lumbar conditions Flashcards
strain
damage to muscle, muscle tendon; typically has faster healing than sprain due to vascularization
sprain
damage to ligament or the stabilizing components of a joint; typically slower healing time due to no vascularity
grade I sprain/strain
mild swelling and point tenderness over ligament, no bruising, <10% fiber damage, mild stretch, no instability; recovery 2-14 days
grade II sprain/strain
mild to moderate swelling, large spectrum of injury that can include partial tearing, 11-90% fiber damage; recovery in 14 days-2 months
grade III sprain/strain
severe deep bruising and swelling, complete tear of multiple ligaments, muscles, and joint capsules; recovery in 1-3 months
clinical presentation of lumbar sprain/strain
low back pain, stiffness, or muscle spasms, prior history of back injury, limited ROM, pain is better with rest, heat, and non-weight bearing, pain aggravated by activity and lifting
clinical presentation of lumbar facet syndrome
dull achy low back pain with sharp, acute episodes, insidious onset with degenerative changes and sudden onset in cases of trauma; pain worse with prolonged standing and lumbar extension, pain relieved when lying supine, sitting, or lumbar flexion
clinical presentation myofascial pain syndrome
deep, aching pain in a muscle, pain that persists or worsens, a tender knot in a muscle, difficulty sleeping due to pain
primary sciatica
direct involvement of the sciatic nerve as a result of underlying neurologic changes such as diabetes, alcoholism, pernicious anemia, B12 deficiency, heavy metal toxicity
secondary sciatica
direct irritation of the sciatic nerve or its nerve roots
clinical presentation of sciatica
ongoing buttock and leg complaints which are self described as sciatica
clinical presentation of piriformis syndrome
chronic pain in buttock that may radiate down into the leg, pain is worse with walking, squatting, or hip internal rotation or adduction, low tolerance for sitting
clinical presentation of degenerative joint and disc disease
dull, achy pain following joint pain, morning stiffness, better with mild activity and rest, worse with excessive activity, prior history of trauma or injury
primary DJD (idiopathic)
abnormal biomechanics or posture, genetic predisposition, metabolic causes
secondary DJD (trauma)
equal gender distribution, childhood anatomic abnormalities, joint trauma or fracture, obesity, repetitive action or joint motion
phase I of degeneration: dysfunctional phase
circumferential tears in the outer annulus, nucleus pulposus loses water and proteoglycan content
phase II degeneration: unstable phase
loss of mechanical integrity, internal disc disruption, resorption and loss of disc height, leading to segmental instability
phase III degeneration: stabilization
further disc resorption, disc space narrowing, endplate destruction, osteophyte formation, possible discogenic pain results
central canal stenosis
may be associated with bulging of the ligamentum flavum or posterior body spurs as well as bulging of the annulus fibrosis
lateral canal stenosis
compression of exiting nerve roots from the lateral recess through the foramen to the extra foraminal zone