M 8 Cognition Flashcards
3 categories of brain injury based on the Glasgow Coma Scale
Mild Moderate Severe
Brain injuries are also classified by the degree of damage to the brain
Concussion
Contusion
Laceration
Ischemia
etc.
4 key event of traumatic brain injurys
Vascular events
Closed head injury
Open head injury
Skull fracture
Antecedents of TBIs
Adequate oxygenation
Tissue perfusion
Nutrition
Good neuro function
Opportunities for growth
Mild TBI S/S
LOC for a few seconds to minutes
Memory problems
Dizziness
Loss of balance
N/V
Mild assessments for TBI
Sensory (vision, hearing, taste)
Light sensitivity
Mood swings
Depression
Moderate TBI S/S
LOC from minutes to hours
Profound confusion
Combativeness
Slurred speech
Moderate assessment for TBI
Loss of coordination
Seizures
Dilation of pupils
Clear fluid from ears/nose
Children’s TBI S/S
Inconsolable crying
Change in eating
Inability to pay attention
Loss of interest in favorite toys
Diagnostics for TBI
CAT
MRI
PET
Glasgow coma scale
Consequences of TBIs
Institutionalization
Poor health
Victimization
Inability to form relationships
Primary TBI interventions
Health promotion
Disease prevention
Education
Secondary TBI interventions
Screening/diagnosis
Occurs after problem has arisen
Tertiary TBI interventions
Rehabilitation
Returning PT to highest level of function possible
TBI Critical skills
Neuro assessment
Communication
Safety assessment/impementation
TBI Collaborative interventions
Med management of Underlying Cause
Physical/occupation/speech therapy
Case manager
Social worker
Post TBI issues
Cerebral herniation
Seizures
^ICP
TBI PaCO2 greater than 50 =
^ICP BAD
TBI
O2 drop or CO2 increase = a/b balance
acidic
^ICP S/S
Early - pupil dilation
Projectile vomit
Muscle weakness
^BP
Late - Eyes roll up
Cushings triad
Irregular resps
Bradycardia
Widening pulse pressure
BAD S/S of ICP
Glasgow coma scale below 8=
Intubate
ICP Less than 15
OK
BP as pt goes from ICP to Neuro shock
First increase
Then decrease
ICP Treatment
HOB at 30 degrees
Propofol - sedative
Mannitol - diuretic
High CO2 means vaso
Low CO2 means vaso
Dilation - ICP
Constriction
Encephalopathy causes
Perfusion disruption
Tumor
Lack of oxygen
Toxins
Infection (meningitis)
Encephalopathy 101
Anything that alters brain structure or function
Encephalopathy key points
Treatment based on PRIMARY CAUSE
Interventions focus on alterations and mental status
Antecedents for encephalopathy
Oxygenation
Perfusion
Nutrition
Neuro function
What does encephalopathy impact in the long term
Interpretation of environment
Communication
Learning
Encephalopathy risk factors
Infection
Liver problems
Kidney problems
Brain tumors
Poor nutrition/alcoholism
Mental manifestations of encephalopathy
Confusion
Poor judgement
Personality change
Nervousness
Sleeping changes
Physical Encephalopathy S/S
Musty or sweet breath
Shaky hands
Slurred speech
Sluggish movement
Diagnostic test for encephalopathy
Mental status
Memory test
Coordination test
Encephalopathy assessments to look for primary cause other than GCS
CBC/blood culture - infection
Altered BP
Metabolic test
Toxin levels
Creatinine
To narrow down encephalopathy area or brain, test
Cranial nerves
Consequences of encephalopathy
Institutionalization/dependence
Poor health
Victimization
Inability to maintain relationships
Primary interventions for encephalopathy
prevention
(alcohol, diet, exercise, exposure to infection)
Secondary interventions for encephalopathy
screening
Minimize toxin exposure
Maintain healthy lifestyle
Tertiary interventions for encephalopathy
Rehabilitation
Returning patient to highest level of function
Critical skills for encephalopathy
Neurologic assessment
Communication
Safety assessment and implementation
Collaborative interventions for encephalopathy
Med management
Physical/occupational/speech therapy
Case manager
Social worker
Postpartum psychosis key points
EMERGENCY due to potential for suicide or infanticide
A form of bipolar disorder
Onset of postpartum psychosis
First 2 weeks
Causes for postpartum psychosis
hormonal loss at birth***
fam hist of bipolar disorder
Hormones that shift and cause postpart psych
estrogen
progesterone
Positive prognosis for postpartum depression is projected based off
acute onset
lack of premorbid debility
Post part psychosis treatment
pills
Antipsychotics
Mood stabilizers
Benzos
Post part psychosis treatment goal
Counseling goal
Timely remission of symptoms VITAL
Follow up appts
Why is remission important
Baby needs contact and attention immediately
Post part risk factors
Hist of bipolar, pt or fam*
Discontinuation of mood stabilizers
First pregnancy
Sleep deprivation
Lack of partner support
Mental status assessment for encephalopathy
Behavioral
Motor
Speech
Post partum psych S/S assessment
Mood
Judgement
Memory
Motivation
Consequences of postpartum psychosis
Institutionalization
Poor health
Victimization
Inability to maintain relationships
Questions for postpartum depression
Difficulty concentrating?
Difficulty sleeping?
Do you have thoughts of harming self or others?
Encourage postpart psych patients to verbalize
Feelings
Post partum psych interventions
Meds and their side effects
Discharge planning BEFORE leaving hospital
Referral to intensive therapy
ECT*
Schizophrenia moa
Altered dopamine levels
Schizophrenia 101
Altered thinking, language, emotions
Abnormal interpretation of reality
Attributes of schizophrenia
Environment interpretation
Communication issues
Learning issues