Lumbar Flashcards
Questionnaires good for self-report with LBP (2)
- Oswestry
2. Roland-Morris
Clinical progression of LBP (3 + time periods)
- Acute (<1 month)
- Subacure (1-2 months)
- Chronic/recurrent (>3 months)
On/off switches at what three levels of the NS?
- Spinal cord
- Brain stem
- Higher brain centers
Gate control theory - “switch off”
at the spinal cord
non-nociceptive A-beta fibers recruit inhibitory neurons in the substantia gelatinosa of the posterior spinal cord
- chemically block ascending A-delta fibers and c-fibers
Four types of neurons in ascending pathways -
- names of fibers
- information carried
- Myelination?
- speed
- A-alpha, proprioception, myelinated
- A-beta, touch, myelinated
- A-delta, pain (mechanical and thermal), myelinated
- C-fibers, pain (mech, therm, chem), NON-myelinated
Spinal cord “switch on” with gate control theory
- theory of chronic spine pain
repetitious c-fiber firing thought to be enhanced by rapidly expressed genes => further increases sensitivity/sensitization of nociceptors = decreases threshold to stimulation and results in a wider receptor field
Brain stem “switch off”
stimulation of periaqueductal gray matter (PAG m.) by descending pathways from HIGHER centers
- PAG is opiodergic (descends to and affects the dorsal horn)
higher brain centers
- interaction with pain (conscious
- spinothalmic tr.
- Modulates neural activity and/or conscious perception of pain
- Numerous cortical regions of spinothalmic tr. produce the multidimensional pain exp.
Chemicals/agents released by higher brain centers for pain management
Dopamine
Serotonin
Endogenous opiates
Cannabinoids
Chemical responses from higher brain centers in presence of placebo, direct interventions, or both?
Clinical research has shown that both placebo and direct interventions may cause the release of multiple substances into the CNS in an opiodergic fashion
Subgroup for Targeted Treatment (STartT) Back Screening Tool
- use?
Promising tool to address homogeneity
- avoiding the one-size-fits/rx-all concept
- used to identify pts with LBP at risk for long term functional limitations
Poor prognosticators for individuals with CLBP (2)
- high pain
2. high disability
T or F: single red flag is predictive for serious dz
- if false, why?
False - many people have single red flags show up, but these should be used in clusters to lead the therapist to concerns for serious pathology - the higher the number of red flags, the greater the concern
T or F: nearly all pts will show one red flag w/o notable serious dz
- if false, why?
True
Two major serious dx to screen for in the outpatient setting
- Metastatic CA
2. Undiagnosed fractures
Key clinical features of metastatic lesions (2)
- hx of CA
2. overall clinician judgement
red flags of metastatic lesions (4)
- previous hx of CA
- insidious onset of symptoms
- older age at onset (>50 y/o)
- failure to recover/improve
red flags of undiagnosed vertebral fractures (CPR developed by Henschke and colleagues)
- > 70 y/o
- significant trauma/injury
- prolonged use of corticosteroids
- female gender
T or F: MRIs are good for identification of serious pathology
- if false, why?
True