lesson 8 Flashcards

1
Q

hunter gatherer diets

A

Diverse, rich in fiber and micronutrients.

Seasonal and varied depending on geography.

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2
Q

agricultural diets

A

Reliance on staple crops (e.g., wheat, rice, maize).

Lower protein, vitamin content; higher risk of deficiencies.

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3
Q

industrial diets

A

Highly processed, high-GI foods.

Increased sugar, fat, and sodium intake.

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4
Q

three compnents of foods

A

proteins, fats (triglycerides), cabohydrates

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5
Q

poteins

A

Coiled chains of amino acids, used for
building and maintaining tissues
* Seldom used for energy

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6
Q

fats (triglycerdes)

A

three long fatty acid molecules bound to a
glycerol
* either stored as energy in the liver or in
adipocytes (in muscle) or immediately
burned
* 1 gram of fat = 9 calories

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7
Q

carbohydrates

A

Two common types:
* Simple sugars: can be burned
immediately or stored as fat, dumped
into blood stream
* Glucose – starchy, not too sweet
* Fructose – very sweet,
metabolized by liver – excess
converted to fat
* Complex sugars (polysaccharides)
broken down by digestion into simple
sugars

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8
Q

insulin

A

hormone produced in
pancreas to regulate blood
sugar levels and delivery of
energy within body

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9
Q

type 2 diabetes

A

cells don’t
respond to insulin, leading to
overproduction of insulin – blood
sugars rise causing heart disease,
vision loss, kidney disease

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10
Q

carrdioovascular disease

A

atherosclerosis (plaque buildup in
arteries)

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11
Q

wrangham cooking hypothesis

A

Cooking enhances nutrient availability, reduces digestion energy costs.

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12
Q

Amylase gene (AMY1)

A

Higher copy number in populations with starch-rich diets.

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13
Q

lactase peristence (LCT gene)

A

Multiple alleles evolved independently in Europe, Africa, and Asia.

Allows adults to digest lactose, conferring survival and reproductive advantages in dairying societies.

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14
Q

FADS genes

A

Influence fat metabolism; selected differently in farming vs. herding populations.

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15
Q

TEE - total energy exppenditure

A

= Basal Metabolic Rate (BMR) + physical activity + thermoregulation + digestion.

Studies show that foragers (e.g., Hadza) and Westerners have similar TEE when controlled for body size.

Obesity likely stems from increased energy intake, not decreased activity.

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16
Q

high-gi foods

A

spike blood sugar → insulin resistance → type 2 diabetes.

17
Q

cardiovascular disease

A

linked to saturated fat, low fiber, and high sugar.

18
Q

foood industrialization

A

increased processing and refined sugar (e.g., HFCS), altering nutritional content.

19
Q

Nutritional Characteristics of Subsistence-level
(small-scale food producing) Human Societies

A

Fluctuations in food availability
* Marginal energy balance
* Limited availability of fat & simple carbohydrates* High level of Energy Expenditure & Physical Activity* No single ‘ideal diet’ – humans can eat almost
anything
* Basic tradeoff between protein fat / carbohydrates– Both, in certain context, can provide nutritious diet

20
Q

dogs and startch adaptation

A

Dogs also adapted genetically to changing
diet
Increased copy number in dog pancreatic
amylase AMY2B relative to wolves
More changes in expression later in the
starch digestion pathway

21
Q

triglycerrides

A

dietary fat found in
meats & dairy
* FADS1 and FADS2 involved in fatty acid
metabolism (plasma lipid and fatty
acid concentration)
* High triglycerides in blood: increased
risk of heart disease, stroke, metabolic
syndrome
* Most-selected allele (rs174546) >
decreased triglyceride levels
* “A critical component of adaptation to
different diets” (Mathieson, 2016)

22
Q

glycemic index

A

measure of how quickly carbohydrates boost blood sugar
- boom bust cycles
- variety, ripeness, cooking methods, processing, length of storage all influence GI

23
Q

fibre

A

encases cells that
contain sugars, delays
absorption, lowers GI –processing reduces fibre

24
Q

a shifting energy balance

A

Traditional interpretations:
Lower activity, greater
intake through recent
human past = obesity

25
BMR
Basal metaboolic rate- BMR typically comprises up to 75% of TEE and is positively correlated with body mass and fat free mass, with fat free mass being the strongest predictor
26
thermic effect of food
(TEF) is the increase in metabolism after a meal and accounts for approximately 10% of total energy expenditure.
27
PAL
Physical activity level (PAL): TEE for 24 hours expressed as a multiple of BMR,TEE/BMR for 24 hours.
28
type 2 diabetes
Characterized by the inability to produce enough insulin * Fat muscle and liver cells become less sensitive (resistant) to insulin * Normally, eating leads to blood glucose increase which causes pancreas to produce insulin that triggers tissues to draw glucose from blood * Resistant cells don’t draw glucose, causing blood levels to rise, trigger more insulin * Excess blood sugar can lead to heart disease, kidney failure, blindness, dementia
29
atherosclerosis
blood vessel walls harden and thicken with age, chronic inflammation, Due to movement of cholesterol and triglicerides – transported in bloodstream by proteins from the liver * Low density lipoproteins (cholesterol carriers) gets stuck on arterial walls * Trigger inflammation and white blood cells which further trap lipoproteins * Coagulates on and narrows arterial walll * arterial blockage can cause failure of region of the heart – Heart Attack * Narrow vessels = Hypertension – high blood pressure
30
double burden of malnutrition
Double burden: Coexistence of undernutrition and overnutrition within individuals, households, or populations. Stunted children with obese parents. Driven by food insecurity, globalization, urbanization. WHO emphasizes reforming food systems to address both forms of malnutrition.