Lung Expansion Therapy Flashcards

1
Q

What is the goal of lung expansion therapy?

A

to prevent or correct atelectasis

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2
Q

What are the 4 types of lung expansion therapy?

A

IPPB, IS, CPAP, PEP

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3
Q

What is absorption atelectasis?

A

Due to lesions or mucus plugs blocking ventilation, gas distal to obstruction is absorbed by blood causing alveolar collapse

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4
Q

What is passive atelectasis

A

caused by persistant small tidal volumes

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5
Q

Who needs lung expansion therapy?

A

Any patient that cannot take a deep breath on their own.

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6
Q

Who is at the most risk to develop atelectasis?

A

Post op pt, especially upper abdominal, history of chronic lung disease, smoker

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7
Q

Clinical signs of atelectasis

A

increased RR, fine late inspiratory crackles, bronchial breath sounds, tachycardia

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8
Q

What is the relationship between RR and atelectasis?

A

the more atelectasis the faster the respiratory rate

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9
Q

Clinical signs on CXR

A

increased radiopacity, mediastinal shift

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10
Q

How does lung expansion therapy work?

A

by increasing transpulmonary pressure gradient

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11
Q

What is transpulmonar pressure?

A

alveolar pressure -pleural pressure; the greater pressure gradient, greater alveolar volume

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12
Q

Which therapy is the most physiological?

A

IS (SMI)

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13
Q

What is compliance?

A

the change in volume per unit of pressure change; chg in V/chg in P

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14
Q

What is elasticity?

A

the property of matter causing it to return to its original state after deformation by some external force; chg in P/chg in V

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15
Q

Average intrapleural pressure

A

-4 to -5 cwp below atmospheric

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16
Q

Intrapleural pressure with stron inspiratory effort

A

Up to -50 cwp

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17
Q

Intrapleural pressure with forced expiration

A

+70 cwp

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18
Q

Transairway pressure

A

P mouth-P alveolar

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19
Q

Transpulmonary pressure

A

P mouth-P intrapleural

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20
Q

alveolar distending pressure

A

P alveolar-P intrapleural

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21
Q

Spontaneous breath before inspiration

A

Flow=0, Ppl = -5, PA =0

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22
Q

When inspiration begins…

A

diaphragm contracts and descends; thoracic volume increases, Ppl becomes neg, alveolar volume increases and PA becomes neg, the change in Ppl cause change in PA

23
Q

During expiration

A

the sequence is reversed, muscles relax, lung recoils, diaphragm ascends

24
Q

What is resting position of lungs and chest

25
When is alveolar pressure 0?
pre inspiration, end inspiration and end exhalation
26
Objective of SMI
increase transpulmonary pressure and inspiratory volumes, improve inspiratory muscle performance
27
Advantages of SMI over IPPB
NO POSITIVE PRESSURE, more effective as prophylactic, time and cost minimized
28
Goals of IS
optimize lung inflation, optimize cough mechanism, early detection of acute lung disease
29
Indications for IS
Presence of atelectasis; presence of conditions predisposing to atelectasis (abd surg), presence of a restrictive lung defect associate with quadriplegic or dysfunctional diagphragm
30
Contraindications for IS
unable to cooperate; cannot follow instructions; unable to generate adequate inspiration;
31
Hazards and Complications of IS
hyperventilation, discomfort(from pain), pulmonary barotrauma, hypoxemia, exacerbation of bronchospasm, fatigue
32
2 types of IS devices
flow oriented, volume oriented
33
3 phases for administering IS or IPPB
planning , implementation, follow up
34
Criteria for SMI
``` cooperative patient VC> 10ml/kg IBW IC>1/3 predicted f<25 bpm no acute lung disease ```
35
Potential outcomes of IS
decreased RR. normal pulse, resolution of abn breaths sounds, improved CXR
36
Administering IS
Set attainable goal; slow and deep; breath hold 5-10 seconds, normal exhalation, rest, repeat 5-10 times/hour
37
IPPB requries
patients carefully chosen; indications be specifically defined, goals understood, properly administered
38
IPPB definition
application of inspiratory positive pressure to a spontaneously breathing patient as an intermittent or short term therapeutic modality
39
Indications for IPPB
need to improve lung expansion, need to deliver aerosol, not responsive to cheaper techniques
40
Contraindications for IPPB
tension pneumo(absolute), ICP>15mmHg, blebs, hemodynamic instability, active hemoptysis, TE fistula, active untreated Tb, facial surgery
41
Signs of pneumothorax
increased HR, decrease BP, absent BS, hyperresonate percussion
42
Hazards and Complications of IPPB
pulmonary barotrauma, respiratory alkalosis, increased RAW, give with bronchodialator
43
Adverse effects positive pressure
hyperinflation with air trapping, pulmonary edema, decreased venous return, increased ICP, arrhythmias, fluid retention
44
GI effects of postive pressure
gastric insuflation- esp with mask tx, abd distention
45
Expiratory retard
modification of pursed lip breating
46
Implementation of IPPB
why ordered, what does it do, how will it feel, expected results, demonstration
47
Settings on IPPB
sensitivity-1-2 cwp, initial pressure 10-15 cwp, flow mod to low, rate 6 bpm, I:E 1:3 or 1:4
48
Any problems with therapy
Stop, stay and stabilize
49
CPAP
elevates and maintains positive airway pressure throughout both inspiration and expiration; PEP and EPAP are positive on expiration only
50
Benifits of CPAP
Recruitment of collapsed alveoli, decrease WOB, improved distribution of ventilation, increased secretion removal
51
CPAP indications
postop atelectasis,
52
Contraindications of CPAP
hypoventilation, hemodynamic unstability, facial trauma, untreated pneumothorax
53
CPAP hazards and complications
increased work of breathing, barotrauma, gastric distention