable increase in inspired oxygen

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4
Q

Why is the oxygen supply from a nasal cannulae unpredictable?

A

mixed in with the air in the room

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5
Q

What is the maximum capacity of oxygen delivered per minute from a nasal cannulae?

A

5 litres per minute (L/min)—delivering an oxygen concentration of

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6
Q

What is the percentage of oxygen in the air delivered from a nasal cannulae?

A

28–44%

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7
Q

What is the percentage of oxygen in the air delivered from a Hudson Mask?

A

around 40%

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8
Q

What is the maximum capacity of oxygen delivered per minute from a non-rebreather mask?

A

flow rate of about 10 to 15 liters/minute (L/min)

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9
Q

What is the oxygen supply from a non-rebreather mask?

A

60 percent to 80 percent oxygen

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10
Q

Reservoir mask

A

Well fitting face mask
Reservoir bag to collect oxygen while patient is breathing out
One way valves to ensure inspiration draws oxygen from reservoir bag and expired breath is directed out of the sides of the mask
Can deliver 80-85% oxygen
Used widely in acutely unwell patients

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11
Q

Controlled oxygen administration

A

Accurate oxygen delivery achieved by high flow of gas, which exceeds patient’s peak inspiratory flow rate
Used for patients who are sensitive to oxygen eg those with severe COPD

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12
Q

pulse oximeter

A

can be blocked from nail varnish

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13
Q

What will circulatory compromise look like in a patient?

A

pale
peripheral capillary refill will be slow
might not be conscious due to lack of blood flow to the brain
increased inspiratory rate

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14
Q

What will circulatory compromise look like in a patient?

A

pale
peripheral capillary refill will be slow
might not be conscious due to lack of blood flow to the brain
increased inspiratory rate

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15
Q

physiology of shock

A

Cardiac Output = Stroke Volume x Heart Rate
If stroke volume is reduced (eg by bleeding) then tachycardia can partially compensate for this.
This is mediated by the sympathetic nervous system which also causes pallor, sweating and diversion of cardiac output towards vital organ systems
Intravenous volume replacement is vital in this situation. It’s also important to stop ongoing losses

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16
Q

What will circulatory compromise look like in a patient?

A
pale
peripheral capillary refill will be slow
might not be conscious due to lack of blood flow to the brain
increased inspiratory rate
cold peripherals
17
Q

Starling curve

A

Explanation of preload improving cardiac output

Need to be cautious with fluid volume resuscitation in the context of poor cardiac function

18
Q

Myocardial Contractility

A
Stroke volume requires good myocardial contractility.  Contractility is impaired by sepsis and cardiac ischaemia amongst other things
Improving contractility needs identification of the cause but treating hypoxia is important.  Cautious intravenous fluid boluses usually help.
Inotropic agents (eg adrenaline) may then be needed
This requires central venous access and intensive care
19
Q

Myocardial Contractility

A
Stroke volume requires good myocardial contractility.  Contractility is impaired by sepsis and cardiac ischaemia amongst other things
Improving contractility needs identification of the cause but treating hypoxia is important.  Cautious intravenous fluid boluses usually help.
Inotropic agents (eg adrenaline) may then be needed
This requires central venous access and intensive care
20
Q

Systemic Vascular Resistance

A

Blood pressure = cardiac output x systemic vascular resistance (V=IR)
A drop in SVR means that there is less resistance to ejection of blood. Even with a normal or high cardiac output, a low SVR (as seen in sepsis and anaphylaxis) will result in a low blood pressure
Intravenous fluid may help in situations of low SVR, but vasopressors may also be needed (eg noradrenaline) which also require central access and intensive care

21
Q

Categories/Causes of Shock

A
Hypovolaemic
Distributive
Obstructive
Cardiogenic
Patients may be suffering from more than one kind of shock
22
Q

Hypovolaemic Shock

A

Haemorrhagic (trauma/GI Bleeding/aortic aneurysm)
Non-haemorrhagic (burns/pancreatitis/GI losses)
Reduced circulating volume reduces ventricular filling, stroke volume and cardiac output

23
Q

Distributive Shock

A
Severe Peripheral Vasodilatation
Septic Shock
Neurogenic Shock
Anaphylactic Shock
These cause hypotension via a reduction in systemic vascular resistance
24
Q

Obstructive Shock

A

Pulmonary Embolism
Tension Pneumothorax
Cardiac Tamponade
These impair ventricular filling which reduces stroke volume and therefore cardiac output. Intravenous fluids may help but other specific treatment may also be necessary

25
Q

Cardiogenic Shock

A
Cardiomyopathies
Cardiac Ischaemia/Infarction
Severe Arrhythmias
Acute heart valve failure
These reduce myocardial contractility and/or cardiac output
26
Q

Cardiogenic Shock

A
Cardiomyopathies
Cardiac Ischaemia/Infarction
Severe Arrhythmias
Acute heart valve failure
These reduce myocardial contractility and/or cardiac output
27
Q

Why is a hypovolaemic patient tachycardic?
Why does an intravenous fluid bolus help in this situation?
What will you look for when reassessing the patient to see if your fluid bolus has improved their circulation?

A

fluid rentention

28
Q

What happens if you overdo it with the IV?

A

oedema

29
Q

Intraosseous access

A

drilling into a bone

usually done in children or babies as a last resort for IV