Lectures 2, 3, & 4 Flashcards

1
Q

Layers of the LGN

A

Layers 1 and 2: Magnocellular (Y-type)
Layers 3-6: Parvocellular (X-type)
IC layers: Koniocellular (input)

Layers 1,4,6: contralateral eye
Layers 2,3,5: ipsilateral eye

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2
Q

Retinotopic mapping in visual cortex

A

This means that the visual field is represented in the brain in specific areas

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3
Q

P and M cells from LGN projection of the visual cortex

A

P cell layers project to layers 2, 3 and 4C in V1
M cell layers project to layers 4B and 4C

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4
Q

Ocular Dominance Columns

A

There is strong seperation in layer 4 of the visual cortex regarding columns in which one eye is more dominant.
The other layers have weaker seperation.

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5
Q

Pathological dominance of one eye (Amblyopia)

A
  • Squinting results in suppression of the input in one eye
  • If this happens during critical period in development, input from that eye occupies less space in V1, the OD columns of that eye become smaller.
  • After critical period this reduced representation remains and amblyopia remains.
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6
Q

Receptive field tuning in V1

A

In V1 the neurons are tuned to orientation and direction of motion.

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7
Q

Orthogonal organization components of primary visual cortex

A
  • Orientation columns
  • Ocular dominance columns
  • CO blobs

These combined are called hypercolumns

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8
Q

How elongated RF’s in V1 are constructed

A

They are constructed from LGN inputs that have their circular RF along a line in visual space.
This creates a Gabor filter which is a sinusoid combined with a Gaussian envelope.

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9
Q

Explanation for white/gold or black/blue dress

A

Colour is not strictly the wavelength but more so how the brain interprets the incoming wavelength.

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10
Q

Colour constancy

A

Perceived colours are not absolute wavelengths but are computed by the brain depending on the wavelengths of the surround. This happens at V4 where the receptive field is large enough to integrate numerous colour opponent signals and discount the illuminant

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11
Q

Achromatopsia

A

Also called cortical colour blindness. This happens when there is a lesion in V4 or V8

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12
Q

Apparent motion

A

When a stimulus goes off in one place and on in another. In this sense the neuron is a coincidence detector because it sort of sees this as one single thing moving.

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13
Q

Reichardt detector model for direction selectivity

A

Neuron in brain receives input from two cells that have spatially seperate RF’s. One of the cells has a delayed input. Only when the stimulus moves in the right direction the cell receives simultaneous input from both cells and will fire.

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14
Q

Direction of motion selectivity

A

V1, V3, MT
Plotted in a polar tuning curve

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15
Q

Aperture problem

A

Detecting motion through an aperture is ambiguous, many motion vectors can yield the same motion through an aperture.
V1 cells suffer from this problem.
Component cells have solved this problem because they encode the plaid motion direction.

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16
Q

Component and pattern cells

A

Component cells do not see real motion and are located in V1, V3a and MT
Pattern cells are tuned for the real perceived motion and are located in MT.

17
Q

MST (medial superior temporal area) neurons

A

Respond selectively to particular motion flow fields. These are often cause by self-motion

18
Q

Biological motion

A

Recognizing species, sex, mood from movement
The STS is selectively activated by biological compared to non-biological motion

19
Q

Akinetopsia

A

Motion blindness.
Caused by lesion in MT, MST, STS

20
Q

Colour processing brain areas

A

Wavelength: V1, V2
Colour constancy (V4)

21
Q

Motion vs colour

A

Motion wins from colour when both are being activated.

22
Q

Dorsal pathway vs ventral pathway

A

Dorsal pathway is Magno dominant
Ventral pathway is Parvo dominant

23
Q

Depth perception cues

A

Monocular:
- Perspective
- Size constancy
- Occlusion
- Cast shadows
- Areal perspective
- Texture
- Motion parallax

Binocular:
- Disparity

24
Q

Disparity

A

When you fixate on a point the eyes converge. Every other stimulus that falls within the same plane of depth (horopter) will be projected as equal distance

25
Q

Correspondence problem

A

There are multiple ways a point can be interpreted by the two eyes. In other words the point can be at different distances. When this problem is solved, binocular fusion is the result, which results in a 3D image.

26
Q

Strabismus

A

Misalignment of the eyes, due to congenital eye-muscle disorders or due to cranial oculomotor nerve disorder.

27
Q

Neurons tuned to specific disparities brain areas

A

V1, V2, V3/V3a
They can be tuned near, far, zero or inhibitory

28
Q

V1 disparity neurons other function

A

These neurons also code for disparity when no depth is perceived

29
Q

IT neurons

A

These neurons encode perceived depth and not just contrast disparity.

30
Q

Depth processing brain areas

A

Disparity: V1, V2, V3/V3A
Perceived depth: IT

31
Q

V2 neurons shape detection

A

These neurons are tuned for the orientation of illusory contours

32
Q

Lateral Occipital Complex (LOC)

A

Responds more strongly to shapes than to scrambled objects.

33
Q

Invariant object coding

A
  • LOC, fusiform and frontal gyri only show activity for identical objects
  • Right anterior and posterior fusiform gyri show activity for identical objects of different sizes
  • Left anterior and posterior fusiform gyri show activity for objects at different viewpoints
  • Broca’s area shows activity for objects within same class
34
Q

Shape coding brain areas

A
  • Orientation: V1, V2, V4
  • Complex shapes: V4, LOC
  • Real world shapes: IT
  • Invariant object coding
35
Q

Cortical areas where position is transformed from retinal to other reference frames

A

VIP, LIP, area 7a

36
Q

Gain modulation

A

VIP neurons change their response as function of eye position

37
Q

Parietal Reach Region (PRR)

A

Has neurons that respond to hand movement relative to eye position