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interpretation > lft interpretation > Flashcards

lft interpretation Flashcards

1
Q

what is seen in acute hepatitis ?

A

ALT/AST in 1000s

mild ALP

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2
Q

in chronic hepatitis?

A

ALT/AST in 100s

decreased bilirubin

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3
Q

cholestatic picture

A

ALP 100S
ALT/AST mildly raised
increased Bilrubin

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4
Q

alcohol

A

decreased platelet/Hb, increased MCV and gama GT

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5
Q

what is specific to liver

A

ALT

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6
Q

what can AST be seen in

A 
liver
heart 
muscles 
kidney 
pancreas
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7
Q

marked increases (1000s)

A

drug induced hep
liver ischamea
hepatitis viral A, B E EBV CMV

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8
Q

modest increase

A

AIH
chronic
alcohol

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9
Q

mild increased

A

HCC
WILSON HAEM
NAFLD
CIRRHOSIS

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10
Q

ratio and cause

A

ALT>AST - cHRONIC LIVER DISEASE

ast/alt in establsshed cirrhosis - or in ALD

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11
Q

alp SEEN WHERE

A

SPECIFIC TO BILARY DUTS AND BONE

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12
Q

what can be ued to confirm source

A

ygt can differentiate bebtwene born and bilary cause

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13
Q

marked increase

A 
PSC
PBC
gall stones
pancreatic ca
drugs
4x normal
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14
Q

moderate increase

A

hepatitis
cirrhosis
hcc

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15
Q

when can ygt be raised

A

enzyme inducing drugs
alcohol
to confirm ALP origin of disease being liver origin

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16
Q

wwhen albumin is low how can protein differentiate

A

increased with protein -myeloma
decrease protein -reduced protein intake,, alcohol, cirrhosis, skin loss etc
with normal protein=infection

17
Q

what can raised INR indicate

A

liver disease
anticogultation
vitamin k deficiency
DIC

18
Q

list cause of all markers:

anti smooth muscle 
anti mitochondrial
anti liver kindey microsomal
ant nuclear
AFP 
ferritin and trasnferin saturation
coper and crulpaslim 
fasting glucose and lpids
anti antitrypsin 
immunoglobulins 
igM
igA
igG
A 
anti smooth muscle  AIH
anti mitochondrial  PBC
anti liver kindey microsomal - AIH hep c and d drug induced hepattis
ant nuclear  - AIH SLE
 AFP  -HCC
ferritin and trasnferin saturation -haemchromatosis
coper and crulpaslim  -wilsons
fasting glucose and lpids -nAFLD
anti antitrypsin (AA defieincy) 
immunoglobulins 
igM PBC
igA ALD
igG - AIH
19
Q

what is bilrubin

A

brbeak down of Hb into globulin and haem (which is broken down nto bilirubin and excreted in bile after coguaiton in liver

20
Q

causes of unconjugated hyperbirubunia

A

increased rcc breakdown
impaired hepatic uptake
impaired conjugation glberts, jaundice neonatal

21
Q

causes of conjugated

A

hepatocellular dysunction

impaired hepatic secretion

interpretation (2 decks)

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