Lecture 5 Flashcards

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1
Q

List the sensory hierarchy

A
Association cortex 
Secondary sensory cortex
Primary sensory cortex
Thalamic nuclei
Receptors
The process begins at the receptors and each layer adds more information or analysis, making the signal more specific and complex.
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2
Q

How is the sensory hierarchy divided?

A

It’s divided into sensation and perception. Sensation involves the first 3 levels of the hierarchy (receptors to PVC) and it’s defined as detecting of the presence of a stimulus. Perception involves the last 2 levels of the hierarchy and it’s defined as integrating, recognising and interpreting the sensation. It’s a high order process. One is able to sense and not perceive and vice versa.

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3
Q

What are the 6 main parts of the eye?

A

Retina, blind spot, pupil/iris, lens, fovea and the optic nerve.

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4
Q

Describe the process of vision in the eye

A

Light enters through the pupil and the iris (a contractile tissue) regulates the size of the pupil. The iris contracts when there is a lot of light and it gives your eye the characteristic colour, e.g. blue eyes. Once it has entered the pupil, it goes through the lens. This focuses the incoming light and turns it all upside down so light from the bottom is projected to the top of the retina and left to right is also opposite. The retina contains the specific receptor cells as well as 4 types of neurons. The retina has a blind spot, where the signals leave the eye and this is filled in by the brain.

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5
Q

Describe the layout of the retina

A

There are 6 layers, the top layers are transparent and the back layers process the light. The light firstly hits the retinal ganglion cells which leaves the eye in one bundle called the optic nerve (where the blind spot is), then there’s the amacrine cells, followed by the bipolar cells, then the horizontal cells. After this is the cone receptors and the rod receptors. When the light reaches the receptors a chain of processes occur resulting in an action potential in the ganglia.

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6
Q

Where is the fovea and what does it do?

A

It’s an area in the centre of the retina and it is specialised for high acuity vision.

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7
Q

What do the rod receptors do?

A

They respond best to faint light and there are 20 times more receptors here compared to cone receptors. Collections of rods converge to one ganglion cell.

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8
Q

What do the cone receptors do?

A

They’re found in and around the fovea. They respond best to bright light and are essential for colour vision. Each cone receptor in the fovea converges to one ganglion cell.

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9
Q

Discuss how rods and cones process light

A

They contain photopigments which release energy. The energy activates secondary messengers which inhibit the receptor cells. This initiates signal transduction across neurons to the brain.

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10
Q

Discuss the signal transmission from the retina to the brain

A

About 90% of the ganglion cells are part of the retina-geniculate-striate pathway. Light from the left visual field in each eye reaches the right visual cortex and vice versa. The axons after the eye cross near the nose, the optic chiasma, apart from the temporal parts, this segregates the visual fields, left visual field in each eye to the right hemisphere and vice versa, the part closest to the nose cross. The axons then travel through the thalamus in the lateral geniculate nucleus to the primary visual cortex, also called V1 or striate cortex. Each level of the system is organised like the retina in that the top of the visual field hits the lower part of the retina and this is maintained throughout the system as different neurons are excited.

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11
Q

What’s another term for edge perception?

How do we see edges?

A

Contrast perception
Lateral inhibition in the retina allows us to see edges. This occurs when areas of light and dark hit the retina. When the horizontal cells are activated by the areas of light, they excite the bipolar cells which inhibits the local area of horizontal cells, therefore inhibiting the bipolar cell and those adjacent to it. This means that the last cells in the area of light are most activated as they only receive inhibition from one adjacent bipolar cell. Also the first cells in the area of dark aren’t excited and are inhibited by the adjacent cells so they’re least activated. This contrast in activation has a heightened action potential because of the inhibited cells, allowing us to perceive edges.

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12
Q

Why do we need to move our eyes?

A

The eye moves about three times per second and these movements are called saccades. It’s essential for a wide angle colour perception. If our eyes don’t move then the retinal images will disappear after a few seconds. This happens because the neurons in the retina respond to signal change so if there is no change, there is no response.

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13
Q

How do we still see our environment as stable even though we have saccades?

A

This is because the visual system temporally integrates the information between saccades. This allows us to have high quality perception and ensures the world doesn’t disappear when we blink. If you fix your eye on one point then your wider vision will decrease in quality because the saccades aren’t occurring.

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14
Q

How do we perceive colour?

A

We are able to see wavelengths between 400 and 700 nm. We perceive colour because of component and opponent processing. Colour is perceived in the cones and different wavelengths correspond to different colours. Component processing is also called trichromatic theory, it involves 3 types of cones. The photopigment in each cone is specialised for either short, medium or long wavelengths. The perception of colour depends on the activity of the cone. There are three cones as one cone would confuse the processing. Opponent processing involves the neurons in the system. Complementary colours cannot exist together (red-green) because the neurons respond antagonistically. For example, a red wavelength would excite one neuron and inhibit the neuron which responds to green.

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15
Q

Discuss colour blindness

A

It’s when you cannot distinguish between certain colours due to the absence of photopigments which respond to a certain wavelength. It’s usually red or green. It’s found on the X chromosome and is therefore mainly found in men. To test for this you can use the Ishihara colour test.

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16
Q

What is colour constancy?

A

We interpret colour using a top down processing method so we receive the information and then use the context to interpret it. Hence optical illusions. We also have brightness, size and shape constancy, for example the optical illusion about the size of a circle surrounded by other circles.

17
Q

Discuss the cortical mechanisms of vision

A

The areas of the brain associated with vision are the striate cortex, prestriate cortex (both part of the PVC), inferotemporal cortex (SVC) and the posterior parietal cortex (Association cortex). The PVC receives information from the thalamus and is the first level of processing. The SVC is composed of different areas, each specialised for a specific type of analysis (colour, motion, shape etc.) Many of the pathways interconnect and connect with the PVC. The dorsal stream begins at the PVC and ends at the posterior parietal cortex. It’s where you analyse where things are. If damaged, you can describe an object but you struggle to reach out and grasp it. The ventral stream begins at the PVC and ends at the inferotemporal C. It allows you to describe what things are and if damaged you struggle to imagine/describe an object.

18
Q

What is blindsight?

A

It says it in the name, it’s when one who has cortical damage and is blind, can still navigate around visual objects and essentially perceive sights.

19
Q

How do you perceive a singular object?

A

This is the binding problem. It’s also a top down process where you begin with simple neurons detecting lines and edges and then the neurons become more and more complex. There is a neuron for a specific object. However, this is the idea of grandmother neurons and our brain isn’t complex enough to have a neuron for every object we have ever perceived. Although there is specific neurons for selected faces. Additionally, there isn’t an area where all the areas converge which is where the grandmother neurons should be found.

20
Q

Give an alternate hypothesis other than the binding problem

A

It’s the idea that processing an object requires simultaneous activity by many neurons. The neurons that are active together are called an assembly and objects are differentiated by the distinct rhythms of neuron activity. This also supports the idea of binding different types of stimuli.

21
Q

What are the three types of receptors associated with touch and pain?

A

Free nerve endings, pacinian corpuscles and Merkel’s disks/ruffini endings.

22
Q

What are the two sensory pathways involved with touch and pain?

A

The first pathway is to do with touch and proprioception (position of the body). The axons ascend on the ipsilateral side of the spinal cord and cross to the other side in the brain stem. The axons then travel to the thalamus which connects with the somatosensory cortex (SI).
The second pathway is to do with pain and temperature. The neurons cross to the contralateral side as they enter the spinal cord. The pathway splits in the brain, half is terminated in the brain stem and the other half goes to the thalamus. This then goes to the SI and the secondary SI (SII) as well as the posterior parietal cortex.

23
Q

Discuss the SI

A

Biologically speaking it is the postcentral gyrus. It’s organised to map the body surface aka homunculus; the places with the most receptors in this system have the largest area, e.g. mouth and hands. The main input is from the contralateral. It’s also organised in horizontal strips and each strip is sensitive to a specific type of sensory input. The output goes to SII and the association cortex.

24
Q

Discuss the SII

Discuss the association cortex

A

The input is mainly from SI and the output is to the association cortex.
The association cortex is biologically called the posterior parietal lobe. It receives input from the SI and SII. If you get damage to the right part, then you can get asomatognosia which is the failure to recognise part of your body. This is often accompanied with anosognosia which is the failure to recognise one’s own symptoms.

25
Q

How do you feel pain?

What are the two main pathways?

A

The neurotransmitters for pain in the spinal cord are glutamate (for mild pain) and glutamate as well as substance P (for strong pain). Psychological variables can increase or decrease the perception of pain.
The thalamus-somatosensory cortex pathway is involved with the sensory aspects of pain. The limbic system and prefrontal cortex pathway is involved with the emotional aspect of pain. This pathway becomes active when you see someone experience pain. The cingulate cortex in the prefrontal cortex has reduced activity during hypnosis/placebos to relieve pain.

26
Q

How does pain relief work?

A

Morphine binds to opiate receptors in the CNS. The way it works is similar to your own mechanism of pain relief. This blocks the relief of substance P. Our body’s own pain relief are endorphines. These are released in the midbrain. The blockage of it supports the gate control theory (non-pain stimuli modifying the intensity of pain). Pleasant and unpleasant stimuli release endorphins (pain and sex), the release is decreased when depressed, explaining why the same injury can hurt more in certain situations