lumbar presentations Flashcards

1
Q

LBP CPGs were created with an impairment-based classification system in mind. The classifications are:
-Acute LBP with___deficits.
-Acute/Chronic LBP w/ movement ___
-Acute LBP w/ related ___ pain
-Acute/Chronic w/ ___ pain
Acute/Chronic w/ related ___/___

A
  1. mobility
  2. movement coordination
  3. relate (referred)
  4. radiating
    related cog/affective
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2
Q

Excessive mobility in the spine or other joints + hx of previous episodes are prognostic indicators for ___pain, not chronic.

A

recurrent

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3
Q

Many of the prognostic indicators for chronic LBP relate to cog factors such as fear/low expectations, distress/depression, and passive coping style. What are 2 more physical indicators?

A

symptoms below the knee and high-intensity pain

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4
Q

Back pain is pretty prevalent in cancer pts- hence, asking re Hx is important. What are the common metastasis origins? (Think lead kettle minus 1)

A

prostate, breast, lung, and kidney

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5
Q

epidural abscesses occur when when bacteria is carried to the space by the blood but it’s often misdx—about___% of the time.

A

50%

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6
Q

Vertebral osteomyelitis and epidural abscesses are often concomitant and similar in that they both present in focal back pain and have nervey s/s but how do HPI differ?

A

EA: likely to occur after a fall-not always though

VM:Hx of infection (esp bladder)

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7
Q

what physical examination findings would support a dx of vertebral osteomyelitis?

A

fever, local tenderness, agg w/ percussion, neurologic s/s (cord/root),

confirmed via lab tests

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8
Q

what physical examination findings would support a dx of epidural abscess?

A

local/focal back pain, radicular s/s, paralysis

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9
Q

Henschke’s 4 Rules for spine fx (think osteoporosis )

A

age >70 years
significant trauma
prolonged corticosteroid use
sensory alt

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10
Q

Roman’s 4 rules for spine fx (think stress or osteoporosis)

A

age>52, no leg pain, BMI </=22, doesn’t exercise reg, female

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11
Q

spondylolysis is a fatigue fracture of the ___ that can be acquired, congenital or developmental. A majority of them are at level___

A

pars interarticularis; L5

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12
Q

Define a flail segment.

A

a bilat pars defect w/ attached multifidi

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13
Q

what are some demographic factors related to spondylolysis & spondylolisthesis

A

Greatest slip in ages 10-15
sports w/ repetitive ext (diving,gymnastics ,weight lifting, etc)
risk + high grade slip in women>men

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14
Q

spondylolysis & spondylolisthesis aggravating fx

A

extension activities, rotation

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15
Q

what physical examination findings would support a dx of spondylolysis & spondylolisthesis?

A

neurologic s/s
focal p!
excessive lordosis w/ possible step-off deformity
hamstring tightness?
+instability and spring test
»often decreased flex/ext via imaging as opposed to instability

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16
Q

When observing spondylolysthesis, we know that CT and MR’s are better than X-rays for confirming the dx. With these images, we are looking for a “______” (think puppy lol)

A

scotty dog sign WITH a collar

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17
Q

This medically/procedurally contracted condition most commonly results from bacterial infection secondary to a discography.

A

iatrogenic discitis

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18
Q

____ or _____ can be complications of iatrogenic discitis.

A

sepsis or epidural abscess

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19
Q

Three primary sources of discogenic pain are:

A

iatrogenic discitis, torsion/rotary injury, internal disc disruption

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19
Q

Falling, repetitive jumping and/or picking up heavy objects (multifidi stress) can lead to axial compression on the vertebral endplates and ultimately fx+ ___ (think nucleus leakage)

A

schmorl’s nodes

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20
Q

Schmorl’s nodes can indicate an interaction between the nucleus pulposus and the body, there is potential for _____of the nucleus matrix (inflammation present) or ____of the pH (inflammation absent). Either way it’s a bad time to be a nucleus.

A

degradation, lowering

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21
Q

Internal disc disruption is typically a result of these two MOI:

A

rotary or end plate injury

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22
Q

The 4 main consequences of an IDD are that:
*The nucleus is less able to bind ___(thus can’t withstand pressure as well) and relies more on ___.
*Discs lose height and may extend to form herniations.
*“_____ effect “as facet joints take on more load.
*_______formation.

A

water
annulus
“tripod effect”
osteophyte formation

23
Q

this is not considered a herniation, but instead a _____. It involves between 50-100% of the disc circumference.

A

bulging disc

24
Herniation: This _____ herniation is considered to be _____ because it involves 25-50% of the disc.
protrusion; broad based
25
Herniation: This _____ herniation is considered to be _____ because it involves <25% of the disc.
protrusion; focal
26
Herniation: This herniation is considered to be _____ because of its shape.
an extrusion
27
Herniation: This herniation is considered to be _____ because of it's free fragment.
sequestered
28
In the same way the the disc can migrate in the transverse plane to impact SC or nerve roots, it can also move ___/____
superiorly/inferiorly
29
talk through torsion MOI step by step. 1. axis of rotation starts @ ____ and lim by _____ 2. Forced rotation changes axis to ____ and the opp ____ rotates backwatds+ shearing force on disc 3. Shear + rotary loading > potential ___
1. posterior 1/3 of disc, z-joint 2. z-joint (both blanks) 3. tearing of annulus
30
While initial trauma is still a factor regardless , pain from disc pathologies can be ___or ___
acute or chronic
31
Pain descriptions for disc pathology are usually: (think type, frequency and location)
dull and constant ache thats recurrent and worsens in intensity over time central but not well localized
32
Aggravating fx of disc pathology include: (hint: R.E.A.D. the room, my back hurts!)
-recumbency -end range rotation -activities that increase compression (sitting, coughing, etc) -day duration/ disk
33
in a physical examination of someone with disc pathology, we might expect to see postural abnormality (ex lat shift), pain in CPAs, LMN s/s and (3)
multidirectional trunk ROM lim directional preference in rep motion (+centralization) +SLR test
34
how might the HPI of radicular pain differ between someone who is acute vs chronic?
acute- likely trauma related to twisting or lifting chronic- insidious but feels like its peripheralizing as the condition progresses
35
describe the symptomatology of radicular pain
shooting band-like pain that is aggravated with closing the foramen
36
what physical examination findings would support a dx of radicular pain?
potential lateral shift and/or disc bulge, lim foramen closing, tenderness/turgor in paraspinals
37
what special test findings would support a dx of radicular pain?
+ slump test +SLR +Well leg raise
38
spinal stenosis is foraminal closing that is often described as degenerative. Structures that may contribute to this are: bulging discs, z joint hypertrophy (or osteophytes), ____(2)
thickening ligaments (potentially PLL), and spondylolisthesis
39
this **calf** and sometimes thigh pain is common in over half of pts with L-spine stenosis.
neurogenic claudification
40
list the common demographics for degenerative spinal stenosis
age >65, chronic LBP
41
A person notes they are experiencing: UMN (**OR LMN**) s/s, pain aggravated by prolonged positioning/relieved w/ recumbency or UE support and pain in both legs that > their LBP. What is at the top of your differential
central canal degen spinal stenosis
42
what physical examination findings would support a dx of degen spinal stenosis
diminished lumbar lordosis painful/lim ext + lat flex ROM that improves w/ flexion shortened HS/tight hip flex UMN/LMN s/s
43
Our CPRs for lumbar stenosis note 4/5 factors are pretty helpful in CONFIRMING a dx. What are the 5 factors?
bilat s/s leg pain>back pain w/ walking and standing alleviating w/ rest age >48
44
T/F: Similar to what occurs in other spinal regions, z-Joint pain typically refers to the buttock and thigh in reliable, defined patterns.
FALSE. honestly they can refer all the way to foot in some cases
45
thes 3 pathologies are contributors to the development of degen z-joint pain
OA, spondyloarthropathy, DDD/disc spondylosis bonus: hx of prior injury can also relate
46
what patient reports would encourage you to make acute z-joint OA part of your diff dx?
sudden oset and/or trauma acute "locked back" pain assoc w/ opening/closing z-joint space
47
what components of your physical examination might cue you into an acute z-joint dx?
-slouched and potentially lat shifted posture -pain and lim ROM (worst w/ ext) -pain w/ spring testing /UPA -tender, guarded paraspinal
48
the insulating fat pads of the spine, ie: meniscoids, move ___ (into/out of) the joint during flexion and ____(into/out of) it during extension. After trauma, they can become highly irritable loose bodies.
flexion: OUT ext: IN
49
NM instability aka muscle imbalance often comes w/ recurrent hx. what other pt reports are characteristic of this dx?
constant LBP, prolong position, quick movement, flexion **+ pop,lock and drop it!** (pop/clunk noises, locking/catching, painful flex /getting up from 'dropping it' lol )
50
what physical examination findings would support a dx of NM instability?
aberrant and or lim trunk AROM joint or muscular hypermobility paraspinal guarding/tenderness
51
what special tests would support a dx of lumbar NM instability?
Prone instability test passive lumbar ext test
52
thoracolumbar fascia fat herniations, aka _____, present as innervated and palpable nodules in the posterior layer of the fascia.Rarely the ONLY issue, they can be treated with______ . They can also be asymptomatic.
back mice; needling and steroid injections
53
LBP is often associated with fatty infiltration and atrophy of the ___. The typical conditions related to these morphologic changes include (3)
multifidi; post-op (fusion!), recurrent unilat LBP, chronic LBP
54
Proposed rationale for why the likelihood of recurrent/ chronic LBP increases with fatty infiltration--in addition to worsening intervention prognosis-- is that...
there is an alteration in muscle recruitment potentially related to reflex inhibition.
55
While a hx of low load, repetitive motions might prioritize trigger points in a differential diagnosis, the 3 requirements of an official dx are:
taut band twitch response local and referred tenderness