List II - Less common 'know of' conditions Flashcards

1
Q

What is a laceration?

A
  • Tearing or splitting of the skin commonly caused by blunt trauma or any incision in the skin caused by a sharp object such as a knife or broken glass
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2
Q

How should a person with a laceration be initially managed in terms of referral?

A
  • Assess the wound to determine the need for admission or referral
  • Admit if the person has signs or symptoms of tetanus (generalised rigidity and spasm of skeletal muscles including lock jaw) and has had a laceration in the previous days or weeks
  • Refer to A and E if:
  • Vascular damage - arterial bleeding from the wound, loss or pulse, or poor perfusion distal to the injury
  • Nerve damage, characterised by loss of light touch or motor function distal to the injury
  • Injury to a tendon, including injury to the sheath
  • Bony injury or fracture
  • Facial laceration for which a good cosmetic repair is important, particularly a laceration that crosses the margins of the lips, nose, or ears
  • Laceration of the palm of the hand with any signs of infection
  • Laceration associated with cellulitis over a joint
  • Laceration is complex, widely gaping, or extensively devitalised

Also refer to A and E if there is tetanus prone wound, which includes:

  • Wounds that require surgical intervention which has been delayed for more than 6 hours
  • Wounds that have a significant degree of devitalised tissue or a puncture-type injury, particularly if there has been contact with material likely to contain tetanus spores (e.g. soil or manure)
  • Wounds containing foreign bodies
  • Compound fractures
  • Wounds in people who have systemic sepsis
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3
Q

How should people with laceration not requiring admission or referral be assessed?

A
  • Decide whether it is infected or at risk of infection
  • Infected laceration may present with general malaise, fever, rigors, erythema spreading from the laceration, and/or lymphadenopathy
  • A laceration is at high risk of infection if it is contaminated with soil, faeces, body fluids, or pus
  • Other risk factors for infection include wound length more than 5 cm, age older than 65 years, DBM, stellate shape or jagged wound margins, and presentation more than 6 hours after injury, risk assessment requires clinical judgement but as a guide:
  • Single risk factor for infection unless unusally severe, is not likely to be at high risk of infection
  • Two or more risk factors, unless these risk factors are unusually mild, is likely to be at high risk of infection
  • Although rare, suspect child maltreatment if a child has lacerations, abrasions or scars and the explanation is inconsistent or unreliable
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4
Q

What is the management for a laceration with low infection risk?

A
  • Clean the wound
  • Disinfect around the skin with antiseptic
  • Keep hair out of the wound - if necessary, clip the hair around the wound with scissors
  • If debriding or exploring the wound, anaesthetise the area - pain from the anaesthetic can be reduced by:
  • Using a 25 gauge needle
  • Warming the anaesthetic before infiltration
  • Infiltrating through the cut edge of the wound into the subdermal tissue
  • Infiltrating slowly
  • Close the wound
  • Suturing with local anaesthetic is preferred for wounds longer than 5 cm or those shorter than 5 cm when:
  • Wound is subject to excessive flexing and tension or wetting
  • Deep dermal sutures are required to allow low tension apposition of the wound edges
  • Tissue adhesives or adhesive strips should be used to close wounds 5 cm or shorter when there are no risk factors for infection, and the wound edges are easily apposed without leaving any dead space and the wound is not subject to excessive flexing, tension or wetting
  • Tissue adhesives are not suitable if any risk factors for infection are present
  • Always use adhesive strips on pre-tibial flaps (not tissue adhesives or sutures)
  • Dress the wound
  • Lacerations with minimal exudate, dress with a clear vapour permeable dressing
  • Lacerations with modest exudate, dress with a low adherence, absorbent, perforated dressing with an adhesive border - these are also available with a plastic film covering to protect dressings from getting wet
  • Check the persons tentanus immunisation status to determine the need for a booster dose of tetanus vaccine
  • Provide verbal and written advice to the person including to:
  • Seek medical attention if they develop signs and symptoms of infection including increasing pain, redness or swelling spreading from the laceration, fever or generalised malaise
  • Take simple analgesia such as paracetamol or ibuprofen if the wound is painful or likely to become painful
  • Keep the wound clean and dry
  • Arrange follow up appointment to remove stitches
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5
Q

What is the advice regarding tentanus vaccine following laceration who is low risk?

A
  • Check immunisation status
  • Fully immunised person will have had a primary course of three vaccines followed by two boosters spaced 10 years apart
  • Administered a booster dose of tetanus vaccine if needed
  • If the person is fully immunised, a booster dose is not needed
  • If primary immunisation is complete but boosters are incomplete but up to date, a booster dose is not required but administer if the next dose is due soon and it is convenient to do so
  • If primary immunisation is incomplete or boosters are not up to date administer a reinforcing dose of the vaccine, and continue with the recommended schedule (to ensure future immunity)
  • If the person is not immunised or the immunisation status is unknown or uncertain, give an immediate dose of vaccine followed, if records confirm, by completion of a full five dose course to ensure future immunity
  • Prophylaxis with tetanus immunoglobulin is not necessary for a person with a wound that is unlikely to become infected
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6
Q

What is the guidance regarding suture removal following laceration?

A

For lacerations closed by sutures, remove stitches after:

  • 3-5 days for wounds on the head
  • 10-14 days for wounds over joints
  • 7-10 days for wounds at other sites
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7
Q

What is the guidance regarding removal of adhesive strips following laceration?

A

Advise the person to remove these themselves after:

  • 3-5 days for wounds on the head
  • 7-10 days for wounds at other sites
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8
Q

What is the advice to patients who have had a laceration closed by tissue adhesives?

A

Advise the person that these do not need to be removed as they will slough off naturally after 7-10 days

  • Dressings should be removed at the same time as sutures or adhesive strips
  • Low adherence absorbent dressings should be replaced if the exudate has cause significant wetting or the dressing
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9
Q

How can facial fractures be classified?

A
  • Lower third
  • Middle third
  • Upper third
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10
Q

What is the lower third of the face?

A
  • Made up of the mandible and its teeth

* Mandible forms a ring with the temporo-mandibular joint and the base of the skull

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11
Q

What are the potential fractures of the lower third of the face?

A
  • Mandible often breaks in more than one place - ring is difficult to break
  • Often has little impact on the airway, unless there is gross displacement
  • Gross displacement can cause bleeding that can cause a large sublingual haematoma producing airway compromise (similar to Ludwigs’ angina)
  • Roots of the teeth act as a natural weak point in the mandible, teeth are often in the fracture line and can become displaced, loosened, or avulsed
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12
Q

What is the middle third of the face?

A
  • Made up of the maxilla, zygoma and lower half of the naso-orbito-ethimoidal complex
  • This part of the face houses the eyes, the nasal airway, maxillary sinuses and maxillary teeth
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13
Q

What are the potential fractures of the middle third of the face?

A
  • Mid face acts as a crumple zone to protect the brain from injury and is frequently involved in facial injuries
  • Can also result in airway compromise
  • Can result in significant haemorrhage and immediate life saving measures may involve placement of Epistats, Rapid Rhinos, Foley catheters or equivalents and bite blocks such as McKesson’s to splint the maxilla and tamponade bleeding points
  • Le Fort classification are common patterns of injury - patterns were produced on cadavers therefore not as precise in vivo
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14
Q

What is Le Fort I?

A
  • Maxilla is fractured from the rest of the face and is often not as freely mobile as one might expect
  • Only in high energy transfer does the maxilla become loose and at risk to the airway
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15
Q

What is Le Fort II?

A
  • Involves the maxilla and nasal complex fracturing from the facial bones and mobility is often more than Le Fort I
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16
Q

What is Le Fort III?

A
  • Injuries are more significant and involve the whole mid-face dissociating from the base of the skull and facial bones
  • Frequently Le Fort injuries will occur in combination and involve the mandible
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17
Q

What is the upper third of the face?

A
  • Made up of the frontal bone, sphenoid and upper half of the naso-orbito-ethimoidal complex
  • Contains the eyes and more of the paranasal sinuses (frontal, anterior and posterior ethmoids and sphenoids)
  • Frontal bone’s weakness is the frontal sinus and the extent of pneumatisation varies considerably between individuals
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18
Q

What are the potential fractures of the upper third of the face?

A
  • When sinuses are large the frontal bone may fracture and involve the anterior skull base
  • These fractures may be associated with dural tears, cerebrospinal fluid leak and, therefore, the risk of ascending infection
  • Upper third fractures often require anaesthetist input because it is commonly necessary for tubes to be placed into the nose (Foley catheters, nasogastric tubes, and temperature probes)
  • Due to fracture displacement and difficult clinical circumstances they can end up in the frontal lobe - it is important to understand that the nasal floor is horizontal when upright and caution is needed
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19
Q

What are the different areas of the shoulder that can be fractured?

A
  • Clavicle fractures - most common and results from a fall
  • Scapula fractures - rare and usually result from high energy trauma such as motor vehicle accidents or far fall
  • Proximal humerus fractures - upper part of the arm and more common in older (>65 years) population
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20
Q

Which classification system can be used for fractures of the proximal humerus?

A
  • Neer classification system
  • Based on the anatomical relationship of 4 segments
  • Greater tuberosity
  • Lesser tuberosity
  • Articular surface
  • Shaft - surgical neck
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21
Q

Which type of shoulder fracture is the most common?

A
  • Proximal humerus

- Often seen in older patients with osteoporotic bone following a simple ground level FOOSH

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22
Q

What are the treatment options for proximal humerus fracture?

A
  • Most are treated with a sling non-operatively
  • Indications for non operative treatment include:
  • Minimally displaced surgical and anatomic neck fractures
  • Greater tuberosity fracture displaced <5 mm
    (>5 mm displacement will result in impingement with loss of abduction and external rotation)
  • Operative
  • Closed reduction percutaneous pinning (CRPP)
  • ORIF
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23
Q

What is the most commonly injured carpal bone in the hand?

A
  • Scaphoid - base of the thumb region
  • Usually the result of a fall onto outstretched hand
  • Most commonly axial load across a hyper-dorsi-flexed, pronated and ulnarly-deviated wrist
  • Common in contact sports
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24
Q

What is the main risk of an untreated scaphoid fracture?

A
  • Avascular necrosis
  • Without treatment the risks are as follows:
  • Proximal 5th AVN rate of 100%
  • Proximal 3rd AVN rate of 33%
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25
Q

Which classification can be used for scaphoid fracture?

A
  • Type A - stable, acute fractures
  • Type B - unstable, acute fractures (distal oblique, complete waist, proximal pole, trans-scaphoid and perilunate associated fractures)
  • Type C - delayed union characterised by cyst formation and fracture widening
  • Type D - non-union
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26
Q

What is a positive clinical test for scaphoid fracture?

A
  • Anatomical snuffbox tenderness dorsally
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27
Q

What is the recommended imaging for scaphoid fracture?

A
  • X-ray in the following views:
  • Neutral rotation and lateral, semi-pronated (45 degree) oblique view
  • Scaphoid view, 30 degree wrist extension, 20 degree ulnar deviation
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28
Q

What is the management if the radiographs are negative (27%) and there is high clinical suspicion of scaphoid fracture?

A
  • Repeat radiographs in 14-21 days
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29
Q

What is the management of scaphoid fracture?

A
  • Non-operative

* Operative

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30
Q

What are the indications for non-operative management of scaphoid fractures?

A
  • Stable non-displaced fracture (majority)
  • If patient has normal radiographs but there is high suspicion can immobilise in thumb spica and re-evaluate in 12 to 21 days
  • Scaphoid fractures with <1 mm displacement have union rate of 90%
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31
Q

What are the indications for operative management of scaphoid fractures?

A
  • Unstable fractures as shown by:
  • Proximal pole fractures
  • Displacement > 1 mm without significant angulation or deformity
  • Non-displaced waist fractures to allow decreased time to union, faster return to work/sport, similar total costs compared to casting
  • Can be fixed with percutaneous screw fixation - union rates 90-95%
32
Q

What are the indications for ORIF for scaphoid fractures?

A
  • Significantly displaced fracture patterns
  • 15 degrees scaphoid humpback deformity
  • Radio-lunate angle >15
  • Intra-scaphoid fractures associated with perilunate dislocation
  • Comminuted fractures
  • Unstable vertical or oblique fractures
  • Prognosis relates to accuracy of reduction which correlates to rate of union
33
Q

How long should cast immobilisation be done for in patients with scaphoid fracture?

A
  • Start immediately
  • Immobilisation should be maintained until radiographic fracture healing is demonstrated, usually no sooner than 8 weeks
  • May be required for up to 12-14 weeks for high risk fracture patterns/patients
  • Athletes should not return to play until imaging shows a healed fracture
34
Q

What is a mallet finger?

A
  • Finger deformity caused by disruption of the terminal extensor tendon distal to the DIP
  • Usually caused by a traumatic injury - impact blow or dorsal laceration
35
Q

What is the classification for a mallet finger?

A
  • Doyle’s classification
  • Type I - closed injury with or without small dorsal avulsion fracture
  • Type II - open injury (laceration)
  • Type III - open injury (deep soft tissue abrasion involving loss of skin and tendon substance)
  • Type IV - Mallet fracture
    A = distal phalanx physeal injury (paediatric)
    B = fracture fragment involving 20% to 50% of articular surface (adult)
    C = fracture fragment >50% of articular surface (adult)
36
Q

What is the typical presentation of a patient with Mallet finger?

A
  • Painful and swollen DIP joint following impaction injury to finger - often ball sports
  • Finger tip at rest ~45 of flexion
  • Lack of active DIP extension
37
Q

What is the non-operative management of Mallet finger?

A
  • Indications:
  • Acute soft tissue injury < 12 weeks
  • Non-displaced bony mallet injury
  • Technique
  • Maintain free movement of the PIP joint
  • Worn for 6-8 weeks
  • Volar spinting has less complications than dorsal splinting
  • Avoid hyper-extension
  • Begin progressive flexion exercises at 6 weeks
38
Q

What are the operative management options for Mallet finger and what are the indications?

A
  • ORIF vs CRPP
  • Absolute indications
  • Volar subluxation of distal phalanx
  • Relative indications
  • > 50% of articular surface involved
  • > 2 mm articular gap
39
Q

What are the soft tissue structures of the knee joint?

A
  • Anterior cruciate ligament (ACL)
  • Posterior cruciate ligament (PCL)
  • Medial collateral ligament (MCL)
  • Lateral collateral ligament (LCL)
  • Medical meniscus
  • Lateral meniscus
40
Q

What is the potential mechanism of injury of the ACL and what is positive for injury of this on physical examination?

A
  • Usually non-contact
  • Landed awkwardly
  • Felt pop
  • Immediate swelling

Physical exam

  • Lachman positive
  • Pivot shift positive
  • Large haemarthrosis
41
Q

What is the Lachman test?

A
  • Anterior draw test - holding the back of the calf and pulling forward
42
Q

What is the potential mechanism of injury of the PCL and what is positive for injury of this on physical examination?

A
  • Struck dash board
  • Fall with PF foot
  • Posterior pain

Physical exam

  • Posterior sag sign
  • Posterior drawer (90 flexion)
  • Quad active test
43
Q

What is the potential mechanism of injury of the MCL and what is positive for injury of this on physical examination?

A
  • Blow to the outside of the knee
  • Medial pain

Physical exam
* Valgus instability

44
Q

What is the potential mechanism of injury of the LCL and what is positive for injury of this on physical examination?

A
  • Varus injury
  • Lateral pain

Physical exam
* Varus instability

45
Q

What is the potential mechanism of injury of the meniscus and what is positive for injury of this on physical examination?

A
  • Mechanical symptoms (catching, locking)
  • Pain at the joint line
  • Delayed swelling

Physical exam

  • Joint line tenderness
  • McMurry positive
46
Q

What is the McMurray test?

A
  • For assessment of meniscus damage
  • Hand on knee
  • Other hand holding heel of the same leg
  • Flex and extend the knee joint with hands in this position
  • Positive test will elicit thud or click with pain
47
Q

How are ankle fractures classified?

A
  • Weber classification
  • Weber A - Infra-syndesmotic
  • Weber B - Trans-syndesmotic
  • Weber C - Supra-syndesmotic
48
Q

Why is classification of ankle fracture important?

A
  • Guides management
  • Essentially refers to the involvement of the syndesmosis
  • Widened syndesmosis (ligament at the bottom of the tibia and fibula) and unequal ankle space mortise indicates talo-shift which is an unstable ankle
49
Q

How should a patient be initially assessed for an ankle fracture?

A

Document all at presentation

  • Mechanism
  • Skin integrity
  • Assessment of circulation and sensation
  • Comorbidities that could influence treatment choice should be documented
  • E.G. pre-existing mobility impairment, diabetes mellitus, peripheral neuropathy, peripheral vascular disease, osteoporosis, renal disease, smoking and alcohol abuse
50
Q

Which management should be performed initially for ankle fracture?

A
  • Reduction and splinting for clinically deformed ankles

* Radiographs before and after reduction unless this will cause unacceptable delay

51
Q

What type of radiographs of the ankle should be taken to assess fracture?

A
  • Centred on the ankle
  • Include a true lateral and a mortise view
  • Additional radiographs of the whole leg are required when clinical examination suggests a more proximal fracture of the fibula (Maisonneuve injury)
  • CT may be indicated in defining fracture configuration in more complex patterns particularly where the posterior malleoulus is involved
52
Q

How should the ankle be managed after reduction and initial radiographs?

A
  • Neurovascular examination repeated and documented

* Adequate reduction must be confirmed by review of repeat radiographs and documented before transfer from ED

53
Q

What is the long term management of stable fractures of the ankle?

A
  • Fractures considered stable should be treated with analgesia, splinting and patients allowed to bear weight as tolerated
  • Further follow up may not be necessary
54
Q

How should patients with ankle fractures where stability is uncertain be managed?

A
  • Where stability is uncertain, patients should be reviewed within 2 weeks with further radiographs (weight bearing if possible) to confirm the position remains acceptable
55
Q

How should patients with unstable ankle fractures be managed?

A
  • Patients with unstable ankle fractures should be managed with early fixation (on the day or day after injury) is recommended for the majority of patients under 60 years when the ankle mortise is unstable
  • Use of external fixation may be rarely indicated in the presence of gross instability associated with soft tissue compromise
  • In patients over 60 years close contact casts are an option if reduction can be maintained
  • Surgery should aim to achieve reduction and stabilisation of the ankle mortise
  • The syndesmosis should then be assessed and stabilised if unstable
  • Intra-operative radiographs should be obtained to confirm reduction
56
Q

What is the guidance regarding weight bearing following ankle stabilisation?

A
  • Most patients should be allowed to bear weight as tolerated in a splint or cast unless there are specific concerns regarding the stability of the fixation or contraindications such as peripheral neuropathy or particular concerns about the status of the soft tissues
57
Q

What is the management of follow up following ankle fracture?

A
  • After surgery follow up in fracture clinic within 6 weeks of surgery to detect complications and confirm maintenance of reduction on radiographs
  • Thromboprophylaxis according to local guidelines
  • All patients should receive information regarding expected functional recovery and rehabilitation including advice about return to normal activities such as working and driving
  • Mechanism should be in place for patients to return to fracture service if progress is not as anticipated
58
Q

How should a pelvic fracture with suspected active bleeding be managed?

A
  • Apply a pelvic binder preferably pre-hospital
  • Patients with suspected pelvic fracture with signs of haemodynamic instability should be transported directly to major trauma centre
  • All patients require IV tranexamic acid as soon as possible, ideally within 1 hour of injury
  • In presence of haemodynamic instability, patients should be urgently resuscitated using blood products according to the major haemorrhage protocol
59
Q

What imaging should be done on patients with suspected pelvic fractures from high energy trauma?

A
  • CT scan with IV contrast including head, chest, abdomen and pelvis on admission
  • Should include a head to toe scanogram
  • All patients with blunt poly trauma undergoing damage control laparotomy should have imaging of the pelvis before surgery (x-ray or CT scan)
  • All patients should have a pelvic binder in situ during surgery and this should not be removed for a post binder pelvic x-ray until the patient is haemodynamically stable
60
Q

How should patients with active bleeding from the pelvis who do not respond to resuscitation be managed?

A
  • Surgical packing of the pelvis or interventional radiology with selective embolisation of active arterial bleeding vessels
  • MTC should have a clear protocol in place for this
61
Q

How should patients be assessed following removal of the pelvic binder?

A
  • All patients should have post binder x-ray after resuscitation because even in the presence of a negative CT scan a well applied binder can mask a catastrophic pelvic ring injury - should be removed within 24 hours of injury
62
Q

What is the early management of pelvic fractures?

A
  • External fixation or traction for displaced vertical fractures should be considered when early definitive surgery cannot be performed
63
Q

When should definitive management of the pelvic fracture be performed?

A
  • Reconstruction of the pelvic ring should be performed within 72 hours of the stabilisation of the patients physiological state if associated injuries allow
64
Q

How are acetabular fractures classified?

A
  • Letournel classification

* Classified as 5 elementary and 5 associated fracture patterns

65
Q

What are the 5 elementary Letournel acetabular fracture patterns?

A
  • Posterior wall
  • Posterior column
  • Anterior wall
  • Anterior column
  • Transverse
66
Q

What are the 5 associated Letournel acetabular fracture patterns?

A
  • Associated both column
  • Transverse and post wall
  • T shaped
  • Anterior column or wall and post hemitransverse
  • Posterior column and posterior wall
67
Q

What is the name of the classification of pelvic ring fractures?

A
  • Young and Burgess
  • Includes 3 basic mechanisms:
  • Anteroposterior compression
  • Lateral compression
  • Vertical shear

(and combined)

68
Q

What are the different features of the anteroposterior compression fractures within the Young and Burgess classification system?

A
  • APC I: Stable
  • Pubic diastasis <2.5 cm
  • APC II: Rotationally unstable, vertically stable
  • Pubic diastasis >2.5 cm
  • Disruption and diastasis of the anterior part of the sacroiliac joint, with intact posterior sacroiliac joint ligaments
  • APC III: Equates to a complete hemi-pelvis separation (but without vertical displacement); unstable
  • Pubic diastasis >2.5 cm
  • Disruption-diastasis of both anterior and posterior sacroiliac joint ligaments with dislocation
69
Q

What are the different features of the lateral compression fractures within the Young and Burgess classification system?

A
  • LC I: stable
    oblique fracture of pubic rami
    ipsilateral anterior compression fracture of the sacral ala
  • LC II: rotationally unstable, vertically stable​
    fracture of pubic rami
    posterior fracture with dislocation of the ipsilateral iliac wing (crescent fracture)
  • LC III: unstable
    ipsilateral lateral compression (LC)
    contralateral anteroposterior compression (APC)
70
Q

What are the different features of the vertical shear fractures within the Young and Burgess classification system?

A

Unstable, associated visceral injuries.

- Vertical displacement of hemi-pelvis, pubic and sacroiliac joint fractures

71
Q

What are the different features of the combined fractures within the Young and Burgess classification system?

A
  • Stability depends on the individual components of this injury.
  • Complex fracture, including a combination of anteroposterior compression (APC), lateral compression (LC), and/or vertical shear (VS)
72
Q

What is pubic diastasis?

A
  • Separation of the pubic symphysis without concomitant fracture
73
Q

What is the Ilizarov frame?

A
  • A circular frame used in orthopaedics to stabilise fractures with the following core biological principles
  • Minimal disturbance of bone and soft tissues
  • Delay before distraction
  • Rate and rhythm of distraction
  • Site of lengthening
  • Stable fixation of the external fixator
  • Functional use of the limb and intense physiotherapy
74
Q

What is distraction osteogenesis?

A
  • Distraction osteogenesis is also called callus distraction, callotasis and osteo-distraction, it is a surgical process used to reconstruct skeletal deformities and lengthen the long bones of the body
  • A corticotomy is used to fracture the bone into two segments, and the two bone ends of the bone are gradually moved apart during the distraction phase, allowing new bone to form in the gap
  • When the desired or possible length is reached, a consolidation phase follows in which the bone is allowed to keep healing
  • Distraction osteogenesis has the benefit of simultaneously increasing bone length and the volume of the surrounding soft tissues
75
Q

In brief, how is Ilizarov frame surgery performed?

A
  • Wires of 1.5 mm or 1.8 mm diameter are passed percutaneously through the skin through bones by means of a drill
  • Protruding ends of these are then fixed to rings with special wire fixation bolts
  • The rings in turn are connected and fixed to one another by threaded rods
  • Once it is fixed, the ilizarov frame affords a stable support to the affected limb
  • A corticotomy is then performed, it is an osteotomy where the periosteum of the bone is preserved
  • Adjustments in the rods produce compression or distraction as desired between the bone ends, and simultaneously deformities are also corrected
  • Ring fixator is removed at the end of the treatment
76
Q

What are the indications for Ilizarov frame surgery?

A
  • Limb lengthening
  • Deformity correction
  • Infected non-unions
  • Congenital pseudo-arthrosis
  • Treatment of joint contractures e.g. resistant congenital talipes, post burn contractures, post traumatic stiffness
  • Fixation of complex fractures
  • Bone transport and osteomyelitis (treatment of missing bone in the limb)
  • Arthrodesis (fusion or joining of two bones across a joint)
  • Peripheral vascular disease e.g. thrombo-angitis obliterans