Levels of Consciousness Flashcards

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1
Q

Ao1: What is Restoration Theory?

A

This was created by Oswald and is a dual function theory about why we sleep. It theorises that we have slow wave sleep (SWS) for the purpose of body repair, as during SWS a growth hormone is produced, and that rapid eye movement sleep (REM) is for the purpose of brain restoration.

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2
Q

Ao1: What 5 predictions come from restoration theory?

A

1) A lack of sleep will cause deficit in cognitive functioning.
2) We will have rebound after lack of sleep, depending on what type of sleep you missed out on.
3) After brain injury or surgery you will have increased REM sleep.
4) After illness or physical injury you will have increased SWS sleep.
5) After exercise you will sleep for longer.

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3
Q

Ao2: What happened to Peter Tripp?

A

He didn’t sleep for 201 hours and during this time he had hypnagogic hallucinations and was incredibly aggressive. He then slept for 24 hours after this and was never the same again.

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4
Q

Ao2: What does Peter Tripp suggest about sleep?

A

Tripp had permanent deficit in cognitive functioning which has lasted for the rest of his life. This shows how important sleep is and how a lack of it can cause permanent problems for our mental health.

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5
Q

Ao2: How does Randy Gardener contradict the conclusion from Peter Tripp?

A

Gardener didn’t sleep for 11 days and afterwards only slept for 14 hours and had no serious deficit in cognitive functioning. This completely contradicts Tripp as it suggests that you can have lack of sleep without a deficit.

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6
Q

Ao2: What is a problem with both Peter Tripp and Randy GArdener?

A

These are both case studies, so only done on one person. This means the conclusions cannot be generalised to the general population as we do not know that everyone would react the same way to both of these.

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7
Q

Ao2: What did Shapiro et al find?

A

They found that marathon runners slept for an extra hour after running a marathon the same day.

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8
Q

Ao2: What does Shapiro et al’s study suggest about sleep?

A

That when we exercise we do need more sleep for body and brain restoration. So restorations predictions may be correct.

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9
Q

Ao2: What did Horne and Minard find and how does this contradict Shapiro et al’s study?

A

They found that when normal people completed exhausting tasks, they did not have any extra sleep they just fell asleep faster.

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10
Q

Ao2: What is good about Horne and Minard’s study?

A

It is far more representative than Shapiro et al as they only looked at marathon runners who are particular types of people who do training like this every day. The people horne and Minard chose should have been more tired as they are not used to the exercise.

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11
Q

Ao2: Why is Shapiro’s study still valid?

A

Marathon runners are going to be a lot more competitive than people doing exhausting tasks which means they would have put a lot more effort and energy in. Running a marathon is also a lot more tiring than an exhausting tasks, therefore this still supports the restoration theories prediction.

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12
Q

Ao1: What is Evolutionary theory?

A

This is based on the process of natural selection to explain why we sleep. This is when an organism has a characteristic which is advantageous in avoiding selection pressures. This helps the organism to survive and pass on the advantageous characteristics to their offspring.

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13
Q

Ao1: what three predictions come from evolutionary theory?

A

1) Sleep is for the conservation of energy.
2) Sleep changes based foraging requirements.
3) Sleep is for predator avoidance.

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14
Q

Ao2: What did Zeppelin and Rechtshaffen find?

A

They found that small animals slept for longer, this would be because they have a higher metabolic rate and therefore need to sleep for longer to conserve their energy.

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15
Q

Ao2: What animal contradicts Zeppelin and Rechtshaffen?

A

The sloth as it sleeps for an average of 20 hours per day and is a large animal. However, it does not have a predator, this shows that the relationship between conservation and predators is a complex one.

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16
Q

Ao2: What did Capellini et al find?

A

Capellini et al did a more a more controlled study or Zeppelin and Rechtshaffen and found a negative correlation between metabolic rate and sleep.

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17
Q

Ao2: How does Capellini et al contradict Zeppelin and Rechtshaffen?

A

Capellini et al found something very different to Z and R which suggests that maybe we are wrong to say that sleep is evolutionary to conserve energy.

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18
Q

Ao2: What is a problem with Capellini et al’s research?

A

It probably should have looked at total hours of non-REM sleep instead of total hours of sleep because of the fact that REM sleep is very tiring as you can see from EEG scans.

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19
Q

Ao2: How long do animals who are exposed usually sleep for and why?

A

They usually sleep for a small amount of time as they need to be alert at all times to avoid predators.

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20
Q

Ao2: What did DeCoursey discover about predator avoidance and sleep?

A

DeCoursey removes the SCN from chipmunks and introduced them to the wild. She found they they were slowly killed by Weasels, their predator, as they were no longer had a circadian rhythm so the Weasels were better able to hear them in their burrows.

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21
Q

Ao2: What does DeCoursey’s investigation suggest about sleep and predator avoidance?

A

That for some animals it is definitely and advantageous behaviour to sleep as it helps them to avoid predators.

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22
Q

Ao2: What evidence contradicts the prediction of predator avoidance?

A

Animals who live socially do not sleep any longer even though their is a reduced level of danger from predators. This makes us realise the the relationship between sleep and predators is very complex and maybe evolutionary theory is far too reductionist to explain sleep on its own.

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23
Q

Ao2: Is restoration theory able to explain the complexities of sleep on its own?

A

It does do a very good job but sometimes sleeping patterns do not coincide with the predictions made of restoration theory.

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24
Q

Ao2: Is evolutionary theory able to explain the complexities of sleep on its own?

A

No, it is far too reductionist, we can clearly see that the sleep and the predictions of evolutionary theory are not as clear cut as that they do intermix.

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25
Q

Ao2: What did Horne propose for theories of sleep?

A

Horne combined both restoration theory and evolutionary theory by creating two types of sleep. Core is SWS sleep and is required for essential body and brain processed. Optional is REM sleep which happens when conserving energy or during unproductive hours.

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26
Q

Ao1: What is Insomnia?

A

Insomnia is described as a changing of sleeping pattern over a certain period of time.

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27
Q

Ao1: What is chronic Insomnia?

A

This is when your sleeping pattern has changed for a month or more.

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28
Q

Ao1: What is transient Insomnia?

A

This is when your sleeping pattern has changed for a temporary amount of time, usually about 2 to 4 weeks.

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29
Q

Ao1: What is secondary insomnia?

A

This is when the insomnia is a result of something in your life, for example you may have insomnia if you are a shift worker, or if the conditions in your house are not good for sleeping in. Insomnia is a symptom.

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30
Q

Ao1: What is primary insomnia?

A

It is not caused by anything in particular, it is not a symptom of something it is just there. It may have had a cause at one point but now that cause has gone.

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31
Q

Ao1: What was Nofzinger et al’s explanation?

A

Nofzinger used PET scans to find there was an overactivity in the brain stem, the hypothalamus and the basal forebrain and they believed this to be the cause of the insomnia.

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32
Q

Ao2: What is good about Nofzinger et al’s findings about insomnia?

A

They have used a scientific method which means that the findings are objective. We know that Nofzinger hasn’t just made his theory up as he has the PET scans to support him.

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33
Q

Ao2: What is a problem with validity arises from Nofzinger et al’s research?

A

It was conducted in a slap lab which will have been an unnatural environment for the participants. This means that it is likely that the insomnia could have been caused by being in this unnatural environment, therefore reducing ecological validity.

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34
Q

Ao2: Why is it hard to make a conclusion from Nofzinger et al’s research into insomnia?

A

Because we don’t know what has actually caused this overactivity in the brain. Is it genes or the environment? Did the insomnia cause the overactivity or did the overactivity cause the insomnia?

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35
Q

Ao1: What was Gontzas et al’s explanation and finding’s?

A

He found high levels of the stress hormones ACTH and cortisol and believed this was the cause for the insomnia.

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36
Q

Ao2: What is good about Gontzas et al’s research?

A

Gonztas used a matched pairs design which meant that participant variables would have been eliminated as they are matched with another person. This also gets rid of things like order effects so the participants cannot work out what the researcher is trying to find.

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37
Q

Ao2: What is a problem with Gonzta’s research?

A

They only had a sample of 11 participants which is very limited therefore making it hard to generalise to the general population as we don’t really know if we would find the same.

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38
Q

Ao2: What practical application has raised from Gonzta’s research?

A

We ended up making Tremazepam which is a relevant used to help insomniac’s.

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39
Q

Ao1: What is narcolepsy?

A

This is a disabling disorder of sleep regulation which affects the control of wake and sleep. There is often an intrusion of REM into wakefulness.

40
Q

Ao1: Between what ages does narcolepsy start?

A

15 and 30.

41
Q

Ao1: What are the symptoms of narcolepsy?

A

Cataplexy, paralysis, hypnagogic hallucinations, excessive sleepiness.

42
Q

Ao1: What was Thanickel et al’s explanation and findings?

A

That there is a mutation to the gene which codes for receptors on the neurotransmitter hypocretin which is found in the hypothalamus (part of the brain which controls wakefulness). They found a 85-95% reduction in narcoleptics.

43
Q

Ao2: What is a problem with Thanickel et al’s research?

A

We do not know if this mutation is passed down through genes or if it is just a problem with DNA replication.

44
Q

Ao2: How does Guillment et al support Thanickel et al?

A

They found that there is a 40% chance of 1st degree relatives of people with narcolepsy developing the disease themselves which shows us that maybe this mutation is passed down through genes.

45
Q

Ao2: What piece of evidence contradicts Guillment et al and Thanickel et al?

A

It has no been discovered that Narcolepsy does not run in families which suggests that it cannot be a genetic cause. However it could be possible that the people in their families just haven’t had the right environmental trigger to set the Narcolepsy off.

46
Q

Ao1: What was Mignot et al’s explanation and findings?

A

The tested 4 narcoleptics and 12 non narcoleptics and found that this reduction in hypocretin was caused by a number of things like brain injury, diet and immune system problems.

47
Q

Ao2: What does Mignot et al’s findings suggest to us about Narcolepsy?

A

That it is a mixture of our genes and our environment that is the cause of the narcolepsy.

48
Q

Ao2: What are 2 problems with Mignot et al’s research?

A

There is a very small sample size of only 4 narcoleptics because it is such a rare disease. This makes it very difficult for us to generalise the findings to any other narcoleptics as we do not know that they would have the same problems. It is also only correlational. Mignot doesn’t actually know for sure that the reduction was caused by these things.

49
Q

Ao2: Does the environment come into play with the cause of Insomnia?

A

It is very hard to conclude anything with the research for insomnia as a lot of the research is inconclusive and fails to show a cause and effect relationship. It is very likely that it is controlled by biology considering tremazepam is a cure but we do not know what causes this change in biology.

50
Q

Ao2: Does the environment come into play with narcolepsy?

A

It is clear from the research that it is controlled by something in our biology, like the reduction in hypocretin which may control wakefulness. However there is a lot of evidence to say that it is started off by the environment. This is known as the diathesis stress model. The person may have a genetic predisposition to this gene mutation, but an environmental trigger must set this mutation off.

51
Q

Ao1: What is primary process thought to Freud?

A

This is innate and instinct driven thought which is pushed to our unconscious as it is unacceptable to our conscious mind and this is down through repression.

52
Q

Ao1: What does primary process thought cause us to do while we sleep?

A

Dream

53
Q

Ao1: What is the tripartite view of the mind?

A

This is the belief that our mind is split into 3 parts, the unconscious (id) the subconscious (super-ego) and the conscious (ego).

54
Q

Ao1: What is a dream to Freud?

A

They are an ego defence mechanism.

55
Q

Ao1: Does Freud think dreams have a meaning?

A

Yes.

56
Q

Ao1: What is dreamwork?

A

This involves a lot of complex processes like condensation and symbolism, to turn latent content (the real meaning of the dream) into manifest content (a more innocuous form, the dream itself.)

57
Q

Ao2: What did Solm’s do and find?

A

He conducted PET scans and found that the cortical area’s of the brain (to do with rational conscious thought) were inactive while the limbic areas (to do with memory and motivation) were active while we slept.

58
Q

Ao2: What does Solm’s research suggest about dreams?

A

That maybe they are an ego defence mechanism as we can see that our rational parts of the mind are inactive so it looks as the id is taking over which is driven by bad memories and motivation.

59
Q

Ao2: What is good about Solm’s research?

A

It has used a scientific measure to collect the findings which is very objective. This means that we can be sure that the conclusion isn’t just something that someone wants to be true.

60
Q

Ao2: What is a problem with Solm’s research and Freud’s theory of dreams?

A

It is completely unfalsifiable. There is no way of being able to prove this theory wrong in any way as we cannot actually see the id at work turning this latent content into manifest content, we just have to take Freud’s word for it meaning it could very easily be wrong.

61
Q

Ao2: Why did Braun et al hate Freud’s theory?

A

He believed that it was completely illogical to say our brain was going through dreamwork while the rational parts of our brain were inactive. This is because dreamwork involves many complex processes to turn latent content into manifest content which Braun thinks would be impossible. This means that there cannot be any meaning to our dreams after all if we cannot even do dreamwork.

62
Q

Ao1: What is Activation-Synthesis?

A

This was a theory of why we dream created by Hobson and McCarley. They stated that random electrical signals are fired from our brainstem (activation) and sent to our basal forebrain. Our forebrain then interprets these signals as this is its natural instinct and also is what happens during the day. (synthesis). Without doing this we would be brain dead, therefore dreaming is just a process which keeps us alive while we sleep.

63
Q

Ao2: What did Braun do and find?

A

They conducted PET scans which found the brain stem was active during REM sleep which suggests this is because it is sending electrical signals to the forebrain.

64
Q

Ao2: What piece of evidence contradicts Braun’s findings?

A

Dreaming is also associated with non-REM sleep. PET scans are very different during Non-REM which makes you think, how could dreams just be a result of random firings of the brain if we still dream when this does’t happen. There must be another reason for why the brain stem is active.

65
Q

Ao2: What did Solms do and find which refutes activation-synthesis theory?

A

Solms conducted PET scans on people with damage to their forebrain. This meant that they could not go through REM however they still report dreaming. This makes the link between an active brain stem and forebrain and the activation-synthesis theory a lot more obscure as it seems unlikely that these have anything to do with dreams if we can still dream while they are not working. Maybe they do actually have meaning.

66
Q

Ao2: Do dreams have meaning?

A

It is far to hard to say yes to this because of the fact theories which say they do are always unfalsifiable as we can not actually see them having a meaning etc.

67
Q

Ao2: Are dreams just random firings of the brain?

A

Definitely not, most the evidence for this is heavily contradicted by other evidence meaning we cannot conclude this safely at all. Maybe it would be better to ask if dreams have a function as both Freud and Hobson and McCarley would agree with this statement.

68
Q

Ao1: What is net-dissociation theory?

A

This is a state theory of hypnosis created by Hilgard. It states that the hypnotists takes control of the executive ego and forms an amnesiac barrier between the two control system: primary consciousness and hidden observer. This means they cannot communicate. The hidden observer is aware of what is going on and does what the hypnotist says and the primary consciousness thinks this is all real and does not know why it is doing what it is doing.

69
Q

Ao1: What is the central executive?

A

This is the control system which controls our awareness.

70
Q

Ao2: What did Hilgard do and find? (ice-water)

A

He had a group of hypnotised people and non hypnotised people and asked them to place their hand in ice cold water and rate their pain. He found that when he asked the hypnotised people if there was another part of them which could experience pain, they reported two levels of pain, one being high and the other being low.

71
Q

Ao2: What does Hilgard’s ice water study suggest about hypnosis?

A

That the hidden observer is responding without awareness and cannot communicate with the primary consciousness, therefore only answers when asked directly, showing this amnesiac barrier has been formed.

72
Q

Ao2: What is a problem with Hilgard’s ice water study?

A

Pain is very subjective meaning that this is an invalid measure as we don’t know how painful it actually is for them, everyone has a different threshold of pain. It also used independent measure design meaning that participant variables would have affected the results greatly. It would have been much more valid if the the same people were used in each condition as they would have had the same threshold of pain.

73
Q

Ao2: What did Hilgard do a and find? (deaf boy)

A

He hypnotised a boy in his class to believe he was deaf. He then asked the boy to hold up a finger if there was another part of him that could hear. The boy raised a finger but was essentially still deaf.

74
Q

Ao2: What does Hilgard’s deaf boy study suggest about hypnosis?

A

That the hidden observe is monitoring everything but only able to respond when asked directly. It shows clearly that the hidden observer is making the body do something and primary consciousness has no idea why. It clearly shows the amnesiac barrier has been formed.

75
Q

Ao2: What is a problem with Hilgard’s deaf boy study?

A

The boy quite easily could have just been complying with Hilgard out of pressure of trying to make his teacher look good. We know from Milgram’s study of obedience that authority can be very daunting for some people and make them do things they do not want to do. This is also a limited sample of one person meaning we cannot generalise to everyone else as we have no idea that they would act in the same way.

76
Q

Ao1: What is social cognitive theory?

A

This is a non-state theory about hypnosis. This states that it is not an altered state of consciousness just people complying. They are responding to expectations. This involves the ESC process. This is when the person develops expectations, employs a strategy and then complies. This theory states that hypnosis is just basically a mixture of demand characteristics and extreme compliance.

77
Q

Ao2: What did Orne do and find?

A

He told people before they were hypnotised that a common feature of hypnotism is stiffening of the hand, even though it is not. He then hypnotised these people and found that 55% of them stiffened their hand compared to none in the control.

78
Q

Ao2: What does Orne’s study suggest about hypnotism?

A

This suggests that the people were just complying to expectations they had before the hypnosis and that it isn’t an altered state otherwise they wouldn’t have stiffened their hand.

79
Q

Ao2: What are two problems with Orne’s research?

A

What about the other 45%, why did they not stiffen their hand? Where they in an altered state of consciousness?? Also, how do we know if the hypnotist has the power to get people to do things said before a hypnotic state has been induced. We have no evidence to say people don’t follow commands given before hypnosis.

80
Q

Ao2: What did Sarbin and Slagle find in 1972?

A

That there is no change in brain activity when in a hypnotic state, this suggests there is no altered state and proves net-dissociation wrong as this theorises that there is a change in the brain.

81
Q

Ao2: What did Kosslyn find in 2000?

A

That there is a change in brain activity when participants were asked to imagine colours on a grey page. This suggests that there may be a different state during hypnosis and change in the brain as this activity is similar to hypnosis. This is better than Sarbin and Slagle because it is much more modern and therefore uses better technology to look at brain activity.

82
Q

Ao2: What is a problem with Kosslyn?

A

We do not know that imagining something is the same as a hypnotic state therefore we cannot draw a link between the two.

83
Q

Ao2: Is hypnosis a unique sate of consciousness or is it just people complying to orders?

A

There is a lot of conflicting evidence to this statement which makes it inconclusive. Dreams have a similar report to hypnosis and cannot be verified suggesting we will probably never know. However, maybe it is all to do with how susceptible someone is as to whether they comply.

84
Q

Ao1: What is a circadian rhythm?

A

This is a biological rhythm which occurs every 24 hours and example of this is the sleep/wake cycle.

85
Q

Ao1: What endogenous pacemaker controls the sleep/wake cycle?

A

The SCN seems to control it. This is a bundle of light sensitive nerves found in the hypothalamus above the optic nerve. The optic nerve picks up the light which travels down the optic nerve to the SCN. This then sends messages to the pineal gland. The pineal gland then produces melatonin which prohibits the production os serotonin. Serotonin is what makes you sleepy. This process is reversed in daylight.

86
Q

Ao2: What did Siffre do and find?

A

They investigated a man who went into a cave for 6 months with only a miners lamp, no other exogenous zeitgabers. They found that the mans sleep/wake cycle increased to about 25 hours instead of 24.

87
Q

Ao2: What does Siffre’s investigation suggest about the impact of endogenous pacemakers and exogenous zeitgabers?

A

That our sleep/wake cycle is clearly regulated by the exogenous zeitgaber of light but that we do not need light to have our cycle. It seems to show that the endogenous pacemaker we have has a larger impact.

88
Q

Ao2: What are 2 problems with Siffre’s research?

A

It is a case study which means we cannot generalise the results to the general population as we do not know we would act in the same way to them. It could also be that the mans loneliness caused a lack of serotonin which is why his cycle changed as it may have been harder for him to sleep. This would suggest that endogenous zeitgabers have a much larger impact.

89
Q

Ao2: What did Morgan do and find?

A

They bred mutant hamsters who had a sleep/wake cycle of 20 hours. They then transplanted their SCN’S into hamsters with a normal sleep/wake cycle. These hamsters ended up having the mutant cycle.

90
Q

Ao2: What does Morgan’s study suggest about the sleep/wake cycle?

A

That it is completely down to the endogenous pacemaker which clearly has a much bigger impact, otherwise the normal hamsters would still have a normal 24 hour sleep wake cycle.

91
Q

Ao2: What is a problem with Morgan’s study?

A

It is conducted on animals which means we cannot generalise the results to humans as we do not know that we would react in the same way as the hamsters did as we are qualitatively different to them.

92
Q

Ao1: What is an infradian rhythm?

A

This is a biological cycle that is longer than 24 hours, for example the menstrual cycle.

93
Q

Ao1: What controls the menstrual cycle?

A

This is controlled mainly by two hormones called oestrogen and progesterone which are thought to be controlled by the pineal gland. Periods usually start during the winter for most women meaning they may also be controlled light and temperature.

94
Q

Ao2: What did Reinberg do and find?

A

They observed a woman who went into a cave for 3 months with no other exogenous zeitgabers than a miners lamp and found that her menstrual cycle shortened by a lot.

95
Q

Ao2: What does Reinberg’s research suggest about the menstrual cycle?

A

That it is heavily influenced by exogenous zeitgabers and has very little impact from edongenous pacemakers.

96
Q

Ao2: What are 2 problems with Reinberg’s research into the menstrual cycle?

A

It is a case study which means we cannot generalise it to the general population as we do not know that we would act in the same way. The lack of people could have caused the woman stress and we already know that stress can affect the menstrual cycle, therefore it is hard to draw a cause and effect relationship here.