Lecture 9 Flashcards

1
Q

There are two types of retinorecipient areas of the brain:

A

1) Image forming areas
2) Non-image-forming areas

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2
Q

Image-forming regions and examples

A
  • The brains representation of the visual field
  • Superior colliculus (SC) : coordination of reflexes and motor planning
  • Dorsal lateral geniculate nucleus (dLGN) : relays information about what and where things are in the visual field
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3
Q

Non-image forming regions and examples

A
  • Controls involuntary reflexes and brain state (arousal and emotions)
    -Suprachiasmatic nucleus (SCN): controls circadian rhythms
    -Medial Terminal Nucleus (MTN) / Nucleus of the Optic Tract (NOT): Optokinetic reflex, image stabilization
  • Olivary Pretectal Nucleus (OPN): pupillary light reflex
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4
Q

Visual pathway into the brain:

A

Optic nerve > Optic Chiasm > dLGN / SC > Visual Cortex

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5
Q

What are the two major visual pathways?

A

1) “Where” pathway (dorsal stream)
2) “What” pathway (ventral stream)

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6
Q

Ventral “what” pathway

A
  • Primary visual Cortex (Occipital Lobe): Edges, orientation, simple shapes
  • Visual Association Cortex (Temporal lobe): object and face recognition (and recognition of objects / places)
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7
Q

What causes lesions in the brain?

A

1) Stroke (ischemia)
2) Traumatic Brain Injury
3) Encephalitis (brain swelling from injury or infection)

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8
Q

Lesions along the visual pathway cause what deficits in the visual field?

A
  • Complete loss of vision
  • Central scotoma
  • Tunnel Vision

2) Bitemporal hemianopia
3) Homonymous hemianopia
4) Quadrantanopia
5-6) Quadrantanopia
with Macular sparing

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9
Q

Why may Macular sparing occur?

A

because the visual cortex that perceives our central vision receives blood from two different arteries.

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10
Q

Lesions to the primary visual area or visual associative areas is known as ____________________.

A

Cortical blindness

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11
Q

Types of visual agnosia:

A

Agnosia: inability to interpret sensations and hence to recognize things, typically as a result of brain damage.

1) simultagnosia
2) prosopagnosia

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12
Q

Simultagnosia

A
  • Balint’s syndrome
  • In this condition, patients can only recognize single object at a time. They fail to comprehend the entire visual scene.
    -a patient with simultanagnosia would be able to name objects in the picture but would not be able to explain what was going on.
  • Most scientists think that there is deficit or injury to the visuospatial processing centers in the parietal cortex (”where” pathways).
  • Some cases have been observed in patients with Alzheimer’s Disease
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13
Q

Prosopagnosia

A
  • “face blindness”
  • Loss of face perception. Patients can see faces but are unable to recognize the faces they see. This includes recognition of one’s own face.
  • Cause by defects Fusiform Face Area (FFA) due to injury (stroke) or congenital (birth defect)
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14
Q

Hemispatial Neglect

A
  • visual neglect
    -In visual neglect, the patient lose attention and awareness to one-side of their visual field.
  • Most cases are the result of stroke in the contralateral parietal or frontal cortex.
  • This affects the dorsal visual pathway.
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15
Q

Synesthesia

A

-A phenomenon where one cortical/sensory pathway is connected to secondary cortical/sensory pathway.

Examples:
* Seeing colors with numbers (Grapheme-color synesthesia)
* Seeing colors with different tones (Chromesthesia)
* Increased grey matter found in Fusiform gyrus (colors) and V4
(number, letter area). Possibly increase in connectivity between
these areas.

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