Lessons 01 - 05 Flashcards
Aggression definition
Range of behaviours that can result in both physical and psychological harm to oneself, other people or objects
Proactive aggression
Based on the term ‘cold blooded’
Planned method
Reactive aggression
Based on the term ‘hot blooded’
Unplanned method angry, impulsive
What are neural mechanisms?
Structures (neurons, neural circuits, regions of the brain) or substances (neural transmitters, hormones) that regulate aggression
Neural explanations
The limbic system - how a person responds to environmental threats
Involves the amygdala, hypothalamus and hippocampus.
Is hierarchical (signals passed from lower systems to higher systems)
The limbic system connects to the cingulate gyrus (responsible for attention on emotionally significant events), and the prefrontal cortex (involved in forward planning)
The hypothalamus regulates the autonomic nervous system, which regulates responses to emotional circumstances. Damage = inappropriate aggressive responses.
The amygdala is responsible for attaching emotional significance to sensory information.
Damage to the prefrontal cortex will reduce the inhibition of the amygdala, resulting in higher levels of aggression
Serotonin
It slows down and calms neural activity (inhibits the firing of the amygdala)
Low levels mean people can’t control their impulsive and aggressive behaviour. Lower levels affect our response to external stimuli, meaning a person can become aggressive easily.
Evaluation of neural mechanisms in aggression
(+) Research support. Kluver and Busy (1939) removed the main areas of the limbic system from Rhesus monkeys. They showed an absence of emotional, motor and vocal reactions, and also lost the social understanding (tried to fight the more dominant members). They became more aggressive
(+) Technological advances. Wong et al. (1997) did MRI scans of 19 violent males, the volume of the amygdala was significantly smaller.
(-) BUT small sample, only done with men (gender bias, specifically beta bias)
(+) Supports the role of neurotransmitters. Ferrari et al. (2003) let 11 male rats fight with each other for 10 days at a specific time. On the 11th day, they didnt, and found that dopamine levels increased by 65% and serotonin levels were reduced by 35%. Changed the rats brain chemistry.
(-) BUT cause and effect? Lowers the validity.
(-) Research uses animals. Can the findings be extrapolated? Despite having similar neural structures, we don’t know if processes are the same
(-) Mann et al. (1990) gave a drug (dexfenfluramine, which reduces serotonin levels) to 35 adults, who then did a questionnaire. Aggression rose amongst males, not females, showing gender differences and the problems of beta bias
Hormonal mechanisms
Testosterone
Male sex hormone, promotes muscle strength, and is responsible for the sex drive. Thought to influence aggression from young adulthood.
Sapolsky (1998) found that removing the source of testosterone resulted in lower levels of aggression.
Men are more aggressive than women (men have more testosterone)
Ages 21-35 is when testosterone concentrations are at the highest, and there is an increase in male-on-male aggression.
Carre and Olmstead (2015) found that testosterone concentrations fluctuate rapidly in the context of changes to the social environment. Changes to levels increases amygdala reactivity.
Hormonal mechanisms
Progesterone
A female ovarian hormone that can increase mood. Is a natural antidepressant. Levels vary during the ovulation cycle.
Ziomkiewicz et al. (2012) found a negative correlation between progesterone levels and self-reported aggression.
Low levels of progesterone are linked to increased aggression.
Evaluation of hormonal mechanisms in aggression
(+) Research support about testosterone. Wagner (1979) castrated mice (removed testes) and their aggression levels went down.
(+) Gianmanco et al. (2005) agrees. In male Rhesus monkeys, there is an increase in testosterone and aggression levels during mating season.
(-) Lack of generalisation in animal studies to humans. Also, studies may only provide support for correlations, nor for cause and effect. Does not confirm if testosterone causes aggression
Genetic factors in Aggression
Genetics
Genes consist of DNA strands, that produce ‘instructions’ for features. Genes are inherited (transferred from parent to offspring).
Most people have 23 pairs of chromosomes, male (XY) or female (XX). Early psychologists believed the genetic cause of aggression was the Y chromosome.
‘Super males’ have XYY. Court-Brown (1965) studied 314 super males, who all had an increased likelihood of aggressive behaviour (he said they should be hospitalised)
More recently, psychologists have used selective breeding in animals to support the idea that aggression is transmitted from parent to offspring
Genetic factors in Aggression
Twin studies
Coccaro et al. (1997) studied male adult twins.
Concordance rates:
Physical assault:
- MZ (100% DNA) = 50%,
- DZ (50% DNA) = 19%
Verbal assault:
- MZ = 28%
- DZ = 7%
Suggests a genetic component
Genetic factors in Aggression
Adoption studies
Rhee and Waldman (2002) carried out a meta-analysis of adoption studies of direct aggression and antisocial behaviour and found genetic influences accounted for 41%
Genetic factors in Aggression
Candidate genes
MAOA gene, responsible for the production of the protein monoamine oxidase, which allows the metabolising of noradrenaline, serotonin and dopamine
Too much adrenaline - hypersensitivity in the fight or flight response.
MAOA-L gene is a low-activity variant, which means people are likely to be aggressive.
Lea and Chambers (2007) found that 56% of Maori men had this (compared to 34% of Caucasians), and they are known for being ferocious warriors
DYSFUNCTIONAL MAOA GENE:
- High levels of dopamine = increased likelihood of feelings of reward when aggression is carried out
- Low levels of serotonin = lack of inhibition over impulsive behaviour
- High levels of noradrenaline = overreaction to perceived threats
Evaluation of the genetic factors in aggression
(+) Abundance of research. Coccaro et al (1997) and MZ/DZ twins study, supporting the role of nature in aggression
(+) Godar et al. did selective breeding by removing the MAOA gene in mice. These mice showed increased aggression and abnormal levels of serotonin, showing the relationship between genes and neurotransmitters. When the mice were given fluoxetine (to raise serotonin levels), their aggression levels went back to normal
(+) Brunner (1993) found that 28 violent criminals possessed the MAOA-L gene, further supporting the genetic explanation
(+) Stuart et al. (2014) found that 97 men who has been involved with severe domestic abuse had the faulty MAOA gene.
(-) Not all research supports the XYY gene causing aggression. Theilgaard (1984) found that XYY causes an increase in height, not in aggression, shows we cannot make generalisations as there is no link shown here
(-) Twin studies lack validity. MZ twins have 100% same DNA and are also reared the same way, and DZ twins may not be. Twin studies may be unintentionally measuring environmental influences also